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Journal Article
Developmental Biology, ISSN 0012-1606, 2011, Volume 352, Issue 1, pp. 58 - 69
Wnt4 and β-catenin are both required for nephrogenesis, but studies using TCF-reporter mice suggest that canonical Wnt signaling is not activated in... 
β-Catenin | Wnt | Peptidomimetic | Kidney | Mesenchymal–epithelial transition | Tubulogenesis | Mesenchymal-epithelial transition | TRANSCRIPTION FACTOR SNAIL | KIDNEY DEVELOPMENT | DEVELOPMENTAL BIOLOGY | CELL GROWTH | EPITHELIAL TRANSFORMATION | NUCLEAR-LOCALIZATION | NF-ATC | SIGNALING PATHWAY | beta-Catenin | MOUSE KIDNEY | HOMEOBOX GENE CUX-1 | Wnt Proteins - pharmacology | Transcription, Genetic - drug effects | Transcriptional Activation - genetics | Humans | Transcriptional Activation - drug effects | Mesoderm - drug effects | Mesoderm - cytology | TCF Transcription Factors - metabolism | Cell Differentiation - genetics | Nephrons - cytology | beta Catenin - chemistry | Kidney Tubules - metabolism | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - metabolism | Nephrons - drug effects | Kidney Tubules - drug effects | Mesoderm - embryology | Nephrons - embryology | Kidney Tubules - embryology | Cells, Cultured | Gene Expression Regulation, Developmental - drug effects | Rats | Genes, Reporter - genetics | Enzyme Activation - drug effects | beta Catenin - metabolism | Kidney Tubules - cytology | Animals | Signal Transduction - drug effects | Calcium Signaling - drug effects | Cell Differentiation - drug effects | Models, Biological | Ionomycin - pharmacology | Morphogenesis - drug effects | Mesoderm - metabolism | Mice | Nephrons - metabolism | Wnt4 Protein | Index Medicus | β-catenin | kidney | peptidomimetic | mesenchymal-epithelial transition | tubulogenesis
Journal Article
Toxicology and Applied Pharmacology, ISSN 0041-008X, 12/2013, Volume 273, Issue 2, pp. 345 - 354
Kidney ischemia–reperfusion (I/R) injury elicits cellular injury in the proximal tubule, and mitochondrial dysfunction is a pathological consequence of I/R.... 
SRT1720 | Proximal tubule | Mitochondrial biogenesis | Mitochondrial dysfunction | Ischemia–reperfusion | Acute kidney injury | Ischemia-reperfusion | PGC-1-ALPHA | EPITHELIAL POLARITY | SIRT1 | ENERGY-EXPENDITURE | OXIDANT INJURY | PHARMACOLOGY & PHARMACY | TOXICOLOGY | PROMOTES RECOVERY | HISTONE DEACETYLASE | SMALL-MOLECULE ACTIVATORS | CALORIE RESTRICTION | Kidney - pathology | Recovery of Function - drug effects | Reperfusion Injury - drug therapy | Male | Heterocyclic Compounds, 4 or More Rings - pharmacology | Kidney - metabolism | Heterocyclic Compounds, 4 or More Rings - therapeutic use | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Recovery of Function - physiology | Homeostasis - drug effects | Kidney Tubules - pathology | Kidney Tubules - metabolism | Reperfusion Injury - metabolism | Kidney - drug effects | Kidney Tubules - drug effects | Reperfusion Injury - pathology | Sirtuin 1 - biosynthesis | Rats | Transcription Factors - biosynthesis | Mitochondria - metabolism | Mitochondria - drug effects | Mitochondria - pathology | Rats, Sprague-Dawley | Animals | Homeostasis - physiology | Index Medicus | HOMEOSTASIS | INJURIES | EPITHELIUM | MITOCHONDRIA | CREATININE | RATS | RECEPTORS | 60 APPLIED LIFE SCIENCES | ATP | POTASSIUM IONS | SODIUM IONS | mitochondrial dysfunction | acute kidney injury | ischemia-reperfusion | proximal tubule | mitochondrial biogenesis
Journal Article
Nature Biotechnology, ISSN 1087-0156, 05/2010, Volume 28, Issue 5, pp. 436 - 440
  There is a paucity of biomarkers that reliably detect nephrotoxicity. The Predictive Safety Testing Consortium (PSTC) faced several challenges in identifying... 
INJURY | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | QUALIFICATION | Biomarkers, Pharmacological - urine | Animals | Kidney Tubules - drug effects | Drug-Related Side Effects and Adverse Reactions | Humans | Kidney Tubules - injuries | Toxicity Tests - standards | Biomarkers | Patient safety | Medical research | Kidney diseases | Toxicity | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2015, Volume 10, Issue 8, pp. e0135083 - e0135083
The chemotherapeutic use of cisplatin is limited by its severe side effects. In this study, by conducting different omics data analyses, we demonstrated that... 
APOPTOSIS | OXIDATIVE STRESS | P53 ACTIVATION | PROTEIN-KINASE | MULTIDISCIPLINARY SCIENCES | TUBULAR EPITHELIAL-CELLS | DOWN-REGULATION | AUDITORY CELLS | DEATH | CANCER | NEPHROTOXICITY | L-Lactate Dehydrogenase - metabolism | Epithelial Cells - metabolism | Reactive Oxygen Species - metabolism | Apoptosis - drug effects | Epithelial Cells - drug effects | Humans | Male | Membrane Potential, Mitochondrial - drug effects | Glycolysis - drug effects | Reactive Oxygen Species - agonists | Tumor Suppressor Protein p53 - genetics | Antineoplastic Agents - toxicity | Dose-Response Relationship, Drug | Cisplatin - toxicity | Tumor Suppressor Protein p53 - agonists | Epithelial Cells - cytology | Kidney Tubules - metabolism | Cell Line | Kidney Tubules - drug effects | Citric Acid Cycle - drug effects | Gene Expression Regulation | Injections, Intraperitoneal | Tumor Suppressor Protein p53 - metabolism | Rats | Rats, Sprague-Dawley | Kidney Tubules - cytology | Animals | L-Lactate Dehydrogenase - genetics | Acetylcysteine - pharmacology | Cell Proliferation - drug effects | Physiological aspects | Reactive oxygen species | Genetic aspects | Research | Cisplatin | Bioengineering | Tricarboxylic acid cycle | Oxidative stress | Correlation | Toxicity | p53 Protein | Genes | Science | Cytotoxicity | Kinases | Ovarian cancer | Mitochondria | Cell growth | Pathways | Metabolites | Rodents | Cell cycle | Inhibition | Adenosine triphosphate | Urine | Abnormalities | Mortality | Data processing | Interdisciplinary aspects | Metabolism | Gene expression | Side effects | Cell death | Glycolysis | Apoptosis | Index Medicus
Journal Article
Nephrology Dialysis Transplantation, ISSN 0931-0509, 09/2015, Volume 30, Issue 9, pp. 1497 - 1506
Journal Article
Kidney International, ISSN 0085-2538, 08/2014, Volume 86, Issue 2, pp. 350 - 357
Journal Article
Journal Article
American Journal of Physiology - Renal Physiology, ISSN 0363-6127, 12/2017, Volume 313, Issue 6, pp. F1209 - F1215
Afferent arteriole (Af-Art) resistance is modulated by two intrinsic nephron feedbacks: 1) the vasoconstrictor tubuloglomerular feedback (TGF) mediated by... 
High salt intake | Afferent arteriole | Connecting tubule glomerular feedback | Tubuloglomerular feedback | Dahl salt-sensitive | SENSITIVE RATS | PHYSIOLOGY | high salt intake | MACULA DENSA CELLS | connecting tubule glomerular feedback | RENAL HEMODYNAMICS | PRESSURE | RENIN-ANGIOTENSIN SYSTEM | ENHANCEMENT | UROLOGY & NEPHROLOGY | DIETARY SALT | afferent arteriole | FILTRATION-RATE | tubuloglomerular feedback | HYPERTENSIVE-RATS | Kidney Glomerulus - blood supply | Kidney Tubules - physiopathology | Solute Carrier Family 12, Member 1 - antagonists & inhibitors | Amiloride - analogs & derivatives | Male | Epithelial Sodium Channels - metabolism | Sodium-Hydrogen Exchangers - metabolism | Epithelial Sodium Channels - drug effects | Time Factors | Arterioles - physiopathology | Renal Circulation - drug effects | Rats, Inbred Dahl | Kidney Tubules - metabolism | Sodium-Hydrogen Exchangers - antagonists & inhibitors | Solute Carrier Family 12, Member 1 - metabolism | Vascular Resistance | Kidney Tubules - drug effects | Sodium Chloride, Dietary - adverse effects | Vasoconstriction | Arterioles - drug effects | Feedback, Physiological | Animals | Arterioles - metabolism | Amiloride - pharmacology | Vasodilation - drug effects | Furosemide - pharmacology | Salt | Blood pressure | Rodents | Medical treatment | Index Medicus
Journal Article
Kidney International, ISSN 0085-2538, 08/2015, Volume 88, Issue 2, pp. 226 - 234
Drug-induced kidney disease is a frequent cause of renal dysfunction; however, there are no standards to identify and characterize the spectrum of these... 
acute kidney injury | hypersensitivity | nephrotoxicity | glomerulonephritis | drugs | adverse reaction | INJURY | UROLOGY & NEPHROLOGY | EXPERIENCE | AKI | Glycosuria - chemically induced | Kidney Tubules - physiopathology | Nephrolithiasis - chemically induced | Humans | Kidney Glomerulus - physiopathology