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FASEB journal : official publication of the Federation of American Societies for Experimental Biology, ISSN 0892-6638, 01/2015, Volume 29, Issue 1, pp. 227 - 238
Journal Article
Gut, ISSN 0017-5749, 03/2012, Volume 61, Issue 3, pp. 416 - 426
ObjectiveMonocyte chemoattractant protein-1 (MCP-1, CCL2), the primary ligand for chemokine receptor C–C chemokine receptor 2 (CCR2), is increased in livers of... 
FIBROSIS | STEATOSIS | INSULIN-RESISTANCE | INFLAMMATION | BONE-MARROW | DISEASE | MONOCYTE SUBSETS | MICE | SPIEGELMER | GASTROENTEROLOGY & HEPATOLOGY | CCR2 | Liver - pathology | Carbon Tetrachloride | Liver Cirrhosis, Experimental - etiology | Bone Marrow Cells - physiology | Male | Chemical and Drug Induced Liver Injury, Chronic - drug therapy | Liver Cirrhosis, Experimental - prevention & control | Non-alcoholic Fatty Liver Disease | Chemokine CCL2 - physiology | Bone Marrow Cells - drug effects | Chemical and Drug Induced Liver Injury - pathology | Chemical and Drug Induced Liver Injury, Chronic - complications | Macrophages - physiology | Aptamers, Nucleotide - pharmacology | Drug Evaluation, Preclinical - methods | Acute Disease | Cytokines - metabolism | Liver - metabolism | Mice, Inbred C57BL | Cells, Cultured | Fatty Liver - prevention & control | Aptamers, Nucleotide - therapeutic use | Chemotaxis - drug effects | Chemical and Drug Induced Liver Injury, Chronic - pathology | Disease Progression | Fatty Liver - drug therapy | Animals | Chemical and Drug Induced Liver Injury - complications | Chemical and Drug Induced Liver Injury - drug therapy | Macrophages - drug effects | Mice | Chemokine CCL2 - antagonists & inhibitors | Fatty Liver - etiology | Care and treatment | Liver diseases | Fibrosis | Models | Research | Macrophages | Chemokine receptors | Studies | Cytokines | Polyethylene glycol | Rodents | Liver | Clinical trials | Tumor necrosis factor-TNF | Inflammation | Diabetes | Metabolic disorders | Chemokines | Immunohistochemistry | CC chemokine receptors | Flow cytometry | Animal models | Leukocyte migration | RNA | Carbon tetrachloride | Oligonucleotides | Blood | Metastases | Interleukin 6 | Fatty liver | CCR2 protein | Degeneration | Lipogenesis | Digestive tract | steatosis | Injuries | Triglycerides | CCL4 protein | Chemotaxis | Diets | Monocytes | gamma -Interferon | Monocyte chemoattractant protein 1 | Index Medicus | Abridged Index Medicus
Journal Article
British Journal of Pharmacology, ISSN 0007-1188, 04/2014, Volume 171, Issue 7, pp. 1706 - 1721
Background and Purpose In contrast to T-cell priming in the periphery, therapeutic strategies targeting the initiation step of T-cell trafficking into the CNS... 
EAE | SHP | cells | CXCR | pioneer | multiple sclerosis | PROTEIN | DENDRITIC CELLS | INTEGRIN | CXCR7 | ARRIVE GUIDELINES | TYROSINE-PHOSPHATASE | PATHOGENESIS | REMITTING MULTIPLE-SCLEROSIS | SHP-2 | DISEASE | P-SELECTIN | PHARMACOLOGY & PHARMACY | pioneer CD8(+) T-cells | EXPRESSION | Protein Tyrosine Phosphatase, Non-Receptor Type 11 - metabolism | Peptide Fragments | Spinal Cord - drug effects | CD8-Positive T-Lymphocytes - transplantation | Encephalomyelitis, Autoimmune, Experimental - immunology | Adoptive Transfer | Quinolines - pharmacology | Chemotaxis, Leukocyte - drug effects | Encephalomyelitis, Autoimmune, Experimental - chemically induced | Time Factors | Receptors, CXCR - metabolism | Female | Cytokines - blood | Anti-Inflammatory Agents - pharmacology | Mice, Inbred C57BL | Cells, Cultured | Enzyme Inhibitors - pharmacology | Protein Tyrosine Phosphatase, Non-Receptor Type 11 - antagonists & inhibitors | Spinal Cord - enzymology | Myelin-Oligodendrocyte Glycoprotein | Biomarkers - blood | CD8-Positive T-Lymphocytes - enzymology | Mice, Knockout | Spinal Cord - immunology | Animals | Encephalomyelitis, Autoimmune, Experimental - enzymology | CD8-Positive T-Lymphocytes - drug effects | Encephalomyelitis, Autoimmune, Experimental - prevention & control | Mice | Protein Tyrosine Phosphatase, Non-Receptor Type 11 - genetics | CD8-Positive T-Lymphocytes - immunology | Receptors, CXCR - drug effects | Genetic engineering | Encephalomyelitis | T cells | Analysis | Cytokines | Rodents | Immune system | Index Medicus | pioneer CD8+ T-cells | Research Paper
Journal Article
Surgery: Official Journal of the Society of University Surgeons, Central Surgical Association, and the American Association of Endocrine Surgeons, ISSN 0039-6060, 2013, Volume 154, Issue 3, pp. 596 - 603
Journal Article
Acta Neuropathologica, ISSN 0001-6322, 3/2013, Volume 125, Issue 3, pp. 395 - 412
The migration of polymorphonuclear granulocytes (PMN) into the brain parenchyma and release of their abundant proteases are considered the main causes of... 
Human | Pathology | Neurosciences | Medicine & Public Health | Mouse | Migration | Neurovascular unit | Polymorphonuclear granulocyte | STROKE TRIAL | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | COLONY-STIMULATING FACTOR | ARTERY OCCLUSION | FOCAL CEREBRAL-ISCHEMIA | PATHOLOGY | NEUROSCIENCES | CLINICAL NEUROLOGY | ENDOTHELIAL GLYCOCALYX | IN-VIVO | P-SELECTIN | CENTRAL-NERVOUS-SYSTEM | BASEMENT-MEMBRANE | Blood Vessels - pathology | Humans | Blood-Brain Barrier - physiopathology | Male | Infarction, Middle Cerebral Artery - immunology | Antigens, CD - metabolism | Blood Vessels - physiopathology | Antigens, Ly - metabolism | Granulocytes - pathology | Disease Models, Animal | Endothelium - pathology | Mice, Inbred C57BL | Cells, Cultured | Gene Expression Regulation - physiology | Functional Laterality | Infarction, Middle Cerebral Artery - pathology | Cell Adhesion Molecules - metabolism | Blood-Brain Barrier - pathology | Glucose - deficiency | Oxygen - administration & dosage | Animals | Models, Biological | Brain - pathology | Hypoxia | Mice | Glucose metabolism | Permeability | Glucose | Ischemia | Proteases | Dextrose | Index Medicus | Neuroimaging | Brain | Cerebral infarction | Stroke | Leukocyte migration | Central nervous system | Parenchyma | Confocal microscopy | Basement membranes | Platelet aggregation | Macrophages | Endothelial cells | Cell adhesion molecules | Reperfusion | Cerebral blood flow | Leukocytes (granulocytic) | Brain injury | Original Paper
Journal Article