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Cell host & microbe, ISSN 1931-3128, 2018, Volume 24, Issue 1, pp. 97 - 108.e4
Journal Article
Journal Article
Free radical biology & medicine, ISSN 0891-5849, 10/2015, Volume 87, pp. 356 - 365
Phospholipids are a major structural component of all cell membranes; their peroxidation represents a severe threat to cellular integrity and their repair is... 
Knock-in mice | Phospholipase A2 | Phospholipid hydroperoxide reductase | Diphenyl-1-pyrenylphosphine | Lipid peroxidation | Thiobarbituric acid reactive substances | Pulmonary microvascular endothelial cells | Pulmonary microvascularendothelialcells | Phospholipid hydroperoxidereductase | Knock-inmice | Thiobarbituric acidreactivesubstances | LIPID-PEROXIDATION | Dipheny1-1-pyrenylphosphine | BIOCHEMISTRY & MOLECULAR BIOLOGY | GLUTATHIONE-PEROXIDASE | MICROVASCULAR ENDOTHELIAL-CELLS | LUNG INJURY | GENE-TARGETED MICE | MECHANISMS | Phospholipase A | MESSENGER-RNA | METABOLISM | ENDOCRINOLOGY & METABOLISM | 1-CYS PEROXIREDOXIN | Endothelial Cells - metabolism | Oxidative Stress - genetics | Phospholipases A2 - metabolism | Lung - cytology | tert-Butylhydroperoxide - administration & dosage | Mice, Knockout | Peroxiredoxin VI - genetics | Peroxiredoxin VI - metabolism | Animals | Lung - drug effects | Lipid Peroxidation - drug effects | Oxidation-Reduction - drug effects | Peroxidases - metabolism | Cell Membrane - metabolism | Lung - metabolism | Mice | Lipid Peroxidation - genetics | Oxidative Stress - drug effects | Cell Membrane - drug effects | Organ Culture Techniques | Endothelial Cells - drug effects | Antioxidants | Lipids | Cell membranes | Iron compounds | Telomerase | Analysis | Oxidative stress | Cell death | Peroxidase | Phospholipases | Endothelium | diphenyl-1 | knock-in mice | phospholipase A2 | thiobarbituric acid reactive substances | pyrenylphosphine | lipid peroxidation | phospholipid hydroperoxide reductase | pulmonary microvascular endothelial cells
Journal Article
Molecular Nutrition & Food Research, ISSN 1613-4125, 04/2017, Volume 61, Issue 4, pp. 1770044 - n/a
Mol. Nutr. Food Res. 2017, 61, 1600858. DOI: 10.1002/mnfr.201600858 Dietary supplementation of LCP, the polyphenols from Lonicera caerulea L. berry, ameliorate... 
Cytokines | Lipid peroxidation | Polyphenols
Journal Article
Neuron (Cambridge, Mass.), ISSN 0896-6273, 04/2013, Volume 78, Issue 1, pp. 57 - 64
Valosin-containing protein (VCP) is a highly expressed member of the type II AAA+ ATPase family. VCP mutations are the cause of inclusion body myopathy,... 
LIPID-PEROXIDATION | SPINAL-CORD PATHOLOGY | MOUSE MODEL | ALS | AMYOTROPHIC-LATERAL-SCLEROSIS | DYSFUNCTION | BONE | NEUROSCIENCES | PAGET-DISEASE | TRANSGENIC MICE | REVEALS | RNA, Small Interfering - genetics | Humans | Middle Aged | Male | Frontotemporal Dementia - metabolism | Neurons - ultrastructure | Muscular Dystrophies, Limb-Girdle - genetics | Adenosine Triphosphate - metabolism | Membrane Potential, Mitochondrial - genetics | Muscular Dystrophies, Limb-Girdle - pathology | NAD - metabolism | Fibroblasts - metabolism | Animals, Newborn | Frontotemporal Dementia - genetics | Magnesium - metabolism | Mitochondria - pathology | Fibroblasts - pathology | Mutation - genetics | Myositis, Inclusion Body - genetics | Osteitis Deformans - pathology | Muscular Dystrophies, Limb-Girdle - metabolism | Analysis of Variance | Luminescent Proteins - genetics | Adenosine Triphosphatases - genetics | Mice | Lipid Peroxidation - genetics | RNA, Small Interfering - metabolism | Valosin Containing Protein | Osteitis Deformans - metabolism | Family Health | Cerebral Cortex - cytology | Case-Control Studies | Osteitis Deformans - genetics | Transfection | Mitochondria - genetics | Cell Cycle Proteins - genetics | Myositis, Inclusion Body - pathology | Adult | Female | Neuroblastoma - pathology | Frontotemporal Dementia - pathology | Adenosine Triphosphatases - deficiency | Mice, Inbred C57BL | Cells, Cultured | Cell Cycle Proteins - deficiency | Mitochondria - metabolism | Animals | Oxygen Consumption - genetics | Myositis, Inclusion Body - metabolism | Aged | Nervous system diseases | Neurosciences | Genes | Amyotrophic lateral sclerosis | Genetic aspects | Adenosine triphosphatase | Dementia | Proteins | Medical research | Phosphorylation | Biomedical research | Disease | Rodents | Respiration | Experiments | Patients | Report
Journal Article
Critical reviews in toxicology, ISSN 1547-6898, 2008, Volume 33, Issue 2, pp. 105 - 136
The use of many halogenated alkanes such as carbon tetrachloride (CCl4), chloroform (CHCl3) or iodoform (CHI3), has been banned or severely restricted because... 
Haloalkylation | Reactive aldehydes | Cytokines | Lipid peroxidation | Fatty degeneration | Lipid homeostasis | Recovery | Necrosis | Antioxidants | Interleukins | Growth factors | Calcium homeostasis | Apoptosis | Hepatoprotectants | reactive aldehydes | antioxidants | lipid homeostasis | apoptosis | haloalkylation | recovery | MESSENGER-RNA LEVELS | RAT-LIVER INJURY | CHEMICAL-INDUCED HEPATOTOXICITY | MITOCHONDRIAL PERMEABILITY TRANSITION | HEPATIC STELLATE CELLS | fatty degeneration | cytokines | TOXICOLOGY | growth factors | calcium homeostasis | NECROSIS-FACTOR-ALPHA | lipid peroxidation | interleukins | GROWTH-FACTOR-BETA | NITRIC-OXIDE PRODUCTION | OXYGEN PARTIAL PRESSURES | necrosis | hepatoprotectants | FREE-RADICAL PRODUCTS | Age Factors | Humans | Hepatocytes - pathology | Carbon Tetrachloride Poisoning - prevention & control | Liver - physiopathology | Halogens - adverse effects | Alkanes - metabolism | Carbon Tetrachloride Poisoning - metabolism | Liver - drug effects | Carbon Tetrachloride - metabolism | Lipid Peroxidation - drug effects | Homeostasis - drug effects | Hepatocytes - drug effects | Cytokines - metabolism | Liver - metabolism | Carbon Tetrachloride - adverse effects | Antioxidants - pharmacology | Alkanes - adverse effects | Chemical and Drug Induced Liver Injury - physiopathology | Lipid Peroxidation - physiology | Drug Synergism | Halogens - metabolism | Methylation - drug effects | Chemical and Drug Induced Liver Injury - metabolism | Homeostasis - physiology | Carbon Tetrachloride Poisoning - physiopathology | Growth Substances - metabolism
Journal Article