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Clinical journal of the American Society of Nephrology, ISSN 1555-905X, 2014, Volume 9, Issue 8, pp. 1461 - 1469
Journal Article
Journal Article
Journal of the American Society of Nephrology, ISSN 1046-6673, 04/2010, Volume 21, Issue 4, pp. 587 - 597
The peak prevalence of ESRD from glomerulosclerosis occurs at 70 to 79 years. To understand why old glomeruli are prone to failure, we analyzed the Fischer 344... 
KIDNEY-DISEASE | GLOMERULOSCLEROSIS | MODULE | CALORIC RESTRICTION | GROWTH | RATS | UROLOGY & NEPHROLOGY | RECEPTOR | PATHOLOGY | SIRTUINS | AGE | Basic Research
Journal Article
Journal of controlled release, ISSN 0168-3659, 2017, Volume 255, pp. 94 - 107
Journal Article
PloS one, ISSN 1932-6203, 2012, Volume 7, Issue 7, p. e40589
Podocyte or endothelial cell VEGF-A knockout causes thrombotic microangiopathy in adult mice. To study the mechanism involved in acute and local injury caused... 
Neuropilin-1 - metabolism | Vascular Endothelial Growth Factor A - metabolism | Proteinuria - pathology | Integrin alphaVbeta3 - metabolism | Gene Knockdown Techniques | Renal Insufficiency - physiopathology | Protein Binding - drug effects | Models, Animal | Renal Insufficiency - complications | Blood Pressure - drug effects | Podocytes - metabolism | Cells, Cultured | Proteinuria - metabolism | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Renal Insufficiency - metabolism | Podocytes - pathology | Down-Regulation - drug effects | Fibronectins - metabolism | Proteinuria - physiopathology | Endothelium - drug effects | Phenotype | Animals | Endothelium - metabolism | Podocytes - drug effects | Signal Transduction - drug effects | Renal Insufficiency - pathology | Doxycycline - pharmacology | Mice | Proteinuria - complications | RNA, Small Interfering - metabolism | Pediatrics | Neuropilin | Activation | mRNA | Fibronectin | Angiogenesis | Cell activation | Ultrastructure | Transgenic animals | Rodents | Extracellular matrix | Vascular endothelial growth factor | Urine | Hypertension | Thrombotic microangiopathy | Kidneys | Permeability | Laminates | Gene expression | Endothelial cells | Endothelium | Medicine | Pathology | Signaling | Lamination | Doxycycline | Renal failure | Glomerulus | Diabetes | Cell size | Proteinuria
Journal Article
Journal Article
Diabetes (New York, N.Y.), ISSN 1939-327X, 2012, Volume 61, Issue 11, pp. 2958 - 2966
...). We then assessed the extent of glomerular dysfunction by measuring proteinuria, glomerular pathology, and glomerular cell apoptosis... 
OVEREXPRESSION | RENAL CHANGES | INHIBITION | ENDOCRINOLOGY & METABOLISM | INJURY | RETINOPATHY | POLYMORPHISM | THROMBOTIC MICROANGIOPATHY | NEPHROPATHY | EXPRESSION | ENDOTHELIAL GROWTH-FACTOR | Kidney Glomerulus - blood supply | Humans | Microvessels - metabolism | Kidney Glomerulus - physiopathology | Microvessels - pathology | Vascular Endothelial Growth Factor A - metabolism | Diabetes Mellitus, Type 1 - complications | Vascular Endothelial Growth Factor A - antagonists & inhibitors | RNA, Messenger - metabolism | Vascular Endothelial Growth Factor A - genetics | Diabetic Nephropathies - physiopathology | Kidney Glomerulus - metabolism | Proteinuria - etiology | Diabetic Nephropathies - urine | Recombinant Proteins - metabolism | Diabetic Nephropathies - pathology | Recombinant Proteins - antagonists & inhibitors | Podocytes - metabolism | Diabetic Nephropathies - metabolism | Cells, Cultured | Mice, Transgenic | Kidney Glomerulus - pathology | Podocytes - pathology | Random Allocation | Disease Progression | Mice, Knockout | Up-Regulation - drug effects | Animals | Endothelium, Vascular - metabolism | Diabetes Mellitus, Type 1 - blood | Endothelium, Vascular - pathology | Hyperglycemia - etiology | Mice | Apoptosis | Diabetic nephropathies | Physiological aspects | Genetic aspects | Research | Kidney glomerulus | Vascular endothelial growth factor | Risk factors | Complications
Journal Article
Journal Article