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Journal of Biosciences, ISSN 0250-5991, 12/2018, Volume 43, Issue 5, pp. 835 - 856
Dysbiosis, or imbalance in the gut microbiome, has been implicated in auto-immune, inflammatory, neurological diseases as well as in cancers. More recently it... 
gut fungal microbiome | Bacterial Keratitis | gut bacterial microbiome | dysbiosis | PROTECTION | BACTEROIDES | 1ST REPORT | SYSTEMIC INFECTION | CHAIN FATTY-ACIDS | BIOLOGY | DIVERSITY | PATIENT | SKIN | EPIDEMIOLOGY | Malassezia - genetics | Kluyveromyces - isolation & purification | Aspergillus - classification | Keratitis - pathology | Clostridiales - classification | DNA, Fungal - genetics | Keratitis - microbiology | Humans | Malassezia - classification | Middle Aged | Male | Gastrointestinal Microbiome - physiology | DNA, Fungal - isolation & purification | Dysbiosis - microbiology | Ruminococcus - isolation & purification | Case-Control Studies | Dysbiosis - diagnosis | Feces - microbiology | Keratitis - diagnosis | Mortierella - isolation & purification | Faecalibacterium - genetics | Aspergillus - isolation & purification | Clostridiales - genetics | Megasphaera - isolation & purification | Adult | Aspergillus - genetics | Female | Kluyveromyces - classification | Megasphaera - genetics | Rhizopus - classification | Malassezia - isolation & purification | DNA, Bacterial - isolation & purification | Faecalibacterium - classification | Faecalibacterium - isolation & purification | Ruminococcus - genetics | Mortierella - genetics | Rhizopus - genetics | Veillonellaceae - classification | DNA, Bacterial - genetics | Megasphaera - classification | Ruminococcus - classification | Veillonellaceae - genetics | Veillonellaceae - isolation & purification | Rhizopus - isolation & purification | Clostridiales - isolation & purification | Dysbiosis - pathology | Mortierella - classification | Kluyveromyces - genetics | Physiological aspects | Keratitis | Research | Microbiota (Symbiotic organisms) | Risk factors | Pathogens | Protocols | Cornea | Dysbacteriosis | Rhizopus | Medical services | Microbiomes | Abundance | Transplantation | Patients | Diseases | Eye | Neurological diseases | Fungi | Probiotics | Blindness | Profiles | Bacteria
Journal Article
Journal Article
Journal of Leukocyte Biology, ISSN 0741-5400, 11/2017, Volume 102, Issue 5, pp. 1159 - 1171
AT‐RvD1 plays a role in controlling herpes stromal keratitis lesions. Stromal keratitis (SK) is a chronic immunopathological lesion of the eye, caused by HSV‐1... 
stromal keratitis | inflammation | resolvins | Resolvins | Stromal keratitis | Inflammation | OCULAR INFECTION | ANGIOGENESIS | RESOLUTION | CD4(+) T-CELLS | LIPID MEDIATORS | IMMUNOLOGY | ACUTE-INFLAMMATION | CELL BIOLOGY | RESPONSES | DISEASE | MICE | HEMATOLOGY | MicroRNAs - antagonists & inhibitors | Chemokine CCL2 - immunology | Humans | Corneal Neovascularization - genetics | Vascular Endothelial Growth Factor A - antagonists & inhibitors | Vascular Endothelial Growth Factor A - genetics | Herpes Simplex - genetics | Herpes Simplex - drug therapy | Th1 Cells - virology | Interleukins - genetics | Th17 Cells - drug effects | Matrix Metalloproteinase 9 - genetics | Keratitis, Herpetic - drug therapy | Corneal Neovascularization - virology | Aspirin - pharmacology | Corneal Stroma - drug effects | Th1 Cells - drug effects | Antiviral Agents - pharmacology | Signal Transduction | Neutrophils - drug effects | Vascular Endothelial Growth Factor A - immunology | MicroRNAs - immunology | Neutrophils - virology | Herpes Simplex - immunology | Interleukins - antagonists & inhibitors | Chemokine CXCL2 - immunology | Herpes Simplex - virology | Aspirin - analogs & derivatives | Th17 Cells - immunology | Mice | Mice, Inbred BALB C | MicroRNAs - genetics | Chemokine CCL2 - antagonists & inhibitors | Herpesvirus 1, Human - pathogenicity | Corneal Neovascularization - immunology | Chemokine CXCL2 - antagonists & inhibitors | Corneal Neovascularization - drug therapy | Corneal Stroma - virology | Matrix Metalloproteinase 9 - immunology | Keratitis, Herpetic - immunology | Th1 Cells - immunology | Th17 Cells - virology | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Herpesvirus 1, Human - growth & development | Interleukins - immunology | Chemokine CXCL2 - genetics | Female | Severity of Illness Index | Keratitis, Herpetic - virology | Gene Expression Regulation | Neutrophils - immunology | Chemokine CCL2 - genetics | Keratitis, Herpetic - genetics | Herpesvirus 1, Human - drug effects | Docosahexaenoic Acids - pharmacology | Animals | Corneal Stroma - pathology | Corneal Stroma - blood supply | Animal models | Cornea | Keratitis | Pathogenesis | Helper cells | Herpes simplex | Chronic infection | Viruses | Infections | Lymphocytes T | Matrix metalloproteinase | Docosahexaenoic acid | Vascularization | Interleukin 6 | Lymphocytes | Rodents | Metalloproteinase | Lesions | Stat1 protein | Vascular endothelial growth factor | Aspirin | Neutrophils | MiRNA | Interleukin 12 | Leukocytes (neutrophilic) | Cell differentiation | Gelatinase B | CD4 antigen | γ-Interferon | Interferon | Monocyte chemoattractant protein 1 | Spotlight on Leading Edge Research
Journal Article
Nature Cell Biology, ISSN 1465-7392, 01/2016, Volume 18, Issue 2, pp. 168 - 180
Chronic inflammation is associated with a variety of pathological conditions in epithelial tissues, including cancer, metaplasia and aberrant wound healing. In... 
FIBROSIS | STEM-CELLS | ACTIVATION | YAP/TAZ | SQUAMOUS METAPLASIA | EXTRACELLULAR-MATRIX | T-CELL | DIFFERENTIATION | SUPPRESSION | CANCER | CELL BIOLOGY | Administration, Ophthalmic | Keratitis - pathology | Epithelial Cells - metabolism | Mechanotransduction, Cellular - drug effects | Epithelial Cells - drug effects | Humans | Corneal Injuries - genetics | Extracellular Matrix - metabolism | Epithelium, Corneal - pathology | Phosphoproteins - metabolism | Stem Cells - metabolism | Epithelium, Corneal - injuries | Corneal Injuries - metabolism | Receptor, Notch1 - deficiency | Transfection | Time Factors | Swine | HEK293 Cells | Inflammation Mediators - metabolism | Epithelium, Corneal - drug effects | Anti-Inflammatory Agents - administration & dosage | Wnt Signaling Pathway | Disease Models, Animal | Keratitis - genetics | Corneal Injuries - pathology | Keratitis - prevention & control | Mice, Inbred C57BL | Epithelial Cells - pathology | Phosphoproteins - genetics | Transcription Factors - genetics | beta Catenin - metabolism | beta Catenin - genetics | Mice, Knockout | Transcription Factors - metabolism | Regeneration - drug effects | Animals | Cell Differentiation - drug effects | Adaptor Proteins, Signal Transducing - genetics | Keratitis - metabolism | Epithelium, Corneal - metabolism | Stem Cells - drug effects | Stem Cells - pathology | Adaptor Proteins, Signal Transducing - metabolism | Receptor, Notch1 - genetics | Chronic Disease | Physiological aspects | Cellular signal transduction | Inflammation | Epithelium | Health aspects
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 02/1999, Volume 189, Issue 4, pp. 663 - 672
Mechanisms responsible for neuroattenuation of herpes simplex virus (HSV) have been defined previously by studies of mutant viruses in cultured cells. The... 
Herpes simplex virus mutants | Mice | Interferons | Pathogenesis | MEDICINE, RESEARCH & EXPERIMENTAL | IMMEDIATE EARLY GENES | TRIGEMINAL GANGLION | DELETION MUTANT | GAMMA-INTERFERON | IMMUNOLOGY | mice | PROTEIN-SYNTHESIS | pathogenesis | RIBONUCLEOTIDE REDUCTASE | herpes simplex virus mutants | interferons | STROMAL KERATITIS | SPONTANEOUS REACTIVATION | LATENCY-ASSOCIATED TRANSCRIPTS | PREMATURE SHUTOFF | Ribonucleotide Reductases - physiology | Simplexvirus - physiology | Virus Replication - genetics | Cercopithecus aethiops | Thymidine Kinase - physiology | Simplexvirus - pathogenicity | Interferon-beta - physiology | Receptor, Interferon alpha-beta | Interferon-alpha - genetics | Virulence - genetics | Viral Proteins - physiology | Interferon-beta - genetics | Immunocompetence | Interferon-gamma - genetics | Interferons - genetics | Interferon-gamma - deficiency | Receptors, Interferon - genetics | Vero Cells | Corneal Injuries | Cornea - virology | Simplexvirus - drug effects | Keratitis, Herpetic - virology | Immediate-Early Proteins - physiology | Keratitis, Herpetic - physiopathology | Thymidine Kinase - genetics | Receptors, Interferon - deficiency | Simplexvirus - genetics | Interferon-gamma - physiology | Viral Proteins - genetics | Ribonucleases | Interferon-alpha - deficiency | Interferons - deficiency | Mice, Knockout | Ubiquitin-Protein Ligases | Phenotype | Animals | Immediate-Early Proteins - genetics | Simplexvirus - isolation & purification | Receptors, Interferon - physiology | Interferons - physiology | Ribonucleotide Reductases - genetics | Trigeminal Ganglion - virology | Interferon-beta - deficiency | Interferon-alpha - physiology
Journal Article
Journal Article