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Journal Article
PLoS ONE, ISSN 1932-6203, 2010, Volume 5, Issue 8, p. e12304
Gliomas are morbid brain tumors that are extremely resistant to available chemotherapy and radiology treatments. Some studies have suggested that... 
BREAST-CANCER CELLS | BRAIN-TUMOR | INHIBITION | MULTIDISCIPLINARY SCIENCES | CA2+-ACTIVATED K+ CHANNELS | GROWTH | CLOTRIMAZOLE | INTERMEDIATE-CONDUCTANCE | EPIDEMIOLOGY | MODULATION | CYCLE | Potassium - metabolism | RNA, Small Interfering - genetics | Large-Conductance Calcium-Activated Potassium Channels - metabolism | Small-Conductance Calcium-Activated Potassium Channels - genetics | Intermediate-Conductance Calcium-Activated Potassium Channels - genetics | Humans | Large-Conductance Calcium-Activated Potassium Channels - deficiency | Molecular Sequence Data | Electric Conductivity | Large-Conductance Calcium-Activated Potassium Channels - antagonists & inhibitors | Intermediate-Conductance Calcium-Activated Potassium Channels - metabolism | RNA, Messenger - metabolism | Gene Knockdown Techniques | Glioblastoma - genetics | Intermediate-Conductance Calcium-Activated Potassium Channels - antagonists & inhibitors | Large-Conductance Calcium-Activated Potassium Channels - genetics | Gene Expression Regulation, Neoplastic - drug effects | Potassium Channel Blockers - pharmacology | Amino Acid Sequence | RNA, Messenger - genetics | Down-Regulation - genetics | Glioblastoma - surgery | Glioblastoma - pathology | Intermediate-Conductance Calcium-Activated Potassium Channels - deficiency | Cell Line, Tumor | Cell Proliferation - drug effects | Chemotherapy | Gliomas | Genes | Brain tumors | Genetic engineering | Potassium channels | Cancer | Cell proliferation | Brain | Biotechnology | Neurosciences | Calcium | Brain cancer | Glioblastoma | Smooth muscle | Potassium conductance | Kinases | Cancer therapies | Channels | Potassium channels (calcium-gated) | Proteins | Cell growth | Surgery | Glioma cells | Cell cycle | Physiology | Calcium channels | Channel gating | Tumor cells | Cultures | siRNA | Pharmacology | Glioblastoma multiforme | Resistance | Studies | Polymerase chain reaction | Gene silencing | Inhibitors | Cell lines | Radiology | Conductance | Glioblastoma cells | Potassium | Prostate cancer | Clotrimazole | Tumors
Journal Article
Journal Article
Journal of Cellular Physiology, ISSN 0021-9541, 09/2017, Volume 232, Issue 9, pp. 2478 - 2488
Glioblastomas (GBMs) are brain tumors characterized by diffuse invasion of cancer cells into the healthy brain parenchyma, and establishment of secondary foci.... 
INVASION | PHYSIOLOGY | BK CHANNELS | MITOCHONDRIAL | GLIOMA | MOUSE | CA2+-ACTIVATED K+ CHANNEL | BETA-SUBUNIT | EXPRESSION | MODULATION | CD133 | CELL BIOLOGY | Up-Regulation | Large-Conductance Calcium-Activated Potassium Channels - metabolism | Neoplastic Stem Cells - drug effects | Humans | Brain Neoplasms - pathology | Gene Expression Regulation, Neoplastic | Large-Conductance Calcium-Activated Potassium Channels - antagonists & inhibitors | Brain Neoplasms - metabolism | Glioblastoma - genetics | Neoplastic Stem Cells - metabolism | Time Factors | Biomarkers, Tumor - metabolism | Large-Conductance Calcium-Activated Potassium Channels - genetics | Neoplastic Stem Cells - pathology | Glioblastoma - metabolism | Tumor Cells, Cultured | Spheroids, Cellular | Potassium Channel Blockers - pharmacology | Signal Transduction | Neoplasm Invasiveness | Brain Neoplasms - genetics | Large-Conductance Calcium-Activated Potassium Channel alpha Subunits - metabolism | Cell Movement - drug effects | Phenotype | Membrane Potentials | AC133 Antigen - metabolism | Glioblastoma - pathology | Cell Line, Tumor | Primary Cell Culture | Potassium channels | Analysis | Brain tumors | Stem cells | Growth conditions | Brain | Cell culture | Calcium channels | Calcium | Leukocyte migration | Invasiveness | Brain cancer | Glioblastoma | Parenchyma | Potassium conductance | Channels | Cell adhesion & migration | Potassium channels (calcium-gated) | Resistance | Calcium conductance | Conductance | Potassium | Cell migration | Cancer | Tumors
Journal Article
Journal Article
Neuropharmacology, ISSN 0028-3908, 05/2015, Volume 92, pp. 8 - 15
We previously showed that activity of the large conductance calcium-activated potassium (Big-K; BK) channels is suppressed in 3xTg Alzheimer disease (AD) model... 
Learning and memory | Excitability | Alzheimer's disease | Large conductance calcium-activated potassium channels | Synaptic plasticity | Excitability Synaptic plasticity | PLAQUES | CEREBROSPINAL-FLUID | Large conductance calcium-activated | NEUROSCIENCES | IMMUNIZATION | TRANSCRANIAL MAGNETIC STIMULATION | potassium channels | A-BETA | MOTOR CORTEX | PHARMACOLOGY & PHARMACY | INTRACELLULAR AMYLOID-BETA | EPILEPSY | PYRAMIDAL CELL EXCITABILITY | BRAIN | Large-Conductance Calcium-Activated Potassium Channels - metabolism | Alzheimer Disease - complications | Humans | Cognition Disorders - metabolism | CA1 Region, Hippocampal - drug effects | Drug Delivery Systems | Long-Term Potentiation - drug effects | Cognition Disorders - etiology | Amyloid beta-Peptides - metabolism | Carboxylic Acids - therapeutic use | Neurotoxins - administration & dosage | Disease Models, Animal | Charybdotoxin - administration & dosage | Peptide Fragments - metabolism | Phenanthrenes - therapeutic use | CA1 Region, Hippocampal - physiology | Gene Expression Regulation - genetics | Mice, Inbred C57BL | Mice, Transgenic | Long-Term Potentiation - genetics | Cognition Disorders - drug therapy | Gene Expression Regulation - drug effects | Patch-Clamp Techniques | Animals | Recognition (Psychology) - drug effects | Mice | Alzheimer Disease - genetics | In Vitro Techniques | Animal experimentation | Potassium channels | Analysis | Enzyme-linked immunosorbent assay
Journal Article