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The New England Journal of Medicine, ISSN 0028-4793, 09/2009, Volume 361, Issue 13, pp. 1249 - 1259
Patients with AML who were between 17 and 60 years of age were randomly assigned to receive induction therapy with the standard dose of daunorubicin or twice... 
CYTOSINE-ARABINOSIDE | MEDICINE, GENERAL & INTERNAL | ONCOLOGY-GROUP | INDUCTION CHEMOTHERAPY | ACUTE NONLYMPHOCYTIC LEUKEMIA | ACUTE MYELOGENOUS LEUKEMIA | ACUTE MYELOCYTIC-LEUKEMIA | GROUP-B | DAUNORUBICIN | REMISSION INDUCTION | UNTREATED ADULT PATIENTS | Antineoplastic Combined Chemotherapy Protocols - administration & dosage | Age Factors | Humans | Middle Aged | Antibiotics, Antineoplastic - adverse effects | Antineoplastic Combined Chemotherapy Protocols - adverse effects | Male | Leukemia, Myelomonocytic, Acute - drug therapy | Stem Cell Transplantation | Young Adult | fms-Like Tyrosine Kinase 3 - genetics | Leukemia, Myelomonocytic, Acute - therapy | Adult | Female | Remission Induction - methods | Daunorubicin - administration & dosage | Leukemia, Myelomonocytic, Acute - genetics | Histone-Lysine N-Methyltransferase | Risk Factors | Kaplan-Meier Estimate | Proportional Hazards Models | Combined Modality Therapy | Cytarabine - administration & dosage | Daunorubicin - adverse effects | Antibiotics, Antineoplastic - administration & dosage | Myeloid-Lymphoid Leukemia Protein - genetics | Adolescent | Infusions, Intravenous | Mutation | Leukemia, Myelomonocytic, Acute - mortality | Anthracyclines | Usage | Daunorubicin | Young adults | Health aspects | Bone marrow | Chemotherapy | Consolidation | Kinases | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 2014, Volume 211, Issue 6, pp. 1093 - 1108
Leukemic stem cells (LSCs) isolated from acute myeloid leukemia (AML) patients are more sensitive to nuclear factor. B (NF-kappa B) inhibition-induced cell... 
MEDICINE, RESEARCH & EXPERIMENTAL | TUMOR-NECROSIS-FACTOR | STEM-CELLS | ACTIVATION | BONE-MARROW | CHEMICAL HEPATOCARCINOGENESIS | NECROTIC CELL-DEATH | JUN NH2-TERMINAL KINASE | IMMUNOLOGY | CANCER | MYELODYSPLASTIC SYNDROMES | FACTOR-ALPHA | Tumor Necrosis Factor-alpha - metabolism | Leukemia, Myelomonocytic, Acute - metabolism | Nitriles - pharmacology | Transcription Factor AP-1 - genetics | Humans | Leukemia, Myeloid, Acute - metabolism | Tumor Necrosis Factor-alpha - genetics | Cell Survival - genetics | JNK Mitogen-Activated Protein Kinases - metabolism | NF-kappa B - metabolism | Sulfones - pharmacology | Transcription Factor AP-1 - metabolism | Leukemia, Promyelocytic, Acute - metabolism | U937 Cells | JNK Mitogen-Activated Protein Kinases - genetics | Receptors, Tumor Necrosis Factor - genetics | Receptors, Tumor Necrosis Factor - metabolism | Cell Survival - drug effects | Leukemia, Myelomonocytic, Acute - genetics | NF-kappa B - antagonists & inhibitors | JNK Mitogen-Activated Protein Kinases - antagonists & inhibitors | Leukemia, Monocytic, Acute - metabolism | Leukemia, Myeloid, Acute - pathology | Cells, Cultured | Gene Expression Regulation, Leukemic - drug effects | Signal Transduction - genetics | Leukemia, Monocytic, Acute - genetics | Reverse Transcriptase Polymerase Chain Reaction | Anthracenes - pharmacology | Blotting, Western | Mice, Knockout | Tumor Necrosis Factor-alpha - pharmacology | Animals | NF-kappa B - genetics | Signal Transduction - drug effects | K562 Cells | Cell Line, Tumor | HL-60 Cells | Mice | Leukemia, Promyelocytic, Acute - genetics | Leukemia, Myeloid, Acute - genetics | Index Medicus
Journal Article
Blood, ISSN 0006-4971, 10/2003, Volume 102, Issue 7, pp. 2395 - 2402
Journal Article
Oncogene, ISSN 0950-9232, 04/2016, Volume 35, Issue 15, pp. 1965 - 1976
The ETS transcription factor ERG has been implicated as a major regulator of both normal and aberrant hematopoiesis. In acute myeloid leukemias harboring t(16;... 
BIOCHEMISTRY & MOLECULAR BIOLOGY | SELF-RENEWAL | RECEPTOR-ALPHA | CELL BIOLOGY | PML-RAR-ALPHA | ONCOLOGY | PROSTATE-CANCER | GENETICS & HEREDITY | GENOME-WIDE ANALYSIS | CD38 ANTIGEN | BINDING-SITES | TRANSCRIPTION FACTOR | HISTONE DEACETYLASE | ACUTE PROMYELOCYTIC LEUKEMIA | RNA, Small Interfering - genetics | Translocation, Genetic | RNA-Binding Protein FUS - antagonists & inhibitors | Humans | Multiprotein Complexes | Neoplasm Proteins - physiology | Hematopoietic Stem Cells - pathology | Chromosomes, Human, Pair 21 - ultrastructure | Oncogene Proteins, Fusion - physiology | RNA Interference | U937 Cells | Neoplastic Stem Cells - pathology | Hematopoiesis - physiology | Neoplasm Proteins - genetics | Chromosomes, Human, Pair 16 - ultrastructure | Dimerization | Retinoid X Receptors - metabolism | Gene Expression Regulation, Neoplastic - genetics | Tretinoin - physiology | Chromosomes, Human, Pair 16 - genetics | Tretinoin - pharmacology | Proto-Oncogene Proteins - metabolism | Promoter Regions, Genetic | Leukemia, Myelomonocytic, Acute - genetics | Leukemia, Myelomonocytic, Acute - physiopathology | Leukemia, Myeloid, Acute - pathology | RNA-Binding Protein FUS - genetics | Receptors, Retinoic Acid - metabolism | Chromosomes, Human, Pair 21 - genetics | Amino Acid Motifs | Protein Interaction Mapping | Transcription Factors - metabolism | Leukemia, Myelomonocytic, Acute - pathology | Enhancer Elements, Genetic | Signal Transduction - drug effects | Oncogene Proteins, Fusion - genetics | Cell Line, Tumor | Protein Binding | RNA-Binding Protein FUS - physiology | Oncogene Proteins, Fusion - antagonists & inhibitors | Signal Transduction - physiology | Trans-Activators - metabolism | Retinoic Acid Receptor alpha | Leukemia, Myeloid, Acute - physiopathology | Leukemia, Myeloid, Acute - genetics | Cellular signal transduction | Genetic aspects | Properties | Gene expression | Health aspects | Oncogenes | Proteins | Genotype & phenotype | Oncology | Leukemia | Index Medicus | Original
Journal Article
Journal Article
Leukemia, ISSN 0887-6924, 2009, Volume 23, Issue 1, pp. 85 - 94
Journal Article
15.