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FEBS Journal, ISSN 1742-464X, 05/2016, Volume 283, Issue 9, pp. 1689 - 1700
Intervertebral discs ( IVD s) provide stability and flexibility to the spinal column; however, IVD s, and in particular the nucleus pulposus ( NP ), undergo a... 
intervertebral disc degeneration | Smad | BMP | mesenchymal stem cells | nucleus pulposus | BMP7 | REGENERATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | MESENCHYMAL STEM-CELLS | TRANSPLANTATION | CROSS-TALK | LUMBAR-SPINE | IN-VITRO | NUCLEUS PULPOSUS CELLS | DISEASE | GROWTH | DIFFERENTIATION | RNA, Small Interfering - genetics | Bone Morphogenetic Protein 7 - genetics | Humans | Extracellular Matrix - metabolism | Intervertebral Disc Degeneration - metabolism | Intervertebral Disc Degeneration - therapy | Collagen Type II - metabolism | Intervertebral Disc Degeneration - pathology | Lentivirus - metabolism | Glucuronosyltransferase - genetics | Mesenchymal Stromal Cells - cytology | Smad1 Protein - genetics | Lentivirus - genetics | Intervertebral Disc - pathology | Chondrocytes - metabolism | Disease Models, Animal | Intervertebral Disc Degeneration - genetics | SOX9 Transcription Factor - metabolism | Chondrocytes - pathology | Rabbits | Genetic Vectors - chemistry | Signal Transduction | Bone Marrow Cells - cytology | Glycosaminoglycans - metabolism | Keratin-8 - genetics | Gene Expression Regulation | Genetic Vectors - metabolism | Mesenchymal Stromal Cells - metabolism | Intervertebral Disc - metabolism | Aggrecans - metabolism | Smad1 Protein - antagonists & inhibitors | Aggrecans - genetics | Collagen Type II - genetics | Keratin-19 - genetics | Animals | Glucuronosyltransferase - metabolism | Keratin-8 - metabolism | Bone Morphogenetic Protein 7 - metabolism | Smad1 Protein - metabolism | Keratin-19 - metabolism | Extracellular Matrix - pathology | SOX9 Transcription Factor - genetics | Bone Marrow Cells - metabolism | Mesenchymal Stem Cell Transplantation | RNA, Small Interfering - metabolism | Physiological aspects | Bone morphogenetic proteins | Cell differentiation | Analysis | Stem cells | Extracellular matrix | Spine | Rodents | Keratins | Therapy | Surgical implants | Smad protein | Mesenchyme | Collagen (type II) | Sox9 protein | Medical services | Differentiation (biology) | Electrochemical machining | Homeostasis | Nucleus pulposus | Remodeling | Intervertebral discs | Nuclei | Flexibility | Bone marrow | Degeneration | Effectiveness | Stability | Markers | Feasibility studies | Implantation | Columns (process) | Survival | Diseases | Overexpression | Disks | Feasibility | Chondroitin sulfate | Sulfate | Aggrecan | Viability | Bone morphogenetic protein 7 | Index Medicus
Journal Article
Molecular Therapy, ISSN 1525-0016, 01/2011, Volume 19, Issue 1, pp. 122 - 132
X-linked chronic granulomatous disease (X-CGD) is a primary immunodeficiency caused by mutations in the gene encoding the phagocyte nicotinamide adenine... 
MEDICINE, RESEARCH & EXPERIMENTAL | NADPH OXIDASE | SUPEROXIDE-PRODUCTION | CHRONIC GRANULOMATOUS-DISEASE | C-MYB | PU.1 | HEMATOPOIETIC-CELLS | GENOMIC INSTABILITY | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | MOUSE MODEL | GENETICS & HEREDITY | HOST-DEFENSE | GENE-THERAPY | Genetic Vectors - administration & dosage | Humans | Myeloid Cells - physiology | Granulomatous Disease, Chronic - genetics | Granulomatous Disease, Chronic - metabolism | NADPH Oxidases - metabolism | Molecular Sequence Data | Stem Cells - metabolism | Recombinant Fusion Proteins - metabolism | DNA Copy Number Variations | Lentivirus - metabolism | Cell Differentiation - genetics | Genes, X-Linked | Retroviridae - metabolism | Retroviridae - genetics | Base Sequence | NADPH Oxidases - genetics | Lentivirus - genetics | Granulocytes - metabolism | Terminal Repeat Sequences | Transgenes | Binding Sites | CCAAT-Enhancer-Binding Proteins - metabolism | Proto-Oncogene Proteins - metabolism | Recombinant Fusion Proteins - biosynthesis | Promoter Regions, Genetic | Cathepsin G - genetics | Proto-Oncogene Proteins c-fes - genetics | Gene Expression Regulation | Mutagenesis - genetics | Genetic Vectors - metabolism | Spleen Focus-Forming Viruses - metabolism | Granulomatous Disease, Chronic - therapy | Hematopoietic Stem Cells - metabolism | Spleen Focus-Forming Viruses - genetics | Genetic Vectors - genetics | Animals | Enhancer Elements, Genetic | Cell Line, Tumor | Myeloid Cells - metabolism | Recombinant Fusion Proteins - genetics | Trans-Activators - metabolism | Granulomatous Disease, Chronic - enzymology | Mice | Receptors, Immunologic - genetics | Receptors, Immunologic - metabolism | Genetic Therapy - methods | Index Medicus | Original
Journal Article
PLoS Biology, ISSN 1544-9173, 05/2012, Volume 10, Issue 5, pp. e1001336 - e1001336
In metazoans, the majority of mRNAs coding for secreted and membrane-bound proteins are translated on the surface of the endoplasmic reticulum (ER). Although... 
RAT-LIVER | TRANSLOCATION | DIRECT ASSOCIATION | MEMBRANE-BOUND RIBOSOMES | MITOCHONDRIAL BIOGENESIS | BUDDING YEAST | BIOCHEMISTRY & MOLECULAR BIOLOGY | BIOLOGY | RECEPTOR | BINDING PROTEIN | POLYSOMES | MAMMALIAN-CELLS | Humans | Alkaline Phosphatase - metabolism | Cercopithecus aethiops | Endoplasmic Reticulum - metabolism | Molecular Sequence Data | Ribosomes - metabolism | RNA, Messenger - metabolism | Calreticulin - genetics | Lentivirus - metabolism | Isoenzymes - metabolism | Base Sequence | Cloning, Molecular | Escherichia coli - metabolism | HEK293 Cells | Plasmids - genetics | Calreticulin - metabolism | Lentivirus - genetics | Lysine - metabolism | Active Transport, Cell Nucleus | Insulin - genetics | Green Fluorescent Proteins - metabolism | Alkaline Phosphatase - genetics | Endoplasmic Reticulum - genetics | Isoenzymes - genetics | RNA, Messenger - genetics | Cell Size | In Situ Hybridization, Fluorescence | Receptors, Cytoplasmic and Nuclear - genetics | Plasmids - metabolism | GPI-Linked Proteins - metabolism | Protein Interaction Mapping | Protein Transport | Proteins - genetics | Insulin - metabolism | Animals | Proteins - metabolism | Escherichia coli - genetics | COS Cells | Digitonin - pharmacology | GPI-Linked Proteins - genetics | Receptors, Cytoplasmic and Nuclear - metabolism | Proteins | Medical research | Polypeptides | Yeast | Insects | Plasmids | Kinases | Gene expression | Experiments | Index Medicus
Journal Article
Human Molecular Genetics, ISSN 0964-6906, 08/2010, Volume 19, Issue 15, pp. 3053 - 3067
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 2017, Volume 292, Issue 37, pp. 15561 - 15576
Preferential expression of the low-activity (dimeric) M2 isoform of pyruvate kinase (PK) over its constitutively active splice variant M1 isoform is considered... 
LUNG-CANCER | METABOLISM | TUMOR SUPPRESSION | PKM2 | BIOCHEMISTRY & MOLECULAR BIOLOGY | GROWTH | PREDICTING SUBCELLULAR-LOCALIZATION | HIGH-TITER LENTIVIRUS | ISOFORM | LKB1 | GENE-TRANSCRIPTION | Mitochondrial Dynamics | AMP-Activated Protein Kinases - metabolism | Humans | Lung Neoplasms - metabolism | Pyruvate Kinase - chemistry | Lung Neoplasms - pathology | Neoplasm Proteins - antagonists & inhibitors | Isoenzymes - chemistry | Neoplasm Proteins - metabolism | Autophagy | Recombinant Fusion Proteins - metabolism | Thyroid Hormones - chemistry | Organelle Biogenesis | RNA Interference | Isoenzymes - metabolism | Adenosine Triphosphate - metabolism | Carcinoma - enzymology | Carrier Proteins - chemistry | Membrane Proteins - metabolism | Carcinoma - pathology | Neoplasm Proteins - genetics | Dimerization | A549 Cells | Lung Neoplasms - enzymology | Pyruvate Kinase - metabolism | Isoenzymes - genetics | Membrane Proteins - genetics | AMP-Activated Protein Kinases - antagonists & inhibitors | Carrier Proteins - antagonists & inhibitors | Neoplasm Proteins - chemistry | Recombinant Fusion Proteins - chemistry | Thyroid Hormones - genetics | Protein Transport | Carrier Proteins - genetics | Pyruvate Kinase - antagonists & inhibitors | Carrier Proteins - metabolism | Energy Metabolism | Membrane Proteins - antagonists & inhibitors | Membrane Proteins - chemistry | Thyroid Hormones - metabolism | Cell Line, Tumor | Carcinoma - metabolism | Mutation | Pyruvate Kinase - genetics | AMP-Activated Protein Kinases - genetics | Isoenzymes - antagonists & inhibitors | Amino Acid Substitution | Apoptosis | Index Medicus | cancer metabolism | cancer therapy | pyruvate kinase M1 | mitochondria | pyruvate kinase M2 | cancer biology | Metabolism | mitochondrial oxidative phosphorylation | energy metabolism | Warburg effect
Journal Article
Journal of Translational Medicine, ISSN 1479-5876, 08/2014, Volume 12, Issue 1, pp. 218 - 218
Journal Article
Cell Reports, ISSN 2211-1247, 03/2016, Volume 14, Issue 8, pp. 1883 - 1891
Journal Article
BBA - Molecular Cell Research, ISSN 0167-4889, 12/2016, Volume 1863, Issue 12, pp. 3040 - 3049
Cardiac ankyrin repeat protein (CARP) is a nuclear transcriptional co-factor that has additional functions in the myoplasm as a component of the muscle... 
Transcription co-factor | Cardiac ankyrin repeat protein | Cardiomyocyte | Hypoxia/reoxygenation | Apoptosis | Bcl-2 family | CELLS | CARP | BIOCHEMISTRY & MOLECULAR BIOLOGY | HEART-FAILURE | DOXORUBICIN | MECHANISMS | DILATED CARDIOMYOPATHY | HYPOXIA | CELL BIOLOGY | CYTOCHROME-C | GENE-EXPRESSION | STRESS | Myocardial Ischemia - genetics | Myocardial Ischemia - metabolism | RNA, Small Interfering - genetics | Proto-Oncogene Proteins c-bcl-2 - agonists | Caspase 3 - metabolism | Male | Repressor Proteins - antagonists & inhibitors | Lentivirus - metabolism | Proto-Oncogene Proteins c-bcl-2 - metabolism | Cell Nucleus - metabolism | Caspase 3 - genetics | Muscle Proteins - metabolism | Adenoviridae - genetics | Lentivirus - genetics | Muscle Proteins - antagonists & inhibitors | Transcription, Genetic | Nuclear Proteins - genetics | Reperfusion Injury - genetics | Reperfusion Injury - metabolism | Repressor Proteins - metabolism | Animals, Newborn | Promoter Regions, Genetic | GATA4 Transcription Factor - metabolism | Genetic Vectors - chemistry | Reperfusion Injury - pathology | Signal Transduction | Mice, Inbred C57BL | Gene Expression Regulation | Genetic Vectors - metabolism | Repressor Proteins - genetics | GATA4 Transcription Factor - genetics | Nuclear Proteins - metabolism | Myocardial Ischemia - pathology | Muscle Proteins - genetics | Myocytes, Cardiac - pathology | Animals | Nuclear Proteins - antagonists & inhibitors | Myocytes, Cardiac - metabolism | Protein Binding | Mice | Adenoviridae - metabolism | Primary Cell Culture | Proto-Oncogene Proteins c-bcl-2 - genetics | RNA, Small Interfering - metabolism | Gene expression | Analysis | Target marketing
Journal Article
MOLECULAR VISION, ISSN 1090-0535, 09/2015, Volume 21, pp. 1071 - 1084
Purpose: Activation of the IL-1/NF-kappa B inflammatory stress pathway and induction of SELE expression in the trabecular meshwork (TBM) is a marker for... 
AQUEOUS OUTFLOW PATHWAY | MATRIX METALLOPROTEINASES | WILD-TYPE | OPEN-ANGLE GLAUCOMA | OXIDATIVE STRESS | PROTEIN | GENE | BIOCHEMISTRY & MOLECULAR BIOLOGY | OPHTHALMOLOGY | INTRAOCULAR-PRESSURE | EXPRESSION | CALORIE RESTRICTION | Cytoskeletal Proteins - genetics | Glaucoma, Open-Angle - genetics | Transforming Growth Factor beta2 - metabolism | Humans | Actins - metabolism | Glycoproteins - metabolism | Glaucoma, Open-Angle - pathology | NF-kappa B - metabolism | Interleukin-1beta - genetics | Actins - genetics | E-Selectin - genetics | Forkhead Transcription Factors - metabolism | Interleukin-1beta - metabolism | Cytoskeletal Proteins - metabolism | Glaucoma, Open-Angle - metabolism | Lentivirus - genetics | Eye Proteins - genetics | Interleukin-6 - metabolism | Fibroblasts - metabolism | Glycoproteins - genetics | Trabecular Meshwork - pathology | Mutagenesis, Site-Directed | Genetic Vectors - chemistry | Interleukin-6 - genetics | Signal Transduction | Endothelial Cells - metabolism | Gene Expression Regulation | Genetic Vectors - metabolism | E-Selectin - metabolism | Inflammation | Fibroblasts - pathology | Forkhead Transcription Factors - genetics | Eye Proteins - metabolism | NF-kappa B - genetics | Models, Biological | Trabecular Meshwork - metabolism | Forkhead Box Protein O1 | Primary Cell Culture | Transforming Growth Factor beta2 - genetics | Endothelial Cells - pathology | Cell Line, Transformed | Amino Acid Substitution | Index Medicus
Journal Article
Stem Cell Research and Therapy, ISSN 1757-6512, 07/2016, Volume 7, Issue 1, pp. 97 - 97
Background: Mesenchymal stromal cells (MSCs) are multipotent progenitor cells used in several cell therapies. MSCs are characterized by the expression of CD73,... 
Mesenchymal stromal cells | Thy-1 | Differentiation | Fibroblast | CD90 | Mesenchymal stem cells | PULP STEM-CELLS | PROGENITOR CELLS | MEDICINE, RESEARCH & EXPERIMENTAL | INTERNATIONAL-SOCIETY | TUMOR-SUPPRESSOR GENE | HUMAN UMBILICAL-CORD | THERAPY POSITION STATEMENT | CELL BIOLOGY | IN-VITRO | NASOPHARYNGEAL CARCINOMA | LUNG FIBROBLASTS | EXTRACELLULAR-MATRIX | RNA, Small Interfering - genetics | Cell Proliferation | Humans | Adipose Tissue - cytology | Adipocytes - cytology | Fetal Proteins - metabolism | Dental Pulp - metabolism | Antigens, CD - genetics | Antigens, CD - metabolism | Lentivirus - metabolism | Adipose Tissue - metabolism | Mesenchymal Stromal Cells - cytology | T-Lymphocytes - metabolism | Amniotic Fluid - metabolism | Thy-1 Antigens - genetics | Hyaluronan Receptors - metabolism | Lentivirus - genetics | Cell Adhesion Molecules, Neuronal - metabolism | Cell Differentiation | Osteoblasts - cytology | Signal Transduction | Amniotic Fluid - cytology | Gene Silencing | Dental Pulp - cytology | Mesenchymal Stromal Cells - metabolism | Immunophenotyping | Hyaluronan Receptors - genetics | Cell Adhesion Molecules, Neuronal - genetics | Thy-1 Antigens - metabolism | T-Lymphocytes - cytology | Adipocytes - metabolism | Fetal Proteins - genetics | Primary Cell Culture | Osteoblasts - metabolism | RNA, Small Interfering - metabolism | Growth | Stem cells | Glycoproteins | Genetic aspects | Properties | Gene expression | Observations | Cell differentiation | Index Medicus
Journal Article
Journal Article