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1. Full Text Krüppel-like factor 4 regulates macrophage polarization
by Liao, Xudong and Sharma, Nikunj and Kapadia, Fehmida and Zhou, Guangjin and Lu, Yuan and Hong, Hong and Paruchuri, Kaavya and Mahabeleshwar, Ganapati H and Dalmas, Elise and Venteclef, Nicolas and Flask, Chris A and Kim, Julian and Doreian, Bryan W and Lu, Kurt Q and Kaestner, Klaus H and Hamik, Anne and Clément, Karine and Jain, Mukesh K
Journal of Clinical Investigation, ISSN 0021-9738, 07/2011, Volume 121, Issue 7, pp. 2736 - 2749
Current paradigms suggest that two macrophage subsets, termed M1 and M2, are involved in inflammation and host defense. While the distinct functions of M1 and... 
MEDICINE, RESEARCH & EXPERIMENTAL | MEDIATED INFLAMMATION | ADIPOSE-TISSUE MACROPHAGES | INSULIN-RESISTANCE | PROINFLAMMATORY ACTIVATION | TUMOR-ASSOCIATED MACROPHAGES | PPAR-GAMMA | ANTIGEN-PRESENTING CELLS | TRANSCRIPTION FACTOR | RAW264.7 MACROPHAGES | MONOCYTE DIFFERENTIATION | Cell Line | Cell Polarity | Gene Expression | Adipose Tissue - physiology | Macrophage Activation - immunology | Humans | Adipose Tissue - cytology | Escherichia coli - immunology | NF-kappa B - metabolism | Obesity - physiopathology | STAT6 Transcription Factor - genetics | Macrophages - cytology | STAT6 Transcription Factor - metabolism | Mice, Knockout | Animals | Insulin Resistance - physiology | Kruppel-Like Transcription Factors - metabolism | Lipopolysaccharides - pharmacology | Macrophages - drug effects | Mice | Wound Healing - physiology | Macrophage Activation - drug effects | Kruppel-Like Transcription Factors - genetics | Macrophages - immunology
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2. Full Text Kruppel-like Factor 4 Regulates Endothelial Inflammation
by Anne Hamik and Zhiyong Lin and Ajay Kumar and Mercedes Balcells and Sumita Sinha and Jonathan Katz and Mark W. Feinberg and Robert E. Gerszten and Elazer R. Edelman and Mukesh K. Jain
Journal of Biological Chemistry, ISSN 0021-9258, 05/2007, Volume 282, Issue 18, pp. 13769 - 13779
The vascular endothelium plays a critical role in vascular homeostasis. Inflammatory cytokines and non-laminar blood flow induce endothelial dysfunction and... 
CELL-ADHESION MOLECULE-1 | GROWTH-FACTOR-BETA | ABDOMINAL AORTIC-ANEURYSM | NITRIC-OXIDE SYNTHASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | FINGER TRANSCRIPTION FACTOR | LAMINAR SHEAR-STRESS | GENE-EXPRESSION | KRUPPEL-LIKE FACTOR | NF-KAPPA-B | PLASMINOGEN-ACTIVATOR INHIBITOR | Inflammation - pathology | Vasculitis - metabolism | Nitric Oxide Synthase Type II - biosynthesis | Humans | Nitric Oxide Synthase Type III - biosynthesis | Vasculitis - pathology | Stress, Mechanical | Thrombomodulin - biosynthesis | Inflammation - metabolism | Vasculitis - genetics | Kruppel-Like Transcription Factors - metabolism | Inflammation Mediators - metabolism | Kruppel-Like Transcription Factors - deficiency | Cell Adhesion - genetics | Endothelial Cells - metabolism | Gene Expression Regulation - genetics | Cells, Cultured | Thrombosis - pathology | Thrombosis - metabolism | Animals | Endothelium, Vascular - metabolism | Inflammation - genetics | Blood Coagulation - genetics | Endothelium, Vascular - pathology | Thrombosis - genetics | Mice | Endothelial Cells - pathology
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3. Full Text Endothelial Kruppel-like factor 4 protects against atherothrombosis in mice
by Zhou, Guangjin and Hamik, Anne and Nayak, Lalitha and Tian, Hongmei and Shi, Hong and Lu, Yuan and Sharma, Nikunj and Liao, Xudong and Hale, Andrew and Boerboom, Lauren and Feaver, Ryan E and Gao, Huiyun and Desai, Amar and Schmaier, Alvin and Gerson, Stanton L and Wang, Yunmei and Atkins, G. Brandon and Blackman, Brett R and Simon, Daniel I and Jain, Mukesh K
Journal of Clinical Investigation, ISSN 0021-9738, 12/2012, Volume 122, Issue 12, pp. 4727 - 4731
The endothelium regulates vascular homeostasis, and endothelial dysfunction is a proximate event in the pathogenesis of atherothrombosis. Stimulation of the... 
MEDICINE, RESEARCH & EXPERIMENTAL | CELLS | ACTIVATION | Vasculitis - metabolism | Human Umbilical Vein Endothelial Cells - metabolism | Humans | Mice, Inbred C57BL | Cells, Cultured | Gene Expression Regulation | Kruppel-Like Transcription Factors - physiology | Mice, Transgenic | Atherosclerosis - metabolism | p300-CBP Transcription Factors - metabolism | Thrombosis - metabolism | Animals | Kruppel-Like Transcription Factors - metabolism | Protein Binding | Thrombosis - genetics | Mice | Protein Interaction Domains and Motifs | Kruppel-Like Transcription Factors - genetics | Prevention | Atheroembolism | Genetic aspects | Research | Gene expression | Thrombosis | Risk factors | Blood clot | Endothelium | Brief Report
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4. Full Text Krüppel-like factor 4 (KLF4): What we currently know
by Ghaleb, Amr M and Yang, Vincent W
Gene, ISSN 0378-1119, 05/2017, Volume 611, pp. 27 - 37
Krüppel-like factor 4 (KLF4) is an evolutionarily conserved zinc finger-containing transcription factor that regulates diverse cellular processes such as cell... 
Transcription | Organs | Inflammation | Proliferation | KLF4 | Gene expression | Tissues | Diseases | Epithelial-to-mesenchymal transition | Cell cycle | Stem cells | Differentiation | Cancer | INDUCED DNA-DAMAGE | TM4 SERTOLI-CELLS | TRANSCRIPTION FACTOR KLF4 | HUMAN GASTRIC-CANCER | CANCER STEM-CELLS | EPITHELIAL-MESENCHYMAL TRANSITION | GROWTH-FACTOR-BETA | MOUSE EMBRYONIC FIBROBLASTS | GENETICS & HEREDITY | ZINC-FINGER PROTEIN | INDUCED GUT INJURY | Neoplasms - metabolism | Cell Proliferation - genetics | Humans | Gene Expression Regulation | Sequence Homology, Amino Acid | Cell Differentiation - genetics | Animals | Neoplasms - genetics | Kruppel-Like Transcription Factors - metabolism | Base Sequence | Neoplasms - pathology | Kruppel-Like Transcription Factors - genetics | Induced Pluripotent Stem Cells - metabolism | Blood circulation disorders | Medical colleges | Physiological aspects | DNA binding proteins | Cell differentiation | stem cells | organs | differentiation | transcription | tissues | proliferation | cell cycle | diseases | epithelial-to-mesenchymal transition | inflammation | cancer | gene expression
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5. Full Text Kruppel-like factor 4 regulates keratinocyte senescence
by Panatta, Emanuele and Lena, Anna Maria and Mancini, Mara and Affinati, Michela and Smirnov, Artem and Annicchiarico-Petruzzelli, Margherita and Piro, Maria Cristina and Campione, Elena and Bianchi, Luca and Mazzanti, Cinzia and Melino, Gerry and Candi, Eleonora
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 05/2018, Volume 499, Issue 2, pp. 389 - 395
Keratinocyte replicative senescence has an important role in time-related changes of epidermis. Previous studies demonstrated that miRNAs play key roles in... 
Keratinocytes | Epidermis | Senescence | Klf4 | Skin aging | miR-34a | CELLS | APOPTOSIS | PROTEIN | BIOCHEMISTRY & MOLECULAR BIOLOGY | MICRORNAS | BIOPHYSICS | TRANSACTIVATION | TUMOR-SUPPRESSION | SKIN | DIFFERENTIATION | KLF4 GENE | Cell Line | Humans | RNA, Messenger - genetics | Gene Silencing | 3' Untranslated Regions - genetics | MicroRNAs - metabolism | Keratinocytes - cytology | Skin Aging | RNA, Messenger - metabolism | Down-Regulation - genetics | Cellular Senescence | Keratinocytes - metabolism | Kruppel-Like Transcription Factors - metabolism | Base Sequence | MicroRNAs - genetics | Kruppel-Like Transcription Factors - genetics | RNA | Squamous cell carcinoma | Skin
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6. Full Text Interferon Regulatory Factor 4 Inhibits Neointima Formation by Engaging Krüppel-Like Factor 4 Signaling
by Cheng, Wen-Lin and She, Zhi-Gang and Qin, Juan-Juan and Guo, Jun-Hong and Gong, Fu-Han and Zhang, Peng and Fang, Chun and Tian, Song and Zhu, Xue-Yong and Gong, Jun and Wang, Zhi-Hua and Huang, Zan and Li, Hongliang
Circulation, ISSN 0009-7322, 10/2017, Volume 136, Issue 15, pp. 1412 - 1433
BACKGROUND:The mechanisms underlying neointima formation remain unclear. Interferon regulatory factors (IRFs), which are key innate immune regulators, play... 
Kruppel-like transcription factors | coronary restenosis | smooth muscle cell | cell proliferation | interferon regulatory factor-4 | PRESSURE-OVERLOAD | CARDIAC & CARDIOVASCULAR SYSTEMS | DOWN-REGULATION | PATHOLOGICAL CARDIAC-HYPERTROPHY | VASCULAR INJURY | SM22 ALPHA | INSULIN-RESISTANCE | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | MACROPHAGE POLARIZATION | T-CELLS | DIFFERENTIATION MARKERS | Muscle, Smooth, Vascular - metabolism | Oligonucleotide Array Sequence Analysis | Humans | Gene Expression Regulation | Interferon Regulatory Factors - metabolism | Interferon Regulatory Factors - genetics | Rats | Neointima - metabolism | Mice, Knockout | Muscle, Smooth, Vascular - pathology | Neointima - genetics | Animals | Kruppel-Like Transcription Factors - metabolism | Neointima - pathology | Mice | Kruppel-Like Transcription Factors - genetics
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7. Full Text Kruppel-like factor 4 (KLF4) is required for maintenance of breast cancer stem cells and for cell migration and invasion
by Yu, F and Li, J and Chen, H and Fu, J and Ray, S and Huang, S and Zheng, H and Ai, W
Oncogene, ISSN 0950-9232, 05/2011, Volume 30, Issue 18, pp. 2161 - 2172
Kruppel-like factor 4 (KLF4) is highly expressed in more than 70% of breast cancers and functions as an oncogene. However, an exact mechanism by which KLF4... 
migration | breast cancer | cancer stem cell | invasion | KLF4 | TRANSFORMATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | SELF-RENEWAL | IDENTIFICATION | TUMORIGENICITY | P53 | CELL BIOLOGY | EPITHELIAL-MESENCHYMAL TRANSITION | ONCOLOGY | GENETICS & HEREDITY | GENE-EXPRESSION | TUMOR-SUPPRESSOR | DIFFERENTIATION | TRANSCRIPTION FACTOR | Neoplasm Metastasis | Neoplastic Stem Cells - cytology | Signal Transduction | Breast Neoplasms - pathology | Neoplasm Invasiveness | Receptors, Notch - metabolism | Humans | Cell Line, Tumor | Kruppel-Like Transcription Factors - physiology | Breast Neoplasms - physiopathology | Kruppel-Like Transcription Factors - genetics | Gene Knockdown Techniques | Transcription factors | Cancer cells | Physiological aspects | Development and progression | Breast cancer | Research | Risk factors | Cell migration | Proteins | Signal transduction | Gene expression | Stem cells | Cell adhesion & migration | Severe combined immunodeficiency | Colonies | Diabetes mellitus | Tumorigenicity | Tumor cell lines | Aldehyde dehydrogenase | KLF4 protein | Notch protein | Tumorigenesis | Mammary gland | Surface markers | Oncogenes | Tumors
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8. Full Text Kruppel-like Factor 4 (Klf4) Prevents Embryonic Stem (ES) Cell Differentiation by Regulating Nanog Gene Expression
by Peilin Zhang and Rose Andrianakos and Yang Yang and Chunming Liu and Wange Lu
Journal of Biological Chemistry, ISSN 0021-9258, 03/2010, Volume 285, Issue 12, pp. 9180 - 9189
Transcription factor Kruppel-like factor 4 (Klf4) is essential for somatic cell reprogramming. In addition, Klf4 seems to play a redundant role along with... 
SOX2 | OCT4 | ACTIVATION | INHIBITION | BIOCHEMISTRY & MOLECULAR BIOLOGY | MOUSE EMBRYOS | STAT3 | C-MYC | SELF-RENEWAL | HUMAN FIBROBLASTS | PLURIPOTENCY | Embryonic Stem Cells - cytology | Nanog Homeobox Protein | Signal Transduction | Homeodomain Proteins - metabolism | Humans | Gene Expression Regulation | Bone Morphogenetic Protein 4 - metabolism | Leukemia Inhibitory Factor - metabolism | Animals | Kruppel-Like Transcription Factors - metabolism | Models, Biological | Cell Differentiation | Mice | STAT3 Transcription Factor - metabolism | Gene Regulation | Gene Transcription | Kruppel-like Factor 4 | Self-renewal | shRNA | Nanog | Differentiation | Embryonic Stem Cell | Chromatin Immunoprecipitation (ChiP) | Pluripotency | Cell Biology
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9. Full Text Platelet factor 4 mediates vascular smooth muscle cell injury responses
by Shi, Guanfang and Shi, Guanfang and Field, David J and Field, David J and Long, Xiaochun and Long, Xiaochun and Mickelsen, Deanne and Mickelsen, Deanne and Ko, Kyung-Ae and Ko, Kyung-ae and Ture, Sara and Ture, Sara and Korshunov, Vyacheslav A and Korshunov, Vyacheslav A and Miano, Joseph M and Miano, Joseph M and Morrell, Craig N and Morrell, Craig N
Blood, ISSN 0006-4971, 05/2013, Volume 121, Issue 21, pp. 4417 - 4427
Activated platelets release many inflammatory molecules with important roles in accelerating vascular inflammation. Much is known about platelet and... 
ALLOGRAFT-REJECTION | PLATELETS | IN-VIVO | GENE-EXPRESSION | ATHEROSCLEROSIS | PROLIFERATION | MICE | KLF4 | HEMATOLOGY | T-CELLS | MONOCYTE DIFFERENTIATION | Blood Platelets - immunology | Vasculitis - physiopathology | Humans | Mice, Inbred C57BL | Vasculitis - pathology | Cells, Cultured | Muscle, Smooth, Vascular - immunology | Male | Muscle, Smooth, Vascular - cytology | Carotid Arteries - cytology | Vasculitis - immunology | Muscle, Smooth, Vascular - injuries | Animals | Ligation | Kruppel-Like Transcription Factors - metabolism | Carotid Arteries - immunology | Carotid Artery Injuries - immunology | Mice | Platelet Factor 4 - metabolism | Kruppel-Like Transcription Factors - genetics | Carotid Artery Injuries - metabolism | Carotid Artery Injuries - pathology | Disease Models, Animal | Vascular Biology
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10. Full Text Kruppel-like factor 4 is critical for transcriptional control of cardiac mitochondrial homeostasis
by Liao, Xudong and Zhang, Rongli and Lu, Yuan and Prosdocimo, Domenick A and Sangwung, Panjamaporn and Zhang, Lilei and Zhou, Guangjin and Anand, Puneet and Lai, Ling and Leone, Teresa C and Fujioka, Hisashi and Ye, Fang and Rosca, Mariana G and Hoppel, Charles L and Christian Schulze, P and Dale Abel, E and Stamler, Jonathan S and Kelly, Daniel P and Jain, Mukesh K
Journal of Clinical Investigation, ISSN 0021-9738, 09/2015, Volume 125, Issue 9, pp. 3461 - 3476
Mitochondrial homeostasis is critical for tissue health, and mitochondrial dysfunction contributes to numerous diseases, including heart failure. Here, we have... 
Mitochondria, Heart - metabolism | Heart Diseases - metabolism | Humans | Rats | Transcription Factors - genetics | PPAR gamma - metabolism | Mice, Knockout | Transcription Factors - metabolism | Animals | Kruppel-Like Transcription Factors - metabolism | Oxygen Consumption - genetics | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | HEK293 Cells | Myocytes, Cardiac - metabolism | Transcription, Genetic | Mice | Autophagy - genetics | Mitochondria, Heart - genetics | Kruppel-Like Transcription Factors - genetics | Heart Diseases - genetics | PPAR gamma - genetics | Physiological aspects | Homeostasis | Genetic aspects | Mitochondrial DNA | Research | Heart cells | Studies | Heart failure | Mitochondria | Morphology | Biosynthesis | Metabolism | Arrays | Gene expression | Autophagy
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11. Full Text Myeloid Krüppel-Like Factor 4 Deficiency Augments Atherogenesis in ApoE−/− Mice—Brief Report
by Sharma, Nikunj and Lu, Yuan and Zhou, Guangjin and Liao, Xudong and Kapil, Parul and Anand, Puneet and Mahabeleshwar, Ganapati H and Stamler, Jonathan S and Jain, Mukesh K
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 12/2012, Volume 32, Issue 12, pp. 2836 - 2838
OBJECTIVE—To investigate the role of Krüppel-like factor 4 (KLF4), an essential transcriptional regulator of macrophage polarization (M1/M2), in the... 
atherosclerosis | macrophage | inflammation | Krüppel-like factor 4 | MACROPHAGES | PERIPHERAL VASCULAR DISEASE | IDENTIFICATION | HEMATOLOGY | Kruppel-like factor 4 | Foam Cells - physiology | Macrophages - physiology | Atherosclerosis - pathology | Apolipoproteins E - deficiency | Macrophages - pathology | Atherosclerosis - physiopathology | Kruppel-Like Transcription Factors - physiology | Mice, Knockout | Phenotype | Animals | Apolipoproteins E - genetics | Apolipoproteins E - physiology | Mice | Kruppel-Like Transcription Factors - deficiency | Foam Cells - pathology | Kruppel-Like Transcription Factors - genetics | Disease Models, Animal
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12. Full Text Hemodynamic Disturbed Flow Induces Differential DNA Methylation of Endothelial Kruppel-Like Factor 4 Promoter In Vitro and In Vivo
by Jiang, Yi-Zhou and Jiménez, Juan M and Ou, Kristy and McCormick, Margaret E and Zhang, Ling-Di and Davies, Peter F
Circulation Research, ISSN 0009-7330, 06/2014, Volume 115, Issue 1, pp. 32 - 43
RATIONALE:Hemodynamic disturbed flow (DF) is associated with susceptibility to atherosclerosis. Endothelial Kruppel-Like Factor 4 (KLF4) is an important... 
atherosclerosis | DNA methylation | epigenetics | hemodynamics | ATHEROSUSCEPTIBLE ENDOTHELIUM | CARDIAC & CARDIOVASCULAR SYSTEMS | NITRIC-OXIDE SYNTHASE | TRANSCRIPTION | KAPPA-B | HISTONE DEACETYLASES | SHEAR-STRESS | CARDIOVASCULAR-DISEASE | TUMOR-SUPPRESSOR | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | EXPRESSION | Promoter Regions, Genetic | DNA (Cytosine-5-)-Methyltransferases - physiology | MEF2 Transcription Factors - genetics | Humans | Cells, Cultured | Nitric Oxide Synthase Type III - genetics | Atherosclerosis - etiology | DNA Methylation | Animals | Endothelium, Vascular - metabolism | Swine | CpG Islands | Hemodynamics | Kruppel-Like Transcription Factors - genetics | Blood Circulation | athergenesis | KLF4 | Atherosusceptible endothelium
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