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Blood, ISSN 0006-4971, 02/2013, Volume 121, Issue 6, pp. 1008 - 1015
The majority of peripheral serotonin is stored in platelets, which secrete it on activation. Serotonin releases Weibel-Palade bodies (WPBs) and we asked... 
PULMONARY-HYPERTENSION | PERMEABILITY | 5-HYDROXYTRYPTAMINE | ENDOTHELIAL-CELLS | MAST-CELLS | P-SELECTIN | RECEPTOR | SMOOTH-MUSCLE-CELLS | HEMATOLOGY | VON-WILLEBRAND-FACTOR | SELECTIN-DEFICIENT MICE | Blood Platelets - immunology | Endothelium, Vascular - drug effects | Male | Shock, Septic - immunology | Lipopolysaccharides | Serotonin - blood | Inflammation - metabolism | L-Selectin - immunology | Leukocytosis - metabolism | Weibel-Palade Bodies - metabolism | Chemotaxis - immunology | Neutrophils - metabolism | Serotonin - immunology | Serotonin Uptake Inhibitors - immunology | Endothelium, Vascular - immunology | Neutrophil Infiltration - immunology | Fluoxetine - pharmacology | Acute Disease | Neutrophils - drug effects | Leukocytosis - immunology | Chemotaxis - drug effects | Mice, Knockout | Blood Platelets - metabolism | Endothelium, Vascular - metabolism | Mice | Serotonin Uptake Inhibitors - pharmacology | Leukocyte Rolling - drug effects | Weibel-Palade Bodies - immunology | Histamine - immunology | L-Selectin - metabolism | Flow Cytometry | Tryptophan Hydroxylase - deficiency | Blood Platelets - drug effects | Fluoxetine - immunology | Shock, Septic - genetics | Leukocyte Rolling - genetics | Histamine - pharmacology | Shock, Septic - chemically induced | Mice, Inbred C57BL | Kaplan-Meier Estimate | Neutrophils - immunology | Inflammation - immunology | Leukocyte Rolling - immunology | Tryptophan Hydroxylase - genetics | Weibel-Palade Bodies - drug effects | Animals | Serotonin - metabolism | Inflammation - genetics | Leukocytosis - genetics | Phagocytes, Granulocytes, and Myelopoiesis | Platelets and Thrombopoiesis
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 10/2007, Volume 293, Issue 4, pp. 2210 - 2218
Targeting cannabinoid-2 (CB2) receptors with selective agonists may represent a novel therapeutic avenue in various inflammatory diseases, but the mechanisms... 
Adhesion molecules | Inflammation | RhoA | Endothelial activation | C-REACTIVE PROTEIN | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | MOLECULE EXPRESSION | ATHEROSCLEROSIS | NECROSIS-FACTOR-ALPHA | KNOCKOUT MICE | endothelial activation | CANNABINOID RECEPTORS | inflammation | adhesion molecules | NITRIC-OXIDE | THERAPEUTIC TARGETS | PERIPHERAL VASCULAR DISEASE | POTENTIAL ROLE | CB2 RECEPTOR ACTIVATION | Inflammation - chemically induced | Leukocyte Rolling - drug effects | Tumor Necrosis Factor-alpha - metabolism | Receptor, Cannabinoid, CB2 - agonists | Humans | Male | Monocytes - metabolism | NF-kappa B - metabolism | rhoA GTP-Binding Protein - metabolism | Aorta - metabolism | RNA, Messenger - metabolism | Receptor, Cannabinoid, CB2 - genetics | Coronary Vessels - metabolism | Lipopolysaccharides | Dose-Response Relationship, Drug | Inflammation - metabolism | Anti-Inflammatory Agents - therapeutic use | Chemokine CCL2 - metabolism | Disease Models, Animal | Coronary Vessels - drug effects | Anti-Inflammatory Agents - pharmacology | Endothelial Cells - metabolism | Aorta - drug effects | Mice, Inbred C57BL | Cells, Cultured | Cannabinoids - pharmacology | Monocytes - drug effects | Receptor, Cannabinoid, CB1 - metabolism | Receptor, Cannabinoid, CB2 - metabolism | Cannabinoids - therapeutic use | Intercellular Adhesion Molecule-1 - metabolism | Animals | Signal Transduction - drug effects | Inflammation - prevention & control | Mice | Vascular Cell Adhesion Molecule-1 - metabolism | Endothelial Cells - drug effects
Journal Article
Blood, ISSN 0006-4971, 03/2013, Volume 121, Issue 12, pp. 2301 - 2310
Integrin activation is essential for the function of leukocytes. Impaired integrin activation on leukocytes is the hallmark of the leukocyte adhesion... 
ISCHEMIA-REPERFUSION INJURY | GAMMA | EMIGRATION | ADHESION | AFFINITY | INFLAMMATION | ACUTE-RENAL-FAILURE | CONFORMATION | HEMATOLOGY | BETA INTEGRINS | NEUTROPHIL RECRUITMENT | Leukocyte Rolling - drug effects | Guanine Nucleotide Exchange Factors - physiology | Blood Vessels - metabolism | Blood Vessels - pathology | Humans | E-Selectin - pharmacology | Lymphocyte Function-Associated Antigen-1 - metabolism | Acute Kidney Injury - genetics | Leukocytes - physiology | Integrins - metabolism | Blood Vessels - immunology | Chemotaxis, Leukocyte - genetics | Chemotaxis, Leukocyte - drug effects | Guanine Nucleotide Exchange Factors - metabolism | Intercellular Adhesion Molecule-1 - pharmacology | Macrophage-1 Antigen - metabolism | Reperfusion Injury - complications | Macrophage-1 Antigen - physiology | Reperfusion Injury - genetics | Acute Kidney Injury - etiology | Neutrophil Infiltration - drug effects | Lymphocyte Function-Associated Antigen-1 - physiology | Guanine Nucleotide Exchange Factors - genetics | Leukocyte Rolling - genetics | Reperfusion Injury - pathology | Acute Kidney Injury - pathology | Mice, Inbred C57BL | Cells, Cultured | Mice, Transgenic | Neutrophil Infiltration - genetics | Animals | Lymphocyte Function-Associated Antigen-1 - genetics | Macrophage-1 Antigen - genetics | HL-60 Cells | Leukocytes - drug effects | Mice | Leukocytes - metabolism
Journal Article
Journal Article
BLOOD, ISSN 0006-4971, 09/2010, Volume 116, Issue 11, pp. 1924 - 1931
Journal Article
Journal Article
Journal Article
British Journal of Pharmacology, ISSN 0007-1188, 10/2006, Volume 149, Issue 4, pp. 345 - 354
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2014, Volume 9, Issue 2, p. e88316
Objective: Activated platelets release serotonin at sites of inflammation where it acts as inflammatory mediator and enhances recruitment of neutrophils.... 
STIMULATION | INTERFERON-GAMMA | INFLAMMATION | PLATELETS | MULTIDISCIPLINARY SCIENCES | IN-VIVO | NECROSIS-FACTOR-ALPHA | SMOOTH-MUSCLE-CELLS | SEROTONIN | RELEASE | VON-WILLEBRAND-FACTOR | Endothelium, Vascular - cytology | Leukocyte Rolling - drug effects | Fluoxetine - pharmacology | Endothelial Cells - metabolism | Mice, Inbred C57BL | E-Selectin - metabolism | Peritonitis - pathology | Serotonin - blood | Animals | Endothelial Cells - cytology | Tryptophan Hydroxylase - deficiency | Cell Communication - drug effects | Lipopolysaccharides - pharmacology | Peritonitis - blood | Leukocytes - drug effects | Cell Membrane - metabolism | Mice | Fluoxetine - therapeutic use | Peritonitis - drug therapy | Leukocytes - metabolism | Cell Membrane - drug effects | Tryptophan Hydroxylase - metabolism | Endothelial Cells - drug effects | Leukocytes - cytology | Tryptophan | Fluoxetine | Mitogens | Analysis | Enzyme-linked immunosorbent assay | Endothelium | Heart | Plasma | Flow cytometry | Adhesion tests | Smooth muscle | Serotonin uptake inhibitors | Stimulation | Leukocytes | Blood | Recruitment | Blood platelets | Antidepressants | Rodents | Extravasation | Tumor necrosis factor-TNF | Pretreatment | Cardiology | Acute effects | Peritonitis | Medical research | Serotonin | Neutrophils | Leukocytes (neutrophilic) | Inflammation | Mesentery | Thrombosis | Velocity | Adhesion | Endothelial cells | White blood cells | E-selectin | Cytometry | Microscopy | Platelets | Cell migration | Leukocyte rolling
Journal Article
Journal Article
Circulation, ISSN 0009-7322, 07/2014, Volume 130, Issue 1, pp. 35 - 50
Background-The relevance of the dissociation of circulating pentameric C-reactive protein (pCRP) to its monomeric subunits (mCRP) is poorly understood. We... 
Myocardial infarction | Microcirculation | Inflammation | C-reactive protein | Atherosclerosis | atherosclerosis | CRP | ANTIBODIES | CARDIAC & CARDIOVASCULAR SYSTEMS | COMPLEMENT ACTIVATION | myocardial infarction | PHOSPHOLIPASE A | RELEASE | MICROPARTICLES | microcirculation | REDUCTION | inflammation | LYSOPHOSPHATIDYLCHOLINE | APOPTOTIC CELLS | PERIPHERAL VASCULAR DISEASE | EXPRESSION | Chemotaxis, Leukocyte | Leukocyte Rolling - drug effects | Complement Activation | Rats, Wistar | Humans | Male | Muscle, Skeletal - metabolism | Myositis - chemically induced | Phosphorylcholine - analogs & derivatives | Biopolymers | Phospholipase A2 Inhibitors - pharmacology | Inflammation - metabolism | Hexanes - pharmacology | Inflammation - drug therapy | Myocardial Infarction - pathology | Phosphorylcholine - pharmacology | Protein Structure, Quaternary | Anti-Inflammatory Agents - therapeutic use | Carrier Proteins - chemistry | Cell Membrane - metabolism | Myositis - pathology | Reperfusion Injury - metabolism | Cell Membrane - drug effects | Muscle, Skeletal - blood supply | Carrier Proteins - physiology | Hexanes - therapeutic use | Phospholipase A2 Inhibitors - therapeutic use | Lipopolysaccharides - toxicity | Receptors, IgG - physiology | Reperfusion Injury - pathology | Anti-Inflammatory Agents - pharmacology | Endothelial Cells - metabolism | Myositis - metabolism | Lysophosphatidylcholines - metabolism | Rats | Phospholipases A2 - metabolism | Myocardial Infarction - metabolism | Random Allocation | C-Reactive Protein - chemistry | C-Reactive Protein - physiology | Cell Adhesion - drug effects | Inflammation - etiology | Animals | Membrane Lipids - metabolism | Endothelial Cells - drug effects | Phosphorylcholine - therapeutic use | Care and treatment | Usage | Anti-inflammatory drugs | Physiological aspects | Development and progression | Research | Health aspects
Journal Article
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 05/2012, Volume 16, Issue 5, pp. 1094 - 1105
Journal Article