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Cell, ISSN 0092-8674, 02/2012, Volume 148, Issue 3, pp. 434 - 446
Journal Article
Journal Article
Scientific Reports, ISSN 2045-2322, 12/2017, Volume 7, Issue 1, pp. 3153 - 11
Formyl peptide receptor 2 (FPR2), a classical chemoattractant receptor of G-protein-coupled receptors, is reported to be involved in invasion and metastasis of... 
INVASIVENESS | ACTIVATE | ANNEXIN A1 EXPRESSION | PROTEIN | TUMOR PROGRESSION | MULTIDISCIPLINARY SCIENCES | DISEASE | FORMYL PEPTIDE-RECEPTOR | IDENTIFICATION | HELICOBACTER-PYLORI | LIPOXIN A RECEPTOR | RNA, Small Interfering - genetics | Phosphorylation | Prognosis | Cadherins - metabolism | Vimentin - metabolism | Humans | Middle Aged | Receptors, Formyl Peptide - antagonists & inhibitors | Receptors, Lipoxin - antagonists & inhibitors | Gene Expression Regulation, Neoplastic | Male | Stomach Neoplasms - pathology | Neoplasm Recurrence, Local - mortality | Antigens, CD - genetics | Antigens, CD - metabolism | Neoplasm Recurrence, Local - pathology | Neoplasm Grading | Vimentin - genetics | Mitogen-Activated Protein Kinase 1 - genetics | Epithelial-Mesenchymal Transition | Receptors, Formyl Peptide - metabolism | Biomarkers, Tumor - metabolism | Female | Receptors, Lipoxin - genetics | Cadherins - genetics | Receptors, Lipoxin - metabolism | Stomach Neoplasms - genetics | Stomach Neoplasms - diagnosis | Mitogen-Activated Protein Kinase 3 - genetics | Signal Transduction | Lymphatic Metastasis | Neoplasm Recurrence, Local - diagnosis | Xenograft Model Antitumor Assays | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | Mice, Nude | Survival Analysis | Cell Line, Tumor | Neoplasm Recurrence, Local - genetics | Aged | Biomarkers, Tumor - genetics | Mice | Neoplasm Staging | Stomach Neoplasms - mortality | Receptors, Formyl Peptide - genetics | Mitogen-Activated Protein Kinase 1 - metabolism | RNA, Small Interfering - metabolism | Vimentin | G protein-coupled receptors | Mesenchyme | Extracellular signal-regulated kinase | Metastasis | Multivariate analysis | Stomach cancer | E-cadherin | Lymph nodes | Metastases | Clonal deletion | Medical prognosis | Down-regulation | Ligands | Gastric cancer
Journal Article
Journal of Virology, ISSN 0022-538X, 2016, Volume 90, Issue 24, pp. 11020 - 11031
Lipoxins are host anti-inflammatory molecules that play a vital role in restoring tissue homeostasis. The efficacy of lipoxins and their analog epilipoxins in... 
VIROLOGY | KSHV | TARGET-CELLS | INFLAMMATION | GENE-EXPRESSION | INFECTION | LATENCY | RECEPTORS | IDENTIFICATION | MICRORNA | Arachidonate 5-Lipoxygenase - genetics | Humans | NF-kappa B - immunology | Lipoxins - pharmacology | Lymphoma, Primary Effusion - virology | Proto-Oncogene Proteins c-akt - genetics | Arachidonate 5-Lipoxygenase - immunology | Herpesvirus 8, Human - immunology | RNA, Viral - genetics | Herpesvirus 8, Human - pathogenicity | Cyclooxygenase 2 - genetics | Mitogen-Activated Protein Kinase 1 - genetics | Adaptor Proteins, Signal Transducing - immunology | Sarcoma, Kaposi - immunology | Lymphoma, Primary Effusion - genetics | Cyclooxygenase 2 - immunology | Cell Line | Mitogen-Activated Protein Kinase 3 - immunology | Mitogen-Activated Protein Kinase 1 - immunology | Mitogen-Activated Protein Kinase 3 - genetics | Signal Transduction | Anti-Inflammatory Agents - pharmacology | Gene Expression Regulation | Herpesvirus 8, Human - genetics | Lymphoma, Primary Effusion - pathology | Inflammation | Sarcoma, Kaposi - virology | MicroRNAs - immunology | Sarcoma, Kaposi - pathology | Endothelial Cells - immunology | Host-Pathogen Interactions | Proto-Oncogene Proteins c-akt - immunology | Lymphoma, Primary Effusion - immunology | NF-kappa B - genetics | Adaptor Proteins, Signal Transducing - deficiency | Adaptor Proteins, Signal Transducing - genetics | CRISPR-Cas Systems | RNA, Viral - immunology | MicroRNAs - genetics | Endothelial Cells - pathology | Sarcoma, Kaposi - genetics | Endothelial Cells - drug effects
Journal Article
The Journal of Rheumatology, ISSN 0315-162X, 11/2007, Volume 34, Issue 11, pp. 2144 - 2153
Journal Article
Nutrition, ISSN 0899-9007, 2016, Volume 32, Issue 2, pp. 174 - 178
Abstract Flavonoid resveratrol modulates the transcription factor NF-κB; inhibits the cytochrome P450 isoenzyme CYP1 A1; suppresses the expression and activity... 
Gastroenterology and Hepatology | Obesity | Flavonoids | Microbiota | Type 2 diabetes mellitus | Alzheimer's disease | Resveratrol | CELLS | OXIDATIVE STRESS | RISK-FACTORS | MITOCHONDRIAL-FUNCTION | ENDOPLASMIC-RETICULUM STRESS | INDUCTION | AUTOPHAGY | SIRT1 | NUTRITION & DIETETICS | INSULIN-RESISTANCE | MOUSE MODEL | Autistic Disorder - chemically induced | Tumor Necrosis Factor-alpha - metabolism | Brain-Derived Neurotrophic Factor - genetics | Apoptosis - drug effects | Humans | Tumor Necrosis Factor-alpha - genetics | Cytochrome P-450 CYP1A1 - antagonists & inhibitors | Hypoxia-Inducible Factor 1, alpha Subunit - antagonists & inhibitors | Lipoxins - pharmacology | NF-kappa B - metabolism | Vascular Endothelial Growth Factor A - metabolism | Stilbenes - pharmacology | Metabolic Syndrome - chemically induced | Vascular Endothelial Growth Factor A - genetics | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Brain-Derived Neurotrophic Factor - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Diabetes Mellitus, Type 2 - prevention & control | Gastrointestinal Tract - microbiology | Interleukin-17 - genetics | Gastrointestinal Tract - drug effects | Antioxidants - pharmacology | Autistic Disorder - prevention & control | Transcription Factors - genetics | Cytochrome P-450 CYP1A1 - metabolism | Transcription Factors - metabolism | Interleukin-17 - metabolism | Gastrointestinal Microbiome - drug effects | Interleukin-17 - antagonists & inhibitors | NF-kappa B - genetics | Cell Differentiation - drug effects | Metabolic Syndrome - prevention & control | Cell Proliferation - drug effects | Diabetes Mellitus, Type 2 - chemically induced | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Type 2 diabetes | COX-2 inhibitors | Bisphenol-A | Endothelial growth factors | Microbiota (Symbiotic organisms) | Cytochrome P-450 | Schizophrenia | Bipolar disorder | T cells | Cell differentiation | Fatty acids | Autism | Stem cells | Tumor proteins | Pathogenesis | p53 Protein | Lipids | Proteins | Antioxidants | Signal transduction | Lymphocytes | Rodents | Oxidation | Growth factors | Cytochromes P450 | Diabetes mellitus | Rapamycin | Metabolism | Bisphenol A | Alloxan | Brain-derived neurotrophic factor | Insulin resistance | Hypoxia | Diabetes | Alzheimers disease | Autoimmune diseases | Metabolic disorders | Dementia | Apoptosis | Tumors
Journal Article
Journal of Leukocyte Biology, ISSN 0741-5400, 11/2015, Volume 98, Issue 6, pp. 897 - 912
Journal Article
Nutrition, ISSN 0899-9007, 2016, Volume 35, pp. 61 - 80
Abstract Objective The aim of this study was to observe whether polyunsaturated fatty acids (PUFAs) can protect rat insulinoma (RIN5 F) cells against... 
Gastroenterology and Hepatology | Antioxidants | Lipoxin A4 | Polyunsaturated fatty acids | Diabetes mellitus | Inflammation | Arachidonic acid | Streptozotocin | ALLOXAN-INDUCED CYTOTOXICITY | INTERLEUKIN-6 | LIPOCALIN-2 | TUMOR-NECROSIS-FACTOR | OBESITY | NUTRITION & DIETETICS | INSULIN-RESISTANCE | CLINICAL-IMPLICATIONS | NITRIC-OXIDE | POLYUNSATURATED FATTY-ACIDS | Tumor Necrosis Factor-alpha - metabolism | Glucose Transporter Type 2 - genetics | Diabetes Mellitus, Experimental - drug therapy | Rats, Wistar | Apoptosis - drug effects | Homeodomain Proteins - metabolism | Pancreas - cytology | Tumor Necrosis Factor-alpha - genetics | Lipoxins - pharmacology | NF-kappa B - metabolism | Glucose Transporter Type 2 - metabolism | Eicosapentaenoic Acid - pharmacology | Cyclooxygenase 2 - genetics | Trans-Activators - genetics | NF-E2-Related Factor 2 - genetics | Diabetes Mellitus, Experimental - chemically induced | Cell Line | Pancreas - drug effects | Rats | Homeodomain Proteins - genetics | Diabetes Mellitus, Type 1 - drug therapy | Docosahexaenoic Acids - pharmacology | Animals | NF-kappa B - genetics | Nitric Oxide Synthase Type II - genetics | NF-E2-Related Factor 2 - metabolism | Arachidonic Acid - pharmacology | Cyclooxygenase 2 - metabolism | gamma-Linolenic Acid - pharmacology | Trans-Activators - metabolism | Blood Glucose - metabolism | Diabetes Mellitus, Type 2 - drug therapy | Nitric Oxide Synthase Type II - metabolism | Type 2 diabetes | Unsaturated fatty acids | Streptozocin | Essential fatty acids | Obesity | Free radicals | Metabolites | Cytokines | Rodents | Lipids | Cytotoxicity | Tumor necrosis factor-TNF | Diabetes | Fatty acids
Journal Article
American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 04/2012, Volume 302, Issue 7, pp. 968 - 978
Lipoxins are formed by leukocytes during cell-cell interactions with epithelial or endothelial cells. Native lipoxin A4 (LXA4) binds to the G protein-coupled... 
Endothelium, Vascular - cytology | Tumor Necrosis Factor-alpha - metabolism | Human Umbilical Vein Endothelial Cells - metabolism | Humans | Receptors, Interleukin-1 - genetics | Tumor Necrosis Factor-alpha - genetics | Submandibular Gland - drug effects | Cell Communication - physiology | Endothelium, Vascular - drug effects | Lipoxins - pharmacology | Interleukin-1beta - genetics | CD4-Positive T-Lymphocytes - immunology | E-Selectin - genetics | Interleukin-1beta - metabolism | Receptors, Formyl Peptide - metabolism | Vascular Cell Adhesion Molecule-1 - genetics | Receptors, Lipoxin - genetics | Receptors, Lipoxin - metabolism | Endothelium, Vascular - immunology | Receptors, Tumor Necrosis Factor - genetics | Receptors, Tumor Necrosis Factor - metabolism | Cell Adhesion - genetics | Jurkat Cells | CD4-Positive T-Lymphocytes - cytology | CD4-Positive T-Lymphocytes - metabolism | Cells, Cultured | Submandibular Gland - metabolism | E-Selectin - metabolism | Cell Communication - genetics | Receptors, Leukotriene - genetics | Receptors, Leukotriene - metabolism | Receptors, Interleukin-1 - metabolism | Up-Regulation - drug effects | Intercellular Adhesion Molecule-1 - metabolism | Cell Adhesion - physiology | Endothelium, Vascular - metabolism | Intercellular Adhesion Molecule-1 - genetics | Adaptor Proteins, Signal Transducing - genetics | Submandibular Gland - cytology | Adaptor Proteins, Signal Transducing - metabolism | Submandibular Gland - immunology | Vascular Cell Adhesion Molecule-1 - metabolism | Receptors, Formyl Peptide - genetics | E-selectin | intercellular cell adhesion molecule-1 | Translational Physiology | ALX receptor | vascular cell adhesion molecule-1
Journal Article
The FASEB Journal, ISSN 0892-6638, 07/2014, Volume 28, Issue 7, pp. 3090 - 3102
Journal Article
PLoS ONE, ISSN 1932-6203, 12/2016, Volume 11, Issue 12, p. e0168418
In high grade glioma (HGG), extensive tumor cell infiltration of normal brain typically precludes identifying effective margins for surgical resection or... 
GROWTH-FACTOR RECEPTOR | MULTIDISCIPLINARY SCIENCES | RESISTANCE | TUMOR | UROKINASE RECEPTOR | MICE | UPAR | PLASMINOGEN-ACTIVATOR RECEPTOR | EGF RECEPTOR | MUTANT EGFR | CANCER STEM-CELLS | Cell Survival - drug effects | Receptor, Epidermal Growth Factor - genetics | Cell Proliferation - genetics | Receptors, G-Protein-Coupled - metabolism | Humans | Cell Survival - genetics | Cell Movement - genetics | Glioma - metabolism | Glioma - genetics | Cell Movement - drug effects | Receptor, Epidermal Growth Factor - metabolism | Receptors, Formyl Peptide - metabolism | Cell Line, Tumor | Cell Proliferation - drug effects | Receptors, G-Protein-Coupled - genetics | Receptors, Lipoxin - genetics | Receptors, Lipoxin - metabolism | Microscopy, Fluorescence | Receptors, Formyl Peptide - genetics | Pertussis Toxin - pharmacology | Care and treatment | Gliomas | Physiological aspects | Development and progression | Genetic aspects | Research | Metastasis | Pertussis toxin | Vimentin | Cell proliferation | Brain | Membranes | G protein-coupled receptors | Mesenchyme | Radiation | Metastases | Cell adhesion & migration | Proteins | Pertussis | Urokinase | Receptors | Epidermal growth factor | Glioma cells | Surgery | Neurospheres | Inhibition | Cell survival | Epidermal growth factor receptors | Gene expression | Whooping cough | U-Plasminogen activator | Pathology | Signaling | Gene amplification | Inhibitors | Stem cells | Irradiation | Infiltration | Cell migration | Cancer | Neutralization
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 12/2014, Volume 111, Issue 52, pp. 18685 - 18690
Sepsis is characterized by overlapping phases of excessive inflammation temporally aligned with an immunosuppressed state, defining a complex clinical scenario... 
Receptors | Monocytes | Cytokines | Neutrophils | Sepsis | Bacteria | Agonists | Mice | Inflammation | Phagocytosis | Annexin peptide | Therapeutic innovation | Resolution of inflammation | ALX | Cardiac dysfunction | LIPOXIN A | FORMYL-PEPTIDE RECEPTOR | CLINICAL-TRIAL | MULTIDISCIPLINARY SCIENCES | RESOLUTION | MONOCLONAL-ANTIBODY | annexin peptide | resolution of inflammation | 2/LIPOXIN A RECEPTOR | EXPERIMENTAL INFLAMMATION | SEPTIC SHOCK | DOUBLE-BLIND | cardiac dysfunction | therapeutic innovation | BACTERIAL LOAD | Tumor Necrosis Factor-alpha - metabolism | Humans | Tumor Necrosis Factor-alpha - genetics | Monocytes - metabolism | Chemokine CXCL1 - genetics | Peritoneum - pathology | Sepsis - pathology | Time Factors | Sepsis - metabolism | Monocytes - pathology | Receptors, Formyl Peptide - metabolism | Granulocytes - pathology | Chemokine CCL2 - metabolism | Granulocytes - metabolism | Chemokine CXCL1 - metabolism | Disease Models, Animal | Signal Transduction | Chemokine CCL2 - genetics | Sepsis - genetics | Mice, Knockout | Animals | Adaptor Proteins, Signal Transducing - genetics | Adaptor Proteins, Signal Transducing - metabolism | Peritoneum - metabolism | Receptors, Formyl Peptide - genetics | Microbiological research | Microbiota (Symbiotic organisms) | Host-parasite relationships | Physiological aspects | Research | Health aspects | Biological Sciences
Journal Article
Journal Article