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Journal of Biological Chemistry, ISSN 0021-9258, 03/2011, Volume 286, Issue 11, pp. 9856 - 9864
Journal Article
Kidney International, ISSN 0085-2538, 2014, Volume 85, Issue 2, pp. 276 - 288
Although the protective effect of lipopolysaccharide (LPS) pretreatment on renal ischemia/reperfusion injury is known, a link to hypoxia-inducible factors... 
ischemia/reperfusion | kidney | nitric oxide | HIF-2α | LPS | ACUTE KIDNEY INJURY | ISCHEMIA/REPERFUSION INJURY | HIF-2 alpha | RELAXING FACTOR | TUMOR ANGIOGENESIS | ACUTE-PHASE RESPONSE | ENDOTHELIAL-CELLS | ENDOTOXIN TOLERANCE | UROLOGY & NEPHROLOGY | TNF-ALPHA | NF-KAPPA-B | HIF-1-ALPHA EXPRESSION | Kidney - blood supply | Human Umbilical Vein Endothelial Cells - metabolism | Humans | Human Umbilical Vein Endothelial Cells - immunology | Male | NF-kappa B - metabolism | Acute Kidney Injury - genetics | Microcirculation - drug effects | Kidney - metabolism | Transfection | Basic Helix-Loop-Helix Transcription Factors - metabolism | RNA Interference | Time Factors | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Nitric Oxide Synthase Type II - antagonists & inhibitors | Bone Marrow Transplantation | Renal Circulation - drug effects | Nitric Oxide Donors - pharmacology | Nitric Oxide Synthase Type III - metabolism | Reperfusion Injury - genetics | Reperfusion Injury - metabolism | Basic Helix-Loop-Helix Transcription Factors - deficiency | Cytoprotection | Disease Models, Animal | Basic Helix-Loop-Helix Transcription Factors - genetics | Human Umbilical Vein Endothelial Cells - drug effects | Kidney - drug effects | Signal Transduction | Endothelial Cells - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Cells, Cultured | Enzyme Inhibitors - pharmacology | Acute Kidney Injury - physiopathology | Acute Kidney Injury - prevention & control | Mice, Knockout | Nitric Oxide Synthase Type III - antagonists & inhibitors | Endothelial Cells - immunology | Animals | Reperfusion Injury - prevention & control | Warm Ischemia | Lipopolysaccharides - pharmacology | Reperfusion Injury - physiopathology | Mice | Acute Kidney Injury - metabolism | Nitric Oxide - metabolism | Endothelial Cells - drug effects | Nitric Oxide Synthase Type II - metabolism | Index Medicus
Journal Article
Journal Article
Journal of Thermal Biology, ISSN 0306-4565, 10/2019, Volume 85
The injection of repeated doses of lipopolysaccharide (LPS) results in attenuation of the immune response, which is an important mechanism to prevent... 
PGE2 | Cytokines | Endotoxin | LPS | Fever
Journal Article
Gastroenterology, ISSN 0016-5085, 2007, Volume 133, Issue 5, pp. 1627 - 1636
Background & Aims: Persistent inflammation contributes to progression of liver damage in chronic HCV (cHCV) infection. Repeated exposure to toll-like receptor... 
Gastroenterology and Hepatology | GAMMA | CYTOKINES | HEPATITIS-C VIRUS | COLONY-STIMULATING FACTOR | ENDOTOXIN TOLERANCE | KUPFFER CELLS | LIVER-DISEASE | GASTROENTEROLOGY & HEPATOLOGY | NF-KAPPA-B | CORE PROTEIN | EXPRESSION | Endotoxins - blood | Tumor Necrosis Factor-alpha - metabolism | Inflammation - pathology | Kupffer Cells - pathology | Humans | Middle Aged | Male | Monocytes - metabolism | NF-kappa B - metabolism | Case-Control Studies | Hepacivirus - metabolism | Inflammation - metabolism | Monocytes - pathology | Adult | Female | Kupffer Cells - metabolism | Viral Core Proteins - pharmacology | Macrophages - pathology | Toll-Like Receptors - immunology | Viral Core Proteins - blood | Hepatitis C, Chronic - blood | Kupffer Cells - drug effects | Monocytes - drug effects | Macrophages - metabolism | Toll-Like Receptor 4 - blood | Hepatitis C, Chronic - immunology | Lipopolysaccharides - pharmacology | Toll-Like Receptor 2 - blood | Ligands | Macrophages - drug effects | Macrophage Activation - drug effects | Endotoxins - pharmacology | Interferon-gamma - blood | Interferon-gamma - pharmacology | Biological response modifiers | Hepatitis C | Macrophages | Tumor necrosis factor | Health aspects | Index Medicus | Abridged Index Medicus | IFNγ | monocytes | TNFα | NFκB | inflammation | HCV core protein | Kupffer cells | MyD88 | LPS
Journal Article
Gut, ISSN 0017-5749, 12/2012, Volume 61, Issue 12, pp. 1701 - 1707
Background Obesity is associated with accumulation of macrophages in white adipose tissue (WAT), which contribute to the development of insulin resistance.... 
LPS MODIFICATIONS | INFLAMMATION | ESCHERICHIA-COLI | TISSUE | TOLL-LIKE RECEPTORS | RESISTANCE | ENDOTOXEMIA | MICROBIOTA | DIET-INDUCED OBESITY | GASTROENTEROLOGY & HEPATOLOGY | INNATE IMMUNITY | Glucose Intolerance - metabolism | Immunohistochemistry | Inflammation - pathology | Adipose Tissue, White - immunology | Adipose Tissue, White - metabolism | Glucose Intolerance - microbiology | Lipopolysaccharides - metabolism | Immunoblotting | Male | Glucose Intolerance - pathology | Glucose Intolerance - immunology | Inflammation - metabolism | Flow Cytometry | Escherichia coli - metabolism | Biomarkers - metabolism | Adipose Tissue, White - pathology | Inflammation - microbiology | Insulin Resistance | Inflammation - immunology | Reverse Transcriptase Polymerase Chain Reaction | Macrophages - metabolism | Animals | Intestines - microbiology | Germ-Free Life | Mice | Glucose metabolism | Research | Microbiota (Symbiotic organisms) | Glucose intolerance | Macrophages | Obesity | Insulin resistance | Glucose | Laboratories | Rodents | Metabolic disorders | Index Medicus | Abridged Index Medicus | cardiovascular disease | metabolic inflammation, inflammation | colonic microflora | lipid metabolism | gnotobiotic mice | inflammatory diseases | prebiotic | Endotoxin | gut microbiota | intestinal permeability | macrophages, obesity | E coli | intestinal epithelium | intestinal tract | 1506 | Original | energy metabolism | obesity | mucosal immunity | microbiota | toll-like receptors | Gastroenterologi | endotoxemia | escherichia-coli | tissue | diet-induced obesity | lps | resistance | innate immunity | inflammation | Gastroenterology and Hepatology | modifications
Journal Article
American Journal of Reproductive Immunology, ISSN 1046-7408, 02/2019, Volume 81, Issue 2, pp. e13080 - n/a
Problem Placental infection induces increased levels of pro-inflammatory cytokines, which have been implicated in the pathogenesis of pre-term labor. Endotoxin... 
pre‐term labor | placenta | inflammation | endotoxin tolerance | LPS | pre-term labor | CHORIOAMNIONITIS | IMMUNOLOGY | AMNIOTIC-FLUID | IL-10 | PREGNANCY | REPRODUCTIVE BIOLOGY | BACTERIAL CLEARANCE | CARBON-MONOXIDE | INFECTION | INDUCED PRETERM LABOR | EXPRESSION | Explants | Pregnancy | Phenotypes | Placenta | Cytokines | Tumor necrosis factor | Secretion | Fetuses | Inflammation | Lipopolysaccharides
Journal Article
Journal of Immunology, ISSN 0022-1767, 04/2014, Volume 192, Issue 7, pp. 3143 - 3155
The molecular basis of TNF tolerance is poorly understood. In human monocytes we detected two forms of TNF refractoriness, as follows: absolute tolerance was... 
TUMOR-NECROSIS-FACTOR | C/EBP-BETA | UBIQUITIN LIGASE | LPS TOLERANCE | ENDOTOXIN TOLERANCE | GENE-EXPRESSION | CROSS-TOLERANCE | IMMUNOLOGY | NF-KAPPA-B | LIPOPOLYSACCHARIDE TOLERANCE | FACTOR-ALPHA | Oligonucleotide Array Sequence Analysis | Transcription Factor AP-1 - genetics | Humans | NF-kappa B - immunology | Transcription Factor AP-1 - immunology | Monocytes - metabolism | NF-kappa B - metabolism | Monocytes - immunology | Transcriptome - immunology | Glycogen Synthase Kinase 3 - immunology | Transcription Factor AP-1 - metabolism | Transcription Factor RelA - genetics | Protein Phosphatase 1 - immunology | Dose-Response Relationship, Drug | Signal Transduction - immunology | I-kappa B Kinase - metabolism | Time Factors | Protein Phosphatase 1 - genetics | Tumor Necrosis Factor-alpha - immunology | Transcription Factor RelA - immunology | Phosphorylation - immunology | Phosphorylation - drug effects | Drug Tolerance - immunology | Cells, Cultured | Signal Transduction - genetics | Transcriptome - drug effects | I-kappa B Kinase - genetics | Glycogen Synthase Kinase 3 - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Monocytes - drug effects | Tumor Necrosis Factor-alpha - pharmacology | Protein Phosphatase 1 - metabolism | Glycogen Synthase Kinase 3 - genetics | NF-kappa B - genetics | Signal Transduction - drug effects | Transcription Factor RelA - metabolism | Cell Line, Tumor | I-kappa B Kinase - immunology | HeLa Cells | Index Medicus | Abridged Index Medicus
Journal Article
The Journal of Immunology, ISSN 0022-1767, 12/2000, Volume 165, Issue 12, pp. 7096 - 7101
A family of Toll-like receptor (TLR) mediates the cellular response to bacterial cell wall components; murine TLR2 and TLR4 recognize mycoplasmal lipopeptides... 
CYTOKINE PRODUCTION | LIPOPOLYSACCHARIDE | DEFECTIVE INTERLEUKIN-1 | FACTOR-KAPPA-B | PERITONEAL-MACROPHAGES | MICE | IMMUNOLOGY | LIPOPROTEINS | BACTERIAL-DNA | LPS | CUTTING EDGE | Toll-Like Receptor 2 | Dose-Response Relationship, Immunologic | Macrophage Activation - genetics | Toll-Like Receptor 4 | Membrane Glycoproteins - biosynthesis | Receptors, Cell Surface - antagonists & inhibitors | Interleukin-10 - physiology | Signal Transduction - immunology | Toll-Like Receptors | Membrane Glycoproteins - physiology | Membrane Glycoproteins - antagonists & inhibitors | Time Factors | Immune Tolerance - immunology | Cell Membrane - metabolism | Receptors, Cell Surface - biosynthesis | Receptors, Cell Surface - physiology | Lymphocyte Antigen 96 | Drosophila Proteins | Interleukin-10 - deficiency | Mice, Inbred C57BL | Antigens, Surface - biosynthesis | Macrophages, Peritoneal - immunology | Signal Transduction - genetics | Mice, Inbred ICR | Mice, Knockout | Drug Synergism | Animals | Interleukin-10 - genetics | Lipopolysaccharides - pharmacology | Immune Tolerance - genetics | Cell Membrane - immunology | Mice | Macrophages, Peritoneal - metabolism | Oligopeptides - pharmacology | Lipopeptides | Tumor Necrosis Factor-alpha - biosynthesis | Toll-like receptors | MALP2 protein | TLR4 protein | TLR2 protein | Index Medicus | Abridged Index Medicus
Journal Article