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PloS one, ISSN 1932-6203, 08/2016, Volume 11, Issue 8, p. e0160312
Heat shock proteins (Hsps) are known to associate with estrogen receptors (ER) and regulate ER-mediated cell proliferation... 
MITOCHONDRIAL LOCALIZATION | GLUCOCORTICOID-RECEPTOR | HORMONE | NUCLEAR RECEPTORS | DNA | MULTIDISCIPLINARY SCIENCES | PROTEASOME | BREAST-CANCER-CELLS | BINDING | EXPRESSION | STEROID-RECEPTORS | Chromatin - metabolism | Humans | Cytoplasm - metabolism | Tacrolimus Binding Proteins - metabolism | Heat-Shock Proteins - genetics | Cell Nucleus - metabolism | MCF-7 Cells | Prostaglandin-E Synthases - genetics | HEK293 Cells | Estrogen Receptor alpha - metabolism | Proteomics - methods | Estradiol - pharmacology | Chromatin - chemistry | Gene Expression | Prostaglandin-E Synthases - metabolism | Heat-Shock Proteins - metabolism | Ubiquitin-Protein Ligases - metabolism | HSP70 Heat-Shock Proteins - genetics | HSP70 Heat-Shock Proteins - metabolism | Protein Interaction Mapping | Tacrolimus Binding Proteins - genetics | Estrogen Receptor alpha - genetics | Protein Binding | Cell Nucleus - drug effects | Ubiquitin-Protein Ligases - genetics | Cytoplasm - drug effects | Cell proliferation | Ubiquitin | Cell culture | Hsp90 protein | Chromatin | Transcription | Estrogens | Estrogen | Estrogen receptors | 17β-Estradiol | Nuclei | Estradiol | Proteins | Receptors | Historical account | Rodents | Penicillin | Trends | Prostaglandin E | Localization | Ubiquitin-protein ligase | Peptidylprolyl isomerase | Starvation | Heat shock proteins | Breast cancer | Hsp70 protein | Sex hormones | Isoforms | Phenols | Ligands | Hsc70 protein | Nuclei (cytology) | Endoplasmic reticulum | Endocrinology | Cytoplasm | Heat shock
Journal Article
Clinical cancer research, ISSN 1078-0432, 10/2017, Volume 23, Issue 20, pp. 6138 - 6151
Purpose: FGFR1 amplification occurs in approximately 15% of estrogen receptor-positive (ER... 
AMPLIFICATION | PROTEIN | ONCOLOGY | THERAPEUTIC TARGET | GENES | ENDOCRINE THERAPY | FACTOR RECEPTOR-1 | FIBROBLAST-GROWTH-FACTOR | SIGNALING MODULE | EXPRESSION | NUCLEAR TRANSLOCATION | Humans | Gene Expression Regulation, Neoplastic | Receptor, Fibroblast Growth Factor, Type 1 - metabolism | Fibroblast Growth Factors - genetics | Molecular Targeted Therapy | Receptor, Fibroblast Growth Factor, Type 1 - genetics | Breast Neoplasms - metabolism | Fibroblast Growth Factors - metabolism | Estrogen Receptor alpha - metabolism | Female | Transcription, Genetic | Disease Models, Animal | Estrogen Receptor alpha - antagonists & inhibitors | Breast Neoplasms - drug therapy | Receptor, Fibroblast Growth Factor, Type 1 - antagonists & inhibitors | Estrogen Receptor Modulators - pharmacology | Protein Transport | Drug Resistance, Neoplasm - genetics | Animals | Breast Neoplasms - genetics | Estrogen Receptor alpha - genetics | Gene Amplification | Signal Transduction - drug effects | Breast Neoplasms - pathology | Cell Line, Tumor | Mice | Protein Kinase Inhibitors - pharmacology | Neoplasm Staging | Cell proliferation | Fibroblast growth factor | Transcription | Genes | Estrogens | Estrogen | Estrogen receptors | Antagonists | Kinases | Nuclei | Fulvestrant | Gene sequencing | E2F protein | Surgery | Xenografts | Inhibition | Fibroblast growth factor receptor 1 | Protein-tyrosine kinase | Deoxyribonucleic acid--DNA | Binding | Tyrosine | Deprivation | Breast cancer | siRNA | Gene expression | Ribonucleic acid--RNA | Patients | Polymerase chain reaction | Amplification | Experimental design | Ligands | Nuclei (cytology) | Tumors | Cancer | Fibroblast growth factor receptors | breast cancer | drug resistance | fulvestrant | FGFR1
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 0027-8424, 3/1999, Volume 96, Issue 7, pp. 3999 - 4004
Estrogen receptor (ER) modulators produce distinct tissue-specific biological effects, but within the confines of the established models of ER action it is difficult to understand why... 
Bacteriophages | Receptors | Conjugated estrogens | Estrogen receptors | Ligands | Agonists | Medical sciences | Estrogen receptor modulators | Nuclear receptors | Modulated signal processing | BREAST-CANCER | COACTIVATOR | ACTIVATION | ANTIESTROGENS | PHARMACOKINETICS | PHARMACOLOGY | MULTIDISCIPLINARY SCIENCES | MECHANISMS | POSTMENOPAUSAL WOMEN | BINDING | NUCLEAR RECEPTOR | Research | Biological response modifiers | Estrogen
Journal Article
Nature (London), ISSN 1476-4687, 2015, Volume 523, Issue 7560, pp. 313 - 317
Journal Article
Molecular pharmacology, ISSN 1521-0111, 2016, Volume 90, Issue 5, pp. 674 - 688
Journal Article
Journal Article
PloS one, ISSN 1932-6203, 11/2014, Volume 9, Issue 11, p. e113175
Journal Article
PloS one, ISSN 1932-6203, 2008, Volume 3, Issue 4, pp. e2069 - e2069
Journal Article
The Journal of biological chemistry, ISSN 0021-9258, 11/2000, Volume 275, Issue 46, pp. 35986 - 35993
It has been proposed that tissue-specific estrogenic and/or antiestrogenic actions of certain xenoestrogens may be associated with alterations in the tertiary structure of estrogen receptor (ER... 
ANTIESTROGENS | MESSENGER-RNA | NUCLEAR HORMONE-RECEPTOR | BIOCHEMISTRY & MOLECULAR BIOLOGY | TRANSCRIPTIONAL ACTIVATION FUNCTIONS | LIGAND | PHYTOESTROGENS | IDENTIFICATION | BINDING | HETERODIMERS | RAT PITUITARY
Journal Article