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BBA - Molecular and Cell Biology of Lipids, ISSN 1388-1981, 06/2016, Volume 1861, Issue 6, pp. 491 - 500
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 09/2009, Volume 284, Issue 38, pp. 25593 - 25601
Glucocorticoids are important regulators of lipid homeostasis, and chronically elevated glucocorticoid levels induce hypertriglyceridemia, hepatic steatosis,... 
OBESITY | PLASMA | GENE | CHROMATIN | ANGIOGENESIS | DIET | BIOCHEMISTRY & MOLECULAR BIOLOGY | HYPERLIPIDEMIA | MICE | TRANSCRIPTIONAL REGULATION | PROTEIN-4 | Transcription, Genetic - drug effects | Hypertriglyceridemia - genetics | Humans | Dexamethasone - adverse effects | Receptors, Glucocorticoid - metabolism | Hepatocytes - metabolism | Fatty Liver - chemically induced | Adipose Tissue - metabolism | Angiopoietin-like 4 Protein | Dexamethasone - pharmacology | Glucocorticoids - genetics | Glucocorticoids - metabolism | Response Elements - genetics | Lipoprotein Lipase - metabolism | Glucocorticoids - adverse effects | Fatty Liver - genetics | Angiopoietins - metabolism | Fatty Liver - metabolism | Lipoprotein Lipase - genetics | Liver - metabolism | Rats | Hypertriglyceridemia - metabolism | Mice, Knockout | Triglycerides - metabolism | Adipose Tissue, White | Hypertriglyceridemia - chemically induced | Animals | Receptors, Glucocorticoid - genetics | Cell Line, Tumor | Dexamethasone - metabolism | Glucocorticoids - pharmacology | Mice | Transcription, Genetic - genetics | Triglycerides - genetics | Angiopoietins - genetics | Index Medicus | Lipids and Lipoproteins | Metabolism, Regulation, and Signaling | Transcription | Dermatologi och venereologi | Dermatology and Venereal Diseases | Dexamethasone/adverse effects/metabolism/pharmacology | Hypertriglyceridemia/chemically induced/genetics/metabolism | Receptors | Adipose Tissue/metabolism | Liver/metabolism | Tumor | Adipose Tissue | Cell Line | Fatty Liver/chemically induced/genetics/metabolism | Triglycerides/genetics/ metabolism | Response Elements/genetics | Angiopoietins/genetics/ metabolism | Knockout | Lipoprotein Lipase/genetics/metabolism | White | Hepatocytes/metabolism | Genetic/drug effects/genetics | Glucocorticoids/adverse effects/genetics/ metabolism/pharmacology | Glucocorticoid/genetics/ metabolism
Journal Article
American Journal of Physiology - Endocrinology and Metabolism, ISSN 0193-1849, 07/2016, Volume 311, Issue 1, pp. E224 - E235
Increased availability of lipids may conserve muscle protein during catabolic stress. Our study was designed to define 1) intracellular mechanisms leading to... 
Human | Protein breakdown | Obesity | Fasting | Subcutaneous adipose tissue | Lipolysis | Skeletal muscle | ADIPOSE TRIGLYCERIDE LIPASE | PHYSIOLOGY | HORMONE-SENSITIVE LIPASE | MUSCLE | fasting | sub-cutaneous adipose tissue | AMINO-ACID | protein breakdown | GROWTH-HORMONE | FAT-CELLS | ENDOCRINOLOGY & METABOLISM | WHOLE-BODY | LEUCINE TURNOVER | lipolysis | skeletal muscle | human | obesity | DECREASED EXPRESSION | 1-Acylglycerol-3-Phosphate O-Acyltransferase - genetics | Phosphorylation | TOR Serine-Threonine Kinases - metabolism | Humans | Sterol Esterase - metabolism | Male | Muscle, Skeletal - metabolism | Autophagy-Related Protein-1 Homolog - metabolism | Intracellular Signaling Peptides and Proteins - metabolism | RNA, Messenger - metabolism | Obesity - genetics | Case-Control Studies | Lipolysis - genetics | Young Adult | Adipose Tissue - metabolism | 1-Acylglycerol-3-Phosphate O-Acyltransferase - metabolism | Forearm | Time Factors | Muscle Proteins - metabolism | Adult | Lipid Metabolism - genetics | Urea - metabolism | Fasting - metabolism | Intracellular Signaling Peptides and Proteins - genetics | Cell Cycle Proteins - metabolism | Ubiquitin-Protein Ligases - metabolism | Lipase - metabolism | Obesity - metabolism | Cross-Over Studies | Tripartite Motif Proteins - metabolism | Autophagy-Related Protein-1 Homolog - genetics | Lipase - genetics | Protein metabolism | Physiological aspects | Physiological research | Lipid metabolism | Research | Biological control systems | Index Medicus
Journal Article
Science, ISSN 0036-8075, 11/2011, Volume 334, Issue 6057, pp. 809 - 813
Phospholipase A₂ (PLA₂) enzymes are considered the primary source of arachidonic acid for cyclooxygenase (COX)-mediated biosynthesis of prostaglandins. Here,... 
Datasets | Brain | Enzymes | Prostaglandins | Cytokines | Neurodegenerative diseases | REPORTS | Eicosanoids | Lipids | Endocannabinoids | Vehicles | SYSTEM | INHIBITION | CYCLOOXYGENASE-2 | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | MOUSE MODEL | INJURY | LIPOPOLYSACCHARIDE | PHOSPHOLIPASE A | MICE | PARKINSONS-DISEASE | Cannabinoid Receptor Modulators - metabolism | Inflammation - pathology | Metabolomics | Arachidonic Acid - metabolism | Monoacylglycerol Lipases - antagonists & inhibitors | Monoacylglycerol Lipases - genetics | Brain - metabolism | Parkinsonian Disorders - metabolism | Monoacylglycerol Lipases - metabolism | Arachidonic Acids - metabolism | Inflammation - metabolism | Neuroprotective Agents - pharmacology | Piperidines - pharmacology | Inflammation Mediators - pharmacology | Lung - metabolism | Cytokines - metabolism | Signal Transduction | Liver - metabolism | Mice, Inbred C57BL | Enzyme Inhibitors - pharmacology | Phospholipases A2 - metabolism | Prostaglandins - biosynthesis | Glycerides - metabolism | Brain - drug effects | Hydrolysis | Animals | Parkinsonian Disorders - pathology | Eicosanoids - metabolism | Lipopolysaccharides - pharmacology | Brain - pathology | Mice | Cyclooxygenase 1 - metabolism | Benzodioxoles - pharmacology | Phospholipases A2 - genetics | Prostaglandins - metabolism | Physiological aspects | Inflammation | Research | Risk factors | Hydrology | Neurology | Inflammatory diseases | Animal models | Index Medicus
Journal Article
Journal of Cell Science, ISSN 0021-9533, 06/2009, Volume 122, Issue 11, pp. 1834 - 1841
Lipid droplets (LDs) are cytoplasmic organelles that store neutral lipids for use as an energy supply in times of nutrient deprivation and for membrane... 
Lipid homeostasis | ATGL | Lipid droplet | ADIPOSE TRIGLYCERIDE LIPASE | CELLS | GOLGI | CELL BIOLOGY | FAT STORAGE | LIPOLYSIS | LIPODYSTROPHY | ENDOPLASMIC-RETICULUM | BREFELDIN-A | DIFFERENTIATION-RELATED PROTEIN | RNA, Small Interfering - genetics | Fluorescence Recovery After Photobleaching | Humans | Adaptor Proteins, Vesicular Transport - genetics | Endoplasmic Reticulum - metabolism | Lipids | Protein Transport - physiology | Intracellular Signaling Peptides and Proteins - metabolism | Pregnancy Proteins - metabolism | Coat Protein Complex I - metabolism | Adaptor Proteins, Vesicular Transport - metabolism | Recombinant Fusion Proteins - metabolism | Endoplasmic Reticulum - ultrastructure | DNA-Binding Proteins - metabolism | Organelles - ultrastructure | Guanine Nucleotide Exchange Factors - metabolism | ADP-Ribosylation Factor 1 - metabolism | Membrane Proteins - metabolism | ADP-Ribosylation Factor 1 - genetics | Golgi Apparatus - ultrastructure | Vesicular Transport Proteins | Guanine Nucleotide Exchange Factors - genetics | Lipase - metabolism | Protein Synthesis Inhibitors - metabolism | Brefeldin A - metabolism | Coatomer Protein - metabolism | Recombinant Fusion Proteins - genetics | Golgi Apparatus - metabolism | HeLa Cells | Organelles - metabolism | Perilipin-3 | Perilipin-2 | Lipase - genetics | RNA, Small Interfering - metabolism | Index Medicus
Journal Article
Cell Metabolism, ISSN 1550-4131, 05/2012, Volume 15, Issue 5, pp. 691 - 702
Numerous studies in humans link a nonsynonymous genetic polymorphism (I148M) in adiponutrin (ADPN) to various forms of fatty liver disease and liver cirrhosis.... 
ADIPOSE TRIGLYCERIDE LIPASE | COMPARATIVE GENE IDENTIFICATION-58 | LIPID-METABOLISM | MESSENGER-RNA EXPRESSION | FATTY LIVER-DISEASE | FAMILY-MEMBERS | ENZYMES | ENDOCRINOLOGY & METABOLISM | PNPLA3 | CONGENITAL GENERALIZED LIPODYSTROPHY | CHANARIN-DORFMAN-SYNDROME | CELL BIOLOGY | 1-Acylglycerol-3-Phosphate O-Acyltransferase - genetics | Lysophospholipids - metabolism | Up-Regulation | Lipids - genetics | Humans | Cercopithecus aethiops | Acyl Coenzyme A - genetics | Male | Lipids - biosynthesis | Acyltransferases - metabolism | Acyltransferases - genetics | 1-Acylglycerol-3-Phosphate O-Acyltransferase - metabolism | Cysteine Endopeptidases - metabolism | Liver - drug effects | Lipid Metabolism - genetics | Membrane Proteins - metabolism | Dietary Sucrose - metabolism | Phospholipids - metabolism | Acyl Coenzyme A - metabolism | CHO Cells | Fatty Liver - genetics | Phosphatidic Acids - genetics | Cricetinae | Fatty Liver - metabolism | Membrane Proteins - genetics | Liver - metabolism | Models, Molecular | Phospholipids - genetics | Hep G2 Cells | Mice, Knockout | Polymorphism, Genetic | Triglycerides - metabolism | Lysophospholipids - genetics | Animals | Cysteine Endopeptidases - genetics | Mice | Phosphatidic Acids - metabolism | Triglycerides - genetics | COS Cells | Physiological aspects | Medical colleges | Disease susceptibility | Liver diseases | Liver cirrhosis | Index Medicus
Journal Article
Nature, ISSN 0028-0836, 10/2017, Volume 550, Issue 7674, pp. 119 - 123
Catecholamine-induced lipolysis, the first step in the generation of energy substrates by the hydrolysis of triglycerides(1), declines with age(2,3). The... 
MONOAMINE-OXIDASE | CELLS | OBESITY | ACTIVATION | MULTIDISCIPLINARY SCIENCES | ACCUMULATION | IDENTIFICATION | GENE ONTOLOGY | DEFICIENCY | ADIPOSE-TISSUE | AGE | Inflammasomes - metabolism | Lipolysis - drug effects | Catecholamines - metabolism | Growth Differentiation Factor 3 - metabolism | Sterol Esterase - metabolism | Aging - drug effects | Adipose Tissue - cytology | Caspase 1 - metabolism | Gene Expression Profiling | Growth Differentiation Factor 3 - deficiency | Lipolysis - genetics | Adipose Tissue - metabolism | Aging - genetics | Monoamine Oxidase Inhibitors - pharmacology | NLR Family, Pyrin Domain-Containing 3 Protein - metabolism | Lipase - metabolism | NLR Family, Pyrin Domain-Containing 3 Protein - deficiency | Gene Expression Regulation - drug effects | Macrophages - metabolism | Animals | Norepinephrine - metabolism | Growth Differentiation Factor 3 - genetics | Adipocytes - metabolism | Mice | Adipose Tissue - drug effects | Catecholamines - pharmacology | Aging - metabolism | Monoamine Oxidase - metabolism | Aging | Observations | Catecholamine metabolism | Health aspects | Elderly people | Adipose tissue | Oxidase | Genomes | Adipocytes | Kinases | Macrophages | Lipase | Caspase-1 | Reduction | Energy resources | Clonal deletion | Deletion | Noradrenaline | Age | Enzymes | Starvation | Fasting | Body temperature | Aging (artificial) | Glycerol | Caspase | Principal components analysis | Triglycerides | Inflammation | Bioavailability | Metabolism | Gene expression | Sympathetic nervous system | Catecholamine | Fatty acids | Substrates | Lipolysis | Amine oxidase (flavin-containing) | Signaling | Catabolism | Norepinephrine | Elderly | Geriatrics | Index Medicus
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 07/2006, Volume 116, Issue 7, pp. 1983 - 1993
Many homeostatic processes, including appetite and food intake, are controlled by neuroendocrine circuits involving the CNS. The CNS also directly regulates... 
MEDICINE, RESEARCH & EXPERIMENTAL | SKELETAL-MUSCLE | AGOUTI-RELATED PROTEIN | PERIPHERAL INSULIN-RESISTANCE | LIPID-METABOLISM | BODY-WEIGHT GAIN | ENERGY HOMEOSTASIS | FOOD-INTAKE | BROWN ADIPOSE-TISSUE | NORMAL RATS | NEUROPEPTIDE-Y | Brain - anatomy & histology | Fatty Acid Synthases - metabolism | Ion Channels | Rats, Wistar | Adipose Tissue - cytology | Homeostasis | Adipocytes - cytology | Male | Neuropeptide Y - genetics | Mitochondrial Proteins | Peptide Hormones - genetics | Acetyl-CoA Carboxylase - genetics | Brain - metabolism | Neuropeptide Y - metabolism | Peptide Hormones - metabolism | Adipose Tissue - metabolism | Ghrelin | Membrane Proteins - metabolism | Fatty Acid Synthases - genetics | Lipoprotein Lipase - metabolism | Peptide Hormones - administration & dosage | Lipoprotein Lipase - genetics | Membrane Proteins - genetics | Rats | Stearoyl-CoA Desaturase - genetics | Mice, Knockout | Carrier Proteins - genetics | Eating - drug effects | Animals | Carrier Proteins - metabolism | Adipocytes - metabolism | Glucose - metabolism | Stearoyl-CoA Desaturase - metabolism | Mice | Uncoupling Protein 1 | Uncoupling Protein 3 | Acetyl-CoA Carboxylase - metabolism | Energy Metabolism - drug effects | Adipose tissues | Medical research | Central nervous system | Cell metabolism | Medicine, Experimental | Research | Index Medicus | Abridged Index Medicus
Journal Article
Plant Cell, ISSN 1040-4651, 10/2014, Volume 26, Issue 10, pp. 4119 - 4134
Triacylglycerol (TAG) metabolism is a key aspect of intracellular lipid homeostasis in yeast and mammals, but its role in vegetative tissues of plants remains... 
DIACYLGLYCEROL ACYLTRANSFERASE | CONTACT SITES | SHORT-CHAIN | CHLOROPLAST INNER ENVELOPE | BIOCHEMISTRY & MOLECULAR BIOLOGY | PHOSPHOLIPID-SYNTHESIS | CARRIER PROTEIN | PLANT SCIENCES | CELL BIOLOGY | OIL CATABOLISM | METABOLISM | ENDOPLASMIC-RETICULUM | PHOSPHATIDIC-ACID | Homeostasis | Acyltransferases - metabolism | Acyltransferases - genetics | Arabidopsis Proteins - metabolism | Lipid Droplets - ultrastructure | ATP-Binding Cassette Transporters - genetics | Membrane Transport Proteins - genetics | beta-Glucosidase - genetics | Plants, Genetically Modified | Gene Expression Regulation, Plant | ATP-Binding Cassette Transporters - metabolism | beta-Glucosidase - metabolism | Membrane Transport Proteins - metabolism | Carboxylic Ester Hydrolases - genetics | Lipid Droplets - metabolism | Fatty Acids - metabolism | Microscopy, Electron, Transmission | Arabidopsis Proteins - genetics | Oxidation-Reduction | Lipase - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Peroxisomes - metabolism | Arabidopsis - metabolism | Triglycerides - metabolism | Arabidopsis - genetics | Plant Leaves - genetics | Plant Leaves - metabolism | Models, Biological | Membrane Lipids - metabolism | Mutation | Microscopy, Fluorescence | Plant Leaves - ultrastructure | Carboxylic Ester Hydrolases - metabolism | Lipase - genetics | Index Medicus
Journal Article
Science, ISSN 0036-8075, 5/2006, Volume 312, Issue 5774, pp. 734 - 737
Fat tissue is the most important energy depot in vertebrates. The release of free fatty acids (FFAs) from stored fat requires the enzymatic activity of... 
Adipose tissues | Fasting | Liver | Nonesterified fatty acids | Myocardium | Lipids | Reports | Triglycerides | Mice | Insulin | Lipolysis | LIPOTOXICITY | CELLS | HORMONE-SENSITIVE LIPASE | MOBILIZATION | INSULIN-RESISTANCE | MULTIDISCIPLINARY SCIENCES | TISSUE | HUMAN OBESITY | ACCUMULATION | EXPRESSION | DEFICIENCY | Testis - metabolism | Lipolysis - drug effects | Homeostasis | Adipocytes - cytology | Male | Insulin - blood | Adipose Tissue - metabolism | Fatty Acids, Nonesterified - metabolism | Kidney - metabolism | Lipids - blood | Myocardium - metabolism | Fatty Acids, Nonesterified - blood | Female | Carboxylic Ester Hydrolases - genetics | Lipase - deficiency | Adipose Tissue - anatomy & histology | Myocytes, Cardiac - cytology | Carboxylic Ester Hydrolases - deficiency | Oxygen Consumption | Cell Size | Myocardium - pathology | Heart Failure - pathology | Lipase - metabolism | Adipose Tissue, Brown - enzymology | Ventricular Dysfunction, Left - physiopathology | Triglycerides - metabolism | Animals | Thermogenesis | Energy Metabolism | Adipocytes - metabolism | Isoproterenol - pharmacology | Myocytes, Cardiac - metabolism | Adipose Tissue - enzymology | Blood Glucose - metabolism | Carboxylic Ester Hydrolases - metabolism | Lipase - genetics | Physiological aspects | Research | Metabolism | Heart | Enzymes | Body fat | Rodents | Glucose | Index Medicus
Journal Article
Molecular and Cellular Endocrinology, ISSN 0303-7207, 09/2013, Volume 377, Issue 1-2, pp. 147 - 158
The molecular mechanisms underlying the effects of selective ER subtype activation on lipogenesis, adipogenesis, lipid utilization and storage as well as... 
Obesity | Glucose uptake | Lipid metabolism | Lipogenesis | Genistein | Estrogen receptor subtype-selective agonist | PEROXISOME-PROLIFERATOR | DIETARY ISOFLAVONES | QUANTITATIVE-ANALYSIS | CELL BIOLOGY | RESPONSE ELEMENT | GLUCOSE-HOMEOSTASIS | ESTROGEN-RECEPTOR-ALPHA | ENDOCRINOLOGY & METABOLISM | REPLACEMENT THERAPY | POSTMENOPAUSAL WOMEN | PHYTOESTROGEN GENISTEIN | ADIPOSE-TISSUE | Rats, Wistar | Insulin - blood | PPAR gamma - metabolism | RNA, Messenger - metabolism | fas Receptor - metabolism | Obesity - genetics | Estrogen Receptor beta - metabolism | Adipose Tissue - metabolism | Estrogen Receptor alpha - agonists | Estrogen Receptor beta - agonists | Liver - drug effects | fas Receptor - genetics | Isotope Labeling | Estrogen Receptor alpha - metabolism | Female | Homeostasis - drug effects | Energy Metabolism - genetics | Muscles - metabolism | Lipoprotein Lipase - metabolism | Estradiol - pharmacology | Sterol Regulatory Element Binding Protein 1 - metabolism | PPAR gamma - genetics | Lipoprotein Lipase - genetics | Liver - metabolism | RNA, Messenger - genetics | Insulin Resistance | Rats | Obesity - metabolism | Triglycerides - metabolism | Gene Expression Regulation - drug effects | Animals | Sterol Regulatory Element Binding Protein 1 - genetics | Muscles - drug effects | Genistein - pharmacology | Blood Glucose - metabolism | Adipose Tissue - drug effects | Energy Metabolism - drug effects | Homeostasis - genetics | Glucose metabolism | Glucose | Dextrose | Index Medicus | Utilization | Muscles | Lipids | Females | Insulin
Journal Article