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ENDOCRINOLOGY, ISSN 0013-7227, 09/2018, Volume 159, Issue 9, pp. 3143 - 3157
Journal Article
Circulation research, ISSN 0009-7330, 2013, Volume 112, Issue 10, pp. 1310 - 1322
A chromosomal haplotype producing cardiac overexpression of dipeptidyl peptidase-like protein-6 (DPP6) causes familial idiopathic ventricular fibrillation. The... 
Molecular electrophysiology | Ventricular tachycardia arrhythmia | Genetic arrhythmia syndromes | Sudden death | ECG | Cardiac arrhythmia mechanisms | Potassium channels | CELLS | CARDIAC & CARDIOVASCULAR SYSTEMS | molecular electrophysiology | K+-CURRENTS | CURRENT I | sudden death | BRUGADA-SYNDROME | potassium channels | genetic arrhythmia syndromes | PERIPHERAL VASCULAR DISEASE | cardiac arrhythmia mechanisms | LONG QT SYNDROME | IONIC MECHANISMS | GENE KNOCKDOWN | CONGESTIVE-HEART-FAILURE | HEMATOLOGY | EXPRESSION | CANINE | ventricular tachycardia arrhythmia | Tetraethylammonium - pharmacology | Cricetulus | Humans | Middle Aged | Potassium Channels - physiology | Male | Kv Channel-Interacting Proteins - genetics | Gene Knockdown Techniques | Shal Potassium Channels - drug effects | Shal Potassium Channels - genetics | Transfection | Ventricular Fibrillation - physiopathology | Adult | Female | Kv Channel-Interacting Proteins - physiology | Dipeptidyl-Peptidases and Tripeptidyl-Peptidases - genetics | Heart Ventricles - pathology | Potassium Channel Blockers - pharmacology | CHO Cells | Disease Models, Animal | Dipeptidyl-Peptidases and Tripeptidyl-Peptidases - physiology | Models, Theoretical | Shal Potassium Channels - physiology | Cricetinae | Nerve Tissue Proteins - physiology | Kv Channel-Interacting Proteins - drug effects | Cells, Cultured | Potassium Channels - drug effects | Nerve Tissue Proteins - genetics | Potassium Channels - genetics | Purkinje Fibers - pathology | Patch-Clamp Techniques | Animals | Heart Ventricles - physiopathology | Dogs | In Vitro Techniques | Purkinje Fibers - physiology | Index Medicus
Journal Article
Psychopharmacology, ISSN 0033-3158, 6/2015, Volume 232, Issue 11, pp. 1995 - 2006
Chronic alcohol-induced cognitive impairments and maladaptive plasticity of glutamatergic synapses are well-documented. However, it is unknown if prolonged... 
Neurosciences | CA1 pyramidal neurons | Chronic alcohol | Biomedicine | Back-propagating action potentials | Kv4.2 channels | KChIP3 | Dendritic calcium transients | Pharmacology/Toxicology | Psychiatry | CHRONIC INTERMITTENT ETHANOL | EMERGENCY ROOM | BLOOD-ALCOHOL | PSYCHIATRY | D-ASPARTATE RECEPTORS | SYNAPTIC PLASTICITY | NEUROSCIENCES | NUCLEUS-ACCUMBENS | NMDA-RECEPTOR | PHARMACOLOGY & PHARMACY | LONG-TERM POTENTIATION | K+ CHANNELS | Dendrites - drug effects | Pyramidal Cells - metabolism | Shal Potassium Channels - physiology | Kv Channel-Interacting Proteins - drug effects | Calcium - metabolism | Calcium Signaling - physiology | Neuronal Plasticity - drug effects | Rats | Hippocampus - drug effects | Rats, Sprague-Dawley | Action Potentials - physiology | Shal Potassium Channels - drug effects | Animals | Neuronal Plasticity - physiology | Synapses - metabolism | Calcium Signaling - drug effects | Dendrites - physiology | Female | Kv Channel-Interacting Proteins - physiology | Alcoholism - physiopathology | Hippocampus - physiopathology | Action Potentials - drug effects | Neuroplasticity | Alcoholics | Physiological aspects | Genetic aspects | Research | Potassium channels | Brain | Alcohol | Psychopharmacology | Index Medicus | back-propagating action potentials | dendritic calcium transients | chronic alcohol
Journal Article
Toxicology, ISSN 0300-483X, 2015, Volume 333, pp. 107 - 117
Abstract 4-Aminopyridine (4-AP) is a potassium channel blocker used for the treatment of neuromuscular disorders. Otherwise, it has been described to produce a... 
Emergency | GABA | Rats | KChIP1 | 4-Aminopyridine | Cell death | Kv 4.3 | APOPTOSIS | RAT | STATUS EPILEPTICUS | CONDITIONED FEAR | CULTURED CORTICAL-NEURONS | IN-VITRO | SEIZURES | MULTIPLE-SCLEROSIS | DOUBLE-BLIND | PHARMACOLOGY & PHARMACY | TOXICOLOGY | INHIBITS PROLIFERATION | Caspase 7 - metabolism | Rats, Wistar | Apoptosis - drug effects | gamma-Aminobutyric Acid - metabolism | Caspase 3 - metabolism | Hippocampus - drug effects | Kv Channel-Interacting Proteins - genetics | Kv Channel-Interacting Proteins - metabolism | Dose-Response Relationship, Drug | Shal Potassium Channels - genetics | Neuroprotective Agents - pharmacology | Transfection | RNA Interference | Time Factors | Semicarbazides - pharmacology | Synaptic Transmission - drug effects | Potassium Channel Blockers - toxicity | 4-Aminopyridine - pharmacology | Potassium Channel Blockers - pharmacology | Cytoprotection | Cell Survival - drug effects | Hippocampus - embryology | Kv Channel-Interacting Proteins - drug effects | Cells, Cultured | Neuroprotective Agents - toxicity | Shal Potassium Channels - antagonists & inhibitors | Hippocampus - metabolism | Shal Potassium Channels - metabolism | Animals | 4-Aminopyridine - toxicity | Primary Cell Culture | Genetic research | Potassium channels | Gene expression | Analysis | Index Medicus | Proteins | Toxicology | Toxic | Exposure | Potassium | Protective | Channels
Journal Article
Circulation Journal, ISSN 1346-9843, 2006, Volume 70, Issue 5, pp. 605 - 609
Journal Article
Journal Article
Autonomic neuroscience : basic & clinical, 12/2008, Volume 144, Issue 1-2, pp. 22 - 29
Nerve sprouting in healed myocardial infarction has been associated with increased incidences of ventricular tachyarrhythmia and sudden cardiac death. However,... 
Ventricular Fibrillation - metabolism | Rats, Wistar | Nerve Regeneration - physiology | Sympathetic Fibers, Postganglionic - metabolism | Male | Extracellular Signal-Regulated MAP Kinases - metabolism | Cyclic AMP Response Element-Binding Protein - drug effects | Ventricular Fibrillation - etiology | Kv Channel-Interacting Proteins - metabolism | Potassium Channels - metabolism | Shal Potassium Channels - drug effects | Neuroprotective Agents - pharmacology | Up-Regulation - physiology | Tachycardia, Ventricular - metabolism | Catechols - pharmacology | Ventricular Fibrillation - physiopathology | Phosphorylation - drug effects | Sympathetic Fibers, Postganglionic - drug effects | Disease Models, Animal | Kv Channel-Interacting Proteins - drug effects | Potassium Channels, Inwardly Rectifying - drug effects | Tachycardia, Ventricular - etiology | Nerve Growth Factor - agonists | Rats | Potassium Channels - drug effects | Sympathetic Fibers, Postganglionic - physiopathology | Up-Regulation - drug effects | Myocardial Infarction - complications | Shal Potassium Channels - metabolism | Tachycardia, Ventricular - physiopathology | Animals | Growth Cones - metabolism | Growth Cones - ultrastructure | Cyclic AMP Response Element-Binding Protein - metabolism | Nerve Regeneration - drug effects | Growth Cones - drug effects | Potassium Channels, Inwardly Rectifying - metabolism | Index Medicus
Journal Article
Journal of Cellular Biochemistry, ISSN 0730-2312, 04/2014, Volume 115, Issue 4, pp. 678 - 689
Journal Article
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 04/2013, Volume 433, Issue 4, pp. 508 - 512
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 06/2001, Volume 21, Issue 12, pp. 4154 - 4161
Journal Article
Biological Psychiatry, ISSN 0006-3223, 2015, Volume 77, Issue 2, pp. 95 - 105
Abstract Background Previous studies have implicated the cyclic adenosine monophosphate/protein kinase A pathway as well as FosB and dynorphin-B expression... 
Psychiatry | FosB | Phospho-GluR1 | Dopaminergic denervation | Abnormal involuntary movements | Dynorphin-B | Parkinson’s disease | Parkinson's disease | NMDA RECEPTOR | HEMIPARKINSONIAN MICE | AMPA RECEPTOR TRAFFICKING | PSYCHIATRY | L-3,4-DIHYDROXYPHENYLALANINE-INDUCED DYSKINESIA | SYNAPTIC PLASTICITY | NEUROSCIENCES | GENE-EXPRESSION | TRANSCRIPTIONAL REPRESSOR | CA2+ HOMEOSTASIS | PARKINSONS-DISEASE | Immunohistochemistry | Motor Activity - physiology | Phosphorylation | Antiparkinson Agents - pharmacology | Dyskinesia, Drug-Induced - physiopathology | Motor Activity - drug effects | Oxidopamine | Kv Channel-Interacting Proteins - genetics | Kv Channel-Interacting Proteins - metabolism | Parkinsonian Disorders - drug therapy | Acetylation | Dynorphins - metabolism | Receptors, AMPA - metabolism | Repressor Proteins - metabolism | Levodopa - pharmacology | Parkinsonian Disorders - physiopathology | Corpus Striatum - physiopathology | Proto-Oncogene Proteins c-fos - metabolism | Repressor Proteins - genetics | Antiparkinson Agents - adverse effects | Blotting, Western | Mice, Knockout | Endorphins - metabolism | Rotarod Performance Test | Animals | Levodopa - adverse effects | Corpus Striatum - drug effects | Histones - metabolism | Resveratrol | Phenols | Genetic engineering | Chemical properties | Binding proteins | Movement disorders | Calmodulin | Protein binding | Index Medicus
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 06/2010, Volume 30, Issue 22, pp. 7575 - 7586
Glutamate-induced excitotoxicity has been implicated in the etiology of stroke, epilepsy, and neurodegenerative diseases. NMDA receptors (NMDARs) play a... 
TRANSCRIPTIONAL REPRESSOR DREAM | CALCIUM-SENSOR PROTEINS | POTASSIUM CHANNELS | CALMODULIN | GENE-EXPRESSION | D-ASPARTATE RECEPTOR | SPLICE VARIANTS | BINDING | NEUROSCIENCES | ALPHA-ACTININ | KINASE-II | Xenopus | Protein Binding - genetics | Embryo, Mammalian | Humans | Peptides - genetics | Receptors, N-Methyl-D-Aspartate - metabolism | Green Fluorescent Proteins - genetics | Protein Transport - drug effects | Kv Channel-Interacting Proteins - genetics | Neuroprotective Agents - metabolism | N-Methylaspartate - pharmacology | Mutation - physiology | Neuroprotective Agents - pharmacology | Time Factors | Protein Binding - drug effects | Kv Channel-Interacting Proteins - physiology | Disease Models, Animal | Brain Ischemia - complications | Membrane Potentials - drug effects | Brain Edema - etiology | Rats | Repressor Proteins - genetics | Excitatory Amino Acid Agonists - pharmacology | Protein Transport - genetics | Brain Infarction - etiology | Analysis of Variance | Brain Ischemia - drug therapy | Hypoxia | Mice | Mice, Inbred BALB C | L-Lactate Dehydrogenase - metabolism | Membrane Potentials - genetics | Immunoprecipitation | Cricetulus | Brain Infarction - metabolism | Brain Ischemia - metabolism | Oocytes | Patch-Clamp Techniques - methods | Receptors, N-Methyl-D-Aspartate - genetics | Dose-Response Relationship, Drug | Repressor Proteins - physiology | Transfection - methods | Neurons - physiology | Neurons - drug effects | CHO Cells | Cricetinae | Brain Edema - metabolism | RNA, Small Interfering - pharmacology | Cells, Cultured | Gene Expression Regulation - physiology | Hippocampus - cytology | Gene Expression Regulation - drug effects | Glucose - deficiency | Animals | Cell Count - methods | Biotinylation - methods | Peptides - therapeutic use | Index Medicus
Journal Article