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1. Full Text Myeloid Krüppel-Like Factor 4 Deficiency Augments Atherogenesis in ApoE−/− Mice—Brief Report
by Sharma, Nikunj and Lu, Yuan and Zhou, Guangjin and Liao, Xudong and Kapil, Parul and Anand, Puneet and Mahabeleshwar, Ganapati H and Stamler, Jonathan S and Jain, Mukesh K
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1079-5642, 12/2012, Volume 32, Issue 12, pp. 2836 - 2838
atherosclerosis | macrophage | inflammation | Krüppel-like factor 4 | Peripheral Vascular Disease | Life Sciences & Biomedicine | Hematology | Cardiovascular System & Cardiology | Science & Technology | Cardiology. Vascular system | Heart | Diseases of the peripheral vessels. Diseases of the vena cava. Miscellaneous | Atherosclerosis (general aspects, experimental research) | Biological and medical sciences | Medical sciences | Coronary heart disease | Blood and lymphatic vessels | Foam Cells - physiology | Macrophages - physiology | Atherosclerosis - pathology | Apolipoproteins E - deficiency | Macrophages - pathology | Atherosclerosis - physiopathology | Kruppel-Like Transcription Factors - physiology | Mice, Knockout | Phenotype | Animals | Apolipoproteins E - genetics | Apolipoproteins E - physiology | Mice | Kruppel-Like Transcription Factors - deficiency | Foam Cells - pathology | Kruppel-Like Transcription Factors - genetics | Disease Models, Animal | Index Medicus
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2. Full Text Krüppel-like factor 4 (KLF4): What we currently know
by Ghaleb, Amr M and Yang, Vincent W
Gene, ISSN 0378-1119, 05/2017, Volume 611, pp. 27 - 37
Transcription | Organs | Inflammation | Proliferation | KLF4 | Gene expression | Tissues | Diseases | Epithelial-to-mesenchymal transition | Cell cycle | Stem cells | Differentiation | Cancer | Life Sciences & Biomedicine | Genetics & Heredity | Science & Technology | Neoplasms - metabolism | Cell Proliferation - genetics | Humans | Gene Expression Regulation | Sequence Homology, Amino Acid | Cell Differentiation - genetics | Animals | Neoplasms - genetics | Kruppel-Like Transcription Factors - metabolism | Base Sequence | Neoplasms - pathology | Kruppel-Like Transcription Factors - genetics | Induced Pluripotent Stem Cells - metabolism | Blood circulation disorders | Medical colleges | Physiological aspects | DNA binding proteins | Cell differentiation | Index Medicus | stem cells | organs | differentiation | transcription | tissues | proliferation | cell cycle | diseases | epithelial-to-mesenchymal transition | inflammation | cancer | gene expression
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3. Full Text Krüppel-like factor 4 regulates macrophage polarization
by Liao, Xudong and Sharma, Nikunj and Kapadia, Fehmida and Zhou, Guangjin and Lu, Yuan and Hong, Hong and Paruchuri, Kaavya and Mahabeleshwar, Ganapati H and Dalmas, Elise and Venteclef, Nicolas and Flask, Chris A and Kim, Julian and Doreian, Bryan W and Lu, Kurt Q and Kaestner, Klaus H and Hamik, Anne and Clément, Karine and Jain, Mukesh K
The Journal of clinical investigation, ISSN 0021-9738, 07/2011, Volume 121, Issue 7, pp. 2736 - 2749
Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Cell Line | Cell Polarity | Gene Expression | Adipose Tissue - physiology | Macrophage Activation - immunology | Humans | Adipose Tissue - cytology | Escherichia coli - immunology | NF-kappa B - metabolism | Obesity - physiopathology | STAT6 Transcription Factor - genetics | Macrophages - cytology | STAT6 Transcription Factor - metabolism | Mice, Knockout | Animals | Insulin Resistance - physiology | Kruppel-Like Transcription Factors - metabolism | Lipopolysaccharides - pharmacology | Macrophages - drug effects | Mice | Wound Healing - physiology | Macrophage Activation - drug effects | Kruppel-Like Transcription Factors - genetics | Macrophages - immunology | Index Medicus | Abridged Index Medicus
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4. Full Text Kruppel-like € factor 4 suppresses serine/ threonine kinase 33 activation and metastasis of gastric cancer through reversing epithelial–mesenchymal transition
by Kong, Fanyang and Sun, Tao and Kong, Xiangyu and Xie, Dacheng and Li, Zhaoshen and Xie, Keping
Clinical cancer research, ISSN 1078-0432, 05/2018, Volume 24, Issue 10, pp. 2440 - 2451
Life Sciences & Biomedicine | Oncology | Science & Technology | Immunohistochemistry | Prognosis | Humans | Stomach Neoplasms - metabolism | Male | Stomach Neoplasms - pathology | Gene Expression Profiling | Epithelial-Mesenchymal Transition - genetics | Case-Control Studies | Cell Movement - genetics | Neoplasm Metastasis | Heterografts | Kruppel-Like Transcription Factors - metabolism | Female | Gene Expression Regulation, Neoplastic - drug effects | Genes, Reporter | Protein-Serine-Threonine Kinases - metabolism | Disease Models, Animal | Stomach Neoplasms - genetics | Protein-Serine-Threonine Kinases - genetics | Animals | Cell Line, Tumor | Mice | Protein Kinase Inhibitors - pharmacology | Enzyme Activation | Neoplasm Staging | Kruppel-Like Transcription Factors - genetics | Stomach Neoplasms - mortality | Cell proliferation | Mesenchyme | Transcription | Threonine | Therapeutic applications | Metastasis | Tissues | Kinases | Stomach cancer | Lymph nodes | Molecular chains | Metastases | Signal transduction | Biological effects | KLF4 protein | Protein-serine/threonine kinase | Inhibition | Gastric cancer | Cell migration | Tumors | Cancer | Kruppel protein | Index Medicus | STK33 | invasion | gastric cancer | KLF4 | metastasis
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5. Full Text GINS complex subunit 4, a prognostic biomarker and reversely mediated by Krüppel‐like factor 4, promotes the growth of colorectal cancer
by Rong, Zeyin and Luo, Zai and Zhang, Jianming and Li, Tengfei and Zhu, Zhonglin and Yu, Zhilong and Fu, Zhongmao and Qiu, Zhengjun and Huang, Chen
Cancer science, ISSN 1347-9032, 04/2020, Volume 111, Issue 4, pp. 1203 - 1217
growth | colorectal cancer | biomarker | KLF4 | GINS4 | Life Sciences & Biomedicine | Oncology | Science & Technology | Cell Cycle - genetics | Cell Proliferation - genetics | Prognosis | Colorectal Neoplasms - genetics | Humans | Middle Aged | Carcinogenesis - genetics | Apoptosis - genetics | Male | Chromosomal Proteins, Non-Histone - genetics | Disease-Free Survival | Cell Line, Tumor | DNA Replication - genetics | Female | Aged | Biomarkers, Tumor - genetics | Colorectal Neoplasms - pathology | Neoplasm Staging | Kruppel-Like Transcription Factors - genetics | Gene Expression Regulation, Neoplastic - genetics | Transcription | Lung cancer | Colorectal carcinoma | Replication initiation | Colorectal cancer | Metastasis | Radiation therapy | Gene expression | Kinases | Stomach cancer | DNA biosynthesis | Proteins | Cell growth | Chemotherapy | Molecular modelling | DNA microarrays | KLF4 protein | S phase | Cell cycle | Tumorigenesis | Apoptosis | Index Medicus | Original
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6. Full Text Haploinsufficiency of Krüppel-like factor 4 promotes adenomatous polyposis coli-dependent intestinal tumorigenesis
by Ghaleb, Amr M and McConnell, Beth B and Nandan, Mandayam O and Katz, Jonathan P and Kaestner, Klaus H and Yang, Vincent W
Cancer research (Chicago, Ill.), ISSN 0008-5472, 08/2007, Volume 67, Issue 15, pp. 7147 - 7154
Life Sciences & Biomedicine | Oncology | Science & Technology | Gastroenterology. Liver. Pancreas. Abdomen | Stomach. Duodenum. Small intestine. Colon. Rectum. Anus | Pharmacology. Drug treatments | Biological and medical sciences | Medical sciences | Antineoplastic agents | Tumors | Adenoma - genetics | Cyclins - genetics | Male | Loss of Heterozygosity | RNA, Messenger - metabolism | Adenomatous Polyposis Coli - pathology | Immunoenzyme Techniques | Adenoma - metabolism | Intestinal Polyps - genetics | Cyclins - metabolism | Cell Transformation, Neoplastic - genetics | Kruppel-Like Transcription Factors - metabolism | Female | Cyclin D | Adenomatous Polyposis Coli - genetics | Intestinal Neoplasms - genetics | Intestinal Neoplasms - metabolism | Mice, Inbred C57BL | Cell Transformation, Neoplastic - metabolism | beta Catenin - metabolism | Adenomatous Polyposis Coli - metabolism | Intestinal Neoplasms - pathology | Animals | Intestinal Polyps - pathology | Adenoma - pathology | Mice | Cell Transformation, Neoplastic - pathology | Intestinal Polyps - metabolism | Kruppel-Like Transcription Factors - genetics | Index Medicus
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7. Full Text ORAI1 Activates Proliferation of Lymphatic Endothelial Cells in Response to Laminar Flow Through Krüppel-Like Factors 2 and 4
by Choi, Dongwon and Park, Eunkyung and Jung, Eunson and Seong, Young Jin and Hong, Mingu and Lee, Sunju and Burford, James and Gyarmati, Georgina and Peti-Peterdi, Janos and Srikanth, Sonal and Gwack, Yousang and Koh, Chester J and Boriushkin, Evgenii and Hamik, Anne and Wong, Alex K and Hong, Young-Kwon
Circulation research, ISSN 0009-7330, 04/2017, Volume 120, Issue 9, pp. 1426 - 1439
Cell proliferation | lymphatic vessels | Calcium channel | vascular endothelial growth factor A | Capillary | Cardiac & Cardiovascular Systems | Peripheral Vascular Disease | Life Sciences & Biomedicine | Hematology | Cardiovascular System & Cardiology | Science & Technology | Cyclin-Dependent Kinase Inhibitor p57 - metabolism | Cell Proliferation | Cyclin-Dependent Kinase Inhibitor p57 - genetics | Human Umbilical Vein Endothelial Cells - metabolism | Humans | Stress, Mechanical | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - genetics | Receptor, Fibroblast Growth Factor, Type 3 - metabolism | Kruppel-Like Transcription Factors - metabolism | Endothelium, Lymphatic - metabolism | Kruppel-Like Transcription Factors - deficiency | Lymphangiogenesis | Endothelium, Lymphatic - pathology | ORAI1 Protein - metabolism | Receptor, Fibroblast Growth Factor, Type 3 - genetics | ORAI1 Protein - genetics | Endothelial Cells - metabolism | Gene Expression Regulation | Genotype | Vascular Endothelial Growth Factor C - genetics | Mice, Knockout | ORAI1 Protein - deficiency | Mechanotransduction, Cellular | Phenotype | Animals | Endothelium, Vascular - metabolism | Vascular Endothelial Growth Factor C - metabolism | Kruppel-Like Transcription Factors - genetics | Endothelium, Lymphatic - physiopathology | Calcium channels | Animal models | Lymphatic system | Fluid flow | Regulatory sequences | Orai1 protein | Embryos | Blood | Endothelial cells | Cell growth | KLF4 protein | Rodents | Shear stress | Skin | Lung Kruppel-like factor | Vascular endothelial growth factor | Fibroblast growth factor receptors | Index Medicus | endothelial cell growth | lymphatic capillary | Signal Transduction | Growth Factors | Cytokines | Vascular Biology | lymphatic development | KLF4 | KLF2 | Endothelial shear stress | vascular endothelial growth factor | Cell Signaling | ORAI1 | Developmental Biology
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8. Full Text Endothelial Kruppel-like factor 4 protects against atherothrombosis in mice
by Zhou, Guangjin and Hamik, Anne and Nayak, Lalitha and Tian, Hongmei and Shi, Hong and Lu, Yuan and Sharma, Nikunj and Liao, Xudong and Hale, Andrew and Boerboom, Lauren and Feaver, Ryan E and Gao, Huiyun and Desai, Amar and Schmaier, Alvin and Gerson, Stanton L and Wang, Yunmei and Atkins, G. Brandon and Blackman, Brett R and Simon, Daniel I and Jain, Mukesh K
The Journal of clinical investigation, ISSN 0021-9738, 12/2012, Volume 122, Issue 12, pp. 4727 - 4731
Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Vasculitis - metabolism | Human Umbilical Vein Endothelial Cells - metabolism | Humans | Mice, Inbred C57BL | Cells, Cultured | Gene Expression Regulation | Kruppel-Like Transcription Factors - physiology | Mice, Transgenic | Atherosclerosis - metabolism | p300-CBP Transcription Factors - metabolism | Thrombosis - metabolism | Animals | Kruppel-Like Transcription Factors - metabolism | Protein Binding | Thrombosis - genetics | Mice | Protein Interaction Domains and Motifs | Kruppel-Like Transcription Factors - genetics | Prevention | Atheroembolism | Genetic aspects | Research | Gene expression | Thrombosis | Risk factors | Blood clot | Endothelium | Index Medicus | Abridged Index Medicus | Competition | Atherogenesis | Animal models | Gene flow | Cytokines | KLF4 protein | Homeostasis | Inflammation | Brief Report
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9. Full Text Kruppel-like factor 4 (KLF4) is required for maintenance of breast cancer stem cells and for cell migration and invasion
by Yu, F and Li, J and Chen, H and Fu, J and Ray, S and Huang, S and Zheng, H and Ai, W
Oncogene, ISSN 0950-9232, 05/2011, Volume 30, Issue 18, pp. 2161 - 2172
migration | breast cancer | cancer stem cell | invasion | KLF4 | Biochemistry & Molecular Biology | Oncology | Genetics & Heredity | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Fundamental and applied biological sciences. Psychology | Gynecology. Andrology. Obstetrics | Mammary gland diseases | Biological and medical sciences | Cell physiology | Molecular and cellular biology | Medical sciences | Cell transformation and carcinogenesis. Action of oncogenes and antioncogenes | Tumors | Neoplasm Metastasis | Neoplastic Stem Cells - cytology | Signal Transduction | Breast Neoplasms - pathology | Neoplasm Invasiveness | Receptors, Notch - metabolism | Humans | Cell Line, Tumor | Kruppel-Like Transcription Factors - physiology | Breast Neoplasms - physiopathology | Kruppel-Like Transcription Factors - genetics | Gene Knockdown Techniques | Transcription factors | Cancer cells | Physiological aspects | Development and progression | Breast cancer | Research | Risk factors | Cell migration | Proteins | Signal transduction | Gene expression | Stem cells | Cell adhesion & migration | Severe combined immunodeficiency | Colonies | Diabetes mellitus | Tumorigenicity | Tumor cell lines | Aldehyde dehydrogenase | KLF4 protein | Notch protein | Tumorigenesis | Mammary gland | Surface markers | Oncogenes | Index Medicus
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