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1. Full Text Lrrk2 and Lewy body disease
by Ross, Owen A and Toft, Mathias and Whittle, Andrew J and Johnson, Joseph L and Papapetropoulos, Spiridon and Mash, Deborah C and Litvan, Irene and Gordon, Mark F and Wszolek, Zbigniew K and Farrer, Matthew J and Dickson, Dennis W
Annals of Neurology, ISSN 0364-5134, 02/2006, Volume 59, Issue 2, pp. 388 - 393
Objective: The Lrrk2 kinase domain G2019S substitution is the most common genetic basis of familial and sporadic parkinsonism. Patients harboring the G2019S... 
TARGET | DEMENTIA | AUTOSOMAL-DOMINANT PARKINSONISM | GENE | MUTATION | BODIES | ALPHA-SYNUCLEIN | PATHOLOGY | NEUROSCIENCES | CLINICAL NEUROLOGY | Glycine - genetics | Humans | Brain Stem - metabolism | Protein-Serine-Threonine Kinases - genetics | Models, Molecular | Male | DNA Mutational Analysis - methods | Leucine-Rich Repeat Serine-Threonine Protein Kinase-2 | Proteins - genetics | Lewy Body Disease - genetics | Brain Stem - pathology | Aged, 80 and over | Female | Aged | Mutation | Index Medicus
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2. Full Text Neuropathology of prodromal Lewy body disease
by Iranzo, Alex and Gelpi, Ellen and Tolosa, Eduard and Molinuevo, José Luis and Serradell, Mónica and Gaig, Carles and Santamaria, Joan
Movement Disorders, ISSN 0885-3185, 03/2014, Volume 29, Issue 3, pp. 410 - 415
BackgroundCurrent evidence suggests that there is a prodromal stage in Parkinson disease characterized by a variety of nonmotor symptoms. Methods and ResultsA... 
dementia with Lewy bodies | prodromal stage | Lewy body disease | incidental Lewy body disease | Parkinson disease | neuropathology | rapid eye movement sleep behavior disorder | Incidental Lewy body disease | Neuropathology | Rapid eye movement sleep behavior disorder | Prodromal stage | Dementia with Lewy bodies | CRITERIA | DIAGNOSIS | DEMENTIA | SYMPTOMS | PATHOLOGY | MILD COGNITIVE IMPAIRMENT | CLINICAL NEUROLOGY | SLEEP BEHAVIOR DISORDER | BODIES | COHORT | IDIOPATHIC PARKINSONS-DISEASE | Depression - diagnosis | Parkinson Disease - pathology | REM Sleep Behavior Disorder - etiology | Follow-Up Studies | Humans | Lewy Body Disease - complications | Lewy Body Disease - pathology | Aged, 80 and over | Depression - complications | Male | Aged | REM Sleep Behavior Disorder - pathology | Sleep | Parkinson's disease | Depression, Mental | Index Medicus
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3. Full Text Passive immunization reduces behavioral and neuropathological deficits in an alpha-synuclein transgenic model of lewy body disease
by Masliah, Eliezer and Rockenstein, Edward and Mante, Michael and Crews, Leslie and Spencer, Brian and Adame, Anthony and Patrick, Christina and Trejo, Margarita and Ubhi, Kiren and Rohn, Troy T and Mueller-Steiner, Sarah and Seubert, Peter and Barbour, Robin and McConlogue, Lisa and Buttini, Manuel and Games, Dora and Schenk, Dale
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 4, pp. e19338 - e19338
Dementia with Lewy bodies (DLB) and Parkinson's Disease (PD) are common causes of motor and cognitive deficits and are associated with the abnormal... 
IN-VITRO | A-BETA COMPONENT | PROTEIN | IMMUNOTHERAPY | ALZHEIMERS-DISEASE | MOUSE MODEL | BIOLOGY | CEREBROSPINAL-FLUID | AGGREGATION | PARKINSONS-DISEASE | BILAYER CHARGE | Immunization, Passive - methods | Lewy Body Disease - immunology | Rats | Mice, Transgenic | Lewy Body Disease - therapy | Nerve Degeneration - pathology | Behavior, Animal | Immunohistochemistry - methods | Lewy Body Disease - genetics | Animals | Lysosomes - metabolism | Maze Learning | Cell Line, Tumor | Mice | alpha-Synuclein - genetics | Antibodies, Monoclonal - metabolism | Disease Models, Animal | Monoclonal antibodies | Calpain | Immunization | Genetic engineering | Research | Gene therapy | Huntingtons disease | Brain | Cell culture | Neurosciences | Parkinson's disease | Neuropathology | Pathogenesis | Central nervous system | Parkinsons disease | Cognitive ability | Transgenic | Synuclein | Autophagy | Accumulation | Proteins | Neurodegeneration | Rodents | Immunotherapy | Dementia disorders | Movement disorders | Immunoglobulins | Neurodegenerative diseases | Lewy body disease | C-Terminus | Lewy bodies | Studies | Disease prevention | Axons | Pathology | Insects | In vivo methods and tests | Immunization (passive) | Alzheimers disease | Synapses | Pharmaceuticals | Index Medicus
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4. Full Text Imaging amyloid deposition in lewy body diseases
by Gomperts, S.N and Rentz, D.M and Moran, E and Becker, J.A and Locascio, J.J and Klunk, W.E and Mathis, C.A and Elmaleh, D.R and Shoup, T and Fischman, A.J and Hyman, B.T and Growdon, J.H and Johnson, K.A
Neurology, ISSN 0028-3878, 09/2008, Volume 71, Issue 12, pp. 903 - 910
Background: Extrapyramidal motor symptoms precede dementia in Parkinson disease (PDD) by many years, whereas dementia occurs early in dementia with Lewy bodies... 
DIAGNOSIS | POSITRON-EMISSION-TOMOGRAPHY | DEMENTIA | PRECLINICAL ALZHEIMERS-DISEASE | COGNITIVE IMPAIRMENT | BODIES | PATHOLOGY | PITTSBURGH COMPOUND-B | CEREBRAL-CORTEX | CLINICAL NEUROLOGY | PARKINSONS-DISEASE | Dementia - diagnostic imaging | Brain - diagnostic imaging | Diagnosis, Differential | Movement | Aniline Compounds | Humans | Middle Aged | Dementia - diagnosis | Male | Cognition | Lewy Body Disease - psychology | Positron-Emission Tomography | Parkinson Disease - physiopathology | Parkinson Disease - psychology | Brain - metabolism | Neuropsychological Tests | Tissue Distribution | Dementia - etiology | Dementia - metabolism | Amyloid beta-Peptides - metabolism | Thiazoles | Female | Aged | Lewy Body Disease - physiopathology | Index Medicus | Abridged Index Medicus
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5. Full Text Neuropathologically mixed Alzheimer's and Lewy body disease: burden of pathological protein aggregates differs between clinical phenotypes
by Walker, L and McAleese, KE and Thomas, AJ and Johnson, M and Martin-Ruiz, C and Parker, C and Colloby, SJ and Jellinger, K and Attems, J
ACTA NEUROPATHOLOGICA, ISSN 0001-6322, 05/2015, Volume 129, Issue 5, pp. 729 - 748
Multiple different pathological protein aggregates are frequently seen in human postmortem brains and hence mixed pathology is common. Mixed dementia on the... 
Mixed dementia | Lewy body disease | NEUROFIBRILLARY PATHOLOGY | ASSOCIATION GUIDELINES | ALPHA-SYNUCLEIN | PATHOLOGY | APOLIPOPROTEIN-E GENOTYPE | NEUROSCIENCES | CLINICAL NEUROLOGY | SUBCORTICAL TAU PATHOLOGY | NATIONAL INSTITUTE | BRAINNET EUROPE CONSORTIUM | AMYLOID ANGIOPATHY | DIAGNOSTIC GUIDELINES | Quantitative neuropathology | Alzheimer's disease | PARKINSONS-DISEASE | Protein Aggregates | Microtubule-Associated Proteins - metabolism | Humans | tau Proteins - metabolism | Dementia - physiopathology | Male | Alzheimer Disease - pathology | Brain - metabolism | Dementia - metabolism | Amyloid beta-Peptides - metabolism | Aged, 80 and over | Female | Parkinson Disease - metabolism | Parkinson Disease - complications | Alzheimer Disease - physiopathology | Parkinson Disease - pathology | Dementia - pathology | Comorbidity | Lewy Body Disease - pathology | Parkinson Disease - physiopathology | Lewy Body Disease - metabolism | Phenotype | Alzheimer Disease - metabolism | Brain - pathology | Aged | alpha-Synuclein - metabolism | Lewy Body Disease - physiopathology | Brain | Genetic aspects | Diagnosis | Index Medicus
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6. Full Text Parkinson disease and incidental Lewy body disease: Just a question of time?
by Iacono, Diego and Geraci-Erck, Maria and Rabin, Marcie L and Adler, Charles H and Serrano, Geidy and Beach, Thomas G and Kurlan, Roger
Neurology, ISSN 0028-3878, 11/2015, Volume 85, Issue 19, pp. 1670 - 1679
OBJECTIVE:To quantify the loss of pigmented neurons in the substantia nigra (SN) of autopsy-confirmed Parkinson disease (PD) and incidental Lewy body disease... 
DEMENTIA | ALPHA-SYNUCLEIN PATHOLOGY | INVOLVEMENT | DISORDERS | BODIES | NEUROPATHOLOGY | SUBSTANTIA-NIGRA | BRAIN | CLINICAL NEUROLOGY | AUTONOMIC NERVOUS-SYSTEM | DEGENERATION | Neurons - pathology | Parkinson Disease - pathology | Substantia Nigra - pathology | Time Factors | Humans | Lewy Body Disease - pathology | Aged, 80 and over | Female | Male | Aged | Longitudinal Studies | Index Medicus | Abridged Index Medicus | 165
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7. Full Text Parkinson's Disease Dementia and Lewy Body Disease
by Sezgin, Mine and Bilgic, Basar and Tinaz, Sule and Emre, Murat
Seminars in Neurology, ISSN 0271-8235, 04/2019, Volume 39, Issue 2, pp. 274 - 282
Abstract Dementia with Lewy bodies (DLB) and Parkinson's disease dementia (PD-D) are Lewy body-related neurodegenerative disorders sharing common clinical and... 
Review Article | dementia with Lewy bodies | synucleinopathy | imaging | Parkinson's disease dementia | AUTONOMIC DYSFUNCTION | ALZHEIMERS-DISEASE | TEMPORAL-LOBE ATROPHY | MILD COGNITIVE IMPAIRMENT | GLUCOCEREBROSIDASE MUTATIONS | MENTAL DYSFUNCTION | CLINICAL NEUROLOGY | SLEEP BEHAVIOR DISORDER | POSITRON-EMISSION-TOMOGRAPHY | FLUID ALPHA-SYNUCLEIN | DIAGNOSTIC-CRITERIA | Dementia - diagnostic imaging | Parkinson Disease - diagnostic imaging | Dementia - drug therapy | Humans | Dementia - physiopathology | Parkinson Disease - drug therapy | Parkinson Disease - physiopathology | Lewy Body Disease - diagnostic imaging | Parkinson Disease - cerebrospinal fluid | Dementia - cerebrospinal fluid | Lewy Body Disease - cerebrospinal fluid | Lewy Body Disease - drug therapy | Lewy Body Disease - physiopathology
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8. Full Text Selective molecular alterations in the autophagy pathway in patients with lewy body disease and in models of α-synucleinopathy
by Crews, Leslie and Spencer, Brian and Desplats, Paula and Patrick, Christina and Paulino, Amy and Rockenstein, Edward and Hansen, Lawrence and Adame, Anthony and Galasko, Douglas and Masliah, Eliezer
PLoS ONE, ISSN 1932-6203, 02/2010, Volume 5, Issue 2, pp. e9313 - e9313
Background: Lewy body disease is a heterogeneous group of neurodegenerative disorders characterized by alpha-synuclein accumulation that includes dementia with... 
WILD-TYPE | CHAPERONE-MEDIATED AUTOPHAGY | ALZHEIMERS-DISEASE | MOUSE MODEL | FRONTOTEMPORAL DEMENTIA | BIOLOGY | AGGREGATE-PRONE PROTEINS | NEURONAL EXPRESSION | PARKINSONS-DISEASE | TRANSGENIC MICE | GAUCHER-DISEASE | Pyramidal Cells - metabolism | Humans | Nerve Degeneration - genetics | Male | Autophagy | Nerve Degeneration - metabolism | Alzheimer Disease - pathology | Brain - metabolism | Lewy Body Disease - genetics | RNA Interference | Brain - ultrastructure | Aged, 80 and over | Female | alpha-Synuclein - genetics | Pyramidal Cells - ultrastructure | Disease Models, Animal | Cell Line | Ubiquitin-Activating Enzymes - genetics | Signal Transduction | Lewy Body Disease - pathology | Mice, Transgenic | Microscopy, Electron | Ubiquitin-Activating Enzymes - metabolism | Blotting, Western | Nerve Degeneration - pathology | Lewy Body Disease - metabolism | Microscopy, Confocal | Autophagy-Related Protein 7 | Animals | Alzheimer Disease - metabolism | Brain - pathology | Cell Line, Tumor | Pyramidal Cells - pathology | Aged | Mice | Alzheimer Disease - genetics | alpha-Synuclein - metabolism | TOR protein | Neurosciences | Animal models | Parkinson's disease | Pathogenesis | Toxicity | Parkinsons disease | Phagosomes | Nervous system | Synuclein | Neuromodulation | Accumulation | Modulators | Proteins | Infusion | Alterations | Neurodegeneration | Dementia disorders | Alzheimer's disease | Movement disorders | Enzymes | Neurodegenerative diseases | Neurons | Immunomodulation | Lewy body disease | Transgenic mice | Rapamycin | Patients | Lewy bodies | Studies | Pathology | Mutation | Alzheimers disease | Phagocytosis | Apoptosis | Dementia | Index Medicus
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9. Full Text Rapidly progressive diffuse Lewy body disease
by Gaig, Carles and Valldeoriola, Francesc and Gelpi, Ellen and Ezquerra, Mario and Llufriu, Sara and Buongiorno, Mariateresa and Rey, Maria Jesús and Martí, Maria Jose and Graus, Francesc and Tolosa, Eduardo
Movement Disorders, ISSN 0885-3185, 06/2011, Volume 26, Issue 7, pp. 1316 - 1323
Background: Lewy body syndromes (mainly Parkinson's disease and dementia with Lewy bodies) share many clinical features and usually have a slowly progressive... 
dementia with Lewy bodies | rapidly progressive dementia | diffuse Lewy body disease | Lewy body syndromes | Creutzfeldt‐Jakob disease | Rapidly progressive dementia | Creutzfeldt-Jakob disease | Dementia with Lewy bodies | Diffuse Lewy body disease | DIFFERENTIAL-DIAGNOSIS | DEMENTIA | MARKER | ALZHEIMERS-DISEASE | PATHOLOGY | VARIANT | NEURODEGENERATIVE DISEASES | CLINICAL NEUROLOGY | ACCURACY | BODIES | CREUTZFELDT-JAKOB-DISEASE | Delirium - pathology | Severity of Illness Index | Parkinsonian Disorders - physiopathology | Delirium - etiology | Humans | Lewy Body Disease - complications | Lewy Body Disease - pathology | Medical Records | Male | Hallucinations - physiopathology | Hallucinations - etiology | Parkinsonian Disorders - etiology | Disease Progression | Myoclonus - physiopathology | Myoclonus - pathology | Parkinsonian Disorders - pathology | Myoclonus - etiology | Fatal Outcome | Brain - pathology | Female | Hallucinations - pathology | Aged | Delirium - physiopathology | Lewy Body Disease - physiopathology | Index Medicus
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10. Full Text Tau Positron Emission Tomographic Imaging in the Lewy Body Diseases
by Gomperts, Stephen N and Locascio, Joseph J and Makaretz, Sara J and Schultz, Aaron and Caso, Christina and Vasdev, Neil and Sperling, Reisa and Growdon, John H and Dickerson, Bradford C and Johnson, Keith
JAMA Neurology, ISSN 2168-6149, 11/2016, Volume 73, Issue 11, pp. 1334 - 1341
IMPORTANCE: The causes of cognitive impairment in dementia with Lewy bodies (DLB) and Parkinson disease (PD) are multifactorial. Tau pathologic changes are... 
DEMENTIA | ALZHEIMERS-DISEASE | BODIES | ALPHA-SYNUCLEIN | PATHOLOGY | MILD COGNITIVE IMPAIRMENT | DIAGNOSTIC-CRITERIA | BURDEN | BRAIN | CLINICAL NEUROLOGY | PARKINSONS-DISEASE | Carbolines | Parkinson Disease - diagnostic imaging | Cross-Sectional Studies | Aniline Compounds | Cognitive Dysfunction - metabolism | Humans | Middle Aged | Neocortex - diagnostic imaging | tau Proteins - metabolism | Male | Positron-Emission Tomography - methods | Cognitive Dysfunction - physiopathology | Parkinson Disease - physiopathology | Lewy Body Disease - diagnostic imaging | Lewy Body Disease - metabolism | Magnetic Resonance Imaging | Neocortex - metabolism | Amyloid beta-Peptides - metabolism | Thiazoles | Female | Aged | Parkinson Disease - metabolism | Cognitive Dysfunction - diagnostic imaging | Lewy Body Disease - physiopathology | Index Medicus | Abridged Index Medicus
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11. Full Text Evidence that incidental Lewy body disease is pre-symptomatic Parkinson's disease
by Dickson, Dennis W and Fujishiro, Hiroshige and DelleDonne, Anthony and Menke, Joshua and Ahmed, Zeshan and Klos, Kevin J and Josephs, Keith A and Frigerio, Roberta and Burnett, Melinda and Parisi, Joseph R and Ahlskog, J. Eric
Acta Neuropathologica, ISSN 0001-6322, 04/2008, Volume 115, Issue 4, pp. 437 - 444
Lewy bodies, the histologic hallmark of Parkinson's disease (PD), are detected in the brains of about 10% of clinically normal people over the age of 60 years.... 
Heart | Immunohistochemistry | Striatum | Tyrosine hydroxylase | Parkinson's disease | Incidental Lewy bodies | OLFACTORY DYSFUNCTION | SCINTIGRAPHY | FP-CIT-SPECT | SYMPTOMATIC THRESHOLD | DOPAMINE TRANSPORTERS | PATHOLOGY | BRAIN PATHOLOGY | NEUROSCIENCES | heart | CLINICAL NEUROLOGY | tyrosine hydroxylase | SLEEP BEHAVIOR DISORDER | striatum | incidental Lewy bodies | ALPHA-SYNUCLEIN PATHOLOGY | immunohistochemistry | NEUROPATHOLOGY | AUTONOMIC NERVOUS-SYSTEM | Tyrosine 3-Monooxygenase - metabolism | Parkinson Disease - pathology | Humans | Lewy Body Disease - pathology | Male | Chi-Square Distribution | Lewy Body Disease - epidemiology | Lewy Body Disease - etiology | Incidence | Brain Stem - pathology | Myocardium - metabolism | Cell Death | Aged, 80 and over | Parkinson Disease - diagnosis | Parkinson Disease - epidemiology | Female | Aged | Index Medicus
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12. Full Text Mapping of brain acetylcholinesterase alterations in Lewy body disease by PET
by Shimada, H and Hirano, S and Shinotoh, H and Aotsuka, A and Sato, K and Tanaka, N and Ota, T and Asahina, M and Fukushi, K and Kuwabara, S and Hattori, T and Suhara, T and Irie, T
Neurology, ISSN 0028-3878, 2009, Volume 73, Issue 4, pp. 273 - 278
Objective: To characterize brain cholinergic deficits in Parkinson disease (PD), PD with dementia (PDD), and dementia with Lewy bodies (DLB). Methods:... 
DIAGNOSIS | POSITRON-EMISSION-TOMOGRAPHY | DEMENTIA | ALZHEIMERS-DISEASE | BODIES | CLINICAL NEUROLOGY | PARKINSONS-DISEASE | Immunohistochemistry | Humans | Middle Aged | Basal Nucleus of Meynert - diagnostic imaging | Cerebral Cortex - pathology | Lewy Body Disease - enzymology | Cholinergic Fibers - enzymology | Neural Pathways - pathology | Occipital Lobe - physiopathology | Cerebral Cortex - metabolism | Cerebral Cortex - physiopathology | Acetylcholine - deficiency | Neural Pathways - physiopathology | Occipital Lobe - diagnostic imaging | Occipital Lobe - metabolism | Biomarkers - metabolism | Diagnosis, Differential | Parkinson Disease - pathology | Parkinson Disease - diagnostic imaging | Biomarkers - analysis | Lewy Body Disease - pathology | Lewy Body Disease - diagnostic imaging | Cerebral Cortex - diagnostic imaging | Disease Progression | Cholinergic Fibers - pathology | Acetylcholinesterase - analysis | Down-Regulation - physiology | Occipital Lobe - pathology | Basal Nucleus of Meynert - pathology | Neural Pathways - diagnostic imaging | Parkinson Disease - enzymology | Radionuclide Imaging | Brain Mapping | Neural Pathways - metabolism | Aged | Basal Nucleus of Meynert - enzymology | Acetylcholinesterase - metabolism | Cohort Studies | Index Medicus | Abridged Index Medicus
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