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Circulation Research, ISSN 0009-7330, 03/2015, Volume 116, Issue 6, pp. 991 - 1006
Excess weight gain, especially when associated with increased visceral adiposity, is a major cause of hypertension, accounting for 65% to 75% of the risk for... 
blood pressure | kidney | chronic renal insufficiency | melanocortins | leptin | mineralocorticoids | CARDIAC & CARDIOVASCULAR SYSTEMS | SYMPATHETIC-NERVOUS-SYSTEM | NATRIURETIC PEPTIDES | CARDIOVASCULAR FUNCTIONS | BLOOD-PRESSURE | MELANOCORTIN SYSTEM | BODY-MASS-INDEX | RENIN-ANGIOTENSIN SYSTEM | ARTERIAL-PRESSURE | SELECTIVE LEPTIN RESISTANCE | PERIPHERAL VASCULAR DISEASE | CHRONIC KIDNEY-DISEASE | HEMATOLOGY | Natriuresis - physiology | Antihypertensive Agents - pharmacology | Intra-Abdominal Fat - physiopathology | Prevalence | Dyslipidemias - complications | Humans | Sodium - metabolism | Sympathetic Nervous System - physiopathology | Pro-Opiomelanocortin - physiology | Hypertension - etiology | Leptin - physiology | Insulin Resistance - physiology | Metabolic Syndrome - physiopathology | Models, Animal | Parasympathetic Nervous System - physiopathology | Hypertension - epidemiology | Kidney - physiopathology | Models, Cardiovascular | Obesity - complications | Aldosterone - physiology | Obesity - physiopathology | Renin-Angiotensin System - physiology | Antihypertensive Agents - therapeutic use | Organ Specificity | Pressure | Renal Insufficiency, Chronic - physiopathology | Hypertension - physiopathology | Metabolic Syndrome - complications | Animals | Heart Conduction System - physiopathology | Receptors, Leptin - physiology | Obesity - epidemiology | Renin-Angiotensin System - drug effects | Hemodynamics | Dyslipidemias - physiopathology | chronic kidney disease | sympathetic activity | Blood pressure | renin-angiotensin system
Journal Article
Circulation research, ISSN 1524-4571, 2015, Volume 116, Issue 6, pp. 937 - 959
Until recently, significant advances in our understanding of the mechanisms of blood pressure regulation arose from studies of monogenic forms of hypertension... 
blood pressure | uromodulin | exome | hypertension | natriuretic peptide, brain | genome-wide association study | COMMON VARIANTS | ALDOSTERONE-PRODUCING ADENOMAS | CARDIAC & CARDIOVASCULAR SYSTEMS | KELCH-LIKE 3 | SECONDARY HYPERTENSION | SOMATIC MUTATIONS | MINERALOCORTICOID RECEPTOR | NATRIURETIC-PEPTIDE | MISSING HERITABILITY | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | AMBULATORY BLOOD-PRESSURE | GENOME-WIDE ASSOCIATION | Kidney Diseases - physiopathology | Paraganglioma - physiopathology | Oligonucleotide Array Sequence Analysis | Sodium, Dietary - adverse effects | Blood Pressure - genetics | Humans | Hypertension - drug therapy | Male | Sympathetic Nervous System - physiopathology | Hypotension - genetics | Kidney Diseases - genetics | Pheochromocytoma - physiopathology | Hypertension, Pregnancy-Induced - genetics | Bartter Syndrome - genetics | Hyperaldosteronism - physiopathology | Glucocorticoids - physiology | Female | Blood Pressure - physiology | Neoplastic Syndromes, Hereditary - genetics | Hypertension - genetics | Mineralocorticoids - physiology | Genetic Predisposition to Disease | Genome-Wide Association Study | Models, Cardiovascular | Paraganglioma - genetics | Hyperaldosteronism - genetics | Hypertension, Pregnancy-Induced - physiopathology | Renin-Angiotensin System - genetics | Renin-Angiotensin System - physiology | Hypertension - physiopathology | Pregnancy | Adrenal Gland Neoplasms - physiopathology | Sodium, Dietary - pharmacokinetics | Neoplastic Syndromes, Hereditary - physiopathology | Pheochromocytoma - genetics | Bartter Syndrome - physiopathology | Polymorphism, Single Nucleotide | Mutation | Adrenal Gland Neoplasms - genetics
Journal Article
European heart journal, ISSN 1522-9645, 2014, Volume 35, Issue 23, pp. 1515 - 1525
Vascular calcifications (VCs) are actively regulated biological processes associated with crystallization of hydroxyapatite in the extracellular matrix and in... 
Vascular molecular biology and genetics | CALCIUM-PHOSPHATE DEPOSITION | ATHEROSCLEROTIC LESIONS | FETUIN-A LEVELS | NONINVASIVE EVALUATION | CARDIAC & CARDIOVASCULAR SYSTEMS | PSEUDOXANTHOMA ELASTICUM | EXTRACELLULAR CALCIUM | MONCKEBERGS SCLEROSIS | Vascular function | ARTERY CALCIFICATION | SMOOTH-MUSCLE-CELLS | CHRONIC KIDNEY-DISEASE | Vascular calcifications | Monckeberg's media sclerosis | Tunica Media - pathology | Prognosis | Monckeberg Medial Calcific Sclerosis - physiopathology | Humans | Tunica Intima - physiopathology | Male | Monckeberg Medial Calcific Sclerosis - therapy | Diabetic Angiopathies - physiopathology | Monckeberg Medial Calcific Sclerosis - pathology | Diabetic Angiopathies - pathology | Vascular Calcification - physiopathology | Adult | Female | Biomarkers - metabolism | Arteriosclerosis - physiopathology | Terminology as Topic | Vascular Calcification - pathology | Phosphates - physiology | Renal Insufficiency, Chronic - physiopathology | Diabetes Mellitus, Type 2 - physiopathology | Tunica Intima - pathology | Calcium-Binding Proteins - physiology | Diabetes Mellitus, Type 2 - pathology | Vascular Calcification - therapy | Hyperphosphatemia - physiopathology | Tunica Media - physiopathology | Arteriosclerosis - pathology | Index Medicus | Life Sciences | Human health and pathology | Cardiology and cardiovascular system | Mönckeberg's media sclerosis | Reviews
Journal Article
Circulation research, ISSN 1524-4571, 2015, Volume 116, Issue 12, pp. 2005 - 2019
Afferent and efferent cardiac neurotransmission via the cardiac nerves intricately modulates nearly all physiological functions of the heart (chronotropy,... 
arrhythmias, cardiac | physiopathology | autonomic nervous system | death, sudden cardiac | POLYMORPHIC VENTRICULAR-TACHYCARDIA | CARDIAC & CARDIOVASCULAR SYSTEMS | death | SPINAL-CORD | sudden cardiac | ATRIAL-FIBRILLATION | RENAL SYMPATHETIC DENERVATION | arrhythmias | HEART-RATE-VARIABILITY | ACUTE MYOCARDIAL-INFARCTION | PERIPHERAL VASCULAR DISEASE | GROWTH-FACTOR | TREATMENT-RESISTANT HYPERTENSION | HEMATOLOGY | T-WAVE ALTERNANS | cardiac | AUTONOMIC NERVOUS-SYSTEM | Humans | Myocardial Contraction - physiology | Afferent Pathways - physiopathology | Death, Sudden, Cardiac - etiology | Hypertension, Renal - physiopathology | Autonomic Nervous System - physiopathology | Arrhythmias, Cardiac - physiopathology | Nerve Growth Factor - physiology | Heart Diseases - complications | Cell Transdifferentiation | Hypertension, Renal - surgery | Diabetic Neuropathies - physiopathology | Disease Models, Animal | Heart - innervation | Autonomic Nervous System Diseases - complications | Heart Diseases - physiopathology | Translational Medical Research | Nerve Regeneration | Feedback, Physiological | Animals | Autonomic Nervous System Diseases - physiopathology | Heart Conduction System - physiopathology | Semaphorin-3A - physiology | Kidney - innervation | Mice | Hemodynamics | Denervation | sudden cardiac death | Autonomic nervous system | arrhythmia | pathophysiology
Journal Article
Journal of the American College of Cardiology, ISSN 0735-1097, 2014, Volume 64, Issue 21, pp. 2281 - 2293
Journal Article