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Journal of Neuroscience, ISSN 0270-6474, 06/2010, Volume 30, Issue 22, pp. 7575 - 7586
Glutamate-induced excitotoxicity has been implicated in the etiology of stroke, epilepsy, and neurodegenerative diseases. NMDA receptors (NMDARs) play a... 
TRANSCRIPTIONAL REPRESSOR DREAM | CALCIUM-SENSOR PROTEINS | POTASSIUM CHANNELS | CALMODULIN | GENE-EXPRESSION | D-ASPARTATE RECEPTOR | SPLICE VARIANTS | BINDING | NEUROSCIENCES | ALPHA-ACTININ | KINASE-II | Xenopus | Protein Binding - genetics | Embryo, Mammalian | Humans | Peptides - genetics | Receptors, N-Methyl-D-Aspartate - metabolism | Green Fluorescent Proteins - genetics | Protein Transport - drug effects | Kv Channel-Interacting Proteins - genetics | Neuroprotective Agents - metabolism | N-Methylaspartate - pharmacology | Mutation - physiology | Neuroprotective Agents - pharmacology | Time Factors | Protein Binding - drug effects | Kv Channel-Interacting Proteins - physiology | Disease Models, Animal | Brain Ischemia - complications | Membrane Potentials - drug effects | Brain Edema - etiology | Rats | Repressor Proteins - genetics | Excitatory Amino Acid Agonists - pharmacology | Protein Transport - genetics | Brain Infarction - etiology | Analysis of Variance | Brain Ischemia - drug therapy | Hypoxia | Mice | Mice, Inbred BALB C | L-Lactate Dehydrogenase - metabolism | Membrane Potentials - genetics | Immunoprecipitation | Cricetulus | Brain Infarction - metabolism | Brain Ischemia - metabolism | Oocytes | Patch-Clamp Techniques - methods | Receptors, N-Methyl-D-Aspartate - genetics | Dose-Response Relationship, Drug | Repressor Proteins - physiology | Transfection - methods | Neurons - physiology | Neurons - drug effects | CHO Cells | Cricetinae | Brain Edema - metabolism | RNA, Small Interfering - pharmacology | Cells, Cultured | Gene Expression Regulation - physiology | Hippocampus - cytology | Gene Expression Regulation - drug effects | Glucose - deficiency | Animals | Cell Count - methods | Biotinylation - methods | Peptides - therapeutic use | Index Medicus
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 12/2014, Volume 9, Issue 12, p. e115731
Normal and painful stimuli are detected by specialized subgroups of peripheral sensory neurons. The understanding of the functional differences of each... 
CAPSAICIN RECEPTOR | PERIPHERAL SENSORY NEURONS | GENE-RELATED PEPTIDE | PRIMARY AFFERENT NEURONS | MULTIDISCIPLINARY SCIENCES | INFLAMMATORY PAIN | PROSTATIC ACID-PHOSPHATASE | VANILLOID RECEPTOR-1 | DORSAL-ROOT GANGLION | KINASE-II | MOLECULAR-MECHANISMS | Receptors, G-Protein-Coupled - metabolism | Capsaicin - pharmacology | TRPV Cation Channels - agonists | Cells, Cultured | Paracrine Communication - genetics | Rats | Male | Gene Expression Profiling | Rats, Sprague-Dawley | Sequence Analysis, DNA | TRPV Cation Channels - genetics | Potassium Channels - metabolism | TRPV Cation Channels - metabolism | Animals | Pain - genetics | Prostaglandin D2 - metabolism | Base Sequence | Phosphatidylinositols - metabolism | Nociceptors - metabolism | Prostaglandin D2 - biosynthesis | Ganglia, Spinal - metabolism | Proteins | Somatotropin | RNA | Neurons | Analysis | Enrichment | Intensive care | Calcium | Genomics | Centrifugation | Genes | Paracrine signalling | Capsaicin receptors | Genomes | Arthritis | Kinases | Subgroups | Gene sequencing | Calcium signalling | Phosphatidylinositol | Receptors | Pain | Prostaglandin D2 synthase | Sensory neurons | Genetics | Ion channels | Bioinformatics | Sensitizing | Adenosine | Pain perception | Gene expression | Ribonucleic acid--RNA | Ganglia | Medicine | Studies | DNA microarrays | Microscopy | Anesthesiology | Binding sites | Ribonucleic acid
Journal Article
The Journal of Physiology, ISSN 0022-3751, 12/2013, Volume 591, Issue 23, pp. 5923 - 5937
Key points •  Previous studies have suggested that the K+ channel auxiliary subunit K+ channel‐interacting protein 2 (KChIP2) serves as a regulator of cardiac... 
Animals | Mice, Inbred C57BL | Heart Failure - physiopathology | Male | Mice | Kv Channel-Interacting Proteins - physiology | Arrhythmias, Cardiac - physiopathology | Mice, Knockout | Heart failure | Proteins | Cardiac arrhythmia | Protein expression | Rodents | Cardiovascular
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2015, Volume 10, Issue 5, p. e0127776
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2014, Volume 9, Issue 3, p. e93315
Reactive oxygen species (ROS) regulate ion channels, modulate neuronal excitability, and contribute to the etiology of neurodegenerative disorders. ROS... 
CEREBELLAR GRANULE NEURONS | BUTYL HYDROPEROXIDE | OXIDATIVE STRESS | MOLECULAR PHYSIOLOGY | GATED K+ CHANNELS | MULTIDISCIPLINARY SCIENCES | N-TYPE INACTIVATION | CLOSED-STATE INACTIVATION | FUNCTIONAL-CHARACTERIZATION | 2-IODOMELATONIN PREVENTS APOPTOSIS | BETA-SUBUNIT | Glutathione - metabolism | Humans | Reactive Oxygen Species - pharmacology | Molecular Sequence Data | Diamide - pharmacology | Kv Channel-Interacting Proteins - genetics | Kv Channel-Interacting Proteins - metabolism | Protein Subunits - metabolism | Potassium Channels - metabolism | Oocytes - cytology | Dipeptidyl-Peptidases and Tripeptidyl-Peptidases - metabolism | Shal Potassium Channels - genetics | Transfection | Cattle | Oocytes - drug effects | Female | tert-Butylhydroperoxide - pharmacology | Action Potentials - drug effects | Dipeptidyl-Peptidases and Tripeptidyl-Peptidases - genetics | Repressor Proteins - metabolism | Dithiothreitol - pharmacology | Protein Subunits - genetics | Amino Acid Sequence | Gene Expression | Oocytes - metabolism | Xenopus laevis | Repressor Proteins - genetics | Nerve Tissue Proteins - genetics | Shal Potassium Channels - antagonists & inhibitors | Potassium Channels - genetics | Nerve Tissue Proteins - metabolism | Sequence Homology, Amino Acid | Patch-Clamp Techniques | Shal Potassium Channels - metabolism | Animals | Protein Subunits - antagonists & inhibitors | Cysteine | Cystine | Oxidative stress | Reactive oxygen species | Neurosciences | Butyl hydroperoxide | Serine | Oxidizing agents | Inactivation | Proteins | Oxidants | Etiology | Rodents | Ion channels | Oxidation | Potassium channels (voltage-gated) | Dithiothreitol | Glutathione | Immunoglobulins | Deactivation | Neurodegenerative diseases | Excitability | Medicine | Sensitivity | Mutation | Laboratory animals | Potassium | Apoptosis
Journal Article
Autonomic neuroscience : basic & clinical, 12/2008, Volume 144, Issue 1-2, p. 22
Nerve sprouting in healed myocardial infarction has been associated with increased incidences of ventricular tachyarrhythmia and sudden cardiac death. However,... 
Ventricular Fibrillation - metabolism | Rats, Wistar | Nerve Regeneration - physiology | Sympathetic Fibers, Postganglionic - metabolism | Male | Extracellular Signal-Regulated MAP Kinases - metabolism | Cyclic AMP Response Element-Binding Protein - drug effects | Ventricular Fibrillation - etiology | Kv Channel-Interacting Proteins - metabolism | Potassium Channels - metabolism | Shal Potassium Channels - drug effects | Neuroprotective Agents - pharmacology | Up-Regulation - physiology | Tachycardia, Ventricular - metabolism | Catechols - pharmacology | Ventricular Fibrillation - physiopathology | Phosphorylation - drug effects | Sympathetic Fibers, Postganglionic - drug effects | Disease Models, Animal | Kv Channel-Interacting Proteins - drug effects | Potassium Channels, Inwardly Rectifying - drug effects | Tachycardia, Ventricular - etiology | Nerve Growth Factor - agonists | Rats | Potassium Channels - drug effects | Sympathetic Fibers, Postganglionic - physiopathology | Up-Regulation - drug effects | Myocardial Infarction - complications | Shal Potassium Channels - metabolism | Tachycardia, Ventricular - physiopathology | Animals | Growth Cones - metabolism | Growth Cones - ultrastructure | Cyclic AMP Response Element-Binding Protein - metabolism | Nerve Regeneration - drug effects | Growth Cones - drug effects | Potassium Channels, Inwardly Rectifying - metabolism
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2013, Volume 8, Issue 2, p. e56179
Journal Article