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Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2013, Volume 110, Issue 22, pp. 8882 - 8887
Journal Article
The Journal of clinical investigation, ISSN 1558-8238, 2017, Volume 127, Issue 11, pp. 4059 - 4074
Overconsumption of high-fat diet (HFD) and sugar-sweetened beverages are risk factors for developing obesity, insulin resistance, and fatty liver disease. Here... 
MEDICINE, RESEARCH & EXPERIMENTAL | BODY-WEIGHT | LIPID-METABOLISM | INDUCED METABOLIC SYNDROME | DE-NOVO LIPOGENESIS | FATTY LIVER-DISEASE | RESISTANCE | SUGAR-SWEETENED BEVERAGES | NONALCOHOLIC STEATOHEPATITIS | MICE | DIETARY FRUCTOSE | Up-Regulation | Liver - enzymology | Insulin - physiology | Humans | Transcriptional Activation | Transcriptome | Diet, High-Fat - adverse effects | Non-alcoholic Fatty Liver Disease - etiology | Male | Glucose Intolerance | Liver - drug effects | Fructose - pharmacology | Non-alcoholic Fatty Liver Disease - enzymology | Mice, Inbred C57BL | Insulin Resistance | Enzyme Induction | Glucose - pharmacology | Transcription Factors - genetics | Fructokinases - metabolism | Transcription Factors - metabolism | Animals | Signal Transduction - drug effects | Lipogenesis - drug effects | Adolescent | Fructokinases - genetics | Obesity - enzymology | Fatty Acids - biosynthesis | RNA sequencing | Glucose metabolism | Obesity | Care and treatment | Usage | Liver diseases | Insulin resistance | Research | Diagnosis | Health aspects | Drinking water | Transcription factors | Lipids | Glucose | Metabolic syndrome | Risk factors | High fat diet | Ketohexokinase | Disease resistance | Fatty liver | Hepatology | Teenagers | Supplementation | Beverages | Lipogenesis | Sugar | Enzymes | Nucleotide sequence | Dietary supplements | Ribonucleic acid--RNA | Fatty acids | Insulin | Fructose | Steatosis | Glucose tolerance | Intolerance | Diet | Diabetes
Journal Article
PloS one, ISSN 1932-6203, 2012, Volume 7, Issue 2, p. e31812
The pathogenesis of fatty liver is not understood in detail, but lipid overflow as well as de novo lipogenesis (DNL) seem to be the key points of hepatocyte... 
DE-NOVO LIPOGENESIS | INSULIN-RESISTANCE | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | SENSITIVITY | CARBOHYDRATE | RECEPTOR | MICE | ACID SYNTHESIS | HEPATIC STEATOSIS | ALPHA-FETOPROTEIN | Organ Specificity - drug effects | Sterol Regulatory Element Binding Protein 1 - chemistry | Transcription, Genetic - drug effects | Acetyltransferases - metabolism | Adipocytes - secretion | Liver - pathology | Liver - enzymology | Fatty Liver - pathology | Humans | Male | Adipocytes - drug effects | Lipogenesis - genetics | Liver - drug effects | Body Composition - drug effects | Intra-Abdominal Fat - drug effects | Fatty Acids - metabolism | Cytokines - blood | Fatty Liver - genetics | Protein Structure, Tertiary | Insulin - pharmacology | Adiposity - drug effects | Fatty Liver - blood | Liver - metabolism | Cytokines - secretion | Insulin Resistance | Mice, Transgenic | Intra-Abdominal Fat - metabolism | Gene Expression Regulation - drug effects | Organ Size - drug effects | Phenotype | Animals | Sterol Regulatory Element Binding Protein 1 - genetics | Lipogenesis - drug effects | Stearoyl-CoA Desaturase - metabolism | Mice | Intra-Abdominal Fat - pathology | Type 2 diabetes | Enzymes | Obesity | Liver | Genes | Albumin | Amino acids | Triglycerides | Genetic transcription | Fatty acids | Fatty liver | Analysis | Physiological aspects | Insulin resistance | Protein binding | Transcription factors | Lipid composition | Adipose tissue | Body fat | Pathogenesis | Homeostasis | Lipids | Biochemistry | Adipocytes | Medical diagnosis | Accumulation | Overflow | Sterols | Transgenic animals | Rodents | Atherosclerosis | Sterol regulatory element-binding protein | Lipid metabolism | Lipogenesis | Desaturation | Hypertension | Carbohydrates | Phenotypes | Liver diseases | Internal medicine | Nutrition | Metabolism | Gene expression | Insulin | Overexpression | Epigenetics | Growth hormones | Diabetes | Metabolic disorders
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 11/2016, Volume 126, Issue 11, pp. 4372 - 4386
Obese, insulin-resistant states are characterized by a paradoxical pathogenic condition in which the liver appears to be selectively insulin resistant.... 
RAT-LIVER | MEDICINE, RESEARCH & EXPERIMENTAL | METABOLIC SYNDROME | DE-NOVO LIPOGENESIS | TRANSCRIPTION FACTOR FOXO1 | FATTY LIVER-DISEASE | OB/OB MICE | ELEMENT-BINDING PROTEIN | HEPATIC STEATOSIS | ADIPOSE-TISSUE | PLASMA TRIGLYCERIDES | Glucose Intolerance - metabolism | Glucose-6-Phosphatase - genetics | Fatty Liver - pathology | Humans | Male | Glucose - biosynthesis | Glucose Intolerance - pathology | Fatty Liver - chemically induced | Glycolysis - drug effects | Glycolysis - genetics | Lipogenesis - genetics | Female | Glucose Intolerance - chemically induced | Insulin - genetics | Nuclear Proteins - genetics | Fatty Liver - genetics | Forkhead Box Protein O1 - metabolism | Glucose Intolerance - genetics | Fatty Liver - metabolism | Fructose - toxicity | Insulin Resistance | Glucose - genetics | Nuclear Proteins - metabolism | Signal Transduction - genetics | Transcription Factors - genetics | Glucose-6-Phosphatase - metabolism | Mice, Knockout | Transcription Factors - metabolism | Insulin - metabolism | Animals | Signal Transduction - drug effects | Lipogenesis - drug effects | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - genetics | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - metabolism | Mice | Forkhead Box Protein O1 - genetics | Obesity | Analysis | Insulin resistance | Genetic aspects | Genetic transcription | Research | Risk factors | Protein binding | Glucose | Metabolites | Rodents | Liver
Journal Article
Nutrients, ISSN 2072-6643, 2015, Volume 7, Issue 11, pp. 9453 - 9474
Journal Article