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Nature Cell Biology, ISSN 1465-7392, 03/2010, Volume 12, Issue 3, pp. 299 - 305
For most types of cancers, the cell at the origin of tumour initiation is still unknown. Here, we used mouse genetics to identify cells at the origin of basal... 
POPULATION | STEM-CELLS | SONIC HEDGEHOG | EPIDERMIS | HAIR FOLLICLE BULGE | KERATINOCYTES | CANCER | EXPRESSION | HUMAN HOMOLOG | MOUSE SKIN | CELL BIOLOGY | Epithelial Cells - metabolism | Cadherins - metabolism | Receptors, G-Protein-Coupled - metabolism | Skin - metabolism | Cell Count | Ear, External - pathology | Integrin beta4 - metabolism | Tail - pathology | Hair Follicle - pathology | Hedgehog Proteins - genetics | Neoplastic Stem Cells - metabolism | Kruppel-Like Transcription Factors - metabolism | Smoothened Receptor | Neoplastic Stem Cells - pathology | Cell Differentiation | Patched Receptors | Skin - pathology | Keratin-10 - metabolism | Epidermis - metabolism | Epidermis - pathology | RNA, Untranslated | Bacterial Proteins - genetics | Receptors, Cell Surface - metabolism | Epithelial Cells - pathology | Genes, Reporter - genetics | Mice, Transgenic | Carcinoma, Basal Cell - pathology | Mice, Inbred Strains | Clone Cells - metabolism | Keratin-14 - genetics | Carcinoma, Basal Cell - metabolism | Keratin-15 - metabolism | Keratin-19 - genetics | Proteins - genetics | Cell Lineage | Animals | Clone Cells - pathology | Proteins - metabolism | Models, Biological | Hair Follicle - metabolism | Bacterial Proteins - metabolism | Luminescent Proteins - genetics | Mice | Receptors, G-Protein-Coupled - genetics | Integrases - genetics | Keratin-15 - genetics | Luminescent Proteins - metabolism | Basal cell carcinoma | Stem cells | Genetic aspects | Cellular signal transduction | Research | Health aspects | Risk factors
Journal Article
Cell, ISSN 0092-8674, 06/2012, Volume 149, Issue 6, pp. 1207 - 1220
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2017, Volume 12, Issue 11, p. e0188398
Epithelial and stromal stem cells are required to maintain corneal transparency. The aim of the study was to develop a new method to isolate and grow both... 
PROGENITOR CELLS | THERAPY | NEURAL CREST DERIVATIVES | MULTIDISCIPLINARY SCIENCES | CULTURE-CONDITIONS | TISSUE | NICHE | PAX6 | EXPRESSION | TRANSPLANTATION | Cell Proliferation | Humans | Adipocytes - drug effects | Spheroids, Cellular - cytology | PAX6 Transcription Factor - genetics | Limbus Corneae - drug effects | Corneal Stroma - cytology | Polycomb Repressive Complex 1 - genetics | Limbus Corneae - metabolism | Epithelium, Corneal - drug effects | Cell Differentiation | Neurons - metabolism | Culture Media - chemistry | Neoplasm Proteins - genetics | Fibroblasts - metabolism | Biomarkers - metabolism | Corneal Stroma - drug effects | SOX9 Transcription Factor - metabolism | Gene Expression | Corneal Stroma - metabolism | Spheroids, Cellular - metabolism | Keratinocytes - drug effects | Keratinocytes - metabolism | Fibroblasts - drug effects | Fibroblasts - cytology | Chondrocytes - cytology | Epithelial Cells - metabolism | Epithelium, Corneal - cytology | Polycomb Repressive Complex 1 - metabolism | Epithelial Cells - drug effects | Adipocytes - cytology | Neurons - cytology | Stem Cells - cytology | Neoplasm Proteins - metabolism | Stem Cells - metabolism | ATP Binding Cassette Transporter, Sub-Family G, Member 2 - metabolism | Chondrocytes - drug effects | Nestin - genetics | Spheroids, Cellular - drug effects | Epithelial Cells - cytology | Neurons - drug effects | Chondrocytes - metabolism | PAX6 Transcription Factor - metabolism | Keratinocytes - cytology | Cell Separation - methods | Nestin - metabolism | ATP Binding Cassette Transporter, Sub-Family G, Member 2 - genetics | Adipocytes - metabolism | Culture Media - pharmacology | Epithelium, Corneal - metabolism | Stem Cells - drug effects | Primary Cell Culture | Limbus Corneae - cytology | SOX9 Transcription Factor - genetics | Physiological aspects | Cornea | Genetic aspects | Research | Stem cells | Vimentin | Bioengineering | Cell culture | Nestin | Transparency | Media (culture) | Transplants & implants | Sox9 protein | Osteocytes | Adipocytes | Scraping | Biomedical materials | Epidermal growth factor | Fibroblasts | Biocompatibility | Chondrogenesis | Explants | Second harmonic generation | Colonies | Fibrils | Forming | Collagenase | Sox10 protein | Studies | Pax6 protein | Microscopy | Biopsy | Collagen | Chondrocytes | Differentiation | Methods | Life Sciences | Cellular Biology
Journal Article
Journal of Investigative Dermatology, ISSN 0022-202X, 11/2008, Volume 128, Issue 11, pp. 2625 - 2630
The critical role of IL-17 has recently been reported in a variety of conditions. Since IL-17 deeply participates in the pathogenesis of psoriasis and... 
INTERFERON-GAMMA | CHEMOKINE PRODUCTION | T(H)17 | IL-17 | GENE-EXPRESSION | GROWTH-FACTOR | DIFFERENTIATION | HUMAN KERATINOCYTES | T-LYMPHOCYTES | SKIN INFLAMMATION | DERMATOLOGY | Th1 Cells - pathology | Tumor Necrosis Factor-alpha - metabolism | Granulocyte-Macrophage Colony-Stimulating Factor - metabolism | Humans | Middle Aged | Male | Vascular Endothelial Growth Factor A - metabolism | Interleukin-17 - pharmacology | CD4-Positive T-Lymphocytes - pathology | Case-Control Studies | Interleukins - metabolism | Th1 Cells - metabolism | Psoriasis - pathology | Dermatitis, Atopic - etiology | Psoriasis - metabolism | Adult | Female | T-Lymphocytes, Helper-Inducer - pathology | Interleukin-8 - metabolism | Child | Th2 Cells - pathology | Dermis - metabolism | T-Lymphocytes, Helper-Inducer - metabolism | CD4-Positive T-Lymphocytes - metabolism | Dermatitis, Atopic - pathology | Th2 Cells - metabolism | Interleukin-17 - metabolism | Adolescent | Dermis - pathology | Aged | Dermatitis, Atopic - metabolism | Cell Movement | Flow cytometry | Psoriasis | Dermatology | Granulocyte-macrophage colony-stimulating factor | Helper cells | Dermis | Interleukin 22 | Keratinocytes | Lymphocytes T | Interleukin 17 | Atopic dermatitis | CXCL10 protein | Eczema | Peripheral blood | gamma -Interferon | Skin diseases | Internet | Vascular endothelial growth factor | Interleukin 8 | Tumor necrosis factor- alpha
Journal Article
eLife, ISSN 2050-084X, 2014, Volume 3
SHARPIN regulates immune signaling and contributes to full transcriptional activity and prevention of cell death in response to TNF in vitro. The inactivating... 
mouse | stem cells | cell biology | dermatitis | inflammation | developmental biology | apoptosis | TNF signaling | LUBAC | ubiquitin | APOPTOSIS | CHRONIC PROLIFERATIVE DERMATITIS | RIP3 KINASE | BIOLOGY | MIXED LINEAGE KINASE | CHAIN ASSEMBLY COMPLEX | DOMAIN-LIKE | LYMPHOTOXIN-ALPHA | NLRP3 INFLAMMASOME | KAPPA-B ACTIVITY | CUTTING EDGE | Receptor-Interacting Protein Serine-Threonine Kinases - metabolism | Inflammation - pathology | Liver - pathology | Nerve Tissue Proteins - deficiency | Caspase 3 - metabolism | Caspase 8 - metabolism | Spleen - drug effects | Receptors, Tumor Necrosis Factor, Type I - metabolism | Inflammation - metabolism | Ubiquitination - drug effects | Liver - drug effects | Receptors, Tumor Necrosis Factor, Type II - metabolism | Cytoprotection - drug effects | Cell Death - drug effects | Spleen - pathology | Fibroblasts - metabolism | Mice, Inbred C57BL | Cells, Cultured | Ubiquitin-Protein Ligases - metabolism | Fibroblasts - pathology | Receptors, Interleukin-1 - metabolism | Nerve Tissue Proteins - metabolism | Dermatitis - pathology | Tumor Necrosis Factor-alpha - pharmacology | Keratinocytes - pathology | Macrophages - metabolism | Animals | Keratinocytes - drug effects | Keratinocytes - metabolism | Fibroblasts - drug effects | Myeloid Cells - metabolism | Heterozygote | Macrophages - drug effects | Receptor-Interacting Protein Serine-Threonine Kinases - deficiency | Myeloid Cells - pathology | Chronic Disease | Dermatitis - metabolism | Tumor necrosis factor receptors | Splenomegaly | Transcription | Liver | Lethality | Kinases | Dermatitis | Inflammatory diseases | Proteins | Genotype & phenotype | Immunology | Clonal deletion | Interleukin 1 | Tumor necrosis factor-TNF | Peyer's patches | Medical research | Cytokines | Glycerol | Caspase | Inflammation | Heterozygosity | Caspase-8 | Tumor necrosis factor | Cell death | Cancer | Apoptosis
Journal Article
Journal Article
Molecular Biology of the Cell, ISSN 1059-1524, 09/2007, Volume 18, Issue 9, pp. 3607 - 3619
Journal Article
Nature, ISSN 0028-0836, 2014, Volume 513, Issue 7516, pp. 90 - 94
Necroptosis has emerged as an important pathway of programmed cell death in embryonic development, tissue homeostasis, immunity and inflammation(1-8). RIPK1 is... 
CHRONIC INTESTINAL INFLAMMATION | CELLS | KAPPA-B ACTIVATION | NECROSIS-FACTOR RECEPTOR | MULTIDISCIPLINARY SCIENCES | IN-VIVO | KINASE | ALPHA-DEPENDENT APOPTOSIS | MICE | SKIN INFLAMMATION | DELETION | Receptor-Interacting Protein Serine-Threonine Kinases - metabolism | Inflammation - pathology | Skin - cytology | Epithelial Cells - metabolism | Skin - metabolism | Caspase 8 - metabolism | Homeostasis | Paneth Cells - pathology | Male | Intestines - metabolism | Receptors, Tumor Necrosis Factor, Type I - metabolism | Necrosis | Receptors, Tumor Necrosis Factor, Type I - deficiency | Inflammation - metabolism | Female | Epithelial Cells - cytology | Skin - pathology | Paneth Cells - metabolism | Intestines - pathology | Cell Survival | Fas-Associated Death Domain Protein - metabolism | Fas-Associated Death Domain Protein - deficiency | Epithelial Cells - pathology | Mice, Knockout | Keratinocytes - pathology | Receptor-Interacting Protein Serine-Threonine Kinases - genetics | Animals | Keratinocytes - metabolism | Mice | Receptor-Interacting Protein Serine-Threonine Kinases - deficiency | Myeloid Differentiation Factor 88 - metabolism | Apoptosis | Intestines - cytology | Physiological aspects | Physiological research | Inflammation | Research | Protein kinases | Pathology | Antibiotics | Rodents | Skin | Kinases
Journal Article
JOURNAL OF IMMUNOLOGY, ISSN 0022-1767, 08/2015, Volume 195, Issue 4, pp. 1744 - 1752
Clinical improvement of psoriasis induced by IL-4 treatment has been ascribed to changes in dermal inflammatory cells, such as activation of Th2 cells and... 
ATOPIC-DERMATITIS | LESIONAL SKIN | ENHANCED EXPRESSION | ORGAN-CULTURE SYSTEM | INCREASED EXPRESSION | GENE-EXPRESSION | TNF-ALPHA | IMMUNOLOGY | AUTOIMMUNE-DISEASE | T-CELLS | HUMAN KERATINOCYTES | GATA3 Transcription Factor - genetics | Leukocytes, Mononuclear - metabolism | Humans | STAT6 Transcription Factor - genetics | Th2 Cells - immunology | Interleukin-1beta - genetics | Interleukin-23 Subunit p19 - metabolism | Interleukin-4 - pharmacology | Psoriasis - genetics | S100 Proteins - genetics | Psoriasis - pathology | Interleukin-1beta - metabolism | Interleukin-23 Subunit p19 - genetics | Inflammation Mediators - metabolism | Psoriasis - metabolism | beta-Defensins - metabolism | Cytokines - genetics | GATA3 Transcription Factor - metabolism | Interleukin-6 - metabolism | Langerhans Cells - drug effects | S100 Calcium Binding Protein A7 | Epidermis - metabolism | beta-Defensins - genetics | Leukocytes, Mononuclear - drug effects | Nerve Growth Factor - metabolism | Psoriasis - drug therapy | Cytokines - metabolism | Interleukin-6 - genetics | Epidermis - drug effects | RNA, Messenger - genetics | Nerve Growth Factor - genetics | S100 Proteins - metabolism | STAT6 Transcription Factor - metabolism | Th2 Cells - metabolism | Gene Expression Regulation - drug effects | Keratinocytes - drug effects | Keratinocytes - metabolism | Langerhans Cells - metabolism
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 11/2012, Volume 122, Issue 11, pp. 3965 - 3976
Psoriasis is a chronic inflammatory disorder of the skin affecting approximately 2% of the world's population. Accumulating evidence has revealed that the... 
GENERALIZED PUSTULAR PSORIASIS | MEDICINE, RESEARCH & EXPERIMENTAL | INFLAMMATORY SKIN-DISEASE | FAMILY-MEMBERS | MOUSE MODEL | DELTA-T-CELLS | NF-KAPPA-B | AUTOINFLAMMATORY DISEASE | INTERLEUKIN-12/23 MONOCLONAL-ANTIBODY | CUTTING EDGE | TRANSGENIC MICE | Cell Communication - immunology | Interleukin-1 - genetics | Membrane Glycoproteins - metabolism | Dermatitis - immunology | Dendritic Cells - immunology | Receptors, Interleukin-1 - genetics | Interleukin-17 - immunology | Dendritic Cells - pathology | Interleukins - metabolism | Interleukin-23 - genetics | Interleukins - genetics | Psoriasis - genetics | Interleukin-23 - metabolism | Psoriasis - pathology | Interleukins - immunology | Toll-Like Receptor 7 - immunology | Interleukin-1 - immunology | Psoriasis - metabolism | Membrane Glycoproteins - immunology | Dendritic Cells - metabolism | Psoriasis - immunology | Toll-Like Receptor 7 - genetics | Interleukin-1 - metabolism | Toll-Like Receptor 7 - metabolism | Dermatitis - genetics | Receptors, Interleukin-1 - immunology | Interleukin-17 - genetics | Cell Communication - genetics | Interleukin-23 - immunology | Keratinocytes - immunology | Membrane Glycoproteins - genetics | Mice, Knockout | Receptors, Interleukin-1 - metabolism | Dermatitis - pathology | Interleukin-17 - metabolism | Keratinocytes - pathology | Animals | Keratinocytes - metabolism | Mice | Dermatitis - metabolism | Psoriasis | Interleukins | Keratinocytes | Genetic aspects | Dosage and administration | Diagnosis | Drug therapy
Journal Article