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Journal of Biological Chemistry, ISSN 0021-9258, 03/2011, Volume 286, Issue 11, pp. 9856 - 9864
Journal Article
Translational Stroke Research, ISSN 1868-4483, 2/2013, Volume 4, Issue 1, pp. 40 - 50
Journal Article
Cellular and Molecular Immunology, ISSN 1672-7681, 09/2011, Volume 8, Issue 5, pp. 388 - 403
Toll-like receptors (TLRs) in innate immune cells are the prime cellular sensors for microbial components. TLR activation leads to the production of... 
microRNA | innate immunity | lipopolysaccharide | LPS tolerance | Toll-like receptor | ACTIVATED PROTEIN-KINASE | COLONY-STIMULATING FACTOR | INNATE IMMUNE-SYSTEM | MONOPHOSPHORYL LIPID-A | IMMUNOLOGY | TOLL-LIKE RECEPTOR-2 | TUMOR-NECROSIS-FACTOR | SERUM-INDEPENDENT PATHWAY | RECEPTOR-ASSOCIATED KINASE | NF-KAPPA-B | MOUSE PERITONEAL-MACROPHAGES | Interleukin-1 Receptor-Associated Kinases - metabolism | Interleukin-1 Receptor-Associated Kinases - genetics | Immune System Diseases - genetics | Humans | MicroRNAs - metabolism | TNF Receptor-Associated Factor 6 - immunology | Signal Transduction - immunology | Bacteria - growth & development | Bacterial Infections - immunology | Interleukin-1 Receptor-Associated Kinases - immunology | Myeloid Differentiation Factor 88 - immunology | TNF Receptor-Associated Factor 6 - genetics | Cytokines - genetics | Bacterial Infections - microbiology | Cytokines - immunology | Cytokines - metabolism | Gene Expression Regulation - immunology | Myeloid Differentiation Factor 88 - genetics | Rats | Toll-Like Receptor 4 - genetics | Signal Transduction - genetics | Toll-Like Receptor 4 - immunology | Immunity, Innate | MicroRNAs - immunology | Toll-Like Receptor 4 - metabolism | Animals | Endotoxins - immunology | TNF Receptor-Associated Factor 6 - metabolism | Immune Tolerance - genetics | Mice | MicroRNAs - genetics | Myeloid Differentiation Factor 88 - metabolism | Endotoxins - pharmacology | Review
Journal Article
Kidney International, ISSN 0085-2538, 2014, Volume 85, Issue 2, pp. 276 - 288
Although the protective effect of lipopolysaccharide (LPS) pretreatment on renal ischemia/reperfusion injury is known, a link to hypoxia-inducible factors... 
ischemia/reperfusion | kidney | nitric oxide | HIF-2α | LPS | HIF-2 alpha | RELAXING FACTOR | ACUTE-PHASE RESPONSE | SMOOTH-MUSCLE | HIF-2-ALPHA | ENDOTHELIAL-CELLS | ENDOTOXIN TOLERANCE | UROLOGY & NEPHROLOGY | TNF-ALPHA | GENE ACTIVATION | HIF-1-ALPHA EXPRESSION | Kidney - blood supply | Human Umbilical Vein Endothelial Cells - metabolism | Humans | Human Umbilical Vein Endothelial Cells - immunology | Male | NF-kappa B - metabolism | Acute Kidney Injury - genetics | Microcirculation - drug effects | Kidney - metabolism | Transfection | Basic Helix-Loop-Helix Transcription Factors - metabolism | RNA Interference | Time Factors | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Nitric Oxide Synthase Type II - antagonists & inhibitors | Bone Marrow Transplantation | Renal Circulation - drug effects | Nitric Oxide Donors - pharmacology | Nitric Oxide Synthase Type III - metabolism | Reperfusion Injury - genetics | Reperfusion Injury - metabolism | Basic Helix-Loop-Helix Transcription Factors - deficiency | Cytoprotection | Disease Models, Animal | Basic Helix-Loop-Helix Transcription Factors - genetics | Human Umbilical Vein Endothelial Cells - drug effects | Kidney - drug effects | Signal Transduction | Endothelial Cells - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Cells, Cultured | Enzyme Inhibitors - pharmacology | Acute Kidney Injury - physiopathology | Acute Kidney Injury - prevention & control | Mice, Knockout | Nitric Oxide Synthase Type III - antagonists & inhibitors | Endothelial Cells - immunology | Animals | Reperfusion Injury - prevention & control | Warm Ischemia | Lipopolysaccharides - pharmacology | Reperfusion Injury - physiopathology | Mice | Acute Kidney Injury - metabolism | Nitric Oxide - metabolism | Endothelial Cells - drug effects | Nitric Oxide Synthase Type II - metabolism
Journal Article
Journal Article
Journal Article
Gastroenterology, ISSN 0016-5085, 2007, Volume 133, Issue 5, pp. 1627 - 1636
Journal Article
Trends in Parasitology, ISSN 1471-4922, 11/2019, Volume 35, Issue 11, pp. 918 - 930
During malaria, the hypothalamic–pituitary–adrenal (HPA) axis is activated and glucocorticoid (GC) levels are increased, but their essential roles have been... 
glucocorticoids | malaria | adrenal hormones | hypothalamic–pituitary–adrenal axis | tolerance | LPS RESISTANCE | SEPTIC SHOCK | IMMUNE REGULATION | GENE-EXPRESSION | NITRIC-OXIDE | DOUBLE-BLIND | PITUITARY-ADRENAL-FUNCTION | INFLAMMATORY CYTOKINES | RAT MODEL | PARASITOLOGY | CEREBRAL MALARIA
Journal Article