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Journal of the American College of Cardiology, ISSN 0735-1097, 2013, Volume 62, Issue 16, pp. 1401 - 1408
Hypercholesterolemia is a major risk factor for cardiovascular diseases, increasing the incidence of myocardial infarction and death. Statin-induced lowering... 
Cardiovascular | Internal Medicine | atherosclerosis | proprotein convertase subtilisin/kexin type 9 | PCSK9 | LDL cholesterol | LDL receptor | atherosclerosis LDL cholesterol LDL receptor PCSK9 proprotein convertase subtilisin/kexin type 9 | NONHUMAN-PRIMATES | LDL-CHOLESTEROL | CARDIAC & CARDIOVASCULAR SYSTEMS | RANDOMIZED-TRIAL | MONOCLONAL-ANTIBODY | DENSITY-LIPOPROTEIN-CHOLESTEROL | SECRETED PCSK9 | RECEPTOR-MEDIATED ENDOCYTOSIS | STATIN THERAPY | HETEROZYGOUS FAMILIAL HYPERCHOLESTEROLEMIA | SERINE-PROTEASE | Proprotein Convertases - antagonists & inhibitors | Dyslipidemias - complications | Humans | Lipoproteins, LDL - genetics | Proprotein Convertases - genetics | Molecular Targeted Therapy - trends | Serine Endopeptidases - genetics | Lipid Metabolism - genetics | Lipoproteins, LDL - metabolism | Molecular Targeted Therapy - methods | Receptors, LDL - genetics | Dyslipidemias - drug therapy | Atherosclerosis - drug therapy | Proprotein Convertases - metabolism | Risk Factors | Receptors, LDL - metabolism | Atherosclerosis - complications | Hypolipidemic Agents - pharmacology | Forecasting | Atherosclerosis - metabolism | Lipid Metabolism - drug effects | Dyslipidemias - metabolism | Serine Endopeptidases - metabolism | Mutation | Proprotein Convertase 9 | Hypertension | Hypercholesterolemia | Low density lipoproteins | Mortality | Atherosclerosis | Drug therapy | Statins | Cholesterol | Protein binding | Studies | Rodents
Journal Article
Journal of Hepatology, ISSN 0168-8278, 2006, Volume 46, Issue 3, pp. 411 - 419
Background/Aims The direct implication of low-density lipoprotein receptor (LDLR) in hepatitis C virus (HCV) infection of human hepatocyte has not been... 
Gastroenterology and Hepatology | Virus receptor | Genome replication | Virus entry | LDL RECEPTOR | CULTURED HUMAN HEPATOCYTES | IN-VITRO INFECTION | virus receptor | genome replication | CANDIDATE RECEPTOR | SCAVENGER RECEPTOR | HUMAN HEPATOMA-CELLS | STRUCTURAL PROTEINS | virus entry | TERM PRIMARY CULTURES | BINDING-SITE | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | CD18 Antigens - physiology | Humans | Middle Aged | Hepacivirus - genetics | Lipoproteins, LDL - physiology | Hepacivirus - pathogenicity | Hepatocytes - pathology | Male | Antibodies - physiology | Scavenger Receptors, Class B - physiology | Viral Load | RNA, Viral - genetics | Virion | Adult | Female | Hepacivirus - physiology | RNA, Viral - metabolism | Receptors, LDL - genetics | Tricarboxylic Acids - pharmacology | Cells, Cultured | Receptors, LDL - immunology | Hydroxycholesterols - pharmacology | Lipoproteins, HDL - physiology | Gene Expression Regulation - drug effects | Bridged Bicyclo Compounds, Heterocyclic - pharmacology | Adolescent | Hepatocytes - virology | Hepatitis C - physiopathology | Aged | Hepatitis C - pathology | Receptors, LDL - physiology | Anticholesteremic Agents - pharmacology | Hepatitis C | Health aspects | Scavenger Receptors, Class B | Receptors, LDL | Antibodies | Bicyclo Compounds, Heterocyclic | Life Sciences | Tricarboxylic Acids | Anticholesteremic Agents | Hépatology and Gastroenterology | Gene Expression Regulation | Human health and pathology | Hepatocytes | Hydroxycholesterols | RNA, Viral | Antigens, CD18 | Lipoproteins, HDL | Hepacivirus | Lipoproteins, LDL
Journal Article
Journal Article
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1524-4636, 2009, Volume 29, Issue 4, pp. 431 - 438
Journal Article
Journal Article
The Journal of biological chemistry, ISSN 1083-351X, 2010, Volume 285, Issue 17, pp. 12882 - 12891
Journal Article
JAMA : the journal of the American Medical Association, ISSN 0098-7484, 01/2019, Volume 321, Issue 4, pp. 364 - 373
IMPORTANCE: Triglycerides and cholesterol are both carried in plasma by apolipoprotein B (ApoB)–containing lipoprotein particles. It is unknown whether... 
Coronary Disease/blood | Cholesterol, LDL/blood | Genetic Predisposition to Disease | Apolipoproteins B/blood | Loss of Function Mutation | Prospective Studies | Humans | Middle Aged | Risk Factors | Male | Case-Control Studies | Lipoprotein Lipase/genetics | Genetic Variation | Metabolic Networks and Pathways | Mendelian Randomization Analysis | Triglycerides/blood | Female | Receptors, LDL/genetics | GEMFIBROZIL | MEDICINE, GENERAL & INTERNAL | METAANALYSIS | INHIBITION | THERAPY | REDUCTION | CARDIOVASCULAR EVENTS | ATHEROSCLEROSIS | DENSITY-LIPOPROTEIN-CHOLESTEROL | WIDE ASSOCIATION | SECONDARY PREVENTION | Coronary Disease - blood | Receptors, LDL - genetics | Lipoprotein Lipase - genetics | Apolipoproteins B - blood | Coronary Disease - genetics | Triglycerides - blood | Cholesterol, LDL - blood | Lipoprotein Lipase - metabolism | Care and treatment | Genetic variation | Cholesterol, LDL | Genetic aspects | Research | Health aspects | Coronary heart disease | Risk factors | Myocardial infarction | Lipoproteins (low density) | Health risks | Risk | Cardiovascular disease | Lipids | Triglycerides | Lipoprotein lipase | LDLR gene | Lipase | Coronary artery disease | Cholesterol | Apolipoprotein B | Lipoproteins | Randomization | Genetic analysis | Lipoprotein (low density) receptors | Infarction | Cardiovascular diseases | Health risk assessment | Heart diseases | Original Investigation
Journal Article
Journal Article