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Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 4/2013, Volume 110, Issue 15, pp. 6021 - 6026
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2012, Volume 7, Issue 4, pp. e33208 - e33208
DDIT3, also known as GADD153 or CHOP, encodes a basic leucine zipper transcription factor of the dimer forming C/EBP family. DDIT3 is known as a key regulator... 
TRANSCRIPTION FACTOR CHOP | IN-VITRO | MYXOID LIPOSARCOMA | INDUCED APOPTOSIS | BIOLOGY | BINDING-PROTEIN | GENE-EXPRESSION | PANCREATIC BETA-CELLS | ENDOPLASMIC-RETICULUM STRESS | AMINO-ACID LIMITATION | C/EBP-HOMOLOGOUS PROTEIN | Transcription Factor CHOP - genetics | Cell Proliferation | Oligonucleotide Array Sequence Analysis | Humans | Cytoplasm - metabolism | Gene Expression Profiling | Green Fluorescent Proteins - genetics | Fibrosarcoma - metabolism | Promoter Regions, Genetic - genetics | Liposarcoma - metabolism | Cell Nucleus - metabolism | Flow Cytometry | Biomarkers, Tumor - metabolism | Real-Time Polymerase Chain Reaction | Fibroblasts - metabolism | Recombinant Proteins - metabolism | Antineoplastic Agents, Hormonal - pharmacology | Liposarcoma - drug therapy | RNA, Messenger - genetics | Cells, Cultured | Liposarcoma - genetics | Recombinant Proteins - genetics | Reverse Transcriptase Polymerase Chain Reaction | Cell Adhesion | Blotting, Western | Fibrosarcoma - genetics | Cell Cycle | Cell Nucleus - genetics | Fibroblasts - drug effects | Tamoxifen - pharmacology | Biomarkers, Tumor - genetics | Fibroblasts - cytology | Cell Nucleus - drug effects | Transcription Factor CHOP - metabolism | Cytoplasm - drug effects | Fibrosarcoma - drug therapy | Cell Movement | Genomes | Genetic transcription | DNA binding proteins | Tamoxifen | Genes | Genomics | Cell culture | Transcription factors | Target recognition | Amino acids | Biochemistry | CCAAT/enhancer-binding protein | Leucine | Kinases | Experiments | Proteins | Cell growth | Annotations | Cell cycle | Fibroblasts | Physiology | Localization | Stress response | Deoxyribonucleic acid--DNA | Translocation | Stresses | Cell survival | Cloning | Forming | Gene expression | Stress | Nuclear transport | Leucine zipper proteins | Pathology | DNA microarrays | Ligands | Prostate | Cell migration | CHOP protein | Apoptosis | Cancer | Index Medicus | cytology | Green Fluorescent Proteins | RNA | Hormonal | Genetic | Fibrosarcoma | Recombinant Proteins | Blotting | Tumor Markers | Western | genetics | Transcription Factor CHOP | pharmacology | Cultured | drug therapy | Promoter Regions | Antineoplastic Agents | drug effects | Messenger | Biological | Cells | Liposarcoma | metabolism | Cancer and Oncology | Cell Nucleus | Cytoplasm | Cancer och onkologi | Deoxyribonucleic acid | DNA
Journal Article
International Journal of Cancer, ISSN 0020-7136, 05/2008, Volume 122, Issue 10, pp. 2233 - 2241
Journal Article
Nature Communications, ISSN 2041-1723, 12/2018, Volume 9, Issue 1, pp. 2765 - 11
Myxofibrosarcoma (MFS) is a common adult soft tissue sarcoma characterized by an infiltrative growth pattern and a high local recurrence rate. Here we report... 
BREAST-CANCER | SURVIVAL | OVEREXPRESSION | DEDIFFERENTIATED LIPOSARCOMAS | MULTIDISCIPLINARY SCIENCES | PANCREATIC NEUROENDOCRINE TUMORS | ATRX | LUNG ADENOCARCINOMA | MUTATIONS | EXPRESSION | NERVE SHEATH TUMORS | Oncogene Proteins, Fusion - metabolism | Epigenesis, Genetic | Humans | Fibrosarcoma - mortality | Gene Expression Regulation, Neoplastic | X-linked Nuclear Protein - metabolism | Fibrosarcoma - metabolism | X-linked Nuclear Protein - genetics | Tumor Suppressor Protein p53 - genetics | Janus Kinase 1 - metabolism | Fibrosarcoma - pathology | Heterografts | Janus Kinase 1 - genetics | Neurofibromin 1 - metabolism | Monosaccharide Transport Proteins - metabolism | Proto-Oncogene Proteins B-raf - metabolism | Monosaccharide Transport Proteins - genetics | Fibroma - mortality | Tumor Suppressor Protein p53 - metabolism | Fibroma - genetics | Fibroma - pathology | Whole Exome Sequencing | Fibrosarcoma - genetics | Animals | Receptor, trkA - metabolism | Fibroma - metabolism | Mice, Nude | Oncogene Proteins, Fusion - genetics | Proto-Oncogene Proteins B-raf - genetics | Survival Analysis | Receptor, trkA - genetics | Mice | Mutation | Neurofibromin 1 - genetics | Genes, cdc | Cohort Studies | Sarcoma | Therapeutic applications | p53 Protein | Genes | Soft tissue sarcoma | Ribonucleic acid--RNA | Gene sequencing | Immune systems | Genetic analysis | Cell cycle | DNA methylation | Epigenetics | Janus kinase | Methylation | Fusion protein | Index Medicus
Journal Article
American Journal of Surgical Pathology, ISSN 0147-5185, 08/2008, Volume 32, Issue 8, pp. 1168 - 1174
Several previous studies have demonstrated the lack of SMARCB1/INI1 protein expression in only the malignant rhabdoid tumor (MRT). Several sarcoma groups are... 
EWS | Extraskeletal myxoid chondrosarcoma | Fusion gene transcript | SMARCB1/INI1 | Rhabdoid | SURGERY | BLADDER-CANCER | fusion gene transcript | AGGRESSIVE-BEHAVIOR | TIME QUANTITATIVE PCR | PATHOLOGY | MALIGNANT RHABDOID TUMORS | extraskeletal myxoid chondrosarcoma | GENE | rhabdoid | IMMUNOHISTOCHEMICAL ANALYSIS | CENTRAL-NERVOUS-SYSTEM | HSNF5/INI1 | MUTATIONS | ATYPICAL TERATOID/RHABDOID TUMORS | Immunohistochemistry | Translocation, Genetic | Neuroectodermal Tumors, Primitive - genetics | Prognosis | Chondrosarcoma - mortality | Humans | Gene Expression Regulation, Neoplastic | Rhabdoid Tumor - mortality | Sarcoma, Clear Cell - chemistry | DNA-Binding Proteins - analysis | Neuroectodermal Tumors, Primitive - chemistry | Rhabdoid Tumor - pathology | SMARCB1 Protein | Sarcoma - mortality | Chondrosarcoma - genetics | Chondrosarcoma - pathology | Liposarcoma, Myxoid - genetics | Sarcoma - genetics | Sarcoma, Clear Cell - genetics | Reproducibility of Results | Sarcoma, Ewing - chemistry | Down-Regulation | RNA-Binding Protein EWS - genetics | Rhabdoid Tumor - chemistry | Chondrosarcoma - chemistry | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Sarcoma - pathology | Chromosomal Proteins, Non-Histone - genetics | Sarcoma - chemistry | Rhabdoid Tumor - genetics | Oncogene Proteins, Fusion - genetics | Chromosomal Proteins, Non-Histone - analysis | Transcription Factors - analysis | Sarcoma, Ewing - genetics | Liposarcoma, Myxoid - chemistry | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 2012, Volume 7, Issue 11, pp. e48983 - e48983
Journal Article
BMC Genomics, ISSN 1471-2164, 03/2007, Volume 8, Issue 1, pp. 73 - 73
Journal Article