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Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 11/2011, Volume 108, Issue 50, pp. 20054 - 20059
We report here that mouse macrophages undergo receptor-interacting kinase-3 (RIP3)-dependent but TNF-α—independent necrosis when Toll-like receptors... 
Reactive oxygen species | Receptors | Cytokines | Cell death | Toll like receptors | Mice | Macrophages | Viability | Necrosis | Apoptosis | Necroptosis | Bone marrow-derived macrophage | Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Protein Structure, Tertiary | Receptor-Interacting Protein Serine-Threonine Kinases - metabolism | Reactive Oxygen Species - metabolism | Macrophages - pathology | Receptor-Interacting Protein Serine-Threonine Kinases - chemistry | NF-kappa B - metabolism | Enzyme Activation - drug effects | Toll-Like Receptor 3 - metabolism | Adaptor Proteins, Vesicular Transport - metabolism | Toll-Like Receptor 4 - metabolism | Mice, Knockout | Signal Transduction - immunology | Models, Immunological | Animals | Signal Transduction - drug effects | Protein Binding - drug effects | Inflammation Mediators - metabolism | Lipopolysaccharides - pharmacology | Interferon Regulatory Factor-3 - metabolism | Macrophages - drug effects | Cytokines - biosynthesis | Macrophages - immunology | Cell receptors | Genetic aspects | Properties | Index Medicus | Tumor necrosis factor receptors | Immune response | Data processing | Inflammation | Interleukin 1 receptors | TLR4 protein | adaptor proteins | TLR3 protein | Lipopolysaccharides | Infection | Signal transduction | beta -Interferon | Toll-like receptors | bone marrow-derived macrophage | Biological Sciences | necroptosis
Journal Article
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 10/2007, Volume 293, Issue 4, pp. 2210 - 2218
Targeting cannabinoid-2 (CB2) receptors with selective agonists may represent a novel therapeutic avenue in various inflammatory diseases, but the mechanisms by which CB2 activation exerts its anti-inflammatory effects... 
Adhesion molecules | Inflammation | RhoA | Endothelial activation | Cardiac & Cardiovascular Systems | Physiology | Peripheral Vascular Disease | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Inflammation - chemically induced | Leukocyte Rolling - drug effects | Tumor Necrosis Factor-alpha - metabolism | Receptor, Cannabinoid, CB2 - agonists | Humans | Male | Monocytes - metabolism | NF-kappa B - metabolism | rhoA GTP-Binding Protein - metabolism | Aorta - metabolism | RNA, Messenger - metabolism | Receptor, Cannabinoid, CB2 - genetics | Coronary Vessels - metabolism | Lipopolysaccharides | Dose-Response Relationship, Drug | Inflammation - metabolism | Anti-Inflammatory Agents - therapeutic use | Chemokine CCL2 - metabolism | Disease Models, Animal | Coronary Vessels - drug effects | Anti-Inflammatory Agents - pharmacology | Endothelial Cells - metabolism | Aorta - drug effects | Mice, Inbred C57BL | Cells, Cultured | Cannabinoids - pharmacology | Monocytes - drug effects | Receptor, Cannabinoid, CB1 - metabolism | Receptor, Cannabinoid, CB2 - metabolism | Cannabinoids - therapeutic use | Intercellular Adhesion Molecule-1 - metabolism | Animals | Signal Transduction - drug effects | Inflammation - prevention & control | Mice | Vascular Cell Adhesion Molecule-1 - metabolism | Endothelial Cells - drug effects | Index Medicus | inflammation | adhesion molecules | endothelial activation
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 09/2007, Volume 282, Issue 36, pp. 26014 - 26025
.... The total fraction of Mycobacterium bovis BCG LM was shown both to induce macrophage activation and pro-inflammatory cytokines through Toll-like receptor 2 (TLR2... 
Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | Mannose-Binding Lectins - metabolism | Mycobacterium bovis - immunology | Toll-Like Receptor 2 - agonists | Membrane Glycoproteins - metabolism | Humans | Lectins, C-Type - immunology | Mycobacterium tuberculosis - immunology | Lipopolysaccharides - metabolism | Cell Adhesion Molecules - immunology | Lipopolysaccharides - immunology | Lectins, C-Type - metabolism | Signal Transduction - immunology | Inflammation - metabolism | Macrophages - secretion | Toll-Like Receptor 1 - agonists | Myeloid Differentiation Factor 88 - immunology | Tumor Necrosis Factor-alpha - immunology | Toll-Like Receptor 4 - agonists | Interleukin-12 Subunit p40 - immunology | Membrane Glycoproteins - immunology | Macrophages - immunology | Acylation | Cell Line | Nitric Oxide - immunology | Immunity, Innate - drug effects | Macrophage Activation - immunology | Receptors, Interleukin-1 - immunology | Tumor Necrosis Factor-alpha - secretion | Receptors, Cell Surface - metabolism | Inflammation - immunology | Toll-Like Receptor 4 - immunology | Toll-Like Receptor 2 - metabolism | Cell Adhesion Molecules - metabolism | Receptors, Cell Surface - immunology | Toll-Like Receptor 4 - metabolism | Mice, Knockout | Receptors, Interleukin-1 - metabolism | Nitric Oxide - secretion | Animals | Mannose-Binding Lectins - immunology | Signal Transduction - drug effects | Lipopolysaccharides - pharmacology | Toll-Like Receptor 2 - immunology | Mice | Toll-Like Receptor 1 - immunology | Macrophage Activation - drug effects | Myeloid Differentiation Factor 88 - metabolism | Index Medicus | Toll-Like Receptor 1 | Toll-Like Receptor 2 | Toll-Like Receptor 4 | Cell Adhesion Molecules | Receptors, Cell Surface | Macrophages | Lipopolysaccharides | Life Sciences | Immunology | Membrane Glycoproteins | Mannose-Binding Lectins | Mycobacterium bovis | Signal Transduction | Lectins, C-Type | Inflammation | Myeloid Differentiation Factor 88 | Nitric Oxide | Tumor Necrosis Factor-alpha | Immunity, Natural | Macrophage Activation | Mycobacterium tuberculosis | Receptors, Interleukin-1 | Interleukin-12 Subunit p40
Journal Article
Gastroenterology (New York, N.Y. 1943), ISSN 0016-5085, 06/2014, Volume 146, Issue 7, pp. 1763 - 1774
.... Toll-like receptors (TLRs) provide the first signal required for activation of the inflammasome and stimulate aerobic glycolysis to generate lactate... 
Gastroenterology and Hepatology | Innate Immune Response | Immune Regulation | Pancreas | Mouse Model | Gastroenterology & Hepatology | Life Sciences & Biomedicine | Science & Technology | Liver - pathology | Inflammasomes - metabolism | Receptors, G-Protein-Coupled - metabolism | NLR Family, Pyrin Domain-Containing 3 Protein | Humans | Male | NF-kappa B - metabolism | Monocytes - immunology | Lipopolysaccharides | Liver - immunology | Liver - drug effects | RNA Interference | Interleukin-1beta - metabolism | Toll-Like Receptors - drug effects | Anti-Inflammatory Agents - administration & dosage | Toll-Like Receptors - metabolism | Cytoprotection | Disease Models, Animal | Galactosamine | Chemical and Drug Induced Liver Injury - prevention & control | Anti-Inflammatory Agents - pharmacology | Down-Regulation | Liver - metabolism | Injections, Intraperitoneal | Pancreas - pathology | Pancreas - metabolism | Pancreas - immunology | Toll-Like Receptor 4 - metabolism | Chemical and Drug Induced Liver Injury - immunology | Monocytes - drug effects | Macrophages - metabolism | Signal Transduction - drug effects | Chemical and Drug Induced Liver Injury - metabolism | Sodium Lactate - pharmacology | beta-Arrestins | Mice | Receptors, G-Protein-Coupled - genetics | RNA, Small Interfering - metabolism | Monocytes - metabolism | Pancreatitis - prevention & control | Arrestins - metabolism | Dose-Response Relationship, Drug | Pancreatitis - genetics | Transfection | Inflammasomes - drug effects | Sodium Lactate - administration & dosage | Pancreatitis - immunology | Chemical and Drug Induced Liver Injury - pathology | Chemical and Drug Induced Liver Injury - etiology | Macrophages - immunology | Cell Line | Immunity, Innate - drug effects | Toll-Like Receptor 4 - drug effects | Mice, Inbred C57BL | Pancreas - drug effects | Chemical and Drug Induced Liver Injury - genetics | Pancreatitis - chemically induced | Animals | Carrier Proteins - metabolism | beta-Arrestin 2 | Inflammasomes - immunology | Macrophages - drug effects | Pancreatitis - pathology | Pancreatitis - metabolism | Ceruletide | Lactates | Gastrointestinal diseases | Inflammation | Index Medicus | Abridged Index Medicus
Journal Article
The Journal of immunology (1950), ISSN 1550-6606, 07/2010, Volume 185, Issue 2, pp. 929 - 942
Journal Article
The Journal of biological chemistry, ISSN 1083-351X, 06/2007, Volume 282, Issue 26, pp. 18810 - 18818
Journal Article
Endocrinology (Philadelphia), ISSN 1945-7170, 07/2011, Volume 152, Issue 7, pp. 2827 - 2836
... in the testicular interstitium, initiate innate immunity through the activation of Toll-like receptors (TLRs... 
Life Sciences & Biomedicine | Endocrinology & Metabolism | Science & Technology | Leydig Cells - cytology | Cholesterol Side-Chain Cleavage Enzyme - metabolism | Testis - metabolism | Male | Leydig Cells - immunology | Leydig Cells - drug effects | Toll-Like Receptor 3 - antagonists & inhibitors | Phosphoproteins - metabolism | RNA, Messenger - metabolism | Leydig Cells - metabolism | Mice, Mutant Strains | c-Mer Tyrosine Kinase | Testis - immunology | Toll-Like Receptors - antagonists & inhibitors | Toll-Like Receptor 4 - agonists | Toll-Like Receptor 3 - genetics | Toll-Like Receptor 4 - antagonists & inhibitors | Toll-Like Receptors - metabolism | Cytokines - genetics | Proto-Oncogene Proteins - metabolism | Cytokines - metabolism | Testis - cytology | Mice, Inbred C57BL | Cells, Cultured | Toll-Like Receptor 4 - genetics | Proto-Oncogene Proteins - genetics | Toll-Like Receptor 3 - agonists | Immunity, Innate | Receptor Protein-Tyrosine Kinases - metabolism | Toll-Like Receptor 3 - metabolism | Toll-Like Receptor 4 - metabolism | Toll-Like Receptors - agonists | Gene Expression Regulation - drug effects | Animals | Receptor Protein-Tyrosine Kinases - genetics | Toll-Like Receptors - genetics | Mice | Steroid 17-alpha-Hydroxylase - metabolism | Immune response | Data processing | Inflammation | TLR4 protein | Immunity | TLR3 protein | Axl protein | Lipopolysaccharides | Infection | Interleukin 6 | beta -Interferon | Interleukin 1 | Leydig cells | Toll-like receptors | Protein-tyrosine kinase receptors | alpha -Interferon | Interferon | Steroidogenesis | Testes | Tumor necrosis factor- alpha | Index Medicus | Abridged Index Medicus
Journal Article
Journal of investigative dermatology, ISSN 0022-202X, 02/2007, Volume 127, Issue 2, pp. 331 - 341
.... Pathogen-associated molecular patterns (PAMPs) are recognized, among others, by Toll-like receptors (TLRs... 
Life Sciences & Biomedicine | Dermatology | Science & Technology | Oligonucleotides - genetics | Phosphorylation | Toll-Like Receptor 5 - genetics | Humans | Toll-Like Receptor 9 - genetics | I-kappa B Proteins - metabolism | RNA, Messenger - metabolism | Flagellin - pharmacology | Tissue Distribution | Cell Nucleus - metabolism | Protein Isoforms - metabolism | Biological Transport | Oligonucleotides - pharmacology | Toll-Like Receptor 3 - genetics | Toll-Like Receptors - metabolism | Chemokine CXCL9 | Toll-Like Receptor 5 - metabolism | Poly I-C - pharmacology | Cytokines - metabolism | Cells, Cultured | Toll-Like Receptor 4 - genetics | Toll-Like Receptor 3 - metabolism | Toll-Like Receptor 4 - metabolism | Chemokine CXCL10 | Transcription Factor RelA - metabolism | Keratinocytes - metabolism | Toll-Like Receptors - genetics | Chemokines, CXC - metabolism | Lipopolysaccharides - pharmacology | CpG Islands | Ligands | Chemokines - metabolism | Toll-Like Receptor 9 - metabolism | CD40 antigen | Oligonucleotides | Flagellin | mRNA | intercellular adhesion molecule 1 | Immunity | Lipopolysaccharides | CXC chemokines | CCL20 protein | Toll-like receptors | CpG islands | TLR7 protein | Keratinocytes | Nuclear transport | Stress | TLR3 protein | TLR9 protein | TLR1 protein | External stimuli | CXCL10 protein | Histocompatibility antigen HLA | Interferon | Internet | Monocyte chemoattractant protein 1 | Tumor necrosis factor- alpha | Index Medicus
Journal Article