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Journal Article
Journal Article
Journal Article
Molecular Neurobiology, ISSN 0893-7648, 9/2017, Volume 54, Issue 7, pp. 4996 - 5007
Voltage-gated calcium channels are key regulators of brain function, and their dysfunction has been associated with multiple conditions and neurodegenerative... 
Calcium signaling | Neurology | Neurosciences | Voltage-gated calcium channels | Biomedicine | Estrogen non-genomic actions | DHP binding site | Neurobiology | Estrogen receptor-independent mechanisms | Estradiol | Cell Biology | CORTICAL SPREADING DEPRESSION | ALZHEIMERS-DISEASE | CA2+ CHANNELS | POTENTIAL INITIATION MECHANISM | NON-GENOMIC ACTIONS | SYNAPTIC PLASTICITY | HIPPOCAMPAL-NEURONS | NEUROSCIENCES | ESTROGEN-RECEPTOR-BETA | NEURONAL EXCITABILITY | ASTROCYTIC CELLS | Estradiol - metabolism | Animals | Signal Transduction - drug effects | Calcium - metabolism | Calcium Channels, L-Type - drug effects | Humans | Neurons - metabolism | Calcium Channels, L-Type - metabolism | Estrogens - metabolism | Neurons - drug effects | Estradiol - pharmacology | Nervous system diseases | Calcium channels | Phenols | Genetic transcription | Calcium-binding proteins | Protein binding | Brain | Regulators | Transcription | Calcium | Computational neuroscience | Estrogens | Estrogen receptors | Intracellular signalling | Kinases | Channels | Calcium influx | Depolarization | Calcium signalling | Proteins | Pathways | Cascades | Modulation | Membrane potential | Drug therapy | Calcium (intracellular) | Neurodegenerative diseases | Calcium channels (voltage-gated) | Gene expression | Neurological diseases | Sex hormones | Brain research | Computer applications | Calcium channels (L-type) | Intracellular | Regulatory proteins
Journal Article
Journal of General Physiology, ISSN 0022-1295, 2014, Volume 143, Issue 6, pp. 679 - 692
Journal Article
Journal of Neurophysiology, ISSN 0022-3077, 11/2004, Volume 92, Issue 5, pp. 2633 - 2641
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 10/2016, Volume 139, Issue S1, pp. 156 - 178
We propose that SN DA neurons possess several feedback and feed‐forward mechanisms to protect and adapt their activity‐pattern and calcium‐homeostasis within a... 
Kir6.2 | SUR | 2 channels | hip3 potassium channels | 1 potassium channels | D2‐autoreceptor‐coupled | GIRK | neuronal calcium sensor | Cav1.3 L‐type/Cav3.1 T‐type calcium channels | ambroxol | A‐type Kv4.3 | NCS | Cav1.3 L-type/Cav3.1 T-type calcium channels | neuronal calcium sensor NCS-1 | A-type Kv4.3/KChip3 potassium channels | Kir6.2/SUR1 potassium channels | D2-autoreceptor-coupled GIRK2 channels | SENSITIVE POTASSIUM CHANNELS | Kir6 | Cav3 | OXIDANT STRESS | Cav1 | K-ATP CHANNELS | BIOCHEMISTRY & MOLECULAR BIOLOGY | CA2+ CHANNELS | A-type Kv4 | GLUCOCEREBROSIDASE MUTATIONS | NEUROSCIENCES | KChip3 potassium channels | 3L-type | D2-AUTORECEPTOR RESPONSES | ALPHA-SYNUCLEIN FORM | SUR1 potassium channels | LOCUS-COERULEUS | MPTP MOUSE MODEL | MITOCHONDRIAL-DNA DELETIONS | 1 T-type calcium channels | Stress, Physiological - physiology | Parkinson Disease - pathology | Animals | Calcium - physiology | Substantia Nigra - pathology | Dopaminergic Neurons - pathology | Dopaminergic Neurons - metabolism | Humans | Ion Channels - physiology | Parkinson Disease - metabolism | Substantia Nigra - metabolism | Health Status | Calcium channels | Stress (Physiology) | Parkinson's disease | Potassium channels | Neurons | Dopamine | Calcium | Animal behavior | Rodents | Bandwidths | Parkinsons disease | Homeostasis | Physiology | Metabolism | Cav3.1 T‐type calcium channels | D2‐autoreceptor‐coupled GIRK2 channels | Cav1.3 L‐type | Jörg B. Schulz and John Hardy | Review | neuronal calcium sensor NCS‐1 | Parkinson Disease. Guest Editors
Journal Article