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Journal of Pathology, ISSN 0022-3417, 2013, Volume 229, Issue 5, pp. 645 - 659
Journal Article
Kidney International, ISSN 0085-2538, 12/2010, Volume 78, Issue 11, pp. 1171 - 1177
Journal Article
Journal Article
Journal of the American Society of Nephrology, ISSN 1046-6673, 10/2009, Volume 20, Issue 10, pp. 2147 - 2153
Renal primary cilia are sensory antennas required for the maintenance of normal epithelial differentiation and proliferation in the kidney, but they also have... 
PATHWAYS | LOCALIZATION | REPAIR | EPITHELIAL-CELLS | POLYCYSTIC KIDNEY-DISEASE | POSTISCHEMIC KIDNEY | INJURY | UROLOGY & NEPHROLOGY | MECHANOSENSATION | FAILURE | Cilia - pathology | Kidney - blood supply | Kidney - pathology | Reperfusion Injury - pathology | Humans | Middle Aged | Male | Kidney Transplantation | Animals | Biopsy | Female | Aged | Mice | Kidney Tubular Necrosis, Acute - pathology | Index Medicus | Basic Research
Journal Article
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 05/2009, Volume 119, Issue 5, pp. 1275 - 1285
The mechanism of mitochondrial damage, a key contributor to renal tubular cell death during acute kidney injury, remains largely unknown. Here, we have... 
MEDICINE, RESEARCH & EXPERIMENTAL | BCL-2 PROTEIN FAMILY | ACUTE-RENAL-FAILURE | DEATH | MECHANISMS | FISSION | CYTOCHROME-C RELEASE | APOPTOTIC PATHWAYS | MAMMALIAN-CELLS | CISPLATIN NEPHROTOXICITY | DRP1 | Adenosine Triphosphate - deficiency | Kidney Tubular Necrosis, Acute - chemically induced | RNA, Small Interfering - genetics | Dynamins - metabolism | Microtubule-Associated Proteins - genetics | Apoptosis - drug effects | Male | GTP Phosphohydrolases - antagonists & inhibitors | Kidney Tubular Necrosis, Acute - prevention & control | Mitochondria - ultrastructure | Imaging, Three-Dimensional | Cell Line | Kidney Tubules, Proximal - cytology | Reperfusion Injury - pathology | Cytochromes c - metabolism | Mice, Inbred C57BL | Cells, Cultured | Enzyme Inhibitors - pharmacology | Dynamins - genetics | Rats | Mitochondria - metabolism | Cisplatin - pharmacology | Microtubule-Associated Proteins - antagonists & inhibitors | Mitochondria - pathology | Caspase Inhibitors | Enzyme Inhibitors - therapeutic use | Animals | Reperfusion Injury - prevention & control | GTP Phosphohydrolases - genetics | Mice | Proto-Oncogene Proteins c-bcl-2 - genetics | Sodium Azide - pharmacology | Kidney Tubular Necrosis, Acute - pathology | Cell culture | Complications and side effects | Physiological aspects | Models | Mitochondrial diseases | Research | Management | Kidney diseases | Drug therapy | Risk factors | Apoptosis | Index Medicus | Abridged Index Medicus
Journal Article
American Journal of Physiology - Renal Physiology, ISSN 0363-6127, 06/2017, Volume 312, Issue 6, pp. F1166 - F1183
Acute kidney injury (AKI) causes severe morbidity, mortality, and chronic kidney disease (CKD). Mortality is particularly marked in the elderly and with... 
Innate immunity | Mitochondria | Chronic kidney disease | Acute kidney injury | Matrix metalloproteinase-2 | matrix metalloproteinase-2 | chronic kidney disease | PHYSIOLOGY | mitochondria | APOE MICE DEFICIENT | HEART-FAILURE | CELL-DEATH | ISCHEMIA-REPERFUSION INJURY | HYPERPOLARIZED C-13 DEHYDROASCORBATE | MURINE MODEL | MITOCHONDRIAL PERMEABILITY TRANSITION | innate immunity | INCREASED SUSCEPTIBILITY | IN-VIVO | UROLOGY & NEPHROLOGY | acute kidney injury | RENAL INJURY | Mitochondria - enzymology | Myocardial Infarction - genetics | Reactive Oxygen Species - metabolism | Age Factors | Kidney Tubular Necrosis, Acute - genetics | Oxidative Stress | Heart Failure - enzymology | Humans | Acute Kidney Injury - genetics | Coronary Artery Disease - enzymology | Mitochondria - ultrastructure | Necrosis | Kidney Tubular Necrosis, Acute - immunology | Myocardial Infarction - pathology | Coronary Artery Disease - pathology | Membrane Potential, Mitochondrial | Acute Kidney Injury - immunology | Kidney Tubules, Proximal - enzymology | Reperfusion Injury - genetics | Disease Models, Animal | Kidney Tubules, Proximal - ultrastructure | Myocardial Infarction - enzymology | Genetic Predisposition to Disease | Kidney Tubules, Proximal - immunology | Reperfusion Injury - pathology | Signal Transduction | Acute Kidney Injury - pathology | Isoenzymes | Kidney Tubular Necrosis, Acute - enzymology | Matrix Metalloproteinase 2 - metabolism | Reperfusion Injury - enzymology | Heart Failure - genetics | Mice, Transgenic | Heart Failure - pathology | Acute Kidney Injury - enzymology | Immunity, Innate | Mice, Knockout | Mitochondrial Degradation | Matrix Metalloproteinase 2 - genetics | Phenotype | Animals | Reperfusion Injury - immunology | Coronary Artery Disease - genetics | Kidney Tubular Necrosis, Acute - pathology | Development and progression | Kidney tubules | Health aspects | Metalloproteins | Acute renal failure | Oxidative stress | Animal models | Syngeneic grafts | Epithelial cells | Matrix metalloproteinase | Proteins | Matrix | Reperfusion | Ischemia | Rodents | Metalloproteinase | Heart diseases | Mortality | Transgenic mice | Permeability | Morbidity | Gelatinase A | Arteriosclerosis | Fibrosis | Kidney diseases | Intracellular | Geriatrics | Kidney transplantation | Index Medicus
Journal Article
Kidney International, ISSN 0085-2538, 02/2017, Volume 91, Issue 2, pp. 387 - 401
Journal Article
Kidney International, ISSN 0085-2538, 02/2017, Volume 91, Issue 2, pp. 352 - 364
Journal Article