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Journal of Virology, ISSN 0022-538X, 10/2010, Volume 84, Issue 19, pp. 9733 - 9748
Journal Article
Behavioural Brain Research, ISSN 0166-4328, 02/2019, Volume 359, pp. 446 - 456
Lethal yellow ( ) mutation causes obesity and type-2 diabetes in mice. Here we studied the effect of the mutation on the brain and behavior. The experiments... 
Brain | Mice | Behavior | MRI | Lethal yellow | DEPRESSION | METABOLIC SYNDROME | PROTEIN | NEUROSCIENCES | OBESITY | INFLAMMATION | BODY-MASS INDEX | BEHAVIORAL SCIENCES | TYROSINE-PHOSPHATASE STEP | EXPRESSION | MELANOCORTIN-4 RECEPTOR | Type 2 diabetes | Tyrosine | Phosphatases | RNA | Genes | Genetic research | Genetic aspects | Gene expression | Life Sciences | Other
Journal Article
Journal Article
Cell, ISSN 0092-8674, 05/2017, Volume 169, Issue 5, pp. 945 - 955.e10
Journal Article
1945, University of California publications in zoology, Volume 53, no. 2, 41-65
Book
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 06/2017, Volume 127, Issue 6, pp. 2392 - 2406
Quiescent and proliferating leukemia cells accumulate highly lethal DNA double-strand breaks that are repaired by 2 major mechanisms: BRCA-dependent homologous... 
GENOMIC INSTABILITY | MEDICINE, RESEARCH & EXPERIMENTAL | POLY(ADP-RIBOSE) POLYMERASE | NHEJ PATHWAYS | HEMATOPOIETIC STEM-CELLS | DOUBLE-STRAND BREAKS | PARP | HOMOLOGOUS RECOMBINATION | DNA-REPAIR | CANCER | CHRONIC MYELOID-LEUKEMIA | Cell Proliferation | Mouse Embryonic Stem Cells - physiology | Humans | Transcriptome | DNA Breaks, Double-Stranded | Genes, Lethal | DNA End-Joining Repair | Genes, BRCA2 | Antineoplastic Agents - pharmacology | Genes, BRCA1 | Leukemia - genetics | Cricetinae | Leukemia - drug therapy | Mice, SCID | Piperazines - pharmacology | Mice, Knockout | Xenograft Model Antitumor Assays | Phthalazines - pharmacology | Poly(ADP-ribose) Polymerase Inhibitors - pharmacology | Animals | Cell Transformation, Neoplastic | Genes, abl | Cell Line, Tumor | Mice, Inbred NOD | Mice | Biotechnology | Pharmacogenetics | Leukemia | Analysis | Genes | Genetic aspects | Genetic transcription | Protein kinases | Sugars | Monosaccharides | Cancer | Cell proliferation | Gene mutations | Cancer cells | Development and progression | Gene expression | Health aspects | BCR protein | Flow cytometry | DNA damage | Homologous recombination | Reverse transcription | Genomes | Lethality | Breast cancer | Drug delivery | Kinases | DNA repair | Polymerase chain reaction | Poly(ADP-ribose) Polymerase 1 | DNA-dependent protein kinase | DNA microarrays | Ribose | Stem cells | DNA methylation | Mutation | Solid tumors
Journal Article