X
Search Filters
Format Format
Format Format
X
Sort by Item Count (A-Z)
Filter by Count
Journal Article (2829) 2829
Book Review (584) 584
Publication (524) 524
Book Chapter (55) 55
Conference Proceeding (23) 23
Dissertation (2) 2
Magazine Article (1) 1
more...
Subjects Subjects
Subjects Subjects
X
Sort by Item Count (A-Z)
Filter by Count
index medicus (2515) 2515
humans (1920) 1920
animals (1373) 1373
cell biology (1091) 1091
lamin type a - genetics (771) 771
proteins (752) 752
mutation (733) 733
lamins (677) 677
mice (619) 619
biochemistry & molecular biology (567) 567
female (560) 560
male (531) 531
lamin type a - metabolism (517) 517
nuclear proteins - metabolism (480) 480
cell nucleus - metabolism (411) 411
nuclear proteins - genetics (395) 395
dreifuss muscular-dystrophy (378) 378
research (369) 369
chromatin (364) 364
genetic aspects (364) 364
nuclear envelope - metabolism (364) 364
gene expression (357) 357
a-type lamins (341) 341
genetics & heredity (341) 341
cells (333) 333
expression (329) 329
phenotype (326) 326
article (321) 321
integumentary system (320) 320
adult (317) 317
analysis (313) 313
lamin (313) 313
molecular sequence data (285) 285
congenital, hereditary, and neonatal diseases and abnormalities (280) 280
nuclear lamina (280) 280
dilated cardiomyopathy (277) 277
lamin a/c (275) 275
nuclear envelope (275) 275
multidisciplinary sciences (268) 268
cell line (265) 265
disease (258) 258
intermediate filament proteins (251) 251
amino acid sequence (247) 247
membrane proteins - metabolism (242) 242
progeria (236) 236
cells, cultured (234) 234
middle aged (232) 232
genes (231) 231
laminopathies (224) 224
nuclear-envelope (222) 222
membrane proteins - genetics (221) 221
progeria - genetics (221) 221
aging (220) 220
envelope (219) 219
hela cells (219) 219
physiological aspects (217) 217
lamin type b - metabolism (216) 216
lamin a (213) 213
lamin type a (213) 213
embryonic structures (209) 209
genetics (207) 207
gene (206) 206
organization (205) 205
biology (203) 203
hutchinson-gilford-progeria (202) 202
mutations (201) 201
cancer (197) 197
lamin type b - genetics (193) 193
phosphorylation (193) 193
cardiomyopathy (187) 187
lamin a/c gene (183) 183
base sequence (178) 178
research article (177) 177
life sciences (175) 175
cellular biology (174) 174
familial partial lipodystrophy (172) 172
lamins - metabolism (171) 171
muscular dystrophy (170) 170
protein binding (170) 170
gene-expression (166) 166
fibroblasts - metabolism (164) 164
lmna (164) 164
fibroblasts (163) 163
mitosis (160) 160
chromatin - metabolism (159) 159
apoptosis (158) 158
differentiation (157) 157
gene expression regulation (156) 156
in-vitro (156) 156
cell cycle (154) 154
oncology (151) 151
lamins - genetics (146) 146
lamin type b (143) 143
pedigree (143) 143
child (142) 142
dna (142) 142
lamin-a (142) 142
signal transduction (139) 139
stem cells (139) 139
biophysics (138) 138
more...
Language Language
Language Language
X
Sort by Item Count (A-Z)
Filter by Count
English (2818) 2818
French (7) 7
German (7) 7
Russian (5) 5
Japanese (3) 3
Czech (1) 1
Italian (1) 1
Polish (1) 1
Portuguese (1) 1
Spanish (1) 1
Swedish (1) 1
more...
Publication Date Publication Date
Click on a bar to filter by decade
Slide to change publication date range


Journal Article
Science, ISSN 0036-8075, 12/2011, Volume 334, Issue 6063, pp. 1706 - 1710
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 3/2009, Volume 106, Issue 9, pp. 3282 - 3287
Journal Article
Molecular Human Reproduction, ISSN 1360-9947, 08/2015, Volume 22, Issue 2, pp. 143 - 157
STUDY HYPOTHESIS: In women with preterm premature rupture of the membranes (PPROM), increased oxidative stress may accelerate premature cellular senescence,... 
Aging | Biomarkers | Oxidative stress | Prematurity | MAPK signaling | ACTIVATION | prematurity | SECRETORY PHENOTYPE | biomarkers | 8-OXOGUANINE DNA GLYCOSYLASE-1 | DEVELOPMENTAL BIOLOGY | OBSTETRICS & GYNECOLOGY | WOMEN | REPAIR | REPRODUCTIVE BIOLOGY | SENESCENCE | aging | FETAL MEMBRANES | NITROTYROSINE | INFLAMMATORY RESPONSE | oxidative stress | PRELABOR RUPTURE | Superoxide Dismutase - genetics | Lamin Type B - genetics | Homeodomain Proteins - metabolism | Humans | Gene Expression Profiling | Fetal Membranes, Premature Rupture - genetics | Fetal Membranes, Premature Rupture - pathology | MAP Kinase Kinase 4 - metabolism | Cellular Senescence | Mitogen-Activated Protein Kinase 1 - genetics | Adult | Female | p38 Mitogen-Activated Protein Kinases - metabolism | Infant, Newborn | Superoxide Dismutase - metabolism | Premature Birth | Mitogen-Activated Protein Kinase 3 - genetics | Fetal Membranes, Premature Rupture - metabolism | Gene Expression Regulation | Oxidative Stress - genetics | p38 Mitogen-Activated Protein Kinases - genetics | Signal Transduction - genetics | Homeodomain Proteins - genetics | Lamin Type B - metabolism | Extraembryonic Membranes - injuries | Pregnancy | Mitogen-Activated Protein Kinase 3 - metabolism | Infant, Premature | Extraembryonic Membranes - metabolism | MAP Kinase Kinase 4 - genetics | DNA Damage | Superoxide Dismutase-1 | Mitogen-Activated Protein Kinase 1 - metabolism | Index Medicus
Journal Article
Nature, ISSN 0028-0836, 11/2015, Volume 527, Issue 7576, pp. 105 - 109
Journal Article
Proceedings of the National Academy of Sciences, ISSN 0027-8424, 04/2015, Volume 112, Issue 15, pp. 4672 - 4677
Embryogenesis requires the precise activation and repression of many transcriptional regulators. The Polycomb group proteins and the associated H3K27me3... 
Polycomb | Long-range chromatin contacts | Hox genes | Nuclear organization | CONTACTS | MULTIDISCIPLINARY SCIENCES | long-range chromatin contacts | GENOME | ORGANIZATION | CHROMOSOME | LAMINA INTERACTIONS | EMBRYONIC STEM-CELLS | ARCHITECTURE | REORGANIZATION | nuclear organization | TRANSITIONS | CHROMATIN INTERACTIONS | Embryonic Stem Cells - metabolism | Multigene Family | Embryonic Stem Cells - cytology | Lamin Type B - genetics | Oligonucleotide Array Sequence Analysis | Chromosomes, Mammalian - genetics | Homeodomain Proteins - metabolism | Male | Gene Expression Profiling | Polycomb-Group Proteins - metabolism | Embryo, Mammalian - metabolism | Gene Expression Regulation, Developmental | Female | Lysine - metabolism | Nuclear Proteins - genetics | Cells, Cultured | Nuclear Proteins - metabolism | Homeodomain Proteins - genetics | Lamin Type B - metabolism | Lysine - genetics | Animals | Histones - genetics | Embryo, Mammalian - embryology | Embryo, Mammalian - cytology | Polycomb-Group Proteins - genetics | Protein Binding | Mice | Models, Genetic | Histones - metabolism | Chromosomes, Mammalian - metabolism | Methylation | Transcriptional coactivators | Influence | Research | Gene expression | Analysis | Proteins | Embryology | Chromatin | Gene loci | Chromosomes | Cells | Index Medicus | Life Sciences | Biological Sciences
Journal Article
Journal Article
Journal Article
The FEBS Journal, ISSN 1742-464X, 06/2018, Volume 285, Issue 12, pp. 2243 - 2262
Lamins are key nuclear proteins which are important for maintaining nuclear structure and function. Mutations in lamins cause a spectrum of genetic diseases... 
nuclear lamina | nuclear organization | ubiquitination | protein degradation | laminopathy | NUCLEAR LAMINS | A-TYPE LAMINS | PROTEASOMAL DEGRADATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | CELL-CYCLE PROGRESSION | GENOMIC INSTABILITY | LAMINOPATHIES | RETINOBLASTOMA PROTEIN | DISEASE