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Journal Article
Development, ISSN 0950-1991, 01/2007, Volume 134, Issue 2, pp. 261 - 272
The transcription factor Gata3 is crucially involved in epidermis and hair follicle differentiation. Yet, little is known about how Gata3 co-ordinates stem... 
Cell Proliferation | Transcription | Hyperplasia | Hyperkeratosis | Transgenic | Genetic | Hair Follicle/abnormalities/cytology/growth & development/metabolism | GATA3 Transcription Factor/deficiency/genetics/metabolism | Bone Morphogenetic Proteins/metabolism | Female | Keratinocytes/cytology/metabolism | Notch1/metabolism | Keratins/metabolism | Signal Transduction | Wnt Proteins/metabolism | Epidermolytic/genetics/metabolism/pathology | Epidermis/growth & development/metabolism | Embryonic Development | Knockout | Pregnancy | Phenotype | Animals | Newborn | Cell Cycle | Receptor | Fibroblast Growth Factors/metabolism | Stem Cells/cytology/metabolism | Mice | Epidermis | Hair follicle | Gata3 | Laser capture microscopy | Mouse | Microarray | Life Sciences & Biomedicine | Developmental Biology | Science & Technology | GATA3 Transcription Factor - genetics | Stem Cells - cytology | Stem Cells - metabolism | Wnt Proteins - metabolism | Epidermis - growth & development | Hair Follicle - abnormalities | Fibroblast Growth Factors - metabolism | Bone Morphogenetic Proteins - metabolism | Keratins - metabolism | Transcription, Genetic | GATA3 Transcription Factor - metabolism | Hyperkeratosis, Epidermolytic - genetics | Animals, Newborn | Epidermis - metabolism | Hyperkeratosis, Epidermolytic - metabolism | Mice, Transgenic | Keratinocytes - cytology | GATA3 Transcription Factor - deficiency | Receptor, Notch1 - metabolism | Mice, Knockout | Hair Follicle - cytology | Hyperkeratosis, Epidermolytic - pathology | Keratinocytes - metabolism | Hair Follicle - metabolism | Hair Follicle - growth & development | Index Medicus
Journal Article
Nature (London), ISSN 1476-4687, 04/2018, Volume 556, Issue 7702, pp. 501 - 504
.... The major effects of itaconate on cellular metabolism during macrophage activation have been attributed to the inhibition of succinate dehydrogenase , yet this inhibition alone is not sufficient... 
Succinates - chemistry | Glutathione - metabolism | Humans | I-kappa B Proteins - metabolism | Inflammation - metabolism | Succinates - administration & dosage | Psoriasis - pathology | Inflammation - drug therapy | Female | Succinates - metabolism | Stress, Physiological - drug effects | Interleukin-6 - metabolism | Cytokines - immunology | Psoriasis - drug therapy | Cytokines - metabolism | Mice, Inbred C57BL | Toll-Like Receptors - immunology | Cells, Cultured | Activating Transcription Factor 3 - metabolism | Gene Expression Regulation - drug effects | Macrophages - metabolism | Animals | Succinates - therapeutic use | Keratinocytes - drug effects | Keratinocytes - metabolism | NF-E2-Related Factor 2 - metabolism | Macrophages - drug effects | Mice | Succinates - pharmacology | Phosphorylation | Transcription factors | Transcription | Macrophages | Interleukin 6 | Proteins | Cell activation | Metabolites | Rodents | Toll-like receptors | Inhibition | Stress response | Glutathione | Immune system | Immune response | Psoriasis | Immunoregulation | Activating transcription factor 3 | Medical treatment | Interleukin 12 | Inflammation | Metabolism | Gene expression | Interleukin 17 | Chromatography | Dimethylitaconate | Tumor necrosis factor | Proteomics | Skin | Autoimmune diseases | Index Medicus
Journal Article
Journal of investigative dermatology, ISSN 0022-202X, 11/2008, Volume 128, Issue 11, pp. 2625 - 2630
Journal Article
Journal Article
Journal Article
Cell reports (Cambridge), ISSN 2211-1247, 12/2015, Volume 13, Issue 9, pp. 1949 - 1964
Journal Article
Journal of investigative dermatology, ISSN 0022-202X, 02/2007, Volume 127, Issue 2, pp. 331 - 341
Keratinocytes are continuously in contact with external stimuli and have the capacity to produce several soluble mediators. Pathogen-associated molecular... 
Life Sciences & Biomedicine | Dermatology | Science & Technology | Oligonucleotides - genetics | Phosphorylation | Toll-Like Receptor 5 - genetics | Humans | Toll-Like Receptor 9 - genetics | I-kappa B Proteins - metabolism | RNA, Messenger - metabolism | Flagellin - pharmacology | Tissue Distribution | Cell Nucleus - metabolism | Protein Isoforms - metabolism | Biological Transport | Oligonucleotides - pharmacology | Toll-Like Receptor 3 - genetics | Toll-Like Receptors - metabolism | Chemokine CXCL9 | Toll-Like Receptor 5 - metabolism | Poly I-C - pharmacology | Cytokines - metabolism | Cells, Cultured | Toll-Like Receptor 4 - genetics | Toll-Like Receptor 3 - metabolism | Toll-Like Receptor 4 - metabolism | Chemokine CXCL10 | Transcription Factor RelA - metabolism | Keratinocytes - metabolism | Toll-Like Receptors - genetics | Chemokines, CXC - metabolism | Lipopolysaccharides - pharmacology | CpG Islands | Ligands | Chemokines - metabolism | Toll-Like Receptor 9 - metabolism | CD40 antigen | Oligonucleotides | Flagellin | mRNA | intercellular adhesion molecule 1 | Immunity | Lipopolysaccharides | CXC chemokines | CCL20 protein | Toll-like receptors | CpG islands | TLR7 protein | Keratinocytes | Nuclear transport | Stress | TLR3 protein | TLR9 protein | TLR1 protein | External stimuli | CXCL10 protein | Histocompatibility antigen HLA | Interferon | Internet | Monocyte chemoattractant protein 1 | Tumor necrosis factor- alpha | Index Medicus
Journal Article