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Kidney International, ISSN 0085-2538, 03/2018, Volume 93, Issue 3, pp. 568 - 579
Renal tubules are the major component of the kidney and are vulnerable to a variety of injuries including hypoxia, proteinuria, toxins, metabolic disorders,... 
chronic kidney disease | acute kidney injury | renal fibrosis | renal tubule | renal inflammation | ACTIVATED PROTEIN-KINASE | CELL-CYCLE ARREST | GLOMERULAR EPITHELIAL-CELLS | ISCHEMIA-REPERFUSION INJURY | TO-MESENCHYMAL TRANSITION | TISSUE GROWTH-FACTOR | RENIN-ANGIOTENSIN SYSTEM | II-INDUCED HYPERTROPHY | UROLOGY & NEPHROLOGY | NF-KAPPA-B | PERICYTE-MYOFIBROBLAST TRANSITION | Cell Proliferation | Epithelial Cells - metabolism | Renal Insufficiency, Chronic - etiology | Humans | Renal Insufficiency, Chronic - metabolism | Acute Kidney Injury - complications | Epithelial-Mesenchymal Transition | Inflammation Mediators - metabolism | Acute Kidney Injury - immunology | Kidney Tubules - pathology | Kidney Tubules - metabolism | Cytokines - immunology | Inflammation Mediators - immunology | Kidney Tubules - immunology | Renal Insufficiency, Chronic - immunology | Cytokines - metabolism | Signal Transduction | Acute Kidney Injury - pathology | Cell Cycle Proteins - metabolism | Epithelial Cells - pathology | Immunity, Innate | Disease Progression | Renal Insufficiency, Chronic - pathology | Cell Cycle Checkpoints | Animals | Energy Metabolism | Epithelial Cells - immunology | Fibrosis | Acute Kidney Injury - metabolism | Senescence | Kidneys | Immune response | Mesenchyme | Epithelial cells | Inflammation | Epithelium | Drug development | Renal tubules | Cell death | Hypoxia | Metabolic disorders | Proteinuria
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 01/2013, Volume 123, Issue 1, pp. 236 - 246
Nephrocalcinosis, acute calcium oxalate (CaOx) nephropathy, and renal stone disease can lead to inflammation and subsequent renal failure, but the underlying... 
KIDNEY-DISEASE | NEPHROLITHIASIS | MEDICINE, RESEARCH & EXPERIMENTAL | DANGER | ACTIVATION | DENDRITIC CELLS | NLRP3 INFLAMMASOME | STONE FORMATION | FAILURE | NEPHROPATHY | NALP3 INFLAMMASOME | Interleukin-18 - immunology | Cytoskeletal Proteins - genetics | NLR Family, Pyrin Domain-Containing 3 Protein | Dendritic Cells - immunology | Nephrocalcinosis - immunology | Receptors, Interleukin-1 - genetics | Interleukin-1beta - genetics | Inflammation - metabolism | Nephrocalcinosis - metabolism | Myeloid Differentiation Factor 88 - immunology | Cytoskeletal Proteins - metabolism | Kidney Tubules - pathology | Immunologic Deficiency Syndromes - immunology | Kidney Tubules - metabolism | Carrier Proteins - immunology | Dendritic Cells - metabolism | Interleukin-1beta - secretion | Kidney Tubules - immunology | Calcium Oxalate - immunology | Receptors, Interleukin-1 - immunology | Myeloid Differentiation Factor 88 - genetics | Interleukin-1beta - immunology | Inflammation - immunology | Calcium Oxalate - metabolism | Mice, Knockout | Receptors, Interleukin-1 - metabolism | Carrier Proteins - genetics | Nephrocalcinosis - pathology | Animals | Apoptosis Regulatory Proteins | Carrier Proteins - metabolism | Cytoskeletal Proteins - immunology | Nephrocalcinosis - genetics | CARD Signaling Adaptor Proteins | Immunologic Deficiency Syndromes - genetics | Immunologic Deficiency Syndromes - metabolism | Inflammation - genetics | Mice | Interleukin-18 - genetics | Myeloid Differentiation Factor 88 - metabolism | Interleukin-18 - metabolism | Phagocytosis
Journal Article
Kidney International, ISSN 0085-2538, 05/2017, Volume 91, Issue 5, pp. 1014 - 1021
Journal Article
Journal Article
Journal of Immunology, ISSN 0022-1767, 02/2011, Volume 186, Issue 3, pp. 1849 - 1860
Journal Article
Journal Article
Journal of clinical investigation, ISSN 0021-9738, 2005, Volume 115, Issue 10, pp. 2894 - 2903
Journal Article
American Journal of Physiology - Renal Physiology, ISSN 0363-6127, 2015, Volume 308, Issue 8, pp. F899 - F909
Nephrotoxicity is a frequent complication of cisplatin-induced chemotherapy, in which T cells are known to promote acute kidney injury (AKI). Apoptosis and... 
Rho-kinase | Cisplatin | AKI | ACTIVATION | PHYSIOLOGY | INJURY | ALPHA | IDENTIFICATION | FAILURE | FASUDIL | INTERLEUKIN-18 | UROLOGY & NEPHROLOGY | cisplatin | INHIBITOR | NF-KAPPA-B | LYMPHOCYTES | T-Lymphocytes - enzymology | Apoptosis - drug effects | Coculture Techniques | NF-kappa B - metabolism | Nephritis - prevention & control | Time Factors | rho-Associated Kinases - metabolism | T-Lymphocytes - drug effects | Nephritis - chemically induced | Acute Kidney Injury - immunology | Kidney Tubules - pathology | Neutrophil Infiltration - drug effects | Cytoprotection | Disease Models, Animal | Amides - pharmacology | Kidney Tubules - drug effects | Acute Kidney Injury - pathology | Anti-Inflammatory Agents - pharmacology | Chemotaxis - drug effects | Acute Kidney Injury - enzymology | Macrophages - enzymology | Signal Transduction - drug effects | Kidney Tubules - enzymology | T-Lymphocytes - immunology | Phosphorylation | Nephritis - genetics | 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine - analogs & derivatives | Acute Kidney Injury - genetics | Nephritis - pathology | rho-Associated Kinases - antagonists & inhibitors | Nephritis - immunology | Inflammation Mediators - metabolism | Acute Kidney Injury - chemically induced | Inflammation Mediators - antagonists & inhibitors | Membrane Proteins - metabolism | T-Lymphocytes - pathology | Macrophages - immunology | Kidney Tubules - immunology | Nephritis - enzymology | Mice, Inbred C57BL | Cells, Cultured | Hepatitis A Virus Cellular Receptor 1 | Acute Kidney Injury - prevention & control | Animals | 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine - pharmacology | Macrophages - drug effects | Protein Kinase Inhibitors - pharmacology | Pyridines - pharmacology | Development and progression | Inflammation | T cells | Phosphotransferases | Health aspects | Identification and classification | Apoptosis | Index Medicus
Journal Article
12.