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Journal of Clinical Investigation, ISSN 0021-9738, 09/2013, Volume 123, Issue 9, pp. 4023 - 4035
Journal Article
American Journal of Physiology - Renal Physiology, ISSN 0363-6127, 03/2010, Volume 298, Issue 3, pp. F662 - F671
Kim HJ, Vaziri ND. Contribution of impaired Nrf2-Keap1 pathway to oxidative stress and inflammation in chronic renal failure. Am J Physiol Renal Physiol 298:... 
Chronic kidney disease progression | Lipid peroxidation | Superoxide dismutase | Anti-inflammatory factors | NAD(P)H oxidase | Glutathione | PHYSIOLOGY | glutathione | anti-inflammatory factors | LOW-DENSITY-LIPOPROTEIN | MASS REDUCTION | GLUTATHIONE SYNTHESIS | lipid peroxidation | chronic kidney disease progression | IN-VITRO | SYNTHASE EXPRESSION | NRF2 | superoxide dismutase | PROTECTIVE ROLE | CULTURED ENDOTHELIAL-CELLS | UROLOGY & NEPHROLOGY | ANTIOXIDANT RESPONSE | NF-KAPPA-B | Reactive Oxygen Species - metabolism | Oxidative Stress | Antioxidants - metabolism | Kidney - enzymology | Kidney - immunology | Male | Inflammation - metabolism | Kidney - metabolism | Nephrectomy | Time Factors | Inflammation Mediators - metabolism | Glutathione - deficiency | Kelch-Like ECH-Associated Protein 1 | Enzymes - metabolism | Disease Models, Animal | Kidney - surgery | Kidney Failure, Chronic - immunology | Signal Transduction | Intracellular Signaling Peptides and Proteins | Rats | Inflammation - immunology | Rats, Sprague-Dawley | Disease Progression | Kidney Failure, Chronic - metabolism | Animals | Proteins - metabolism | NF-E2-Related Factor 2 - metabolism | Lipid Peroxidation | Oxidative stress | Transcription factors | Chronic kidney failure | Development and progression | Inflammation | Diagnosis | Properties | Observations | Antioxidants | Enzymes | Tissue | Rodents | Amino acids | Oxidation | Kidney diseases | Cells | Index Medicus
Journal Article
Atherosclerosis, ISSN 0021-9150, 2015, Volume 239, Issue 2, pp. 393 - 400
Abstract Objective The pathogenic events responsible for accelerated atherosclerosis in patients with chronic renal failure (CRF) are poorly understood. Here... 
Cardiovascular | MnTBAP | chronic renal failure | UCP-1 | Endothelial cells | uncoupling protein 1 | Urea | O-linked-N-acetylglucosamine | manganese tetrakis (4-benzoic acid) porphyrin | CRF | ROS | reactive oxygen species | GlcNAc | Prostacyclin synthase | Reactive oxygen species | Chronic renal failure | Manganese tetrakis (4-benzoic acid) porphyrin | Uncoupling protein 1 | CELLS | OXIDATIVE STRESS | ATHEROSCLEROSIS | INSULIN-RESISTANCE | CARDIOVASCULAR-DISEASE | GENE-EXPRESSION | HIGH GLUCOSE | PERIPHERAL VASCULAR DISEASE | CHRONIC KIDNEY-DISEASE | HYPERGLYCEMIC DAMAGE | Tumor Necrosis Factor-alpha - metabolism | Reactive Oxygen Species - metabolism | Oxidative Stress | Humans | Cytochrome P-450 Enzyme System - metabolism | Endothelium, Vascular - drug effects | Aorta - metabolism | Urea - chemistry | Antigens, CD - metabolism | Endoglin | Atherosclerosis - enzymology | Kidney Failure, Chronic - chemically induced | Chemokine CCL2 - metabolism | Endothelium - enzymology | Intramolecular Oxidoreductases - metabolism | Interleukin-6 - metabolism | Superoxide Dismutase - metabolism | Atherosclerosis - physiopathology | Endothelial Cells - metabolism | Mice, Inbred C57BL | Receptors, Cell Surface - metabolism | Random Allocation | Atherosclerosis - metabolism | Kidney Failure, Chronic - metabolism | Catalase - metabolism | Glyceraldehyde-3-Phosphate Dehydrogenase (Phosphorylating) - metabolism | Animals | Endothelium, Vascular - pathology | Mice | Vascular Cell Adhesion Molecule-1 - metabolism | Chronic kidney failure | Atherosclerosis | Endothelium | Index Medicus
Journal Article
American Journal of Physiology - Renal Physiology, ISSN 1931-857X, 08/2009, Volume 297, Issue 2, pp. 410 - 419
Elevated level of homocysteine (Hcy), known as hyperhomocysteinemia (HHcy), is associated with end-stage renal diseases. Hcy metabolizes in the body to produce... 
Cystathionine-γ lyase | Matrix metalloproteinase | Homocysteine | Cystathionine-β synthase | homocysteine | PHYSIOLOGY | RAT | H2S | cystathionine-beta synthase | GLOMERULAR INJURY | PROTECTS | MURINE MODEL | PODOCYTE INJURY | INDUCED OXIDATIVE STRESS | UROLOGY & NEPHROLOGY | matrix metalloproteinase | cystathionine-gamma lyase | PPAR-GAMMA AGONIST | EXPRESSION | Hydrogen Sulfide - metabolism | Kidney - pathology | Apoptosis - drug effects | Glutathione - metabolism | Antioxidants - metabolism | Hyperhomocysteinemia - metabolism | Male | Glutathione Disulfide - metabolism | Proteinuria - prevention & control | Kidney - metabolism | Matrix Metalloproteinase 9 - metabolism | Nephrectomy | Superoxides - metabolism | Membrane Proteins - metabolism | Sulfides - metabolism | Cystathionine beta-Synthase - genetics | Disease Models, Animal | Sulfides - pharmacology | Kidney - drug effects | Matrix Metalloproteinase 2 - metabolism | Mice, Inbred C57BL | Homocysteine - blood | Proteinuria - metabolism | Antioxidants - pharmacology | Cystathionine beta-Synthase - metabolism | Hydrogen Sulfide - blood | Hyperhomocysteinemia - drug therapy | Kidney Failure, Chronic - metabolism | Mice, Knockout | Kidney Failure, Chronic - genetics | Animals | Kidney Failure, Chronic - prevention & control | Desmin - metabolism | Hyperhomocysteinemia - genetics | Mice | Oxidative Stress - drug effects | Hydrogen sulfide | Proteins | Antioxidants | Plasma | Rodents | Kidney diseases | Apoptosis | Index Medicus | cystathionine-β synthase | cystathionine-γ lyase
Journal Article
Circulation, ISSN 0009-7322, 04/2009, Volume 119, Issue 13, pp. 1785 - 1794
Background-Clinical studies have demonstrated that 50% of individuals with chronic renal disease (CRD) die of cardiovascular causes, including advanced... 
Calcification | Chronic | Kidney failure | Aortic valve | Elastin | Atherosclerosis | atherosclerosis | kidney failure, chronic | aortic valve | ELASTIN PEPTIDES | CARDIAC & CARDIOVASCULAR SYSTEMS | ATHEROSCLEROTIC PLAQUES | VASCULAR CALCIFICATION | elastin | POTENTIAL MECHANISM | FAILURE | IN-VIVO | CYSTATIN-C | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | calcification | CHRONIC KIDNEY-DISEASE | VULNERABLE PLAQUE | Tunica Media - pathology | Osteogenesis - physiology | Cystatin C - blood | Humans | Carotid Artery Diseases - physiopathology | Aorta - metabolism | Aortic Valve - pathology | Nephrectomy | Carotid Artery Diseases - pathology | Mice, Mutant Strains | Tunica Media - metabolism | Phosphates - blood | Calcinosis - metabolism | Carotid Artery Diseases - metabolism | Phosphates - pharmacology | Muscle, Smooth, Vascular - physiology | Kidney Failure, Chronic - physiopathology | Elastin - metabolism | Mice, Inbred C57BL | Osteogenesis - drug effects | Aortic Valve Stenosis - physiopathology | Cells, Cultured | Elastin - pharmacology | Muscle, Smooth, Vascular - cytology | Aortic Valve - metabolism | Kidney Failure, Chronic - metabolism | Aorta - pathology | Tunica Intima - metabolism | Animals | Aortic Valve Stenosis - pathology | Tunica Intima - pathology | Cathepsins - genetics | Cathepsins - metabolism | Apolipoproteins E - genetics | Creatinine - blood | Aortic Valve Stenosis - metabolism | Mice | Calcinosis - pathology | Cathepsins - deficiency | Calcinosis - physiopathology | Cathepsins | Physiological aspects | Research | Chronic kidney failure | Diseases | Index Medicus | Abridged Index Medicus
Journal Article
Journal of Molecular Medicine, ISSN 0946-2716, 8/2014, Volume 92, Issue 8, pp. 871 - 879
Journal Article
Molecular and Cellular Biochemistry, ISSN 0300-8177, 11/2011, Volume 357, Issue 1, pp. 171 - 179
Journal Article
Science Translational Medicine, ISSN 1946-6234, 03/2013, Volume 5, Issue 175, pp. 175ra29 - 175ra29
Urea, the toxic end product of protein catabolism, is elevated in end-stage renal disease (ESRD), although it is unclear whether or how it contributes to... 
Kidney Failure, Chronic - mortality | Amino Acids - blood | Humans | Risk Factors | Kidney Failure, Chronic - metabolism | Serum Albumin - metabolism | MEDICINE, RESEARCH & EXPERIMENTAL | CELL BIOLOGY | Index Medicus
Journal Article