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index medicus (83) 83
lcn2 (77) 77
lipocalin-2 (67) 67
humans (65) 65
gelatinase-associated lipocalin (51) 51
ngal (48) 48
animals (45) 45
male (40) 40
female (33) 33
lipocalins - metabolism (30) 30
expression (29) 29
lipocalins - genetics (28) 28
acute-phase proteins - metabolism (27) 27
acute-phase proteins - genetics (26) 26
mice (26) 26
cancer (23) 23
cell biology (22) 22
oxidative stress (19) 19
proto-oncogene proteins - metabolism (18) 18
up-regulation (18) 18
analysis (17) 17
inflammation (17) 17
iron (17) 17
middle aged (17) 17
proto-oncogene proteins - genetics (17) 17
aged (16) 16
lipocalin 2 (16) 16
proteins (16) 16
apoptosis (15) 15
gene expression (15) 15
rats (15) 15
biochemistry & molecular biology (14) 14
lipocalin (14) 14
urology & nephrology (14) 14
lcn2 protein, human (13) 13
lipocalins - blood (13) 13
original (13) 13
protein (13) 13
article (12) 12
identification (12) 12
lcn2 protein (12) 12
liver (12) 12
mice, inbred c57bl (12) 12
oncogene proteins - genetics (12) 12
reverse transcriptase polymerase chain reaction (12) 12
biomarker (11) 11
human (11) 11
lipocalins (11) 11
proto-oncogene proteins - blood (11) 11
acute kidney injury (10) 10
acute-phase proteins (10) 10
adult (10) 10
gene (10) 10
gene-expression (10) 10
injury (10) 10
neurosciences (10) 10
oncogene proteins - metabolism (10) 10
oncology (10) 10
research (10) 10
abridged index medicus (9) 9
cell line, tumor (9) 9
cells (9) 9
cells, cultured (9) 9
endocrinology & metabolism (9) 9
kidney (9) 9
medicine, research & experimental (9) 9
original article (9) 9
polymerase chain reaction (9) 9
prognosis (9) 9
biomarkers (8) 8
breast-cancer (8) 8
disease models, animal (8) 8
immunology (8) 8
insulin-resistance (8) 8
mouse (8) 8
neoplasm invasiveness (8) 8
neutrophil gelatinase (8) 8
physiological aspects (8) 8
predictive value of tests (8) 8
urology (8) 8
biomarkers - blood (7) 7
blotting, western (7) 7
early urinary biomarker (7) 7
epithelial-cells (7) 7
homeostasis (7) 7
infection (7) 7
ischemia (7) 7
lipocalin-2 - metabolism (7) 7
lipocalins - urine (7) 7
mice, knockout (7) 7
ngal/lcn2 (7) 7
rats, sprague-dawley (7) 7
receptor (7) 7
rna, messenger - genetics (7) 7
time factors (7) 7
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1. Full Text Lipocalin‐2 ameliorates granulocyte functionality
by Schroll, Andrea and Eller, Kathrin and Feistritzer, Clemens and Nairz, Manfred and Sonnweber, Thomas and Moser, Patrizia A and Rosenkranz, Alexander R and Theurl, Igor and Weiss, Guenter
European Journal of Immunology, ISSN 0014-2980, 12/2012, Volume 42, Issue 12, pp. 3346 - 3357
Attraction of neutrophils to sites of infection or tissue injury is an essential prerequisite for an efficient innate immune response. Herein, we provide novel... 
Chemotaxis | Lipocalin‐2 (Lcn2) | polymorphonuclear neutrophils (PMNs) | Lipocalin-2 (Lcn2) | Polymorphonuclear neutrophils (PMNs) | NGAL | HUMAN NEUTROPHIL GELATINASE | PROTEIN | RECEPTOR | IRON | GELATINASE-ASSOCIATED LIPOCALIN | IMMUNOLOGY | ISCHEMIA-REPERFUSION INJURY | ADHESION | INFECTION | EXPRESSION | Oncogene Proteins - genetics | Humans | Salmonella Infections - genetics | Salmonella Infections - immunology | Male | Acute-Phase Proteins - genetics | Paracrine Communication - immunology | Lipocalins - genetics | Proto-Oncogene Proteins - immunology | Chemotaxis - immunology | Oncogene Proteins - immunology | Proto-Oncogene Proteins - pharmacology | Lipocalins - pharmacology | Salmonella typhimurium - immunology | Neutrophils - immunology | Oncogene Proteins - pharmacology | Paracrine Communication - genetics | Proto-Oncogene Proteins - genetics | Inflammation - immunology | Chemotaxis - drug effects | Chemotaxis - genetics | Mice, Knockout | Animals | Lipocalin-2 | Lipocalins - immunology | Paracrine Communication - drug effects | Inflammation - genetics | Mice | Acute-Phase Proteins - pharmacology | Acute-Phase Proteins - immunology | Medical research | Rodents
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2. Full Text Lipocalin 2: A New Mechanoresponding Gene Regulating Bone Homeostasis
by Rucci, Nadia and Capulli, Mattia and Piperni, Sara Gemini and Cappariello, Alfredo and Lau, Patrick and Frings‐Meuthen, Petra and Heer, Martina and Teti, Anna
Journal of Bone and Mineral Research, ISSN 0884-0431, 02/2015, Volume 30, Issue 2, pp. 357 - 368
ABSTRACT Mechanical loading represents a crucial factor in the regulation of skeletal homeostasis. Its reduction causes loss of bone mass, eventually leading... 
LCN2 | UNLOADING | OSTEOCLASTS | ADIPOKINE | MECHANICAL FORCES | OSTEOBLASTS | Unloading | Osteoclasts | Mechanical forces | Adipokine | Osteoblasts | NGAL | PROTEIN | MOUSE | MUSCULAR-DYSTROPHY | IRON | GELATINASE-ASSOCIATED LIPOCALIN | ISCHEMIA-REPERFUSION INJURY | MODELED MICROGRAVITY | INSULIN-RESISTANCE | ENDOCRINOLOGY & METABOLISM | NEUTROPHIL-GELATINASE | Oncogene Proteins - genetics | Humans | Male | Acute-Phase Proteins - genetics | Paralysis - pathology | Lipocalins - blood | Lipocalins - genetics | HEK293 Cells | Adult | Cell Differentiation | Mechanotransduction, Cellular - genetics | Disease Models, Animal | Bed Rest | Osteoclasts - pathology | Mice, Inbred C57BL | Gene Expression Regulation | Proto-Oncogene Proteins - genetics | Hindlimb Suspension | Osteoclasts - metabolism | Osteoblasts - pathology | Bone and Bones - physiopathology | Phenotype | Animals | Lipocalin-2 | Paralysis - physiopathology | Osteoblasts - metabolism | Proto-Oncogene Proteins - blood | Homeostasis - genetics | Osteogenesis | Osteoporosis | Exercise | RNA | Analysis | Genes | Homeostasis | Physiological aspects
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3. Full Text Increased plasma levels of lipocalin 2 in mild cognitive impairment
by Choi, Jihye and Lee, Ho-Won and Suk, Kyoungho
Journal of the Neurological Sciences, ISSN 0022-510X, 2011, Volume 305, Issue 1, pp. 28 - 33
Abstract Background Alzheimer's disease (AD) is a progressive neurodegenerative disorder characterized by an irreversible cognitive decline and neuronal loss... 
Neurology | Alzheimer's disease (AD) | Biomarker | Lipocalin 2 (LCN2) | Inflammation | Mild cognitive impairment (MCI) | ELISA | PROTEIN | NEUTROPHIL GELATINASE | CSF BIOMARKERS | GELATINASE-ASSOCIATED LIPOCALIN | PROINFLAMMATORY CYTOKINES | OVARIAN-CANCER | CEREBROSPINAL-FLUID | NEUROSCIENCES | CLINICAL NEUROLOGY | INFLAMMATORY MARKERS | PRECLINICAL ALZHEIMERS-DISEASE | EXPRESSION | Lipocalins - biosynthesis | Predictive Value of Tests | Encephalitis - diagnosis | Cognition Disorders - blood | Humans | Middle Aged | Male | Acute-Phase Proteins - genetics | Proto-Oncogene Proteins - biosynthesis | Alzheimer Disease - pathology | Lipocalins - blood | Lipocalins - genetics | Female | Encephalitis - genetics | Cognition Disorders - pathology | Proto-Oncogene Proteins - genetics | Cognition Disorders - genetics | Disease Progression | Lipocalin-2 | Aged | Alzheimer Disease - genetics | Proto-Oncogene Proteins - blood | Acute-Phase Proteins - biosynthesis | Encephalitis - blood | Alzheimer Disease - blood | Alzheimer's disease | Nervous system diseases | Amyloid beta-protein
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4. Full Text Perilipin 5 and Lipocalin 2 Expression in Hepatocellular Carcinoma
by Asimakopoulou, A and Vucur, M and Luedde, T and Schneiders, S and Kalampoka, S and Weiss, TS and Weiskirchen, R
CANCERS, ISSN 2072-6694, 03/2019, Volume 11, Issue 3, p. 385
Hepatocellular carcinoma (HCC) is one of the most prevalent and deadly cancers worldwide. Therefore, current global research focuses on molecular tools for... 
NGAL | PROTEIN | TUMOR-MARKERS | liver | HCC | HEPATIC PROGENITOR CELLS | PROLIFERATION | AFP | OVEREXPRESSION | LIVER-INJURY | ONCOLOGY | ROLES | LCN2 | cancer | PLIN5 | ALPHA-FETOPROTEIN
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5. Full Text Light-Induced Lipocalin 2 Facilitates Cellular Apoptosis by Positively Regulating Reactive Oxygen Species/Bim Signaling in Retinal Degeneration
by Tang, Wenyi and Ma, Jun and Gu, Ruiping and Lei, Boya and Ding, Xinyi and Xu, Gezhi and Xu, Gezhi
Investigative ophthalmology & visual science, ISSN 0146-0404, 12/2018, Volume 59, Issue 15, pp. 6014 - 6025
PURPOSE. Lipocalin 2 ( LCN2) is reported to be one of the key regulators of cell survival and death; however, its effect on retinal degeneration is unclear.... 
LCN2 | Retinal degeneration | Oxidative stress | Photoreceptor | Apoptosis | PROTEIN FAMILY | retinal degeneration | ACTIVATION | photoreceptor | INJURY | apoptosis | MACULAR DEGENERATION | RECEPTOR | 24P3 PROTEIN | DEATH | PREVALENCE | OPHTHALMOLOGY | BCL-2 FAMILY | EXPRESSION | oxidative stress
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6. Full Text Over‐expression of lipocalin 2 promotes cell migration and invasion through activating ERK signaling to increase SLUG expression in prostate cancer
by Ding, Guanxiong and Fang, Jie and Tong, Shijun and Qu, Lianxi and Jiang, Haowen and Ding, Qiang and Liu, Jun
The Prostate, ISSN 0270-4137, 06/2015, Volume 75, Issue 9, pp. 957 - 968
BACKGROUND Metastasis is the primary cause of prostate cancer (PCa) lethality and poses a huge clinical obstacle. Lipocalin 2 (LCN2), a member of the lipocalin... 
LCN2 | prostate cancer | SLUG | EMT | ERK | CARCINOMA-CELLS | NEUTROPHIL GELATINASE | GELATINASE-ASSOCIATED LIPOCALIN | EPITHELIAL-MESENCHYMAL TRANSITIONS | E-CADHERIN | BREAST-CANCER | TUMOR-METASTASIS | ENDOCRINOLOGY & METABOLISM | UROLOGY & NEPHROLOGY | UP-REGULATION | PROGRESSION | SNAIL | Lipocalins - biosynthesis | Nitriles - pharmacology | Humans | Extracellular Signal-Regulated MAP Kinases - antagonists & inhibitors | Male | Acute-Phase Proteins - genetics | Extracellular Signal-Regulated MAP Kinases - metabolism | Proto-Oncogene Proteins - biosynthesis | Cell Movement - physiology | MAP Kinase Signaling System | RNA - genetics | Heterografts | Prostatic Neoplasms - genetics | Lipocalins - blood | Lipocalins - genetics | Epithelial-Mesenchymal Transition | Prostatic Neoplasms - blood | Snail Family Transcription Factors | Prostatic Neoplasms - pathology | Butadienes - pharmacology | Neoplasm Invasiveness | Enzyme Inhibitors - pharmacology | Proto-Oncogene Proteins - genetics | Transcription Factors - biosynthesis | Transcription Factors - genetics | RNA - chemistry | Reverse Transcriptase Polymerase Chain Reaction | Animals | Lipocalin-2 | Mice, Nude | Histocytochemistry | Cell Line, Tumor | Mice | Proto-Oncogene Proteins - blood | Acute-Phase Proteins - biosynthesis | Development and progression | Prostate cancer
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7. Full Text A Complex Role for Lipocalin 2 in Bone Metabolism: Global Ablation in Mice Induces Osteopenia Caused by an Altered Energy Metabolism
by Capulli, Mattia and Ponzetti, Marco and Maurizi, Antonio and Gemini‐Piperni, Sara and Berger, Thorsten and Mak, Tak Wah and Teti, Anna and Rucci, Nadia
Journal of Bone and Mineral Research, ISSN 0884-0431, 06/2018, Volume 33, Issue 6, pp. 1141 - 1153
ABSTRACT Lipocalin 2 (Lcn2) is an adipokine that carries out a variety of functions in diverse organs. We investigated the bone phenotype and the energy... 
NGAL | LCN2 | OSTEOBLAST | GLUT1 | ENERGY METABOLISM | BONE METABOLISM | HOMEOSTASIS | HYPERINSULINEMIA | EXPENDITURE | OBESITY | INSULIN-RESISTANCE | ENDOCRINOLOGY & METABOLISM | WEIGHT | Glucose metabolism | Phosphatases | Body weight | Physiological aspects | Glucose | Insulin | Dextrose | Hyperinsulinemia | Alkaline phosphatase | Energy metabolism | Bone (trabecular) | Osteoblasts | Bone growth | Mineralization | Rodents | Bone marrow | Protein transport | Osteopenia | Hyperphagia | Glucose transporter | Phenotypes | Lipocalin | Bone turnover | Metabolism | Glucose tolerance | Calvaria | Stromal cells | Polyuria | Renal cortex | Osteogenesis | Proteinuria
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8. Full Text Lipocalin 2 Upregulation Protects Hepatocytes from IL1-β-Induced Stress
by Hu, Ying and Xue, Jihua and Yang, Ying and Zhou, Xiaotang and Qin, Chaochao and Zheng, Min and Zhu, Haihong and Liu, Yanning and Liu, Weixia and Lou, Guohua and Wang, Jing and Wu, Shanshan and Chen, Zhi and Chen, Feng
Cellular Physiology and Biochemistry, ISSN 1015-8987, 05/2015, Volume 36, Issue 2, pp. 753 - 762
Background: Lipocalin 2 (LCN2), a protein primarily produced by hepatocytes, is highly upregulated under various conditions that induce cellular stress, such... 
Original Paper | LCN2 | Hepatocytes | TNF-α | IL-1β | IL-6 | ACTIVATION | PHYSIOLOGY | TNF-alpha | MACROPHAGES | NEUTROPHIL GELATINASE | IL-1 beta | GELATINASE-ASSOCIATED LIPOCALIN | INDUCTION | CELL BIOLOGY | INHIBITION | LIVER-INJURY | INFLAMMATION | GENE-EXPRESSION | INFECTION | Cell Line | RNA, Small Interfering - genetics | Up-Regulation | Interleukin-6 - genetics | Humans | RNA, Messenger - genetics | Tumor Necrosis Factor-alpha - genetics | Gene Expression Regulation | Interleukin-1beta - immunology | Proto-Oncogene Proteins - genetics | Acute-Phase Proteins - genetics | Hepatocytes - metabolism | Hepatocytes - immunology | RNA Interference | Lipocalin-2 | Lipocalins - genetics | Lipocalins - immunology | Proto-Oncogene Proteins - immunology | Acute-Phase Proteins - immunology
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9. Full Text Intracellular Mycobacterium avium Intersect Transferrin in the Rab11 + Recycling Endocytic Pathway and Avoid Lipocalin 2 Trafficking to the Lysosomal Pathway
by Øyvind Halaas and Magnus Steigedal and Markus Haug and Jane A. Awuh and Liv Ryan and Andreas Brech and Shintaro Sato and Harald Husebye and Gerard A. Cangelosi and Shizuo Akira and Roland K. Strong and Terje Espevik and Trude H. Flo
The Journal of Infectious Diseases, ISSN 0022-1899, 3/2010, Volume 201, Issue 5, pp. 783 - 792
Iron is an essential nutrient for microbes, and many pathogenic bacteria depend on siderophores to obtain iron. The mammalian innate immunity protein lipocalin... 
Transferrins | Mycobacterium tuberculosis | Neutrophils | Lysosomes | Phagosomes | Bacteria | Infections | Iron | Macrophages | Endosomes | INFECTIOUS DISEASES | RAB5 | MACROPHAGES | MICROBIOLOGY | GELATINASE-ASSOCIATED LIPOCALIN | IMMUNOLOGY | MATURATION | MEDIATED IRON ACQUISITION | TUBERCULOSIS PHAGOSOME | INHIBITION | GROWTH | EARLY ENDOSOMES | PROTEINS | Oncogene Proteins - blood | Lysosomes - chemistry | Liver - microbiology | Tuberculosis - microbiology | Blood - microbiology | Colony Count, Microbial | Spleen - microbiology | Lysosomes - metabolism | Lipocalins - blood | Mycobacterium avium - pathogenicity | Oncogene Proteins - immunology | Macrophages - immunology | Macrophages - microbiology | Lipocalins - metabolism | rab GTP-Binding Proteins - metabolism | Acute-Phase Proteins - metabolism | Mice, Inbred C57BL | Oncogene Proteins - metabolism | Mycobacterium avium - immunology | Mycobacterium avium - metabolism | Tuberculosis - immunology | Animals | Lysosomes - microbiology | Lipocalin-2 | Lipocalins - immunology | Mice | Transferrin - metabolism | Mycobacterium avium - growth & development | Acute-Phase Proteins - immunology | Transferrin | Growth | Physiological aspects | Mycobacterium avium | Genetic aspects | Research | mouse | infection | Rab11 | Mycobacteria | transferrin | iron | Lcn2
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10. Full Text Circulating Lipocalin 2 Levels Predict Fracture‐Related Hospitalizations in Elderly Women: A Prospective Cohort Study
by Lim, Wai H and Wong, Germaine and Lim, Ee M and Byrnes, Elizabeth and Zhu, Kun and Devine, Amanda and Pavlos, Nathan J and Prince, Richard L and Lewis, Joshua R
Journal of Bone and Mineral Research, ISSN 0884-0431, 11/2015, Volume 30, Issue 11, pp. 2078 - 2085
Lipocalin 2 (LCN2) or neutrophil gelatinase–associated lipocalin (NGAL) is expressed in a wide range of cells and pathological states. Mounting evidence... 
NGAL | LCN2 | PHYSICAL ACTIVITY | OSTEOPOROSIS | FRACTURE RISK | ELDERLY WOMEN | GELATINASE-ASSOCIATED LIPOCALIN | OBESE WOMEN | DECLINE | INFLAMMATION | ENDOCRINOLOGY & METABOLISM | RESISTANCE | OLDER WOMEN | PHYSICAL-ACTIVITY | BONE MASS | POSTMENOPAUSAL WOMEN | Body Mass Index | Bone Density | Glomerular Filtration Rate | Prospective Studies | Acute-Phase Proteins | Humans | Osteoporotic Fractures - diagnostic imaging | Risk Factors | Linear Models | Hospitalization | Motor Activity | Heel - diagnostic imaging | Lipocalin-2 | Lipocalins - blood | Ultrasonography | Female | Aged | Osteoporotic Fractures - blood | Hip Fractures - complications | Osteoporotic Fractures - physiopathology | Proto-Oncogene Proteins - blood | Accidental Falls | Women | Osteoporosis | Analysis | Bones | Density | Health aspects
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11. Full Text The role of lipocalin‐2 in liver regeneration
by Kienzl‐Wagner, Katrin and Moschen, Alexander R and Geiger, Sabine and Bichler, Alexandra and Aigner, Felix and Brandacher, Gerald and Pratschke, Johann and Tilg, Herbert
Liver International, ISSN 1478-3223, 04/2015, Volume 35, Issue 4, pp. 1195 - 1202
Background & Aims Various immune mediators such as interleukin‐6 (IL‐6) have been implicated in the process of liver regeneration. Lipocalin‐2 (LCN2) has been... 
partial hepatectomy | LCN2 | hepatic proliferation | lipocalin‐2 | biomarker | liver regeneration | Biomarker | Hepatic proliferation | Partial hepatectomy | Lipocalin-2 | Liver regeneration | PARTIAL-HEPATECTOMY | CYTOKINES | ACUTE-PHASE PROTEIN | MOUSE | INJURY | GELATINASE-ASSOCIATED LIPOCALIN | EPITHELIAL-CELLS | MICE | lipocalin-2 | KIDNEY | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | Oncogene Proteins - genetics | Up-Regulation | Humans | Middle Aged | Male | Acute-Phase Proteins - genetics | Liver - physiopathology | Time Factors | Interleukin-6 - blood | Lipocalins - blood | Lipocalins - genetics | Adult | Female | Acute-Phase Proteins - deficiency | Liver - surgery | Lipocalins - metabolism | Liver Regeneration | Hepatectomy - methods | Signal Transduction | Acute-Phase Proteins - metabolism | Liver - metabolism | Oncogene Proteins - metabolism | Biomarkers - blood | Mice, Knockout | Homozygote | Phenotype | Animals | Heterozygote | Oncogene Proteins - deficiency | Aged | Proto-Oncogene Proteins - blood | Liver | Analysis
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12. Full Text Lipocalin 2 binds to membrane phosphatidylethanolamine to induce lipid raft movement in a PKA-dependent manner and modulates sperm maturation
by Watanabe, Hitomi and Takeo, Toru and Tojo, Hiromasa and Sakoh, Kazuhito and Berger, Thorsten and Nakagata, Naomi and Mak, Tak W and Kondoh, Gen
Development (Cambridge), ISSN 0950-1991, 2014, Volume 141, Issue 10, pp. 2157 - 2164
Mammalian sperm undergo multiple maturation steps after leaving the testis in order to become competent for fertilization, but the molecular mechanisms... 
Lipid raft | Phosphatidylethanolamine | Mouse | Sperm | PKA | LCN2 | Oviduct | CONTACT | PROTEIN | FUSION | MOUSE SPERMATOZOA | MAMMALIAN FERTILIZATION | IRON | DEVELOPMENTAL BIOLOGY | INFECTION | MOLECULAR-BASIS | CAPACITATION | ACROSOME REACTION | Oncogene Proteins - genetics | Cricetulus | Membrane Fluidity - genetics | Male | Acute-Phase Proteins - genetics | Fertility - genetics | Lipocalins - genetics | Sperm Maturation - genetics | Female | CHO Cells | Lipocalins - metabolism | Cricetinae | Movement | Acute-Phase Proteins - metabolism | Mice, Inbred C57BL | Oncogene Proteins - metabolism | Membrane Microdomains - chemistry | Mice, Transgenic | Cholesterol - metabolism | Pregnancy | Cyclic AMP-Dependent Protein Kinases - physiology | Animals | Lipocalin-2 | Membrane Microdomains - physiology | Mice | Phosphatidylethanolamines - metabolism | Protein Binding - physiology
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13. Full Text Nitric oxide boosts TLR‐4 mediated lipocalin 2 expression in chondrocytes
by Gómez, Rodolfo and Scotece, Morena and Conde, Javier and Lopez, Veronica and Pino, Jesus and Lago, Francisca and Gómez‐Reino, Juan J and Gualillo, Oreste
Journal of Orthopaedic Research, ISSN 0736-0266, 07/2013, Volume 31, Issue 7, pp. 1046 - 1052
Lipocalin 2 (LCN2) has recently emerged as a novel adipokine involved in different processes including arthritis and chondrocyte inflammatory response.... 
LCN2 | nitric oxide | adipokines | chondrocyte | inflammation | TLR4 | APOPTOSIS | ACTIVATION | GELATINASE-ASSOCIATED LIPOCALIN | INDUCTION | GENE | PATHWAY | TOLL-LIKE RECEPTORS | ARTICULAR CHONDROCYTES | OSTEOARTHRITIS | ORTHOPEDICS | Chondrocytes - cytology | Guanidines - pharmacology | Nitroprusside - pharmacology | Chondrocytes - drug effects | Matrix Metalloproteinase 9 - metabolism | Nitric Oxide Donors - pharmacology | Chondrocytes - metabolism | Lipocalins - metabolism | Cell Line | Cell Survival - drug effects | Lipocalins - pharmacology | Acute-Phase Proteins - metabolism | Cells, Cultured | Oncogene Proteins - metabolism | Oncogene Proteins - pharmacology | Macrophages - cytology | Toll-Like Receptor 4 - metabolism | Macrophages - metabolism | Animals | Lipocalin-2 | Lipopolysaccharides - pharmacology | Macrophages - drug effects | Mice | Nitric Oxide - metabolism | Acute-Phase Proteins - pharmacology | Periodical publishing | Genetic research | Gene expression | Analysis | Nitric oxide
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14. Full Text Thyroid hormone-mediated regulation of lipocalin 2 through the Met/FAK pathway in liver cancer
by Chung, I-Hsiao and Chen, Cheng-Yi and Lin, Yang-Hsiang and Chi, Hsiang-Cheng and Huang, Ya-Hui and Tai, Pei-Ju and Liao, Chia-Jung and Tsai, Chung-Ying and Lin, Syuan-Ling and Wu, Meng-Han and Chen, Ching-Ying and Lin, Kwang-Huei
Oncotarget, ISSN 1949-2553, 2015, Volume 6, Issue 17, pp. 15050 - 15064
LCN2 | Thyroid hormone receptor | Met/FAK cascade
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15. Full Text NFAT3 transcription factor inhibits breast cancer cell motility by targeting the Lipocalin 2 gene
by Fougère, M and Gaudineau, B and Barbier, J and Guaddachi, F and Feugeas, J.-P and Auboeuf, D and Jauliac, S
Oncogene, ISSN 0950-9232, 04/2010, Volume 29, Issue 15, pp. 2292 - 2301
NFAT1 and NFAT5 act as pro-invasive and promigratory transcription factors in breast carcinoma, contributing to the formation of metastases. We report that... 
Breast | LCN2 | Estrogen receptor a | Motility | NFAT3 | NGAL | PROTEIN | BIOCHEMISTRY & MOLECULAR BIOLOGY | estrogen receptor alpha | PANCREATIC-CANCER | GELATINASE-ASSOCIATED LIPOCALIN | IDENTIFICATION | CELL BIOLOGY | INVASION | ONCOLOGY | ESTROGEN-RECEPTOR-ALPHA | GENETICS & HEREDITY | motility | NUCLEAR FACTOR | breast | EXPRESSION | T-CELLS | Neoplasm Invasiveness | Humans | NFATC Transcription Factors - metabolism | Actins - metabolism | Gene Expression Regulation, Neoplastic | Proto-Oncogene Proteins - genetics | Acute-Phase Proteins - genetics | Proto-Oncogene Proteins - deficiency | Breast Neoplasms - metabolism | Breast Neoplasms - genetics | Breast Neoplasms - pathology | Lipocalin-2 | Lipocalins - genetics | Cell Line, Tumor | Protein Binding | Estrogen Receptor alpha - metabolism | Acute-Phase Proteins - deficiency | Cell Movement | NFATC Transcription Factors - genetics | Cellular proteins | Transcription factors | Physiological aspects | Development and progression | Breast cancer | Genetic aspects | Research | Cells | Signal transduction | Gene expression | Estrogen | Cell adhesion & migration | Acute-Phase Proteins | Actins | NFATC Transcription Factors | Breast Neoplasms | Life Sciences | Estrogen Receptor alpha | Lipocalins | Proto-Oncogene Proteins | Cancer
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