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Development, ISSN 0950-1991, 01/2007, Volume 134, Issue 2, pp. 261 - 272
The transcription factor Gata3 is crucially involved in epidermis and hair follicle differentiation. Yet, little is known about how Gata3 co-ordinates stem... 
Cell Proliferation | Transcription | Hyperplasia | Hyperkeratosis | Transgenic | Genetic | Hair Follicle/abnormalities/cytology/growth & development/metabolism | GATA3 Transcription Factor/deficiency/genetics/metabolism | Bone Morphogenetic Proteins/metabolism | Female | Keratinocytes/cytology/metabolism | Notch1/metabolism | Keratins/metabolism | Signal Transduction | Wnt Proteins/metabolism | Epidermolytic/genetics/metabolism/pathology | Epidermis/growth & development/metabolism | Embryonic Development | Knockout | Pregnancy | Phenotype | Animals | Newborn | Cell Cycle | Receptor | Fibroblast Growth Factors/metabolism | Stem Cells/cytology/metabolism | Mice | Epidermis | Hair follicle | Gata3 | Laser capture microscopy | Mouse | Microarray | Life Sciences & Biomedicine | Developmental Biology | Science & Technology | GATA3 Transcription Factor - genetics | Stem Cells - cytology | Stem Cells - metabolism | Wnt Proteins - metabolism | Epidermis - growth & development | Hair Follicle - abnormalities | Fibroblast Growth Factors - metabolism | Bone Morphogenetic Proteins - metabolism | Keratins - metabolism | Transcription, Genetic | GATA3 Transcription Factor - metabolism | Hyperkeratosis, Epidermolytic - genetics | Animals, Newborn | Epidermis - metabolism | Hyperkeratosis, Epidermolytic - metabolism | Mice, Transgenic | Keratinocytes - cytology | GATA3 Transcription Factor - deficiency | Receptor, Notch1 - metabolism | Mice, Knockout | Hair Follicle - cytology | Hyperkeratosis, Epidermolytic - pathology | Keratinocytes - metabolism | Hair Follicle - metabolism | Hair Follicle - growth & development | Index Medicus
Journal Article
Breast cancer research : BCR, ISSN 1465-5411, 09/2011, Volume 13, Issue 5, pp. R87 - R87
Journal Article
Gut, ISSN 0017-5749, 10/2017, Volume 66, Issue 10, pp. 1748 - 1760
ObjectiveEpidemiological and clinical data indicate that patients suffering from IBD with long-standing colitis display a higher risk to develop colorectal... 
COLORECTAL CANCER | IBD | CELL MATRIX INTERACTION | INTEGRINS | INTESTINAL BARRIER FUNCTION | Gastroenterology & Hepatology | Life Sciences & Biomedicine | Science & Technology | Intestinal Mucosa - metabolism | Intestinal Mucosa - physiopathology | Epithelial Cells - metabolism | Colitis - genetics | Colorectal Neoplasms - genetics | B-Lymphocytes | Hemidesmosomes - physiology | Caspase 1 - metabolism | Colitis - pathology | Hemidesmosomes - genetics | Adenocarcinoma - metabolism | Adenocarcinoma - physiopathology | Mucus - metabolism | Adenocarcinoma - genetics | Integrin alpha6beta4 - metabolism | Cytokines - genetics | Colorectal Neoplasms - metabolism | Adaptive Immunity | Severity of Illness Index | Cytokines - metabolism | Signal Transduction | T-Lymphocytes | Lymphocyte Activation | Myeloid Differentiation Factor 88 - genetics | Integrin alpha6 - genetics | Permeability | Colorectal Neoplasms - physiopathology | Basement Membrane - physiopathology | Animals | Colitis - physiopathology | Keratin-8 - metabolism | Keratin-18 - metabolism | Colitis - metabolism | Mice | Homeostasis - genetics | Intestinal Mucosa - pathology | Complications and side effects | Inflammatory bowel diseases | Antibiotics | Colorectal cancer | Dosage and administration | Research | Epidemiology | Risk factors | Adenocarcinoma | Transformation | Animal models | Epithelial cells | Hyperplasia | Colorectal carcinoma | Helper cells | Homeostasis | Genomes | Lymphocytes T | Small intestine | Defects | Metastases | Keratin | Cell activation | Laminin | Intestine | Hemidesmosomes | Tumorigenesis | Immune system | Dysplasia | Cytokines | Inflammation | Patients | Interleukin 18 | Inflammatory bowel disease | Studies | Lymphocytes B | Colitis | Tumors | Index Medicus | Abridged Index Medicus | Life Sciences | Development Biology | Inflammatory Bowel Disease | 1506
Journal Article
Journal Article
Nature medicine, ISSN 1546-170X, 03/2012, Volume 18, Issue 4, pp. 572 - 579
Nat Med. 2012 Mar 4;18(4):572-9. doi: 10.1038/nm.2667. Macrophage-derived Wnt opposes Notch signaling to specify hepatic progenitor cell fate in chronic liver... 
Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Cell Biology | Research & Experimental Medicine | Wnt3A Protein - metabolism | Ethionine - administration & dosage | Keratins, Hair-Specific - genetics | Receptors, Notch - metabolism | Humans | Middle Aged | Liver Diseases - pathology | Cell Communication - physiology | Male | RNA, Messenger - metabolism | Young Adult | Cell Differentiation - genetics | Intercellular Signaling Peptides and Proteins - metabolism | Liver Regeneration - genetics | Biliary Tract - metabolism | Serrate-Jagged Proteins | Adult | Antigens, Differentiation - metabolism | Female | Membrane Proteins - metabolism | Cell Differentiation - physiology | Hepatocytes - physiology | Jagged-1 Protein | Calcium-Binding Proteins - metabolism | Mice, Inbred C57BL | Cells, Cultured | Gene Expression Regulation - physiology | Mice, Transgenic | gamma-Glutamyltransferase - metabolism | Biliary Tract - physiopathology | Cell Communication - genetics | Liver Regeneration - physiology | beta Catenin - genetics | Biliary Tract - pathology | Nerve Tissue Proteins - metabolism | Macrophages - metabolism | Animals | Creatine Kinase - metabolism | Signal Transduction - physiology | Stem Cell Niche - physiology | Stem Cells - physiology | Aged | Mice | Chronic Disease | Liver diseases | Physiological aspects | Development and progression | Genetic aspects | Research | Macrophages | Cell differentiation | Wnt proteins | Signal transduction | Cellular biology | Gene expression | Index Medicus
Journal Article
Development (Cambridge), ISSN 1477-9129, 12/2004, Volume 131, Issue 23, pp. 5871 - 5881
Journal Article