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1. Full Text Pain inhibition by blocking leukocytic and neuronal opioid peptidases in peripheral inflamed tissue
by Schreiter, Anja and Gore, Carmen and Labuz, Dominika and Fournie‐Zaluski, Marie‐Claude and Roques, Bernard P and Stein, Christoph and Machelska, Halina
The FASEB journal, ISSN 0892-6638, 12/2012, Volume 26, Issue 12, pp. 5161 - 5171
immune cells | analgesia | sensory nerves | Immune cells | Analgesia | Ory nerves | Biochemistry & Molecular Biology | Biology | Life Sciences & Biomedicine - Other Topics | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Leucine - pharmacology | Opioid Peptides - pharmacology | Rats, Wistar | Thiorphan - pharmacology | Opioid Peptides - immunology | Male | Enkephalin, Methionine - pharmacology | Leukocytes - enzymology | Enkephalin, Methionine - metabolism | Dose-Response Relationship, Drug | Narcotic Antagonists | Alanine - analogs & derivatives | Enkephalin, Methionine - immunology | Hindlimb - physiopathology | Inflammation - complications | Flow Cytometry | Pain - complications | Pain - enzymology | Enkephalin, Leucine - pharmacology | Antibodies - immunology | Pain Threshold - drug effects | Dynorphins - metabolism | Neurons - drug effects | Leucine - analogs & derivatives | Alanine - pharmacology | Receptors, Opioid - metabolism | Amino Acid Sequence | Enkephalin, Leucine - metabolism | Pain - prevention & control | CD13 Antigens - antagonists & inhibitors | Enzyme Inhibitors - pharmacology | Rats | Hindlimb - innervation | Enkephalin, Leucine - immunology | Neprilysin - metabolism | CD13 Antigens - metabolism | Opioid Peptides - metabolism | Antibodies - pharmacology | Dynorphins - immunology | Animals | Neurons - enzymology | Phosphinic Acids - pharmacology | Neprilysin - antagonists & inhibitors | Hindlimb - drug effects | Leukocytes - drug effects | Dynorphins - pharmacology | Inflammation - prevention & control | Inflammation - enzymology | Index Medicus
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2. Full Text The tyrosine kinase inhibitor bafetinib inhibits PAR2‐induced activation of TRPV4 channels in vitro and pain in vivo
by Grace, M S and Lieu, T and Darby, B and Abogadie, F C and Veldhuis, N and Bunnett, N W and McIntyre, P
British journal of pharmacology, ISSN 0007-1188, 08/2014, Volume 171, Issue 16, pp. 3881 - 3894
Life Sciences & Biomedicine | Pharmacology & Pharmacy | Science & Technology | Leucine - pharmacology | Hyperalgesia - metabolism | Humans | Mice, Inbred C57BL | Receptor, PAR-2 - metabolism | TRPV Cation Channels - agonists | Morpholines - pharmacology | Male | Pyrimidines - pharmacology | Pain - metabolism | Sulfonamides - pharmacology | TRPV Cation Channels - genetics | TRPV Cation Channels - metabolism | Pyrroles - pharmacology | Receptor, PAR-2 - antagonists & inhibitors | Animals | Trypsin - pharmacology | TRPV Cation Channels - antagonists & inhibitors | HEK293 Cells | Receptor, PAR-2 - agonists | Protein Kinase Inhibitors - pharmacology | Oligopeptides - pharmacology | Leucine - analogs & derivatives | Tyrosine | Phenols | Phosphotransferases | Adenosine triphosphatase | Rodents | Index Medicus | Research Papers
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3. Full Text Noradrenaline induces peripheral antinociception by endogenous opioid release
by Romero, Thiago Roberto Lima and Soares Santos, Raquel Rodrigues and Castor, Marina Gomes Miranda e and Petrocchi, Júlia Alvarenga and Guzzo, Luciana Souza and Klein, Andre and Duarte, Igor Dimitri Gama
Pharmacological reports, ISSN 1734-1140, 08/2018, Volume 70, Issue 4, pp. 784 - 788
Adrenoceptors | Noradrenaline | Noradrenergic system | Peripheral antinociception | Life Sciences & Biomedicine | Pharmacology & Pharmacy | Science & Technology | Hyperalgesia - chemically induced | Naltrexone - analogs & derivatives | Norepinephrine - antagonists & inhibitors | Analgesics - pharmacology | Norepinephrine - pharmacology | Leucine - pharmacology | Dinoprostone | Hyperalgesia - prevention & control | Rats | Male | Pain Measurement - drug effects | Cinnamates - pharmacology | Opioid Peptides - metabolism | Yohimbine - pharmacology | Dose-Response Relationship, Drug | Naltrexone - pharmacology | Propranolol - pharmacology | Animals | Prazosin - pharmacology | Morphine Derivatives - pharmacology | Leucine - analogs & derivatives | Index Medicus
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4. Full Text Use of a cAMP BRET Sensor to Characterize a Novel Regulation of cAMP by the Sphingosine 1-Phosphate/G13 Pathway
by Lily I. Jiang and Julie Collins and Richard Davis and Keng-Mean Lin and Dianne DeCamp and Tamara Roach and Robert Hsueh and Robert A. Rebres and Elliott M. Ross and Ronald Taussig and Iain Fraser and Paul C. Sternweis
The Journal of biological chemistry, ISSN 0021-9258, 04/2007, Volume 282, Issue 14, pp. 10576 - 10584
Cell Line | Lysophospholipids - metabolism | Basic-Leucine Zipper Transcription Factors - metabolism | Calcium Signaling - physiology | Humans | RNA, Small Interfering - pharmacology | Prostaglandins E - pharmacology | Adrenergic beta-Agonists - pharmacology | Macrophages - metabolism | Sphingosine - analogs & derivatives | Animals | Complement C5a - pharmacology | Calcium Signaling - drug effects | Isoproterenol - pharmacology | Biosensing Techniques | Mice | Sphingosine - metabolism | Receptors, Lysosphingolipid - metabolism | Uridine Diphosphate - pharmacology | Cyclic AMP - metabolism | Immunologic Factors - pharmacology | Pertussis Toxin - pharmacology | Index Medicus
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5. Full Text Nitrogen source activates TOR (Target of Rapamycin) complex 1 via glutamine and independently of Gtr/Rag proteins
by Stracka, Daniele and Jozefczuk, Szymon and Rudroff, Florian and Sauer, Uwe and Hall, Michael N
The Journal of biological chemistry, ISSN 0021-9258, 2014, Volume 289, Issue 36, pp. 25010 - 25020
Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | Leucine - pharmacology | Proline - metabolism | Saccharomyces cerevisiae - genetics | Glutamine - metabolism | Immunoblotting | Saccharomyces cerevisiae - metabolism | Nitrogen - pharmacology | Proline - pharmacology | Glutamine - pharmacology | Phosphorylation - drug effects | Protein-Serine-Threonine Kinases - metabolism | Leucine - metabolism | Nitrogen - metabolism | Protein-Serine-Threonine Kinases - genetics | Methionine Sulfoximine - pharmacology | Monomeric GTP-Binding Proteins - genetics | Saccharomyces cerevisiae Proteins - genetics | Transcription Factors - genetics | Sirolimus - pharmacology | Transcription Factors - metabolism | Ammonium Compounds - metabolism | Monomeric GTP-Binding Proteins - metabolism | Saccharomyces cerevisiae Proteins - metabolism | Ammonium Compounds - pharmacology | Mutation | Saccharomyces cerevisiae - growth & development | Index Medicus | Nitrogen Metabolism | Amino Acid | Yeast | Kinetics | Target of Rapamycin (TOR) | Cell Biology
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6. Full Text Depletion of substrates for protein prenylation increases apoptosis in human periovulatory granulosa cells
by Rung, Emilia and Friberg, P. Anders and Bergh, Christina and Billig, Håkan and Institutionen för kliniska vetenskaper and Göteborgs universitet and Gothenburg University and Institute of Neuroscience and Physiology, Department of Physiology and Sahlgrenska Academy and Sahlgrenska akademin and Institute of Clinical Sciences and Institutionen för neurovetenskap och fysiologi, sektionen för fysiologi
Molecular reproduction and development, ISSN 1040-452X, 10/2006, Volume 73, Issue 10, pp. 1277 - 1283
ovary | progesterone receptor | progesterone | ovulation | Ovary | Progesterone | Progesterone receptor | Ovulation | Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Developmental Biology | Reproductive Biology | Science & Technology | Cell Biology | Fundamental and applied biological sciences. Psychology | Biological and medical sciences | Hormone metabolism and regulation | Vertebrates: reproduction | Mammalian female genital system | Diterpenes - pharmacology | Protein Prenylation - drug effects | Leucine - pharmacology | Apoptosis - drug effects | Granulosa Cells - physiology | Farnesol - pharmacology | Furans - pharmacology | Humans | Cells, Cultured | Imidazoles - pharmacology | Quinolones - pharmacology | Granulosa Cells - drug effects | Granulosa Cells - metabolism | Receptors, Progesterone - antagonists & inhibitors | Hormone Antagonists - pharmacology | Hydroxymethylglutaryl-CoA Reductase Inhibitors - pharmacology | Estrenes - pharmacology | Female | Farnesyltranstransferase - antagonists & inhibitors | Cholesterol - biosynthesis | Mifepristone - pharmacology | Leucine - analogs & derivatives | Index Medicus | MEDICIN OCH HÄLSOVETENSKAP | Granulosa Cells/drug effects/metabolism/physiology | Receptors | Farnesyltranstransferase/antagonists & inhibitors | Imidazoles/pharmacology | Hydroxymethylglutaryl-CoA Reductase Inhibitors/pharmacology | Progesterone/antagonists & inhibitors | Physiology | Cultured | Estrenes/pharmacology | MEDICAL AND HEALTH SCIENCES | Diterpenes/pharmacology | Farnesol/pharmacology | Hormone Antagonists/pharmacology | Fysiologi | Apoptosis/drug effects | Furans/pharmacology | Cholesterol/biosynthesis | Leucine/analogs & derivatives/pharmacology | Cells | Protein Isoprenylation/drug effects | Mifepristone/pharmacology | Quinolones/pharmacology
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7. Full Text Central antinociception induced by ketamine is mediated by endogenous opioids and μ- and δ-opioid receptors
by Pacheco, Daniela da Fonseca and Romero, Thiago Roberto Lima and Duarte, Igor Dimitri Gama
Brain research, ISSN 0006-8993, 2014, Volume 1562, pp. 69 - 75
Neurology | Endogenous opioid | Ketamine | Central antinociception | Fundamental and applied biological sciences. Psychology | Vertebrates: nervous system and sense organs | Biological and medical sciences | Somesthesis and somesthetic pathways (proprioception, exteroception, nociception); interoception; electrolocation. Sensory receptors | Naltrexone - analogs & derivatives | Receptors, Opioid, delta - metabolism | Analgesics - pharmacology | Leucine - pharmacology | Pain Perception - physiology | Male | Nociceptive Pain - drug therapy | Brain - metabolism | Dose-Response Relationship, Drug | Nociceptive Pain - metabolism | Naltrexone - pharmacology | Pain Perception - drug effects | Receptors, Opioid, delta - antagonists & inhibitors | Aminopeptidases - metabolism | Leucine - analogs & derivatives | Receptors, Opioid, kappa - antagonists & inhibitors | Receptors, Opioid, mu - metabolism | Enzyme Inhibitors - pharmacology | Receptors, Opioid, kappa - metabolism | Aminopeptidases - antagonists & inhibitors | Hot Temperature | Receptors, Opioid, mu - antagonists & inhibitors | Cinnamates - pharmacology | Opioid Peptides - metabolism | Brain - drug effects | Animals | Naloxone - pharmacology | Narcotic Antagonists - pharmacology | Mice | Morphine Derivatives - pharmacology | Ketamine - pharmacology | Index Medicus
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8. Full Text TRPA1 and TRPV4 Activation in Human Odontoblasts Stimulates ATP Release
by Egbuniwe, O and Grover, S and Duggal, A.K and Mavroudis, A and Yazdi, M and Renton, T and Di Silvio, L and Grant, A.D
Journal of dental research, ISSN 0022-0345, 9/2014, Volume 93, Issue 9, pp. 911 - 917
orofacial pain | sensation | dentin | signal transduction | transient receptor potential channel | ion channels | Life Sciences & Biomedicine | Dentistry, Oral Surgery & Medicine | Science & Technology | Acrolein - analogs & derivatives | Transient Receptor Potential Channels - antagonists & inhibitors | Leucine - pharmacology | Capsaicin - pharmacology | Humans | Adenosine Triphosphate - secretion | Nociceptors - physiology | Sensory System Agents - pharmacology | Calcium Channels - physiology | Pyrimidinones - pharmacology | Transient Receptor Potential Channels - physiology | Cell Culture Techniques | Leucine - analogs & derivatives | Isothiocyanates - pharmacology | Acrolein - pharmacology | Cell Line | Nerve Tissue Proteins - antagonists & inhibitors | Acetanilides - pharmacology | Nerve Tissue Proteins - physiology | TRPV Cation Channels - physiology | Purines - pharmacology | Nerve Tissue Proteins - agonists | TRPV Cation Channels - agonists | Dental Pulp - cytology | TRPM Cation Channels - agonists | Sulfonamides - pharmacology | Transient Receptor Potential Channels - agonists | Culture Media, Conditioned | Calcium Signaling - drug effects | Odontoblasts - physiology | Odontoblasts - secretion | TRPA1 Cation Channel | Cell culture | Membranes | Cinnamaldehyde | Immunocytochemistry | Capsaicin | Reverse transcription | Capsaicin receptors | Allyl isothiocyanate | Experiments | Western blotting | Odontoblasts | Sensory neurons | Fura-2 | Infrared imaging systems | Phenotypes | Calcium (intracellular) | Neurons | Hypersensitivity | Teeth | Calcium imaging | Isothiocyanate | Gene expression | Polymerase chain reaction | Councils | Culture media | Agonists | Mechanotransduction | Intracellular | Dental pulp | Index Medicus | Dentistry | Research Reports
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9. Full Text Effect of Phosphoric Acid on the Degradation of Human Dentin Matrix
by Tezvergil-Mutluay, A and Mutluay, M and Seseogullari-Dirihan, R and Agee, K.A and Key, W.O and Scheffel, D.L.S and Breschi, L and Mazzoni, A and Tjäderhane, L and Nishitani, Y and Tay, F.R and Pashley, D.H
Journal of dental research, ISSN 0022-0345, 1/2013, Volume 92, Issue 1, pp. 87 - 91
collagen | demineralized | bonding | cathepsins | MMPs | Life Sciences & Biomedicine | Dentistry, Oral Surgery & Medicine | Science & Technology | Enzyme Activators - pharmacology | Leucine - pharmacology | Phenylmercuric Acetate - pharmacology | Dipeptides - pharmacology | Humans | Peptide Hydrolases - drug effects | Enzyme Precursors - drug effects | Materials Testing | Collagenases - drug effects | Phosphoric Acids - pharmacology | Matrix Metalloproteinase Inhibitors - pharmacology | Dentin - enzymology | Dentin - drug effects | Time Factors | Matrix Metalloproteinases - drug effects | Cysteine Proteinase Inhibitors - pharmacology | Protein Denaturation | Phenylmercuric Acetate - analogs & derivatives | Sulfhydryl Reagents - pharmacology | Collagen Type I - analysis | Leucine - analogs & derivatives | Peptides - analysis | Cathepsins - antagonists & inhibitors | Dentin | Phosphoric acid | Enzymes | Adhesives | Acids | Collagen | Etching | Acetic acid | Binding sites | Index Medicus | Dentistry | Research Reports
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