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PLoS genetics, ISSN 1553-7404, 2011, Volume 7, Issue 3, pp. e1001324 - e1001324
Journal Article
Frontiers in cellular and infection microbiology, ISSN 2235-2988, 2016, Volume 6
Human CARD9 deficiency is an autosomal recessive primary immunodeficiency disorder caused by biallelic mutations in the gene CARD9, which encodes a signaling protein that is found downstream of many C... 
Fungi | Lectins | Innate immunity | Candida | CARD9 | Neutrophils | INDUCED ACTIVATION | RECOGNITION | lectins | MICROBIOLOGY | HUMAN DECTIN-1 | INDUCTION | IMMUNOLOGY | DEFICIENCY | COMBINED IMMUNODEFICIENCY | innate immunity | fungi | HOST-DEFENSE | neutrophils | HOMOZYGOUS CARD9 MUTATION | TH17 CELLS | Physiological aspects | Usage | Cellular signal transduction | Genetic aspects | Drug resistance | Antifungal agents | Phaeohyphomycosis | Lectins, C-Type
Journal Article
Oncogene, ISSN 1476-5594, 2017, Volume 36, Issue 44, pp. 6097 - 6108
The C-type lectin domain containing group 14 family members CLEC14A and CD93 are proteins expressed by endothelium and are implicated in tumour angiogenesis. CD248... 
MOLECULAR CHARACTERIZATION | MARKER | BIOCHEMISTRY & MOLECULAR BIOLOGY | RECEPTOR | TUMOR ANGIOGENESIS | CELL BIOLOGY | IN-VITRO | ELEVATED EXPRESSION | ONCOLOGY | COLORECTAL-CANCER | GROWTH | GENETICS & HEREDITY | SPROUTING ANGIOGENESIS | ENDOSIALIN | Human Umbilical Vein Endothelial Cells | Cell Adhesion Molecules - genetics | Membrane Glycoproteins - metabolism | Humans | Lectins, C-Type - genetics | Antigens, CD - genetics | Antigens, CD - metabolism | Neovascularization, Pathologic - pathology | Receptors, Complement - metabolism | Lectins, C-Type - metabolism | Thrombomodulin - metabolism | Pericytes - pathology | Receptors, Complement - genetics | Antigens, Neoplasm - metabolism | Antigens, Surface - metabolism | Thrombomodulin - genetics | Antigens, Neoplasm - genetics | Endothelial Cells - metabolism | Pancreatic Neoplasms - pathology | Pericytes - metabolism | Antigens, Surface - genetics | Pancreatic Neoplasms - genetics | Cell Adhesion Molecules - metabolism | Membrane Glycoproteins - genetics | Animals | Protein Binding | Ligands | Neovascularization, Pathologic - genetics | Mice | Endothelial Cells - pathology | Care and treatment | Pancreatic cancer | Analysis | Lectins | Extracellular matrix | Diagnosis | Research | Protein-protein interactions | Animal models | Peptides | Cell surface | Endothelial cells | Endothelium | Angiogenesis | Rodents | Fibroblasts | Pericytes | Matrix protein | Thrombomodulin | Protein interaction | Umbilical vein | Tumors | Original
Journal Article
Nature (London), ISSN 1476-4687, 2017, Volume 549, Issue 7670, pp. 111 - 115
The majority of genetic variants associated with common human diseases map to enhancers, non-coding elements that shape cell-type-specific transcriptional programs and responses to extracellular cues(1-3... 
INTERLEUKIN-2 | ANTIBODIES | AUTOIMMUNE | GENE | MULTIDISCIPLINARY SCIENCES | LOCI | EXPRESSION | Antigens, CD - immunology | Antigens, CD - biosynthesis | Humans | Interleukin-2 Receptor alpha Subunit - biosynthesis | Lectins, C-Type - immunology | Autoimmunity - genetics | Lectins, C-Type - biosynthesis | Lectins, C-Type - genetics | Antigens, CD - genetics | Interleukin-2 Receptor alpha Subunit - genetics | Female | Receptors, Antigen, T-Cell - immunology | Th17 Cells - cytology | Cell Differentiation | Clustered Regularly Interspaced Short Palindromic Repeats - genetics | Interleukin-2 Receptor alpha Subunit - immunology | Cell Line | Antigens, Differentiation, T-Lymphocyte - immunology | Gene Expression Regulation - genetics | CRISPR-Cas Systems - genetics | Animals | Antigens, Differentiation, T-Lymphocyte - genetics | Th17 Cells - immunology | Enhancer Elements, Genetic - genetics | Mice | Receptors, Antigen, T-Cell - genetics | Antigens, Differentiation, T-Lymphocyte - biosynthesis | Chromatin - genetics | Genetic research | Genetic aspects | Research | RNA processing | Immunity | Interleukin 2 receptors | CRISPR | Chromatin | Animal models | Disease | Transcription | Helper cells | Lymphocytes T | Inflammation | T cell receptors | Genetic diversity | Gene deletion | Gene expression | Gene sequencing | Studies | CD69 antigen | Immunology | Clonal deletion | Perturbation methods | Lymphocytes | Genetic engineering | Gene mapping | Immune system
Journal Article
The Journal of biological chemistry, ISSN 1083-351X, 2014, Volume 289, Issue 4, pp. 2405 - 2414
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 04/2007, Volume 282, Issue 17, pp. 12397 - 12409
Journal Article
Traffic (Copenhagen, Denmark), ISSN 1398-9219, 2016, Volume 17, Issue 6, pp. 639 - 656
.... In addition to the C‐type lectin DC‐SIGN (Dendritic cell‐specific intercellular adhesion molecule 3‐grabbing non‐integrin... 
DC‐SIGNR | Rift Valley fever virus | endocytic motif | C‐type lectins | uptake | binding | L‐SIGN | phlebovirus | virus receptor | Uukuniemi virus | CD209 | bunyavirus | CD209L | endocytosis | DC‐SIGN | Binding | Virus receptor | Bunyavirus | DC-SIGNR | Uptake | L-SIGN | Endocytic motif | Endocytosis | DC-SIGN | C-type lectins | Phlebovirus | PHOSPHATIDYLSERINE RECEPTORS | HUMAN DENDRITIC CELLS | ENTRY | MAMMALIAN-CELLS | CELL BIOLOGY | DENGUE-VIRUS | RIFT-VALLEY FEVER | CARBOHYDRATE-RECOGNITION DOMAIN | INFECTION | Liver - virology | Cell Adhesion Molecules - genetics | Phlebovirus - pathogenicity | Endothelial Cells - metabolism | Humans | Receptors, Cell Surface - metabolism | Lectins, C-Type - genetics | Cell Adhesion Molecules - metabolism | Virus Internalization | Lectins, C-Type - metabolism | Phlebovirus - physiology | Protein Binding | Liver - cytology | HeLa Cells | Endothelial Cells - virology | Receptors, Cell Surface - genetics | Ammonium compounds | Rift Valley fever | Dendritic cells | Ammonium paratungstate | Analysis | Liver | Lectins | Livestock | Health aspects | Rodents | Phlebovirus/physiology | Endothelial Cells/virology | Phlebovirus/pathogenicity | Liver/virology | Lectins, C-Type/metabolism | Virology | Life Sciences | Cell Adhesion Molecules/metabolism | Liver/cytology | Microbiology and Parasitology | Receptors, Cell Surface/genetics | Endothelial Cells/metabolism | Lectins, C-Type/genetics | Receptors, Cell Surface/metabolism | Cell Adhesion Molecules/genetics
Journal Article
The Journal of biological chemistry, ISSN 1083-351X, 2014, Volume 289, Issue 43, pp. 30052 - 30062
.... Previous studies indicate that both Dectin-3 (also called MCL or Clec4d) and Mincle (also called Clec4e), two C-type lectin receptors, can recognize trehalose 6,6-dimycolate... 
SYK KINASE | Lectin | CELLS | Signal Transduction | RECOGNITION | NF-kappa B (NF-B) | BIOCHEMISTRY & MOLECULAR BIOLOGY | Innate Immunity | NF-B Transcription Factor | INNATE IMMUNE | ANTIFUNGAL IMMUNITY | RESPONSES | CANDIDA-ALBICANS | INFECTION | NF-KAPPA-B | CORD FACTOR | Adjuvants, Immunologic - pharmacology | Humans | CARD Signaling Adaptor Proteins - deficiency | NF-kappa B - metabolism | Lectins, C-Type - genetics | Neoplasm Proteins - metabolism | Promoter Regions, Genetic - genetics | Protein Subunits - metabolism | Lectins, C-Type - metabolism | CARD Signaling Adaptor Proteins - metabolism | Caspases - metabolism | Caspases - deficiency | Membrane Proteins - metabolism | Cord Factors - pharmacology | Lectins, C-Type - deficiency | Membrane Proteins - genetics | Mice, Inbred C57BL | NFATC Transcription Factors - metabolism | Receptors, Immunologic - deficiency | Gene Expression Regulation - drug effects | Animals | Signal Transduction - drug effects | Transcription Factor RelA - metabolism | Models, Biological | Adaptor Proteins, Signal Transducing - deficiency | B-Cell CLL-Lymphoma 10 Protein | Mucosa-Associated Lymphoid Tissue Lymphoma Translocation 1 Protein | Neoplasm Proteins - deficiency | Adaptor Proteins, Signal Transducing - metabolism | Cytokines - biosynthesis | Protein Multimerization - drug effects | Receptors, Immunologic - metabolism | Immunology | NF-κB Transcription Factor | NF-kappa B (NF-κB)
Journal Article
Journal Article
Journal Article