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Journal of the Neurological Sciences, ISSN 0022-510X, 04/2019, Volume 399, pp. 144 - 150
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 4, p. e19338
Journal Article
Neurology, ISSN 0028-3878, 11/2015, Volume 85, Issue 19, pp. 1670 - 1679
OBJECTIVE:To quantify the loss of pigmented neurons in the substantia nigra (SN) of autopsy-confirmed Parkinson disease (PD) and incidental Lewy body disease... 
DEMENTIA | ALPHA-SYNUCLEIN PATHOLOGY | INVOLVEMENT | DISORDERS | BODIES | NEUROPATHOLOGY | SUBSTANTIA-NIGRA | BRAIN | CLINICAL NEUROLOGY | AUTONOMIC NERVOUS-SYSTEM | DEGENERATION | Neurons - pathology | Parkinson Disease - pathology | Substantia Nigra - pathology | Time Factors | Humans | Lewy Body Disease - pathology | Aged, 80 and over | Female | Male | Aged | Longitudinal Studies | 165
Journal Article
ACTA NEUROPATHOLOGICA, ISSN 0001-6322, 05/2015, Volume 129, Issue 5, pp. 729 - 748
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2010, Volume 5, Issue 2, p. e9313
Background: Lewy body disease is a heterogeneous group of neurodegenerative disorders characterized by alpha-synuclein accumulation that includes dementia with... 
WILD-TYPE | CHAPERONE-MEDIATED AUTOPHAGY | ALZHEIMERS-DISEASE | MOUSE MODEL | FRONTOTEMPORAL DEMENTIA | BIOLOGY | AGGREGATE-PRONE PROTEINS | NEURONAL EXPRESSION | PARKINSONS-DISEASE | TRANSGENIC MICE | GAUCHER-DISEASE | Pyramidal Cells - metabolism | Humans | Nerve Degeneration - genetics | Male | Autophagy | Nerve Degeneration - metabolism | Alzheimer Disease - pathology | Brain - metabolism | Lewy Body Disease - genetics | RNA Interference | Brain - ultrastructure | Aged, 80 and over | Female | alpha-Synuclein - genetics | Pyramidal Cells - ultrastructure | Disease Models, Animal | Cell Line | Ubiquitin-Activating Enzymes - genetics | Signal Transduction | Lewy Body Disease - pathology | Mice, Transgenic | Microscopy, Electron | Ubiquitin-Activating Enzymes - metabolism | Blotting, Western | Nerve Degeneration - pathology | Lewy Body Disease - metabolism | Microscopy, Confocal | Autophagy-Related Protein 7 | Animals | Alzheimer Disease - metabolism | Brain - pathology | Cell Line, Tumor | Pyramidal Cells - pathology | Aged | Mice | Alzheimer Disease - genetics | alpha-Synuclein - metabolism | TOR protein | Neurosciences | Animal models | Parkinson's disease | Pathogenesis | Toxicity | Parkinsons disease | Phagosomes | Nervous system | Synuclein | Neuromodulation | Accumulation | Modulators | Proteins | Infusion | Alterations | Neurodegeneration | Dementia disorders | Alzheimer's disease | Movement disorders | Enzymes | Neurodegenerative diseases | Neurons | Immunomodulation | Lewy body disease | Transgenic mice | Rapamycin | Patients | Lewy bodies | Studies | Pathology | Mutation | Alzheimers disease | Phagocytosis | Apoptosis | Dementia
Journal Article
Biochemical Pharmacology, ISSN 0006-2952, 04/2014, Volume 88, Issue 4, pp. 508 - 516
Journal Article
Journal Article