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The Journal of Physiology, ISSN 0022-3751, 12/2013, Volume 591, Issue 23, pp. 5923 - 5937
•  Previous studies have suggested that the K + channel auxiliary subunit K + channel‐interacting protein 2 (KChIP2) serves as a regulator of cardiac... 
Animals | Mice, Inbred C57BL | Heart Failure - physiopathology | Male | Mice | Kv Channel-Interacting Proteins - physiology | Arrhythmias, Cardiac - physiopathology | Mice, Knockout | Heart failure | Proteins | Cardiac arrhythmia | Protein expression | Rodents | Index Medicus | Cardiovascular
Journal Article
Circulation research, ISSN 0009-7330, 2013, Volume 112, Issue 10, pp. 1310 - 1322
A chromosomal haplotype producing cardiac overexpression of dipeptidyl peptidase-like protein-6 (DPP6) causes familial idiopathic ventricular fibrillation. The... 
Molecular electrophysiology | Ventricular tachycardia arrhythmia | Genetic arrhythmia syndromes | Sudden death | ECG | Cardiac arrhythmia mechanisms | Potassium channels | CELLS | CARDIAC & CARDIOVASCULAR SYSTEMS | molecular electrophysiology | K+-CURRENTS | CURRENT I | sudden death | BRUGADA-SYNDROME | potassium channels | genetic arrhythmia syndromes | PERIPHERAL VASCULAR DISEASE | cardiac arrhythmia mechanisms | LONG QT SYNDROME | IONIC MECHANISMS | GENE KNOCKDOWN | CONGESTIVE-HEART-FAILURE | HEMATOLOGY | EXPRESSION | CANINE | ventricular tachycardia arrhythmia | Tetraethylammonium - pharmacology | Cricetulus | Humans | Middle Aged | Potassium Channels - physiology | Male | Kv Channel-Interacting Proteins - genetics | Gene Knockdown Techniques | Shal Potassium Channels - drug effects | Shal Potassium Channels - genetics | Transfection | Ventricular Fibrillation - physiopathology | Adult | Female | Kv Channel-Interacting Proteins - physiology | Dipeptidyl-Peptidases and Tripeptidyl-Peptidases - genetics | Heart Ventricles - pathology | Potassium Channel Blockers - pharmacology | CHO Cells | Disease Models, Animal | Dipeptidyl-Peptidases and Tripeptidyl-Peptidases - physiology | Models, Theoretical | Shal Potassium Channels - physiology | Cricetinae | Nerve Tissue Proteins - physiology | Kv Channel-Interacting Proteins - drug effects | Cells, Cultured | Potassium Channels - drug effects | Nerve Tissue Proteins - genetics | Potassium Channels - genetics | Purkinje Fibers - pathology | Patch-Clamp Techniques | Animals | Heart Ventricles - physiopathology | Dogs | In Vitro Techniques | Purkinje Fibers - physiology | Index Medicus
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 05/2017, Volume 141, Issue 4, pp. 544 - 552
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2015, Volume 10, Issue 7, pp. e0133274 - e0133274
Gradients of the fast transient outward K+ current (I-to,I- f) contribute to heterogeneity of ventricular repolarization in a number of species. Cardiac... 
HEART | HETEROGENEITY | MESSENGER-RNA | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | DOWN-REGULATION | K+ CURRENTS | BETA-SUBUNIT | CANINE | POTASSIUM CHANNEL | REPOLARIZATION | Action Potentials - genetics | Spider Venoms - pharmacology | Cell Membrane - physiology | Kv Channel-Interacting Proteins - genetics | Kv Channel-Interacting Proteins - metabolism | Protein Subunits - metabolism | Shal Potassium Channels - genetics | Cell Membrane - metabolism | Kv Channel-Interacting Proteins - physiology | Protein Subunits - physiology | Action Potentials - drug effects | Cell Membrane - drug effects | Potassium Channel Blockers - pharmacology | Protein Subunits - genetics | Shal Potassium Channels - physiology | Gene Expression | Myocytes, Cardiac - cytology | Mice, Inbred C57BL | Cells, Cultured | Mice, Transgenic | Reverse Transcriptase Polymerase Chain Reaction | Mice, Knockout | Action Potentials - physiology | Patch-Clamp Techniques | Shal Potassium Channels - metabolism | Animals | Myocytes, Cardiac - physiology | Fluorescent Antibody Technique | Myocytes, Cardiac - metabolism | RNA | Heart cells | Heart failure | Heart | Cardiac arrhythmia | Cardiomyocytes | Gene expression | Coronary artery disease | Depolarization | Medicine | Studies | Health care networks | Rodents | Stem cells | Physiology | Potassium channels (voltage-gated) | Ventricle | Cardiology | Heart diseases | Potassium currents | Potassium | Voltage pulses | Pore formation | Index Medicus
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 06/2010, Volume 30, Issue 22, pp. 7575 - 7586
Glutamate-induced excitotoxicity has been implicated in the etiology of stroke, epilepsy, and neurodegenerative diseases. NMDA receptors (NMDARs) play a... 
TRANSCRIPTIONAL REPRESSOR DREAM | CALCIUM-SENSOR PROTEINS | POTASSIUM CHANNELS | CALMODULIN | GENE-EXPRESSION | D-ASPARTATE RECEPTOR | SPLICE VARIANTS | BINDING | NEUROSCIENCES | ALPHA-ACTININ | KINASE-II | Xenopus | Protein Binding - genetics | Embryo, Mammalian | Humans | Peptides - genetics | Receptors, N-Methyl-D-Aspartate - metabolism | Green Fluorescent Proteins - genetics | Protein Transport - drug effects | Kv Channel-Interacting Proteins - genetics | Neuroprotective Agents - metabolism | N-Methylaspartate - pharmacology | Mutation - physiology | Neuroprotective Agents - pharmacology | Time Factors | Protein Binding - drug effects | Kv Channel-Interacting Proteins - physiology | Disease Models, Animal | Brain Ischemia - complications | Membrane Potentials - drug effects | Brain Edema - etiology | Rats | Repressor Proteins - genetics | Excitatory Amino Acid Agonists - pharmacology | Protein Transport - genetics | Brain Infarction - etiology | Analysis of Variance | Brain Ischemia - drug therapy | Hypoxia | Mice | Mice, Inbred BALB C | L-Lactate Dehydrogenase - metabolism | Membrane Potentials - genetics | Immunoprecipitation | Cricetulus | Brain Infarction - metabolism | Brain Ischemia - metabolism | Oocytes | Patch-Clamp Techniques - methods | Receptors, N-Methyl-D-Aspartate - genetics | Dose-Response Relationship, Drug | Repressor Proteins - physiology | Transfection - methods | Neurons - physiology | Neurons - drug effects | CHO Cells | Cricetinae | Brain Edema - metabolism | RNA, Small Interfering - pharmacology | Cells, Cultured | Gene Expression Regulation - physiology | Hippocampus - cytology | Gene Expression Regulation - drug effects | Glucose - deficiency | Animals | Cell Count - methods | Biotinylation - methods | Peptides - therapeutic use | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2017, Volume 12, Issue 4, pp. e0175221 - e0175221
Pathologic electrical remodeling and attenuated cardiac contractility are featured characteristics of heart failure. Coinciding with these remodeling events is... 
HYPERTROPHY | PROTEIN | PRESENILINS | POTASSIUM CHANNELS | CALSENILIN | ALZHEIMERS-DISEASE | MULTIDISCIPLINARY SCIENCES | SINGLE-CHANNEL ACTIVITY | VENTRICULAR MYOCYTES | N-TERMINUS | I-TO | Immunohistochemistry | Animals | Guinea Pigs | Calcium - metabolism | Cells, Cultured | Muscle Cells - metabolism | Ryanodine Receptor Calcium Release Channel - metabolism | Kv Channel-Interacting Proteins - physiology | Kv Channel-Interacting Proteins - genetics | Gene Knockdown Techniques | Myocardial infarction | Heart | Brain | Regulations | Salts | Trafficking | Carbon dioxide | Ryanodine receptors | Nervous system | Biochemistry | Glucose | Inactivation | Fragmentation | Finite element method | Proteins | Degradation | Signal transduction | Temperature effects | Aorta | Genetics | Physiology | Membrane potential | Alzheimer's disease | Failure | Heart failure | Neurodegenerative diseases | Cardiac muscle | Macromolecules | Cultures | Digestion | Myocardium | β-Amyloid | Mice | Parks | Circulation | Neurosciences | Animal models | Calcium | Gene regulation | Action potential | Stimulation | Accessories | Heterogeneity | Rodents | Calcium-binding protein | Modulation | Attenuation | Excitation | Heart diseases | Neurons | Antisense RNA | Light emitting diodes | Biophysics | Medicine | Brain research | Antibiotics | Adenoviruses | N-Terminus | Position (location) | Alzheimers disease | Potassium | Ca2+/calmodulin-dependent protein kinase II | Index Medicus | pH
Journal Article
Psychopharmacology, ISSN 0033-3158, 6/2015, Volume 232, Issue 11, pp. 1995 - 2006
Chronic alcohol-induced cognitive impairments and maladaptive plasticity of glutamatergic synapses are well-documented. However, it is unknown if prolonged... 
Neurosciences | CA1 pyramidal neurons | Chronic alcohol | Biomedicine | Back-propagating action potentials | Kv4.2 channels | KChIP3 | Dendritic calcium transients | Pharmacology/Toxicology | Psychiatry | CHRONIC INTERMITTENT ETHANOL | EMERGENCY ROOM | BLOOD-ALCOHOL | PSYCHIATRY | D-ASPARTATE RECEPTORS | SYNAPTIC PLASTICITY | NEUROSCIENCES | NUCLEUS-ACCUMBENS | NMDA-RECEPTOR | PHARMACOLOGY & PHARMACY | LONG-TERM POTENTIATION | K+ CHANNELS | Dendrites - drug effects | Pyramidal Cells - metabolism | Shal Potassium Channels - physiology | Kv Channel-Interacting Proteins - drug effects | Calcium - metabolism | Calcium Signaling - physiology | Neuronal Plasticity - drug effects | Rats | Hippocampus - drug effects | Rats, Sprague-Dawley | Action Potentials - physiology | Shal Potassium Channels - drug effects | Animals | Neuronal Plasticity - physiology | Synapses - metabolism | Calcium Signaling - drug effects | Dendrites - physiology | Female | Kv Channel-Interacting Proteins - physiology | Alcoholism - physiopathology | Hippocampus - physiopathology | Action Potentials - drug effects | Neuroplasticity | Alcoholics | Physiological aspects | Genetic aspects | Research | Potassium channels | Brain | Alcohol | Psychopharmacology | Index Medicus | back-propagating action potentials | dendritic calcium transients | chronic alcohol
Journal Article