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Publication DateBlood, ISSN 0006-4971, 02/2017, Volume 129, Issue 6, pp. 704 - 714
Life Sciences & Biomedicine | Hematology | Science & Technology | Eosinophilia - therapy | Oncogene Proteins, Fusion - metabolism | Leukemia - pathology | Proto-Oncogene Proteins c-abl - antagonists & inhibitors | Humans | Gene Expression Regulation, Neoplastic | Receptor, Fibroblast Growth Factor, Type 1 - metabolism | fms-Like Tyrosine Kinase 3 | Antineoplastic Agents - therapeutic use | Myeloproliferative Disorders - pathology | Receptor, Fibroblast Growth Factor, Type 1 - genetics | Receptor, Platelet-Derived Growth Factor beta - genetics | Myeloproliferative Disorders - genetics | Transplantation, Homologous | Eosinophilia - pathology | Hypereosinophilic Syndrome - diagnosis | Receptor, Platelet-Derived Growth Factor beta - antagonists & inhibitors | Receptor, Platelet-Derived Growth Factor alpha - antagonists & inhibitors | Janus Kinase 2 - metabolism | Eosinophilia - genetics | Hypereosinophilic Syndrome - pathology | Hypereosinophilic Syndrome - therapy | Eosinophilia - diagnosis | Janus Kinase 2 - antagonists & inhibitors | Leukemia - genetics | Receptor, Platelet-Derived Growth Factor beta - metabolism | Myeloproliferative Disorders - diagnosis | Proto-Oncogene Proteins c-abl - genetics | Receptor, Platelet-Derived Growth Factor alpha - metabolism | Myeloproliferative Disorders - therapy | Janus Kinase 2 - genetics | Hematopoietic Stem Cell Transplantation | Leukemia - therapy | Receptor, Fibroblast Growth Factor, Type 1 - antagonists & inhibitors | Oncogene Proteins, Fusion - genetics | Protein Kinase Inhibitors - therapeutic use | Receptor, Platelet-Derived Growth Factor alpha - genetics | Leukemia - diagnosis | Proto-Oncogene Proteins c-abl - metabolism | Hypereosinophilic Syndrome - genetics | Oncogene Proteins, Fusion - antagonists & inhibitors | Index Medicus | Abridged Index Medicus
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Loading RightsNature (London), ISSN 0028-0836, 05/2017, Volume 545, Issue 7655, pp. 500 - 504
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Transaminases - biosynthesis | Cell Proliferation | Transaminases - genetics | Amino Acids, Branched-Chain - metabolism | Humans | Mice, Inbred C57BL | Male | Disease Progression | Animals | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - pathology | Transaminases - metabolism | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - metabolism | Female | Cell Differentiation | Mice | Enzyme Activation | Blast Crisis | Transaminases - deficiency | RNA-Binding Proteins - metabolism | Nonlymphoid leukemia | Myelocytic leukemia | Drug therapy | Branched chain amino acids | Health aspects | Index Medicus
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Loading RightsJournal of leukocyte biology, ISSN 0741-5400, 07/2014, Volume 96, Issue 1, pp. 83 - 91
Hox | leukemic stem cell | gene‐expression profiling | Leukemic stem cell | Gene-expression profiling | Immunology | Life Sciences & Biomedicine | Hematology | Science & Technology | Cell Biology | Homeodomain Proteins - metabolism | Humans | Leukemia, Myeloid, Acute - metabolism | Neoplasm Proteins - metabolism | Gene Expression Regulation, Leukemic - genetics | Neoplasms, Experimental - pathology | Cell Transformation, Neoplastic - genetics | Neoplasms, Experimental - genetics | Neoplasm Proteins - genetics | Wnt Signaling Pathway | Interleukin-3 - metabolism | Transcription Factor 4 | Signal Transduction | Leukemia, Myeloid, Acute - pathology | Transcription Factors - genetics | Interleukin-3 - genetics | Cell Transformation, Neoplastic - metabolism | beta Catenin - metabolism | Homeodomain Proteins - genetics | beta Catenin - genetics | Transcription Factors - metabolism | Animals | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - genetics | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - metabolism | Mice | Neoplasms, Experimental - metabolism | Cell Transformation, Neoplastic - pathology | Cell Line, Transformed | Leukemia, Myeloid, Acute - genetics | Index Medicus
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Tumor refractoriness to anti- VEGF treatment is mediated by CD11b + Gr1 + myeloid cells
Nature biotechnology, ISSN 1087-0156, 08/2007, Volume 25, Issue 8, pp. 911 - 920
Life Sciences & Biomedicine | Biotechnology & Applied Microbiology | Science & Technology | Fundamental and applied biological sciences. Psychology | Biotechnology | Miscellaneous | Health. Pharmaceutical industry | Immunotherapy | Industrial applications and implications. Economical aspects | Pharmacology. Drug treatments | Biological and medical sciences | Medical sciences | Antineoplastic agents | Mice, Inbred C57BL | Receptors, Chemokine - metabolism | Drug Resistance, Neoplasm | Treatment Outcome | Antineoplastic Agents - administration & dosage | Vascular Endothelial Growth Factor A - antagonists & inhibitors | Leukemia, Myeloid - drug therapy | Animals | Leukemia, Myeloid - metabolism | Neovascularization, Pathologic - drug therapy | Cell Line, Tumor | Mice | Neovascularization, Pathologic - metabolism | CD11b Antigen - metabolism | Cellular biology | Gene expression | Rodents | Tumors | Cancer | Index Medicus
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TGF-Β-FOXO signalling maintains leukaemia-initiating cells in chronic myeloid leukaemia
Nature (London), ISSN 0028-0836, 02/2010, Volume 463, Issue 7281, pp. 676 - 680
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Hematologic and hematopoietic diseases | Leukemias. Malignant lymphomas. Malignant reticulosis. Myelofibrosis | Biological and medical sciences | Medical sciences | Phosphorylation | Neoplastic Stem Cells - drug effects | Humans | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - drug therapy | Antineoplastic Agents - therapeutic use | Cell Nucleus - metabolism | Neoplastic Stem Cells - metabolism | Forkhead Transcription Factors - metabolism | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - pathology | Neoplastic Stem Cells - pathology | Transforming Growth Factor beta - antagonists & inhibitors | Cell Differentiation | Proto-Oncogene Proteins c-akt - metabolism | Forkhead Transcription Factors - deficiency | Disease Models, Animal | Tumor Stem Cell Assay | Mice, Inbred C57BL | Piperazines - therapeutic use | Forkhead Transcription Factors - genetics | Imatinib Mesylate | Protein Transport | Animals | Signal Transduction - drug effects | Protein Kinase Inhibitors - therapeutic use | Pyrimidines - therapeutic use | Cell Line, Tumor | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - metabolism | Mice | Benzamides | Enzyme Activation | Forkhead Box Protein O3 | Transforming Growth Factor beta - metabolism | Apoptosis | Protein-Tyrosine Kinases - antagonists & inhibitors | Index Medicus
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Loading RightsBlood, ISSN 0006-4971, 03/2010, Volume 115, Issue 10, pp. 1976 - 1984
Life Sciences & Biomedicine | Hematology | Science & Technology | Hematologic and hematopoietic diseases | Leukemias. Malignant lymphomas. Malignant reticulosis. Myelofibrosis | Biological and medical sciences | Medical sciences | Immunotherapy, Adoptive - methods | Antigens, CD34 - metabolism | Erythroid Precursor Cells - transplantation | Leukemia, Myeloid, Acute - pathology | Humans | Leukemia, Myeloid, Acute - metabolism | Mutant Proteins - metabolism | Nuclear Proteins - metabolism | Cell Separation - methods | Mice, SCID | Mice, Knockout | Xenograft Model Antitumor Assays | Erythroid Precursor Cells - pathology | Phenotype | Animals | Neoplastic Stem Cells - metabolism | Erythroid Precursor Cells - metabolism | Neoplastic Stem Cells - pathology | ADP-ribosyl Cyclase 1 - metabolism | Mice, Inbred NOD | Mice | Nuclear Proteins - genetics | Leukemia, Myeloid, Acute - therapy | Leukemia, Myeloid, Acute - genetics | Index Medicus | Abridged Index Medicus | Myeloid Neoplasia
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Loading RightsNature, ISSN 0028-0836, 08/2010, Volume 466, Issue 7307, pp. 765 - 768
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Hematologic and hematopoietic diseases | Leukemias. Malignant lymphomas. Malignant reticulosis. Myelofibrosis | Biological and medical sciences | Medical sciences | RNA-Binding Proteins - genetics | Up-Regulation | Oncogene Proteins, Fusion - metabolism | Prognosis | Homeodomain Proteins - metabolism | Humans | Gene Expression Regulation, Neoplastic | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - genetics | Nuclear Pore Complex Proteins - genetics | Cell Differentiation - genetics | RNA-Binding Proteins - biosynthesis | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - pathology | Nerve Tissue Proteins - biosynthesis | Membrane Proteins - metabolism | Blast Crisis - pathology | Signal Transduction | Membrane Proteins - genetics | Nuclear Pore Complex Proteins - metabolism | Mice, Inbred C57BL | Tumor Suppressor Protein p53 - metabolism | Receptor, Notch1 - metabolism | Nerve Tissue Proteins - genetics | Disease Progression | Homeodomain Proteins - genetics | Nerve Tissue Proteins - metabolism | Fusion Proteins, bcr-abl - genetics | Membrane Proteins - biosynthesis | Animals | Oncogene Proteins, Fusion - genetics | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - metabolism | Blast Crisis - genetics | Mice | Blast Crisis - metabolism | Fusion Proteins, bcr-abl - metabolism | RNA-Binding Proteins - metabolism | Myelocytic leukemia | Molecular genetics | Physiological aspects | Development and progression | Nonlymphoid leukemia | Cellular signal transduction | Genetic aspects | Research | Genetic regulation | Gene expression | Medical research | Stem cells | Chronic illnesses | Index Medicus
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Loading RightsThe Journal of biological chemistry, ISSN 0021-9258, 07/2020, Volume 295, Issue 27, pp. 8887 - 8900
cell differentiation | gene transcription | histone acetyltransferase | chromatin accessibility | acute myeloid leukemia (AML) | all-trans-retinoic acid (ATRA) | epigenetics | chromatin immunoprecipitation (ChIP) | CBFA2/RUNX1 partner transcriptional co-repressor 3 (CBFA2T3) | transcriptional corepressor | transcription corepressor | chromatin regulation | nuclear receptor | cancer biology | histone acetylation | Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | Retinoic Acid Receptor alpha - genetics | Gene Expression - drug effects | Gene Expression - genetics | Humans | Myeloid Cells - physiology | Gene Expression Regulation, Leukemic - drug effects | Repressor Proteins - genetics | Receptors, Retinoic Acid - metabolism | Co-Repressor Proteins - genetics | Receptors, Retinoic Acid - genetics | Gene Expression Regulation, Leukemic - genetics | Cell Differentiation - genetics | Tretinoin - metabolism | Tumor Suppressor Proteins - genetics | Myeloid Cells - metabolism | Cell Differentiation - physiology | Repressor Proteins - metabolism | Leukemia, Myeloid, Acute - genetics | Tretinoin - pharmacology | Index Medicus | Editors' Picks | RUNX1 partner transcriptional co-repressor 3 (CBFA2T3) | CBFA2
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