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Free Radical Biology and Medicine, ISSN 0891-5849, 08/2014, Volume 73, pp. 51 - 59
Microglia are the resident immune cells in the brain. Microglial activation is characteristic of several inflammatory and neurodegenerative diseases including... 
Peroxynitrite | Free radicals | Protein radical | Inducible nitric oxide synthase | Lipopolysaccharide | Nitrone adducts | Parkinson disease | Microglia | NADPH OXIDASE | OXIDATIVE STRESS | MACROPHAGES | BIOCHEMISTRY & MOLECULAR BIOLOGY | INOS EXPRESSION | KAPPA-B | LIPOPOLYSACCHARIDE-ACTIVATED MICROGLIA | REACTIVE OXYGEN | ENDOCRINOLOGY & METABOLISM | GENERATION | TYROSINE NITRATION | PARKINSONS-DISEASE | Microglia - metabolism | Metalloporphyrins - pharmacology | Lipopolysaccharides | Thiocarbamates - pharmacology | RNA Interference | Nitric Oxide Synthase Type II - antagonists & inhibitors | Spin Trapping | Acetophenones - pharmacology | Free Radicals - metabolism | Proline - pharmacology | Microglia - cytology | NG-Nitroarginine Methyl Ester - pharmacology | NF-kappa B - antagonists & inhibitors | Proline - analogs & derivatives | Enzyme Inhibitors - pharmacology | Imidazoles - pharmacology | Antioxidants - pharmacology | Neurodegenerative Diseases - metabolism | Coumarins - pharmacology | Animals | Nitric Oxide Synthase Type II - genetics | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Peroxynitrous Acid - metabolism | Amidines - pharmacology | Mice | Pyridines - pharmacology | RNA, Small Interfering | Benzylamines - pharmacology | Cell Line, Transformed | Boronic Acids - pharmacology | Nitric Oxide Synthase Type II - metabolism | Nitration | Nervous system diseases | Multiple sclerosis | Analysis | Nitric oxide | Environmental health | Physiological aspects | Alzheimer's disease | Index Medicus | Inducible nitric | oxide synthase | LPS | Parkinson’s disease
Journal Article
International Journal of Oncology, ISSN 1019-6439, 12/2013, Volume 43, Issue 6, pp. 1787 - 1798
The highly aggressive adult sarcomas are characterized by high levels of matrix metalloproteinase (MMP)-2 and -9, which play crucial roles in tumor invasion... 
inhibitors | synovial sarcoma | inducers | chondrosarcoma | cytokines | liposarcoma | matrix metalloproteinases | fibrosarcoma | Chondrosarcoma | Inhibitors | Matrix metalloproteinases | Cytokines | Synovial sarcoma | Liposarcoma | Fibrosarcoma | Inducers | CANCER-CELLS | VITAMIN-C | TUMOR INVASION | ASCORBIC-ACID | GREEN TEA | ONCOLOGY | NUTRIENT MIXTURE | SERUM-LEVELS | SOFT-TISSUE SARCOMA | ENDOTHELIAL GROWTH-FACTOR | Interleukin-1beta - pharmacology | Tetradecanoylphorbol Acetate - pharmacology | Humans | Fibrosarcoma - metabolism | Liposarcoma - metabolism | Matrix Metalloproteinase 9 - metabolism | Carcinogens - pharmacology | Dexamethasone - pharmacology | Antineoplastic Agents - pharmacology | Catechin - pharmacology | Matrix Metalloproteinase 2 - biosynthesis | Tretinoin - pharmacology | Matrix Metalloproteinase 9 - biosynthesis | Anti-Inflammatory Agents - pharmacology | Matrix Metalloproteinase 2 - metabolism | Sarcoma, Synovial - metabolism | Antioxidants - pharmacology | Sarcoma - metabolism | Matrix Metalloproteinase Inhibitors - pharmacology | Tumor Necrosis Factor-alpha - pharmacology | Chondrosarcoma - metabolism | Lipopolysaccharides - pharmacology | Cell Line, Tumor | Doxycycline - pharmacology | Anti-Bacterial Agents - pharmacology | Catechin - analogs & derivatives | Dactinomycin - pharmacology | Studies | Tea | Cell growth | Medical prognosis | Collagen | Penicillin | Tumor necrosis factor-TNF | Physiology | Metastasis | Growth factors | Cancer
Journal Article
European Journal of Immunology, ISSN 0014-2980, 12/2012, Volume 42, Issue 12, pp. 3334 - 3345
Extracellular ATP, released upon microbial infection, cell damage, or inflammation, acts as an alert signal toward immune cells by activating P2 receptors. The... 
Extracellular ATP | IL‐18 | P2X7 | P2 receptors | Human macrophages | IL-18 | DENDRITIC CELLS | CASPASE-1 | IFN-GAMMA | GAMMA-INDUCING FACTOR | IMMUNOLOGY | POLYMYXIN-B | HUMAN BLOOD | IL-1-BETA | TNF-ALPHA | SECRETION | ATP | Polymyxin B - pharmacology | Interleukin-18 - immunology | Tetrazoles - pharmacology | Humans | 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine - analogs & derivatives | Acetamides - pharmacology | Purinergic P2X Receptor Antagonists - pharmacology | Quinolines - pharmacology | Adenosine Triphosphate - pharmacology | Calcium Ionophores - pharmacology | Cell-Derived Microparticles - metabolism | Receptors, Purinergic P2X7 - immunology | Macrophages - immunology | Receptors, Purinergic P2X4 - immunology | Protein Precursors - immunology | Toll-Like Receptor 4 - immunology | Sulfonamides - pharmacology | Calcimycin - pharmacology | Purinergic P2X Receptor Agonists - pharmacology | Toll-Like Receptor 4 - metabolism | Adenosine Triphosphate - analogs & derivatives | Protein Precursors - metabolism | Macrophages - metabolism | Cell-Derived Microparticles - immunology | 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine - pharmacology | Lipopolysaccharides - pharmacology | Receptors, Purinergic P2X7 - metabolism | Anti-Bacterial Agents - pharmacology | Pyridines - pharmacology | Receptors, Purinergic P2X4 - metabolism | Interleukin-18 - metabolism | Macrophages | Ionophores | Rodents | Index Medicus
Journal Article
British Journal of Pharmacology, ISSN 0007-1188, 08/2015, Volume 172, Issue 15, pp. 3805 - 3816
Journal Article
British Journal of Pharmacology, ISSN 0007-1188, 08/2011, Volume 163, Issue 7, pp. 1533 - 1549
BACKGROUND AND PURPOSE Endocannabinoids have both anti‐inflammatory and neuroprotective properties against harmful stimuli. We previously demonstrated that the... 
monoacylglycerol lipase | peroxisome proliferator‐activated receptor‐γ | nuclear factor κ‐B | endocannabinoids | neuroinflammation | cyclooxygenase‐2 | nuclear factor κ-B | peroxisome proliferator-activated receptor-γ | cyclooxygenase-2 | CB1 CANNABINOID RECEPTORS | 2-ARACHIDONYL GLYCEROL | NUCLEAR RECEPTORS | SYNAPTIC-TRANSMISSION | MICROGLIAL CELLS | KAPPA-B | OXIDATIVE-METABOLISM | peroxisome proliferator-activated receptor-gamma | PROTECTS NEURONS | nuclear factor kappa-B | ACTIVATED-RECEPTOR-GAMMA | PHARMACOLOGY & PHARMACY | Cannabinoid Receptor Modulators - pharmacology | Interleukin-1beta - pharmacology | Monoacylglycerol Lipases - antagonists & inhibitors | NF-kappa B - metabolism | Hippocampus - drug effects | PPAR gamma - metabolism | Excitatory Postsynaptic Potentials - drug effects | Cyclooxygenase 2 - biosynthesis | Inflammation - metabolism | Biphenyl Compounds - pharmacology | Piperidines - pharmacology | Cyclooxygenase 2 - genetics | Prostaglandin D2 - analogs & derivatives | Synaptic Transmission - drug effects | Neurons - metabolism | Phosphorylation - drug effects | Neurons - drug effects | Thiazolidinediones - pharmacology | Endocannabinoids | PPAR gamma - genetics | NF-kappa B - antagonists & inhibitors | Cyclooxygenase 2 Inhibitors - pharmacology | Cells, Cultured | Arachidonic Acids - pharmacology | Hippocampus - metabolism | Receptor, Cannabinoid, CB1 - metabolism | Glycerides - pharmacology | Animals | PPAR gamma - antagonists & inhibitors | Signal Transduction - drug effects | Anilides - pharmacology | Cyclooxygenase 2 - metabolism | Lipopolysaccharides - pharmacology | PPAR gamma - agonists | Mice | Prostaglandin D2 - pharmacology | Receptor, Cannabinoid, CB1 - antagonists & inhibitors | Benzodioxoles - pharmacology | Index Medicus | Research Papers
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 03/1998, Volume 101, Issue 5, pp. 1163 - 1174
Transcription factors of the NF-kappa B/Rel family are critical for inducible expression of multiple genes involved in inflammatory responses, Sulfasalazine... 
Inflammatory bowel disease | NF-κB | Therapy | SW620 | AP1 | RHEUMATOID-ARTHRITIS | MEDICINE, RESEARCH & EXPERIMENTAL | TRANSCRIPTIONAL ACTIVATION | METABOLITES | PHOSPHORYLATION | therapy | ULCERATIVE-COLITIS | ACTIVE MOIETY | ALPHA | CELL-LINES | 5-AMINOSALICYLIC ACID | inflammatory bowel disease | NF-kappa B | SODIUM-SALICYLATE | Phosphorylation | Anti-Infective Agents - pharmacology | Humans | Tumor Necrosis Factor-alpha - genetics | NF-kappa B - metabolism | Sulfasalazine - pharmacology | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Cystine - pharmacology | Thiocarbamates - pharmacology | Transfection | Recombination, Genetic | Phorbol Esters - pharmacology | Proline - pharmacology | Colonic Diseases - drug therapy | Aspirin - pharmacology | Cystine - analogs & derivatives | Fluorescent Antibody Technique, Indirect | NF-kappa B - antagonists & inhibitors | Proline - analogs & derivatives | Anti-Inflammatory Agents - pharmacology | Cells, Cultured | Epithelial Cells | Antioxidants - pharmacology | Recombinant Proteins - pharmacology | Blotting, Western | Sulfapyridine - pharmacology | Tumor Necrosis Factor-alpha - pharmacology | Colonic Diseases - immunology | Immunosuppression | Mesalamine - pharmacology | Transcription Factor AP-1 - antagonists & inhibitors | NF-kappa B - genetics | Plasmids | Lipopolysaccharides - pharmacology | Index Medicus | Abridged Index Medicus
Journal Article
Oncology Reports, ISSN 1021-335X, 03/2017, Volume 37, Issue 3, pp. 1907 - 1913
Brain tumors are highly aggressive, characterized by the secretion of high levels of matrix metalloproteinase (MMP)-2 and MMP-9 that degrade the extracellular... 
Inhibitors | Matrix metalloproteinases | Cytokines | Glioblastoma T-98G | Inducers | VITAMIN-C | GLIOMA-CELLS | inhibitors | CANCER-RELATED INFLAMMATION | glioblastoma T-98G | N-ACETYLCYSTEINE | LINES | GREEN TEA | BRAIN-TUMOR | IN-VITRO | ONCOLOGY | inducers | GROWTH | cytokines | matrix metalloproteinases | TUMOR MICROENVIRONMENT | Enzyme Activators - pharmacology | Gene Expression Regulation, Enzymologic - drug effects | Glioblastoma - enzymology | Interleukin-1beta - pharmacology | Tetradecanoylphorbol Acetate - pharmacology | Humans | Carcinogens - pharmacology | Cattle | Matrix Metalloproteinase 9 - secretion | Antineoplastic Agents - pharmacology | Gene Expression Regulation, Neoplastic - drug effects | Tumor Cells, Cultured | Catechin - pharmacology | Tretinoin - pharmacology | Antioxidants - pharmacology | Matrix Metalloproteinase Inhibitors - pharmacology | Tumor Necrosis Factor-alpha - pharmacology | Animals | Glioblastoma - pathology | Lipopolysaccharides - pharmacology | Doxycycline - pharmacology | Matrix Metalloproteinase 2 - secretion | Anti-Bacterial Agents - pharmacology | Catechin - analogs & derivatives | Glioblastoma - drug therapy | Cytokines - pharmacology | Enzymes | Care and treatment | Proteases | Development and progression | Genetic aspects | Regulation | Health aspects | Glioblastoma multiforme | Angiogenesis | Acids | Collagen | Brain cancer | Penicillin | Tumor necrosis factor-TNF | Nervous system | Metastasis | Cancer therapies | Tumors | Apoptosis | Index Medicus
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 02/2009, Volume 296, Issue 2, pp. 470 - 479
Bacterial endotoxin lipopolysaccharide (LPS) is responsible for the multiorgan dysfunction that characterizes septic shock and is causal in the myocardial... 
Green fluorescent protein- Microtubule-associated protein light chain 3 | Lipopolysaccharide | Oxidative stress | Hl-1 cardiac myocyte | TRANSCRIPTIONAL ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | HL-1 cardiac myocyte | CARDIAC MYOCYTES | NECROSIS-FACTOR-ALPHA | MYOCARDIAL DEPRESSION | PROGRAMMED CELL-DEATH | ENDOTOXEMIC RATS | SCAVENGER RECEPTOR | green fluorescent protein-microtubule-associated protein light chain 3 | SEPTIC SHOCK | PERIPHERAL VASCULAR DISEASE | TNF-ALPHA | lipopolysaccharide | NF-KAPPA-B | oxidative stress | Tumor Necrosis Factor-alpha - metabolism | Mitochondria, Heart - metabolism | Glutathione - metabolism | Mitochondria, Heart - pathology | Nitric Oxide Synthase - antagonists & inhibitors | omega-N-Methylarginine - pharmacology | Mitochondria, Heart - drug effects | Nitroprusside - pharmacology | Autophagy - drug effects | p38 Mitogen-Activated Protein Kinases - metabolism | Nitric Oxide Donors - pharmacology | Cytoprotection | Animals, Newborn | Cells, Cultured | Enzyme Inhibitors - pharmacology | Rats | Mice, Transgenic | Imidazoles - pharmacology | Tyrphostins - pharmacology | Antioxidants - pharmacology | Sirolimus - pharmacology | Hydrogen Peroxide - metabolism | Myocytes, Cardiac - pathology | Animals | Myocytes, Cardiac - drug effects | Signal Transduction - drug effects | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Acetylcysteine - pharmacology | Lipopolysaccharides - pharmacology | Myocytes, Cardiac - metabolism | Mice | Nitric Oxide Synthase - metabolism | Pyridines - pharmacology | Oxidative Stress - drug effects | Nitric Oxide - metabolism | Signal transduction | Bacteria | Oxidation | Biochemistry | Cytokines | Sugar | Index Medicus
Journal Article
British Journal of Pharmacology, ISSN 0007-1188, 2015, Volume 172, Issue 1, pp. 24 - 37
Background and Purpose N-docosahexaenoylethanolamine (DHEA) is the ethanolamine conjugate of the long-chain polyunsaturated n-3 fatty acid docosahexaenoic... 
PLASMA | CONCISE GUIDE | NITRIC-OXIDE SYNTHASE | INFLAMMATION | OXYGENATION | ANANDAMIDE | PHARMACOLOGY & PHARMACY | FATTY-ACIDS | FISH-OIL | KAPPA-B | ETHANOLAMIDE | Endocannabinoids - metabolism | Bornanes - pharmacology | Male | NF-kappa B - metabolism | Receptor, Cannabinoid, CB2 - antagonists & inhibitors | PPAR gamma - metabolism | Piperidines - pharmacology | Thiazolidinediones - pharmacology | Pyrazoles - pharmacology | Cell Line | Indenes - pharmacology | Cyclooxygenase 2 Inhibitors - pharmacology | Dehydroepiandrosterone - pharmacology | Mice, Inbred C57BL | Cells, Cultured | Toll-Like Receptor 3 - metabolism | Toll-Like Receptor 4 - metabolism | Arachidonic Acids - pharmacology | Mice, Knockout | Dinoprostone - metabolism | Receptor, Cannabinoid, CB1 - metabolism | Receptor, Cannabinoid, CB2 - metabolism | Macrophages - metabolism | Animals | PPAR gamma - antagonists & inhibitors | Interferon-beta - metabolism | Anilides - pharmacology | Cyclooxygenase 2 - metabolism | Lipopolysaccharides - pharmacology | Macrophages - drug effects | Mice | Receptor, Cannabinoid, CB1 - antagonists & inhibitors | Nitric Oxide - metabolism | Myeloid Differentiation Factor 88 - metabolism | Androgens | Dehydroepiandrosterone | DNA microarrays | Anti-inflammatory drugs | RNA | Ethanolamines | Macrophages | Gene expression | Fatty acids | Medical research | Rodents | Index Medicus | Research Papers | arrive guidelines | pharmacology | fatty-acids | cannabinoid receptor | prostaglandin e-2 | nitric-oxide synthase | anandamide | fish-oil | concise guide | kappa-b
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 10/2013, Volume 110, Issue 43, pp. 17253 - 17258
Phenotypic polarization of macrophages is regulated by a milieu of cues in the local tissue microenvironment. Although much is known about how soluble factors... 
Phenotypes | Cell growth | Cytokines | Stem cells | Cell lines | Actins | Physiological regulation | Macrophages | Endothelial cells | Cells | FIBER | MATRIX | ACTIVATION | ACTIN | MULTIDISCIPLINARY SCIENCES | IN-VIVO | DIFFERENTIATION | SCAFFOLDS | CULTURE | MESENCHYMAL STEM-CELLS | Mannose-Binding Lectins - metabolism | Arginase - metabolism | Lectins, C-Type - immunology | Cell Shape - immunology | Myosin-Light-Chain Kinase - antagonists & inhibitors | Cytochalasin D - pharmacology | Lectins, C-Type - metabolism | Arginase - immunology | Interleukin-4 - pharmacology | Flow Cytometry | Doxorubicin - analogs & derivatives | Inflammation Mediators - metabolism | Female | Immunophenotyping - methods | Cell Polarity - drug effects | Doxorubicin - metabolism | Macrophages - immunology | Cytokines - immunology | Amides - pharmacology | Biomarkers - metabolism | Inflammation Mediators - immunology | Cell Polarity - immunology | Cytokines - metabolism | Myosin-Light-Chain Kinase - metabolism | Mice, Inbred C57BL | Biomarkers - analysis | Cells, Cultured | Receptors, Cell Surface - metabolism | Macrophages - cytology | Interleukin-13 - pharmacology | Receptors, Cell Surface - immunology | Azepines - pharmacology | Cell Shape - drug effects | Macrophages - metabolism | Animals | Mannose-Binding Lectins - immunology | Lipopolysaccharides - pharmacology | Mice | Pyridines - pharmacology | Doxorubicin - immunology | Interferon-gamma - pharmacology | Microscopy, Fluorescence | Physiological aspects | Microbial genetics | Phenotype | Genetic aspects | Research | Geometry | Genotype & phenotype | Morphology | Index Medicus | Biological Sciences | Physical Sciences
Journal Article
Nature Chemical Biology, ISSN 1552-4450, 2014, Volume 10, Issue 8, pp. 656 - 663
Journal Article