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Toxicology Letters, ISSN 0378-4274, 12/2019, Volume 317, pp. 53 - 58
In 2017, the U.S. Department of Health and Human Services and the White House declared a public health emergency to address the opioid crisis ( ). On average,... 
Traceable Opioid Material§ Kits | Opioid CRM Kit | TOM Kits | Fentanyl analogs | Opioid Certified Reference Material Kit | Opioid | Fentanyl | Fentanyl Analog Screening Kit | FAS Kit | Opioid overdose crisis
Journal Article
Journal Article
2010, ISBN 9780853698616, vii, 228
The United States is seeing a large increase in the number of people assuming more responsibility for their own health care. Essentials of Nonprescription... 
Drugs, Nonprescription -- Government policy -- United States | Self medication | Drugs, Nonprescription | Nursing & Ancillary Services | Drugs | Administration | Trends | Nonprescription drugs
Book
Nature Medicine, ISSN 1078-8956, 2011, Volume 17, Issue 6, pp. 700 - 707
The natural killer (NK) cell receptor NKp30 is involved in the recognition of tumor and dendritic cells (DCs). Here we describe the influence of three NKp30... 
MEDICINE, RESEARCH & EXPERIMENTAL | NATURAL-KILLER-CELLS | HUMAN DENDRITIC CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | IL-10 PRODUCTION | C-KIT | TRIGGERING RECEPTOR | CELL BIOLOGY | NK CELLS | CLASS-III | CYTOTOXICITY RECEPTORS | HUMAN MHC | EXPRESSION | Prognosis | Alternative Splicing - genetics | Humans | Interleukin-12 - physiology | Lysosomal-Associated Membrane Protein 1 - physiology | Interferon-gamma - physiology | Alternative Splicing - physiology | Gastrointestinal Stromal Tumors - physiopathology | Natural Cytotoxicity Triggering Receptor 3 - genetics | Natural Cytotoxicity Triggering Receptor 3 - physiology | Killer Cells, Natural - physiology | Proto-Oncogene Proteins c-kit - physiology | Gastrointestinal Stromal Tumors - diagnosis | Protein Isoforms | Tumor Necrosis Factor-alpha - physiology | Cell Line, Tumor | Polymorphism, Single Nucleotide - genetics | Proto-Oncogene Proteins c-kit - genetics | Gastrointestinal Stromal Tumors - genetics | Dendritic cells | Gastrointestinal tumors | Physiological aspects | Genetic aspects | Research | Gene expression | Interferon | Medical prognosis | Gastrointestinal diseases | Tumors | Cancer | Interferon-gamma | Killer Cells, Natural | Alternative Splicing | Natural Cytotoxicity Triggering Receptor 3 | Interleukin-12 | Proto-Oncogene Proteins c-kit | Tumor Necrosis Factor-alpha | Life Sciences | Lysosomal-Associated Membrane Protein 1 | Immunology | Polymorphism, Single Nucleotide | Gastrointestinal Stromal Tumors
Journal Article
Tetrahedron computer methodology, ISSN 0898-5529, 1988
Journal
Journal Article
Forensic Science International, ISSN 0379-0738, 12/2001, Volume 124, Issue 1, pp. 47 - 54
Journal Article
Cancer, ISSN 0008-543X, 04/2017, Volume 123, Issue 8, pp. 1333 - 1344
BACKGROUND Optimal treatments for vulvar and vaginal melanomas (VVMs) have not been identified. Herein, the authors compare molecular profiles between VVM and... 
genitourinary melanoma | gene profiling | vulvar melanoma | KIT proto‐oncogene receptor tyrosine kinase (KIT) mutation | mucosal melanoma | B‐Raf proto‐oncogene serine/threonine kinase (BRAF) mutation | molecular targets | vaginal melanoma | neuroblastoma rat sarcoma (NRAS) mutation | KIT proto-oncogene receptor tyrosine kinase (KIT) mutation | B-Raf proto-oncogene serine/threonine kinase (BRAF) mutation | threonine kinase (BRAF) mutation | B-Raf proto-oncogene serine | MALIGNANT-MELANOMA | DACARBAZINE | TUMORS | ONCOLOGY | KIT GENE-MUTATIONS | BRAF MUTATIONS | UNCOMMON | EXPRESSION | IPILIMUMAB | Immunohistochemistry | Melanoma - diagnosis | Humans | Middle Aged | Male | Gene Expression Profiling | Biomarkers, Tumor | Vaginal Neoplasms - genetics | Young Adult | Melanoma - genetics | Aged, 80 and over | Adult | Female | Proto-Oncogene Proteins c-kit - genetics | Vulvar Neoplasms - diagnosis | In Situ Hybridization, Fluorescence | Mucous Membrane - metabolism | Mucous Membrane - pathology | Vulvar Neoplasms - genetics | Proto-Oncogene Proteins B-raf - genetics | Aged | High-Throughput Nucleotide Sequencing | Mutation | Neoplasm Staging | Vaginal Neoplasms - diagnosis | Usage | Care and treatment | Gene mutations | Vulvar cancer | Genetic aspects | Research | Biological markers | Drugs | PD-1 protein | Identification methods | Estrogens | Mucosa | Clinical trials | Raf protein | Alkylation | Skin cancer | Platinum | Immunotherapy | Bioindicators | Medical research | Threonine | Melanoma | Vagina | Exploration | Gene expression | Sensitivity | Reviews | Anthracycline | Valine | PD-L1 protein | Biomarkers | ERCC1 protein | Sampling methods | Protein-serine/threonine kinase | Progesterone | Tumors | Cancer
Journal Article
Cancer Research, ISSN 0008-5472, 04/2009, Volume 69, Issue 8, pp. 3563 - 3569
Journal Article
Human Reproduction Update, ISSN 1355-4786, 11/2015, Volume 21, Issue 6, pp. 779 - 786
BACKGROUND: The first small follicles to appear in the mammalian ovaries are primordial follicles. The initial pool of primordial follicles serves as the... 
KIT ligand | Primordial follicle activation | Mechanistic target of rapamycin complex 1 | KIT | Primordial follicle granulosa cells | PI3 kinase signaling | OOCYTE GROWTH | primordial follicle activation | FACTOR RECEPTOR/C-KIT | C-KIT | TRANSCRIPTION FACTOR FOXL2 | ANTI-MULLERIAN HORMONE | RAT OVARIES | primordial follicle granulosa cells | OBSTETRICS & GYNECOLOGY | REPRODUCTIVE BIOLOGY | mechanistic target of rapamycin complex 1 | FEMALE RATS | CLINICAL-IMPLICATIONS | PHYSIOLOGICAL FUNCTIONS | MOUSE OVARY | TOR Serine-Threonine Kinases - metabolism | Humans | Multiprotein Complexes - genetics | Phosphatidylinositol 3-Kinases - metabolism | Stem Cell Factor - genetics | Mechanistic Target of Rapamycin Complex 1 | Multiprotein Complexes - metabolism | TOR Serine-Threonine Kinases - genetics | Mammals - physiology | Ovarian Follicle - physiology | Female | TOR Serine-Threonine Kinases - physiology | Ovarian Follicle - cytology | Ovarian Follicle - metabolism | Stem Cell Factor - physiology | Ovary - metabolism | Stem Cell Factor - metabolism | Signal Transduction | Multiprotein Complexes - physiology | Phosphatidylinositol 3-Kinases - genetics | Animals | Oocytes - physiology | Models, Biological | Phosphatidylinositol 3-Kinases - physiology | Granulosa Cells - cytology | female rats | c-kit | mechanistic target of rapamycin | rat ovaries | v110 | Obstetrics & Gynecology | mouse ovary | p17474 | anti-mullerian hormone | Biokemi och molekylärbiologi | receptor/c-kit | Biochemistry and Molecular Biology | physiological functions | clinical-implications | transcription factor foxl2 | factor | oocyte growth | Reproductive Biology | ates of america
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 11/2014, Volume 20, Issue 22, pp. 5745 - 5755
Purpose: KIT is the major oncogenic driver of gastrointestinal stromal tumors (GIST). Imatinib, sunitinib, and regorafenib are approved therapies; however,... 
EFFICACY | ONCOLOGY | SAFETY | SUNITINIB | C-KIT | MYELOID-LEUKEMIA | MECHANISMS | MUTATIONS | DASATINIB | IMATINIB RESISTANCE | NILOTINIB | Exons | Humans | Molecular Conformation | Imidazoles - chemistry | Tomography, X-Ray Computed | Antineoplastic Agents - therapeutic use | Pyridazines - pharmacology | Proto-Oncogene Proteins c-kit - antagonists & inhibitors | Dose-Response Relationship, Drug | Protein Kinase Inhibitors - chemistry | Gastrointestinal Stromal Tumors - diagnosis | Gastrointestinal Stromal Tumors - pathology | Inhibitory Concentration 50 | Female | Indoles - pharmacology | Proto-Oncogene Proteins c-kit - genetics | Tumor Burden - genetics | Antineoplastic Agents - pharmacology | Benzamides - pharmacology | Imidazoles - therapeutic use | Gastrointestinal Stromal Tumors - genetics | Pyridazines - therapeutic use | Disease Models, Animal | Neoplasm Recurrence, Local | Models, Molecular | Imidazoles - pharmacology | Pyrimidines - pharmacology | Antineoplastic Agents - chemistry | Imatinib Mesylate | Piperazines - pharmacology | Pyridazines - chemistry | Xenograft Model Antitumor Assays | Drug Resistance, Neoplasm - genetics | Pyrroles - pharmacology | Animals | Tumor Burden - drug effects | Gastrointestinal Stromal Tumors - drug therapy | Protein Kinase Inhibitors - therapeutic use | Cell Line, Tumor | Protein Binding | Protein Kinase Inhibitors - pharmacology | Mutation | Proto-Oncogene Proteins c-kit - chemistry | Index Medicus | GIST | ponatinib | tyrosine kinase inhibitor | resistance | KIT
Journal Article