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PLoS genetics, ISSN 1553-7404, 2011, Volume 7, Issue 3, pp. e1001324 - e1001324
Journal Article
Journal of Dental Research, ISSN 0022-0345, 9/2014, Volume 93, Issue 9, pp. 882 - 890
..., osteoporosis, and vitamin D deficiency (Genco and Borgnakke, 2013). The investigation of genetic factors in CP has been based on early twin and family studies (Michalowicz... 
genomics | genetics | bacteria | pathway analysis | inflammation | periodontal disease | PATHWAYS | POPULATION | SUSCEPTIBILITY | ADULTS | RECEPTOR | ATHEROSCLEROSIS RISK | CIRCADIAN CLOCK | DENTISTRY, ORAL SURGERY & MEDICINE | DISEASE | HEALTH | Prospective Studies | Tumor Necrosis Factor Decoy Receptors - genetics | Microtubule-Associated Proteins - genetics | Cytoskeletal Proteins - genetics | Atherosclerosis - genetics | Humans | Middle Aged | Apoptosis - genetics | Male | Monoacylglycerol Lipases - genetics | Receptors, Peptide - genetics | Receptors, Tumor Necrosis Factor, Member 10c | ras GTPase-Activating Proteins - genetics | Chronic Periodontitis - microbiology | Chronic Periodontitis - genetics | Adult | Female | Atherosclerosis - microbiology | Adaptor Protein Complex beta Subunits - genetics | Nuclear Proteins - genetics | Porphyromonas gingivalis - genetics | Genome-Wide Association Study | Genetic Association Studies | Membrane Proteins - genetics | Risk Factors | Linkage Disequilibrium - genetics | Potassium Channels, Tandem Pore Domain - genetics | Chromosome Mapping | Membrane Glycoproteins - genetics | Aggregatibacter actinomycetemcomitans - genetics | Adaptor Protein Complex 3 - genetics | Polymorphism, Single Nucleotide - genetics | Aged | Receptors, G-Protein-Coupled - genetics | Mucins - genetics | Cohort Studies | GPI-Linked Proteins - genetics | Research Reports
Journal Article
Journal of Lipid Research, ISSN 0022-2275, 02/2011, Volume 52, Issue 2, pp. 189 - 206
Journal Article
PLoS genetics, ISSN 1553-7404, 2017, Volume 13, Issue 10, p. e1007079
Journal Article
Gastroenterology, ISSN 0016-5085, 2012, Volume 143, Issue 5, pp. 1244 - 1252.e12
...) infection in genome-wide association (GWA) studies. Only a fraction of patients with chronic HCV infection develop liver fibrosis, a process that might also be affected by genetic factors... 
Gastroenterology and Hepatology | Cirrhosis | Genetic Analysis | Liver Disease | Risk Factors | CLEARANCE | PROTEIN | CHRONIC HEPATITIS-C | SUSCEPTIBILITY | IL28B | INTERFERON-ALPHA | REACTIVE OXYGEN | POLYMORPHISMS | 2 GENE VARIANTS | APOPTOTIC CELLS | GASTROENTEROLOGY & HEPATOLOGY | Humans | Hepatitis C, Chronic - virology | Apoptosis - genetics | Male | Hepatitis C, Chronic - complications | Young Adult | c-Mer Tyrosine Kinase | Adult | Female | Eye Proteins - genetics | Liver Cirrhosis - genetics | Genome-Wide Association Study | Membrane Proteins - genetics | Proportional Hazards Models | Genotype | Logistic Models | Proto-Oncogene Proteins - genetics | Disease Progression | Phenotype | Liver Cirrhosis - virology | Receptor Protein-Tyrosine Kinases - genetics | Polymorphism, Single Nucleotide | Ubiquitin-Protein Ligases - genetics | Hepacivirus | Lipase - genetics | Antiviral agents | Medical research | Medical colleges | Liver | Genomics | Immunodeficiency | Development and progression | Genomes | HIV (Viruses) | Risk factors | Analysis | Fibrosis | Medical genetics | Medicine, Experimental | Hepatitis C virus | Hepatitis C | Health aspects | Liver diseases | Genetic research | Life Sciences | Human health and pathology | Genetics | Hépatology and Gastroenterology | Infectious diseases | Human genetics | risk factors | genetic analysis | liver disease | cirrhosis
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 09/2009, Volume 284, Issue 38, pp. 25593 - 25601
Glucocorticoids are important regulators of lipid homeostasis, and chronically elevated glucocorticoid levels induce hypertriglyceridemia, hepatic steatosis,... 
OBESITY | PLASMA | GENE | CHROMATIN | ANGIOGENESIS | DIET | BIOCHEMISTRY & MOLECULAR BIOLOGY | HYPERLIPIDEMIA | MICE | TRANSCRIPTIONAL REGULATION | PROTEIN-4 | Transcription, Genetic - drug effects | Hypertriglyceridemia - genetics | Humans | Dexamethasone - adverse effects | Receptors, Glucocorticoid - metabolism | Hepatocytes - metabolism | Fatty Liver - chemically induced | Adipose Tissue - metabolism | Angiopoietin-like 4 Protein | Dexamethasone - pharmacology | Glucocorticoids - genetics | Glucocorticoids - metabolism | Response Elements - genetics | Lipoprotein Lipase - metabolism | Glucocorticoids - adverse effects | Fatty Liver - genetics | Angiopoietins - metabolism | Fatty Liver - metabolism | Lipoprotein Lipase - genetics | Liver - metabolism | Rats | Hypertriglyceridemia - metabolism | Mice, Knockout | Triglycerides - metabolism | Adipose Tissue, White | Hypertriglyceridemia - chemically induced | Animals | Receptors, Glucocorticoid - genetics | Cell Line, Tumor | Dexamethasone - metabolism | Glucocorticoids - pharmacology | Mice | Transcription, Genetic - genetics | Triglycerides - genetics | Angiopoietins - genetics | Index Medicus | Lipids and Lipoproteins | Metabolism, Regulation, and Signaling | Transcription | Dermatologi och venereologi | Dermatology and Venereal Diseases | Dexamethasone/adverse effects/metabolism/pharmacology | Hypertriglyceridemia/chemically induced/genetics/metabolism | Receptors | Adipose Tissue/metabolism | Liver/metabolism | Tumor | Adipose Tissue | Cell Line | Fatty Liver/chemically induced/genetics/metabolism | Triglycerides/genetics/ metabolism | Response Elements/genetics | Angiopoietins/genetics/ metabolism | Knockout | Lipoprotein Lipase/genetics/metabolism | White | Hepatocytes/metabolism | Genetic/drug effects/genetics | Glucocorticoids/adverse effects/genetics/ metabolism/pharmacology | Glucocorticoid/genetics/ metabolism
Journal Article
Nature (London), ISSN 1476-4687, 2008, Volume 452, Issue 7186, pp. 429 - 435
Identifying variations in DNA that increase susceptibility to disease is one of the primary aims of genetic studies using a forward genetics approach... 
QUANTITATIVE TRAIT LOCI | SEGREGATING MOUSE-POPULATION | HEART MITOCHONDRIAL PROTEOME | DETERMINING ATHEROSCLEROSIS SUSCEPTIBILITY | MULTIDISCIPLINARY SCIENCES | TISSUE-SPECIFIC EXPRESSION | GENE-EXPRESSION | FAMILIAL COMBINED HYPERLIPIDEMIA | FACTOR-H POLYMORPHISM | APOLIPOPROTEIN-A-II | UPSTREAM TRANSCRIPTION FACTOR-1 | Chromosomes, Mammalian - genetics | Phosphoprotein Phosphatases - metabolism | Apolipoprotein A-II - genetics | Male | Metabolic Syndrome - metabolism | Obesity - genetics | Lod Score | Adipose Tissue - metabolism | Female | Metabolic Syndrome - enzymology | Genetic Predisposition to Disease - genetics | Reproducibility of Results | Gene Regulatory Networks - genetics | Lipoprotein Lipase - genetics | Membrane Proteins - genetics | Ribosomal Proteins - genetics | Liver - metabolism | Linkage Disequilibrium | Obesity - metabolism | Macrophages - metabolism | Phenotype | Animals | Phosphoprotein Phosphatases - genetics | Metabolic Syndrome - genetics | Phosphoprotein Phosphatases - deficiency | Genetic Variation - genetics | Mice | Obesity - enzymology | Quantitative Trait Loci | Quantitative trait loci | Molecular neurobiology | Disease susceptibility | Genetic aspects | Research | Genetic variation | Obesity | Integrated approach | Biomedical research | Gene expression | Genomics | Lipoprotein Lipase | Liver | Apolipoprotein A-II | Metabolic Syndrome X | Macrophages | Gene Regulatory Networks | Life Sciences | Adipose Tissue | Phosphoprotein Phosphatases | Genetic Predisposition to Disease | Biochemistry, Molecular Biology | Variation (Genetics) | Chromosomes, Mammalian | Membrane Proteins | Molecular biology | Ribosomal Proteins
Journal Article
by Wu, Ying and Waite, Lindsay L and Jackson, Anne U and Sheu, Wayne H-H and Buyske, Steven and Absher, Devin and Arnett, Donna K and Boerwinkle, Eric and Bonnycastle, Lori L and Carty, Cara L and Cheng, Iona and Cochran, Barbara and Croteau-Chonka, Damien C and Dumitrescu, Logan and Eaton, Charles B and Franceschini, Nora and Guo, Xiuqing and Henderson, Brian E and Hindorff, Lucia A and Kim, Eric and Kinnunen, Leena and Komulainen, Pirjo and Lee, Wen-Jane and Le Marchand, Loic and Lin, Yi and Lindström, Jaana and Lingaas-Holmen, Oddgeir and Mitchell, Sabrina L and Narisu, Narisu and Robinson, Jennifer G and Schumacher, Fred and Stančáková, Alena and Sundvall, Jouko and Sung, Yun-Ju and Swift, Amy J and Wang, Wen-Chang and Wilkens, Lynne and Wilsgaard, Tom and Young, Alicia M and Adair, Linda S and Ballantyne, Christie M and Bůžková, Petra and Chakravarti, Aravinda and Collins, Francis S and Duggan, David and Feranil, Alan B and Ho, Low-Tone and Hung, Yi-Jen and Hunt, Steven C and Hveem, Kristian and Juang, Jyh-Ming J and Kesäniemi, Antero Y and Kuusisto, Johanna and Laakso, Markku and Lakka, Timo A and Lee, I-Te and Leppert, Mark F and Matise, Tara C and Moilanen, Leena and Njølstad, Inger and Peters, Ulrike and Quertermous, Thomas and Rauramaa, Rainer and Rotter, Jerome I and Saramies, Jouko and Tuomilehto, Jaakko and Uusitupa, Matti and Wang, Tzung-Dau and Boehnke, Michael and Haiman, Christopher A and Chen, Yii-Der I and Kooperberg, Charles and Assimes, Themistocles L and Crawford, Dana C and Hsiung, Chao A and North, Kari E and Mohlke, Karen L
PLoS genetics, ISSN 1553-7404, 2013, Volume 9, Issue 3, p. e1003379
...Introduction Genome-wide association studies (GWAS) have identified many common genetic variants associated with human diseases and complex traits... 
COMMON VARIANTS | DENSITY-LIPOPROTEIN CHOLESTEROL | FUNCTIONAL POLYMORPHISM | GENE | GENOTYPE IMPUTATION | GENETICS & HEREDITY | RISK | LIPASE | COMPLEX TRAITS | CORONARY-ARTERY-DISEASE | GENOME-WIDE ASSOCIATION | Apolipoprotein A-V | European Continental Ancestry Group - genetics | Genome-Wide Association Study | Apolipoproteins A - genetics | Lipoproteins, HDL - genetics | Lipoproteins, HDL - blood | Humans | African Americans - genetics | Cholesterol, HDL - genetics | Lipoproteins, LDL - genetics | Proprotein Convertases - genetics | Cholesterol, LDL - genetics | Serine Endopeptidases - genetics | Triglycerides - blood | Cholesterol, HDL - blood | Cholesterol, LDL - blood | Triglycerides - genetics | Lipoproteins, LDL - blood | Proprotein Convertase 9 | Quantitative genetics | Genetic variation | Physiological aspects | Cholesterol, LDL | Genetic aspects | Research | Population genetics | Health aspects | Medisinsk genetikk: 714 | VDP | Basic medical, dental and veterinary science disciplines: 710 | Medical molecular biology: 711 | Medical genetics: 714 | Medisinske Fag: 700 | Medisinsk molekylærbiologi: 711 | Medical disciplines: 700 | Basale medisinske, odontologiske og veterinærmedisinske fag: 710 | Medical research | Genealogy | Womens health | African Americans | Population | Genomes | Gene loci | Gene expression | Epidemiology | Cholesterol
Journal Article