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Development, ISSN 0950-1991, 02/2008, Volume 135, Issue 4, pp. 609 - 620
Journal Article
Journal Article
Journal Article
Nature Medicine, ISSN 1078-8956, 04/2017, Volume 23, Issue 4, pp. 429 - 438
Journal Article
Transplantation, ISSN 0041-1337, 09/2015, Volume 99, Issue 9, pp. 1807 - 1816
Journal Article
Diabetes, ISSN 0012-1797, 03/2017, Volume 66, Issue 3, pp. 763 - 778
The molecular signaling mechanisms between glomerular cell types during initiation/progression of diabetic kidney disease (DKD) remain poorly understood. We... 
GLOMERULOSCLEROSIS | CELLS | OXIDATIVE STRESS | ANTAGONIST | ATRASENTAN | MICROALBUMINURIA | DNA-DAMAGE | ENDOCRINOLOGY & METABOLISM | MICE | NEPHROPATHY | PODOCYTE DETACHMENT | Humans | Middle Aged | Diabetes Mellitus, Type 1 - metabolism | Male | Albuminuria | Chromatography, High Pressure Liquid | Young Adult | Kidney Glomerulus - ultrastructure | Flow Cytometry | Piperidines - pharmacology | Kidney Glomerulus - metabolism | Mesangial Cells - ultrastructure | Organophosphorus Compounds - pharmacology | Adult | Female | Mice, Inbred DBA | Diabetes Mellitus, Experimental - metabolism | Real-Time Polymerase Chain Reaction | Deoxyguanosine - analogs & derivatives | Microscopy, Electron, Scanning | Disease Susceptibility | DNA, Mitochondrial - metabolism | Deoxyguanosine - urine | Enzyme-Linked Immunosorbent Assay | Signal Transduction | Endothelin-1 - metabolism | Diabetic Nephropathies - metabolism | Kidney Glomerulus - drug effects | Mice, Inbred C57BL | Oxygen Consumption | Mitochondria - metabolism | Antioxidants - pharmacology | Kidney Glomerulus - pathology | Mitochondria - drug effects | Podocytes - pathology | Endothelium - drug effects | Podocytes - ultrastructure | Animals | Endothelium - metabolism | Endothelium - ultrastructure | Mesangial Cells - pathology | Receptor, Endothelin A - metabolism | Aged | Mice | Mitochondria | Reactive oxygen species | Genetic aspects | Research | Genetic transcription | Diabetic nephropathies | Complications | Klinisk medicin | Clinical Medicine
Journal Article
Arthritis & Rheumatology, ISSN 2326-5191, 08/2017, Volume 69, Issue 8, pp. 1636 - 1646
Objective Development of proteinuria in lupus nephritis (LN) is associated with podocyte dysfunction. The NLRP3 inflammasome has been implicated in the... 
CRESCENTIC GLOMERULONEPHRITIS | CELLS | INTERLEUKIN-1 RECEPTOR ANTAGONIST | MAJOR SOURCE | INJURY | MECHANISMS | MICE | RHEUMATOLOGY | KIDNEY | DAMAGE | ERYTHEMATOSUS | Interleukin-1beta - drug effects | Reactive Oxygen Species - metabolism | Humans | Kidney - immunology | Caspase 1 - metabolism | Caspase 1 - immunology | Proteinuria - pathology | Sulfones - pharmacology | Heterocyclic Compounds, 4 or More Rings - pharmacology | Kidney Glomerulus - ultrastructure | Kidney - metabolism | Flow Cytometry | Interleukin-1beta - metabolism | Kidney Glomerulus - metabolism | Lupus Nephritis - metabolism | Lupus Nephritis - pathology | Cell Line | Microscopy, Electron, Transmission | Kidney - drug effects | Inflammasomes | Proteinuria - immunology | Kidney Glomerulus - drug effects | NLR Family, Pyrin Domain-Containing 3 Protein - antagonists & inhibitors | Interleukin-1beta - immunology | Proteinuria - metabolism | Lupus Nephritis - immunology | Blotting, Western | Kidney - ultrastructure | Microscopy, Confocal | Podocytes - ultrastructure | Animals | NLR Family, Pyrin Domain-Containing 3 Protein - immunology | Podocytes - drug effects | Caspase 1 - drug effects | Podocytes - immunology | Mice | Kidney Glomerulus - immunology | Flow cytometry | Reactive oxygen species | Nephritis | Pathogenesis | Fluorescence | Activation | Lupus nephritis | Tissues | Caspase-1 | Ultrastructure | Rodents | Lesions | Activation analysis | Lupus | Kidneys | Caspase | Patients | Feet | Cytometry | Inhibitors | Biopsy | Autoimmune diseases | In vitro methods and tests | Proteinuria | Kidney transplantation | lupus nephritis | NLRP3 inflammasome | IL-1β | podocytes
Journal Article
Journal Article
PROTEOMICS, ISSN 1615-9853, 10/2016, Volume 16, Issue 20, pp. 2706 - 2717
Kidney aging together with related renal disease had become a major clinical problem. Understanding the mechanisms of aging was important for suspending... 
Aging | FHL2 | Animal proteomics | Tubule | Glomerular | Proteomics | APOPTOSIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | BIOCHEMICAL RESEARCH METHODS | PALMITIC ACID | TRANSCRIPTIONAL COACTIVATOR | TBX3 | P53 | EPITHELIAL-CELLS | PATHWAY | LIM-DOMAIN PROTEINS | EXPRESSION | LIM-Homeodomain Proteins - metabolism | Oxidative Stress | Rats, Inbred F344 | Kidney Glomerulus - ultrastructure | Tandem Mass Spectrometry | Cyclin-Dependent Kinase Inhibitor p16 - analysis | Cellular Senescence | Muscle Proteins - metabolism | Chromatography, Liquid | T-Box Domain Proteins - analysis | LIM-Homeodomain Proteins - analysis | Kidney Glomerulus - cytology | Cell Line | Actin Cytoskeleton - metabolism | Kidney Tubules - physiology | Kidney Tubules - ultrastructure | Kidney Glomerulus - physiology | Proteome - analysis | T-Box Domain Proteins - metabolism | Kidney Tubules - cytology | Transcription Factors - metabolism | Animals | Muscle Proteins - analysis | Cyclin-Dependent Kinase Inhibitor p16 - metabolism | Transcription Factors - analysis | Proteome - metabolism | Physiological aspects | Kidney diseases | Muscle proteins | Cell death | FHL2 protein | Oxidative stress | Senescence | Kidneys | Oxidative metabolism | Rats | Galactosidase | Inflammatory response | Inflammation | Metabolism | Renal tubules | Golgi apparatus | Proteins | LIM protein | Actin | Rodents | Glomerulus | Bioinformatics
Journal Article