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Breast Cancer Research, ISSN 1465-5411, 09/2011, Volume 13, Issue 5, pp. R87 - R87
Introduction: Some molecular subtypes of breast cancer have preferential sites of distant relapse. The protein expression pattern of the primary tumor may... 
MOLECULAR SUBTYPES | SURVIVAL | RELAPSE | CELLS | ONCOLOGY | ERBB2 | BRAIN METASTASES | MARKERS | PATTERNS | CARCINOMA | TUMORS | Nestin | Receptors, Estrogen - metabolism | Cadherins - metabolism | Follow-Up Studies | Humans | Lung Neoplasms - metabolism | Glycoproteins - metabolism | Receptor, ErbB-2 - metabolism | Bone Neoplasms - secondary | Proteins - analysis | Brain Neoplasms - metabolism | Antigens, CD - metabolism | Bone Neoplasms - metabolism | Breast Neoplasms - metabolism | Receptors, Progesterone - metabolism | Brain Neoplasms - secondary | Receptor, Epidermal Growth Factor - metabolism | Peptides - metabolism | Lung Neoplasms - secondary | Female | Liver Neoplasms - secondary | Snail Family Transcription Factors | AC133 Antigen | Skin Neoplasms - metabolism | Nerve Tissue Proteins - metabolism | Transcription Factors - metabolism | Proteins - metabolism | Breast Neoplasms - pathology | Cyclooxygenase 2 - metabolism | Finland | Liver Neoplasms - metabolism | Skin Neoplasms - secondary | Keratin-5 - metabolism | Intermediate Filament Proteins - metabolism | Cohort Studies | Immunohistochemistry | Care and treatment | Estrogen | Development and progression | Breast cancer | Research | Gene expression | Keratin | Epidermal growth factor | Genetic aspects | Diagnosis | Progesterone | Tumor proteins | Index Medicus
Journal Article
Molecular Biology of the Cell, ISSN 1059-1524, 05/2010, Volume 21, Issue 10, pp. 1698 - 1713
Journal Article
Gastroenterology, ISSN 0016-5085, 2011, Volume 141, Issue 2, pp. 731 - 741.e4
Background & Aims Animal studies have indicated that pancreatic exocrine acinar cells have phenotypic plasticity. In rodents, acinar cells can differentiate... 
Gastroenterology and Hepatology | Metaplasia | Reprogramming | Exocrine Pancreas | Tissue Engineering | BETA-CELLS | INTRAEPITHELIAL NEOPLASIA | HUMAN EXOCRINE PANCREAS | ADULT HUMAN PANCREAS | IN-VITRO | NOTCH SIGNALING PATHWAY | GENERATION | MICE | GROWTH-FACTOR | DIFFERENTIATION | GASTROENTEROLOGY & HEPATOLOGY | Cell Proliferation | Chymotrypsin - metabolism | Humans | Glycoproteins - metabolism | Carbonic Anhydrase II - metabolism | Ki-67 Antigen - metabolism | Cell Transdifferentiation - genetics | RNA, Messenger - metabolism | Pancreatic Ducts - metabolism | Antigens, CD - metabolism | Cell Transdifferentiation - physiology | Peptides - metabolism | Pancreas, Exocrine - cytology | Cell Lineage - genetics | Genes, Reporter | Hepatocyte Nuclear Factor 1-beta - metabolism | Plant Lectins - pharmacokinetics | Biomarkers - metabolism | SOX9 Transcription Factor - metabolism | Green Fluorescent Proteins - metabolism | Promoter Regions, Genetic | Pancreatic Ducts - cytology | Transduction, Genetic | Cell Survival | Cells, Cultured | Cystic Fibrosis Transmembrane Conductance Regulator - metabolism | Signal Transduction - genetics | AC133 Antigen | Cell Lineage - physiology | Phenotype | Signal Transduction - physiology | Pancreas, Exocrine - metabolism | Keratin-19 - metabolism | Mitogen-Activated Protein Kinases - metabolism | Keratin | Epidermal growth factor | Analysis | Fluorescein | Fluorescence | Cystic fibrosis | Diabetes | Universities and colleges | Research | Protein kinases | Index Medicus | Abridged Index Medicus
Journal Article
Gastroenterology, ISSN 0016-5085, 2010, Volume 138, Issue 5, pp. 1810 - 1822.e2
Background & Aims The molecular mechanism underlying epithelial metaplasia in Barrett's esophagus remains unknown. Recognizing that Hedgehog signaling is... 
Gastroenterology and Hepatology | BMP4 | Hedgehog Signaling | SOX9 | Barrett's Esophagus | CELLS | SONIC HEDGEHOG | ESOPHAGUS | GASTROINTESTINAL-TRACT | INTESTINAL EPITHELIUM | PYLORIC SPHINCTER EPITHELIUM | COLUMNAR | TERMINAL DIFFERENTIATION | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | Precancerous Conditions - etiology | Adenocarcinoma - pathology | Epithelial Cells - metabolism | Humans | Hedgehog Proteins - metabolism | Bile - metabolism | Precancerous Conditions - metabolism | Barrett Esophagus - etiology | Bone Morphogenetic Protein 4 - metabolism | Esophageal Neoplasms - pathology | Adenocarcinoma - metabolism | Barrett Esophagus - pathology | Keratins - metabolism | Transfection | Hedgehog Proteins - genetics | RNA Interference | Esophageal Neoplasms - metabolism | Precancerous Conditions - pathology | Cell Differentiation | Patched Receptors | Disease Models, Animal | SOX9 Transcription Factor - metabolism | Barrett Esophagus - metabolism | Cell Line | Gastroesophageal Reflux - metabolism | Esophagus - metabolism | Receptors, Cell Surface - metabolism | Epithelial Cells - pathology | Mice, Transgenic | Signal Transduction - genetics | Cell Communication - genetics | Metaplasia | Esophagus - pathology | Phenotype | Animals | Gastroesophageal Reflux - complications | Bile Reflux - complications | Bile Reflux - metabolism | Mesoderm - metabolism | Mice | Mesoderm - pathology | Patched-1 Receptor | Hydrogen-Ion Concentration | Receptors, Cell Surface - genetics | RNA | Oncology, Experimental | Brain tumors | Barrett's esophagus | Bone morphogenetic proteins | Research | Universities and colleges | Cancer | Index Medicus | Abridged Index Medicus | Hedgehog signaling
Journal Article
Nature Medicine, ISSN 1078-8956, 2012, Volume 18, Issue 4, pp. 572 - 579
Nat Med. 2012 Mar 4;18(4):572-9. doi: 10.1038/nm.2667. Macrophage-derived Wnt opposes Notch signaling to specify hepatic progenitor cell fate in chronic liver... 
MEDICINE, RESEARCH & EXPERIMENTAL | BIOCHEMISTRY & MOLECULAR BIOLOGY | HUMAN JAGGED1 | CELL BIOLOGY | ALAGILLE-SYNDROME | BILE-DUCT DEVELOPMENT | REGULATES FORMATION | MOUSE MODEL | CHOLINE-DEFICIENT | GENE-EXPRESSION | NUMB | OVAL CELLS | DIFFERENTIATION | Wnt3A Protein - metabolism | Ethionine - administration & dosage | Keratins, Hair-Specific - genetics | Receptors, Notch - metabolism | Humans | Middle Aged | Liver Diseases - pathology | Cell Communication - physiology | Male | RNA, Messenger - metabolism | Young Adult | Cell Differentiation - genetics | Intercellular Signaling Peptides and Proteins - metabolism | Liver Regeneration - genetics | Biliary Tract - metabolism | Serrate-Jagged Proteins | Adult | Antigens, Differentiation - metabolism | Female | Membrane Proteins - metabolism | Cell Differentiation - physiology | Hepatocytes - physiology | Jagged-1 Protein | Calcium-Binding Proteins - metabolism | Mice, Inbred C57BL | Cells, Cultured | Gene Expression Regulation - physiology | Mice, Transgenic | gamma-Glutamyltransferase - metabolism | Biliary Tract - physiopathology | Cell Communication - genetics | Liver Regeneration - physiology | beta Catenin - genetics | Biliary Tract - pathology | Nerve Tissue Proteins - metabolism | Macrophages - metabolism | Animals | Creatine Kinase - metabolism | Signal Transduction - physiology | Stem Cell Niche - physiology | Stem Cells - physiology | Aged | Mice | Chronic Disease | Liver diseases | Physiological aspects | Development and progression | Genetic aspects | Research | Macrophages | Cell differentiation | Wnt proteins | Signal transduction | Cellular biology | Gene expression | Index Medicus
Journal Article
FEBS Journal, ISSN 1742-464X, 05/2016, Volume 283, Issue 9, pp. 1689 - 1700
Intervertebral discs ( IVD s) provide stability and flexibility to the spinal column; however, IVD s, and in particular the nucleus pulposus ( NP ), undergo a... 
intervertebral disc degeneration | Smad | BMP | mesenchymal stem cells | nucleus pulposus | BMP7 | REGENERATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | MESENCHYMAL STEM-CELLS | TRANSPLANTATION | CROSS-TALK | LUMBAR-SPINE | IN-VITRO | NUCLEUS PULPOSUS CELLS | DISEASE | GROWTH | DIFFERENTIATION | RNA, Small Interfering - genetics | Bone Morphogenetic Protein 7 - genetics | Humans | Extracellular Matrix - metabolism | Intervertebral Disc Degeneration - metabolism | Intervertebral Disc Degeneration - therapy | Collagen Type II - metabolism | Intervertebral Disc Degeneration - pathology | Lentivirus - metabolism | Glucuronosyltransferase - genetics | Mesenchymal Stromal Cells - cytology | Smad1 Protein - genetics | Lentivirus - genetics | Intervertebral Disc - pathology | Chondrocytes - metabolism | Disease Models, Animal | Intervertebral Disc Degeneration - genetics | SOX9 Transcription Factor - metabolism | Chondrocytes - pathology | Rabbits | Genetic Vectors - chemistry | Signal Transduction | Bone Marrow Cells - cytology | Glycosaminoglycans - metabolism | Keratin-8 - genetics | Gene Expression Regulation | Genetic Vectors - metabolism | Mesenchymal Stromal Cells - metabolism | Intervertebral Disc - metabolism | Aggrecans - metabolism | Smad1 Protein - antagonists & inhibitors | Aggrecans - genetics | Collagen Type II - genetics | Keratin-19 - genetics | Animals | Glucuronosyltransferase - metabolism | Keratin-8 - metabolism | Bone Morphogenetic Protein 7 - metabolism | Smad1 Protein - metabolism | Keratin-19 - metabolism | Extracellular Matrix - pathology | SOX9 Transcription Factor - genetics | Bone Marrow Cells - metabolism | Mesenchymal Stem Cell Transplantation | RNA, Small Interfering - metabolism | Physiological aspects | Bone morphogenetic proteins | Cell differentiation | Analysis | Stem cells | Extracellular matrix | Spine | Rodents | Keratins | Therapy | Surgical implants | Smad protein | Mesenchyme | Collagen (type II) | Sox9 protein | Medical services | Differentiation (biology) | Electrochemical machining | Homeostasis | Nucleus pulposus | Remodeling | Intervertebral discs | Nuclei | Flexibility | Bone marrow | Degeneration | Effectiveness | Stability | Markers | Feasibility studies | Implantation | Columns (process) | Survival | Diseases | Overexpression | Disks | Feasibility | Chondroitin sulfate | Sulfate | Aggrecan | Viability | Bone morphogenetic protein 7 | Index Medicus
Journal Article
Leukemia, ISSN 0887-6924, 01/2004, Volume 18, Issue 1, pp. 29 - 40
It has been suggested that bone marrow (BM)-derived hematopoietic stem cells transdifferentiate into tissue-specific stem cells (the so-called phenomenon of... 
SDF-1 | CXCR4 | Stem cell mobilization | Stem cell homing | Stem cell plasticity | stem cell plasticity | PROGENITOR CELLS | stem cell mobilization | FUSION | CHEMOKINE RECEPTOR CXCR4 | stem cell homing | PERIPHERAL-BLOOD | FACTOR-I | MICE LACKING | SKELETAL-MUSCLE | EPITHELIAL-CELLS | ONCOLOGY | STEM/PROGENITOR CELLS | HEMATOLOGY | Glial Fibrillary Acidic Protein - genetics | Nestin | Antigens, CD34 - metabolism | MyoD Protein - genetics | Humans | Blood Cells - metabolism | Muscle, Skeletal - metabolism | Glial Fibrillary Acidic Protein - metabolism | RNA, Messenger - metabolism | Blood Cells - cytology | Keratins - metabolism | Bone Marrow - metabolism | Intermediate Filament Proteins - genetics | Muscle Proteins - metabolism | Female | Neurons - metabolism | Hematopoietic Stem Cell Mobilization | DNA-Binding Proteins | alpha-Fetoproteins - metabolism | Hematopoietic Stem Cells - drug effects | Cell Line | Chemokine CXCL12 | alpha-Fetoproteins - genetics | Liver - metabolism | RNA, Messenger - genetics | Trans-Activators | Biomarkers - analysis | Hematopoietic Stem Cells - metabolism | MyoD Protein - metabolism | Nerve Tissue Proteins | Biomarkers - blood | Myogenic Regulatory Factor 5 | Chemokines, CXC - genetics | Receptors, CXCR4 - metabolism | Muscle Proteins - genetics | Granulocyte Colony-Stimulating Factor - pharmacology | Animals | Chemokines, CXC - metabolism | Mice | Mice, Inbred BALB C | Keratins - genetics | Intermediate Filament Proteins - metabolism | Index Medicus
Journal Article
Nature Cell Biology, ISSN 1465-7392, 09/2010, Volume 12, Issue 9, pp. 876 - 885
Keratins 8 and 18 (K8 and K18) are heteropolymeric intermediate filament phosphoglycoproteins of simple-type epithelia. Mutations in K8 and K18 predispose the... 
CELLS | SIGNALING PATHWAYS | MITOTIC ARREST | KINASE-B | O-GLCNAC MODIFICATION | LIVER-DISEASE | NUCLEOCYTOPLASMIC PROTEINS | LINKED N-ACETYLGLUCOSAMINE | INTERMEDIATE-FILAMENTS | TRANSGENIC MICE | CELL BIOLOGY | Liver - pathology | Apoptosis - drug effects | Humans | Caspase 3 - metabolism | Apoptosis - genetics | Glycosylation - drug effects | Pancreas - injuries | Phosphorylation - genetics | Keratins - metabolism | Liver - drug effects | Phosphorylation - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Antibodies, Monoclonal - immunology | Protein-Serine-Threonine Kinases - metabolism | Chemical and Drug Induced Liver Injury - prevention & control | Antibodies, Monoclonal - pharmacology | Liver - metabolism | Enzyme Inhibitors - pharmacology | Pancreas - pathology | Acetylglucosamine - pharmacology | Mice, Transgenic | Pancreas - metabolism | Mice, Knockout | Phenylcarbamates - pharmacology | fas Receptor - agonists | Chemical and Drug Induced Liver Injury - metabolism | Models, Biological | Cytoskeleton - metabolism | Cytosol - metabolism | Mice | Keratins - genetics | Protein Binding - physiology | Cytosol - drug effects | Hepatocytes - metabolism | Proto-Oncogene Proteins c-akt - genetics | fas Receptor - immunology | Acetylglucosamine - analogs & derivatives | Chemical and Drug Induced Liver Injury - pathology | Hepatocytes - drug effects | Keratins - chemistry | Pancreas - drug effects | beta-N-Acetylhexosaminidases - antagonists & inhibitors | Chemical and Drug Induced Liver Injury - genetics | Glycogen Synthase Kinase 3 - metabolism | Mice, Inbred Strains | Streptozocin - pharmacology | Keratin-18 - genetics | HSP70 Heat-Shock Proteins - metabolism | Protein Kinase C-delta - metabolism | Animals | Keratin-8 - metabolism | Keratin-18 - metabolism | Oximes - pharmacology | Amino Acid Substitution - genetics | Keratin | Liver diseases | Gene mutations | Physiological aspects | Glycosylation | Genetic aspects | Research | Health aspects | Risk factors | Index Medicus
Journal Article
Journal Article