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Neuron (Cambridge, Mass.), ISSN 0896-6273, 2003, Volume 40, Issue 3, pp. 501 - 514
Targeted deletion of focal adhesion kinase ( fak) in the developing dorsal forebrain resulted in local disruptions of the cortical basement membrane located... 
NEURONAL MIGRATION | MATRIX METALLOPROTEINASES | TYROSINE PHOSPHORYLATION | ALPHA-6 INTEGRINS | ECTOPIC EXPRESSION | CENTRAL-NERVOUS-SYSTEM | BETA-DYSTROGLYCAN | FOCAL ADHESION KINASE | NEUROSCIENCES | MENINGEAL CELLS | BASEMENT-MEMBRANE | Immunohistochemistry | Membrane Glycoproteins - metabolism | Embryo, Mammalian | Protein-Tyrosine Kinases - metabolism | Homeodomain Proteins - metabolism | Astrocytes - pathology | Cerebral Cortex - pathology | Glial Fibrillary Acidic Protein - metabolism | Muscular Dystrophies - genetics | Protein-Tyrosine Kinases - deficiency | Focal Adhesion Kinase 1 | Calbindins | Neurons - metabolism | Serine Endopeptidases | Disease Models, Animal | Fibroblasts - metabolism | Muscular Dystrophies - metabolism | Muscular Dystrophies - congenital | Silver Staining | Nerve Tissue Proteins | Basement Membrane - metabolism | Blotting, Western | Precipitin Tests | Mice, Knockout | Infection | Staining and Labeling | Mice | Mutation | Dura Mater | Astrocytes - metabolism | Neurons - pathology | Microtubule-Associated Proteins - metabolism | Otx Transcription Factors | Phosphopyruvate Hydratase - metabolism | Basement Membrane - pathology | Dystroglycans | Cerebral Cortex - metabolism | Cerebral Cortex - ultrastructure | Phosphotyrosine - metabolism | DNA-Binding Proteins - metabolism | Protein-Tyrosine Kinases - genetics | Lamins - metabolism | src-Family Kinases - metabolism | Cytoskeletal Proteins - metabolism | Cell Adhesion Molecules, Neuronal - metabolism | Cerebral Cortex - abnormalities | Extracellular Matrix Proteins - metabolism | Fibroblasts - virology | Cells, Cultured | Focal Adhesion Protein-Tyrosine Kinases | Microscopy, Electron | Muscular Dystrophies - pathology | S100 Calcium Binding Protein G - metabolism | Transcription Factors - metabolism | Animals | Carrier Proteins - metabolism | Calbindin 2 | Bacterial Proteins - metabolism | Heterozygote | Proteins | Neurons | Rodents | Protein expression | Kinases | Embryos | Muscular dystrophy | Deoxyribonucleic acid--DNA | Cell adhesion & migration
Journal Article
Journal Article
The Journal of cell biology, ISSN 0021-9525, 03/2013, Volume 200, Issue 5, pp. 605 - 617
Journal Article
Journal Article
Journal Article
The Journal of clinical investigation, ISSN 0021-9738, 01/2013, Volume 123, Issue 1, pp. 19 - 26
Genetic mutations account for a significant percentage of cardiomyopathies, which are a leading cause of congestive heart failure. In hypertrophic... 
MEDICINE, RESEARCH & EXPERIMENTAL | SARCOMERE PROTEIN GENES | COMPLEX | HYPERTROPHIC CARDIOMYOPATHY | POLYMORPHISMS | TITIN | DREIFUSS MUSCULAR-DYSTROPHY | LAMIN-A/C DEFICIENCY | RISK | CONGESTIVE-HEART-FAILURE | MECHANOTRANSDUCTION | Cardiomyopathy, Dilated - pathology | Humans | Heart Failure - physiopathology | Myosin Heavy Chains - genetics | Myosin Heavy Chains - metabolism | Arrhythmias, Cardiac - physiopathology | Sarcomeres - pathology | Arrhythmias, Cardiac - pathology | Mitochondria - genetics | Sarcomeres - genetics | Myocardium - metabolism | Heart Failure - etiology | Arrhythmias, Cardiac - genetics | Cardiac Myosins - metabolism | Arrhythmias, Cardiac - metabolism | Calcium-Binding Proteins - metabolism | Cardiomyopathy, Dilated - genetics | Cytoskeleton - pathology | Cardiomyopathy, Dilated - complications | Sarcomeres - metabolism | Cardiac Myosins - genetics | Cytoskeleton - genetics | Heart Failure - genetics | Myocardium - pathology | Arrhythmias, Cardiac - etiology | Heart Failure - metabolism | Mitochondria - metabolism | Heart Failure - pathology | Mitochondria - pathology | Cardiomyopathy, Dilated - metabolism | Carrier Proteins - genetics | Animals | Carrier Proteins - metabolism | Cardiomyopathy, Dilated - physiopathology | Cytoskeleton - metabolism | Mutation | Calcium-Binding Proteins - genetics | Gene mutations | Cardiomyopathy | Genetic aspects | Congestive heart failure | Diagnosis | Heart diseases | Genetic screening | Methods | Review Series
Journal Article
Nature (London), ISSN 1476-4687, 2015, Volume 527, Issue 7576, pp. 105 - 109
Journal Article
Autophagy, ISSN 1554-8635, 2014, Volume 5, Issue 6, pp. 795 - 804
Journal Article
Aging (Albany, NY.), ISSN 1945-4589, 2017, Volume 9, Issue 5, pp. 1453 - 1469
Nuclear integrity and mechanical stability of the nuclear envelope (NE) are conferred by the nuclear lamina, a meshwork of intermediate filaments composed of... 
Nucleoskeleton | Mitochondria | Emerin | Induced pluripotent stem cells | MKL1 | SIRT7 | Actin cytoskeleton | Lamins | Nesprins | lamins | nesprins | mitochondria | PROGERIA | STRUCTURAL ORGANIZATION | CELL BIOLOGY | GERIATRICS & GERONTOLOGY | actin cytoskeleton | NUCLEAR-ENVELOPE | NESPRIN-2 | emerin | LAMIN-A | DISEASE | nucleoskeleton | induced pluripotent stem cells | SENESCENCE | ACCUMULATION | EXPRESSION | Age Factors | Lamin Type B - genetics | Humans | NF-kappa B - metabolism | Aging, Premature - genetics | Aging, Premature - pathology | Aging - genetics | Time Factors | Cellular Senescence | Membrane Proteins - metabolism | Microfilament Proteins - metabolism | Nuclear Proteins - genetics | Sirtuins - genetics | Aging, Premature - metabolism | Microfilament Proteins - genetics | Induced Pluripotent Stem Cells - metabolism | Induced Pluripotent Stem Cells - pathology | Cell Line | Actin Cytoskeleton - metabolism | Signal Transduction | Membrane Proteins - genetics | Nuclear Proteins - metabolism | Lamin Type A - metabolism | Nerve Tissue Proteins - genetics | Lamin Type B - metabolism | Aging - pathology | Nerve Tissue Proteins - metabolism | Nuclear Envelope - metabolism | Phenotype | Lamin Type A - genetics | Trans-Activators - metabolism | Nuclear Envelope - pathology | Sirtuins - metabolism | Aging - metabolism
Journal Article
Journal Article