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Nature Cell Biology, ISSN 1465-7392, 10/2011, Volume 13, Issue 10, pp. 1214 - 1223
The midbody is a singular organelle formed between daughter cells during cytokinesis and required for their final separation. Midbodies persist in cells long... 
NEURAL PROGENITORS | STEM-CELLS | ASYMMETRIC INHERITANCE | CYTOKINESIS | MEMBRANE | CENTROSOME | DISEASE | DEGRADATION | LITHIUM | DIVISION | CELL BIOLOGY | Embryonic Stem Cells - metabolism | Cell Proliferation | Coculture Techniques | Humans | Recombinant Fusion Proteins - metabolism | Cellular Reprogramming | Lysosomes - metabolism | Transfection | Neoplastic Stem Cells - metabolism | RNA Interference | Time Factors | Cell Transformation, Neoplastic - genetics | Cell Division | Cell Cycle Proteins - genetics | Neoplastic Stem Cells - pathology | Cell Differentiation | Autophagy - genetics | Nuclear Proteins - genetics | Induced Pluripotent Stem Cells - metabolism | Calcium-Binding Proteins - metabolism | Induced Pluripotent Stem Cells - pathology | Cell Line | Chromosomal Proteins, Non-Histone - metabolism | Organelles - pathology | Cell Cycle Proteins - metabolism | Nuclear Proteins - metabolism | Cell Transformation, Neoplastic - metabolism | Centrosome - metabolism | Chromosomal Proteins, Non-Histone - genetics | Proteins - genetics | Animals | Proteins - metabolism | Embryonic Stem Cells - pathology | Mice | HeLa Cells | Cell Transformation, Neoplastic - pathology | Organelles - metabolism | Calcium-Binding Proteins - genetics | Physiological aspects | Cell division | Autophagy (Cytology) | Research | Stem cells | Index Medicus
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 06/2005, Volume 280, Issue 22, pp. 21453 - 21462
In most neurodegenerative disorders, including multiple sclerosis, Parkinson disease, and Alzheimer disease, a massive neuronal cell death occurs as a... 
CEREBELLAR GRANULE NEURONS | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | MICROGLIA | NITRIC-OXIDE SYNTHASE | ALZHEIMERS-DISEASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | 15-DEOXY-DELTA(12,14)-PROSTAGLANDIN J | PPAR-GAMMA | EXPRESSION | ASTROCYTES | BRAIN | Immunohistochemistry | Tumor Necrosis Factor-alpha - metabolism | Models, Chemical | Cyclooxygenase 2 | Lipopolysaccharides - metabolism | Nitrites - metabolism | Glycogen Synthase Kinase 3 beta | PPAR gamma - metabolism | Nitric Oxide Synthase Type II | Brain - metabolism | Dose-Response Relationship, Drug | Staurosporine - metabolism | Transfection | Time Factors | Tretinoin - metabolism | Inflammation - drug therapy | Lithium Chloride - pharmacology | Cell Death | Neurons - metabolism | Neurons - drug effects | Thiazolidinediones - pharmacology | Interleukin-6 - metabolism | Cell Line | Anti-Inflammatory Agents - pharmacology | Glycogen Synthase Kinase 3 - antagonists & inhibitors | Cells, Cultured | Enzyme Inhibitors - pharmacology | Rats | Neurodegenerative Diseases - metabolism | Nitrites - chemistry | Hippocampus - metabolism | Microscopy, Confocal | Animals | Cell-Free System | Prostaglandin-Endoperoxide Synthases - metabolism | Anilides - pharmacology | Neuroglia - metabolism | Glutamic Acid - metabolism | Mice | Nitric Oxide Synthase - metabolism | In Vitro Techniques | Microscopy, Fluorescence | Apoptosis | Astrocytes - metabolism | Index Medicus
Journal Article
Neurobiology of Disease, ISSN 0969-9961, 2009, Volume 37, Issue 3, pp. 596 - 603
Abstract Bipolar disorder (BD) is a progressive psychiatric disorder characterized by recurrent changes of mood and is associated with cognitive decline. There... 
Neurology | Caspase | Synaptophysin | Drebrin | Bipolar disorder | BDNF | Apoptosis | CYTOSOLIC PHOSPHOLIPASE A | ALZHEIMERS-DISEASE | ARACHIDONIC-ACID CASCADE | BINDING PROTEIN | NEUROSCIENCES | POSITRON-EMISSION-TOMOGRAPHY | RAT FRONTAL-CORTEX | GENE-EXPRESSION | MOOD STABILIZERS | LITHIUM | ADMINISTRATION INCREASES | Bipolar Disorder - metabolism | Arachidonic Acid - metabolism | Humans | Middle Aged | Prefrontal Cortex - physiopathology | Bipolar Disorder - physiopathology | Male | RNA, Messenger - metabolism | Brain - metabolism | Synaptophysin - genetics | Encephalitis - physiopathology | Synapses - metabolism | Encephalitis - metabolism | Up-Regulation - physiology | Atrophy - genetics | Aged, 80 and over | Apoptosis Regulatory Proteins - genetics | Adult | Female | Neuropeptides - genetics | Synaptophysin - metabolism | Biomarkers - metabolism | Atrophy - physiopathology | Encephalitis - genetics | Brain - physiopathology | Biomarkers - analysis | Neuropeptides - metabolism | Bipolar Disorder - genetics | Nerve Tissue Proteins - genetics | Disease Progression | Apoptosis Regulatory Proteins - metabolism | Down-Regulation - physiology | Nerve Tissue Proteins - metabolism | Prefrontal Cortex - metabolism | Aged | Apoptosis - physiology | Atrophy - metabolism | Unsaturated fatty acids | Messenger RNA | Lymphomas | Peptide hormones | Arachidonic acid | Growth factors | Index Medicus
Journal Article
Circulation Research, ISSN 0009-7330, 09/2007, Volume 101, Issue 6, pp. 581 - 589
The aberrant differentiation of pericytes along the adipogenic, chondrogenic, and osteogenic lineages may contribute to the development and progression of... 
Vascular disease | Pericytes | Wnt signaling | Chondrogenesis | Differentiation | CELLS | CARDIAC & CARDIOVASCULAR SYSTEMS | differentiation | MESENCHYMAL PROGENITORS | pericytes | ARTERY WALL | CHONDROCYTE DIFFERENTIATION | BETA | STEM | ATHEROSCLEROTIC PLAQUE DEVELOPMENT | IN-VITRO | vascular disease | CALCIFICATION | PERIPHERAL VASCULAR DISEASE | chondrogenesis | HEMATOLOGY | EXPRESSION | LDL-Receptor Related Proteins - metabolism | Transcription, Genetic - drug effects | Chondrogenesis - drug effects | Chondrogenesis - genetics | Adipogenesis - drug effects | RNA, Messenger - metabolism | Wnt Proteins - metabolism | Transforming Growth Factor beta3 - metabolism | Collagen Type II - metabolism | TCF Transcription Factors - metabolism | Wnt Proteins - genetics | Cattle | Lithium Chloride - pharmacology | Adipogenesis - genetics | Transduction, Genetic | High Mobility Group Proteins - metabolism | Glycosaminoglycans - metabolism | Pericytes - metabolism | Cells, Cultured | Frizzled Receptors - metabolism | Proteoglycans - metabolism | Vascular Diseases - genetics | Lipid Metabolism | Signal Transduction - genetics | Vascular Diseases - physiopathology | Aggrecans - metabolism | beta Catenin - metabolism | beta Catenin - genetics | Transcription Factors - metabolism | Animals | Signal Transduction - drug effects | Wnt3 Protein | SOX9 Transcription Factor | Vascular Diseases - metabolism | Index Medicus
Journal Article
Journal Article
Journal Article
Cancer Letters, ISSN 0304-3835, 2015, Volume 371, Issue 2, pp. 347 - 353
Journal Article
Planta, ISSN 0032-0935, 2/2008, Volume 227, Issue 3, pp. 659 - 669
Journal Article
Journal of Cell Science, ISSN 0021-9533, 11/2012, Volume 125, Issue 22, pp. 5564 - 5577
Nitric oxide (NO) has been shown to play a crucial role in bone formation in vivo. We sought to determine the temporal effect of NO on murine embryonic stem... 
Brachyury | Primitive streak | Embryonic stem cell | Osteoblast | β-catenin | Nitric oxide | beta-catenin | GROWTH-INHIBITION | NITRIC-OXIDE SYNTHASE | PROTEIN-KINASE | CELL BIOLOGY | SIGNALING PATHWAY | BONE-FORMATION | ENDOTHELIAL-CELLS | FUNCTIONAL-ANALYSIS | SMOOTH-MUSCLE-CELLS | IN-VITRO DIFFERENTIATION | NF-KAPPA-B | Embryonic Stem Cells - metabolism | Embryonic Stem Cells - cytology | Cell Count | Humans | Nitric Oxide Synthase - antagonists & inhibitors | Fetal Proteins - metabolism | RNA, Messenger - metabolism | Tumor Suppressor Protein p53 - genetics | Time Factors | Lithium Chloride - pharmacology | Minerals - metabolism | Pluripotent Stem Cells - cytology | Mice, Inbred C57BL | Osteogenesis - drug effects | Phosphatidylserines - metabolism | Primitive Streak - embryology | RNA, Messenger - genetics | Enzyme Inhibitors - pharmacology | Gene Expression Regulation, Developmental - drug effects | Tumor Suppressor Protein p53 - metabolism | Primitive Streak - metabolism | Primitive Streak - cytology | Up-Regulation - genetics | beta Catenin - metabolism | T-Box Domain Proteins - metabolism | Pluripotent Stem Cells - metabolism | Up-Regulation - drug effects | Animals | Cyclic GMP - metabolism | Embryonic Stem Cells - drug effects | Signal Transduction - drug effects | Cell Differentiation - drug effects | Pluripotent Stem Cells - drug effects | Mice | Nitric Oxide Synthase - metabolism | Nitric Oxide - metabolism | Index Medicus
Journal Article
Science, ISSN 0036-8075, 5/2005, Volume 308, Issue 5725, pp. 1181 - 1184
β-Catenin is a multifunctional protein that mediates Wnt signaling by binding to members of the T cell factor (TCF) family of transcription factors. Here, we... 
T lymphocytes | Oxidative stress | Animals | HEK293 cells | Life span | Small interfering RNA | Nematode larvae | Journalism | Reports | Gene expression regulation | Insulin | ACTIVATION | PATHWAY | RAS | MULTIDISCIPLINARY SCIENCES | DAF-16 | GENE-EXPRESSION | CELL-CYCLE | C-ELEGANS | NEMATODE CAENORHABDITIS-ELEGANS | FORKHEAD TRANSCRIPTION FACTORS | Immunoprecipitation | Oxidative Stress | Cytoskeletal Proteins - genetics | Humans | Caenorhabditis elegans Proteins - metabolism | Intracellular Signaling Peptides and Proteins - metabolism | Trans-Activators - chemistry | DNA-Binding Proteins - metabolism | Caenorhabditis elegans - physiology | Transfection | Lithium Chloride - pharmacology | Receptor, Insulin - genetics | Trans-Activators - genetics | Cytoskeletal Proteins - metabolism | Intracellular Signaling Peptides and Proteins - genetics | Superoxide Dismutase - metabolism | Cell Line | Insulin - pharmacology | Caenorhabditis elegans - metabolism | Signal Transduction | beta Catenin | Animals, Genetically Modified | Caenorhabditis elegans - genetics | Hydrogen Peroxide - pharmacology | Cytoskeletal Proteins - chemistry | Longevity | Cyclin-Dependent Kinase Inhibitor p27 | Transcription Factors - metabolism | Carrier Proteins - genetics | Carrier Proteins - metabolism | Cell Cycle | Cell Line, Tumor | Receptor, Insulin - metabolism | Trans-Activators - metabolism | Forkhead Box Protein O1 | Mice | Mutation | Caenorhabditis elegans Proteins - genetics | Forkhead Transcription Factors | Index Medicus
Journal Article
Molecular Neurobiology, ISSN 0893-7648, 7/2016, Volume 53, Issue 5, pp. 2954 - 2968
Creatine has been proposed to exert beneficial effects in the management of depression, but the cell signaling pathways implicated in its antidepressant... 
Antidepressant | Neurology | Neurosciences | Biomedicine | Neurobiology | Heme oxygenase-1 | Glycogen kinase 3 | mTOR | Akt | Creatine | Cell Biology | ACTIVATED PROTEIN-KINASE | TAIL SUSPENSION TEST | NMDA RECEPTORS | HIPPOCAMPAL-NEURONS | SUICIDE VICTIMS | NEUROSCIENCES | 5-HT1A RECEPTOR | MICE | MAJOR DEPRESSIVE DISORDER | TRANSCRIPTION FACTOR | Cyclic AMP-Dependent Protein Kinases - metabolism | Creatine - pharmacology | Heme Oxygenase-1 - metabolism | Ribosomal Protein S6 Kinases, 70-kDa - metabolism | TOR Serine-Threonine Kinases - metabolism | Glycogen Synthase Kinase 3 - antagonists & inhibitors | Disks Large Homolog 4 Protein - metabolism | Male | Phosphatidylinositol 3-Kinases - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Glycogen Synthase Kinase 3 - metabolism | Hippocampus - metabolism | Animals | Substrate Specificity - drug effects | Signal Transduction - drug effects | Protein Kinase C - metabolism | Intracellular Space - metabolism | Antidepressive Agents - pharmacology | Mice | Protein Kinase Inhibitors - pharmacology | Phosphorylation - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Glycogen | Antidepressants | Depression, Mental | Heme | Cellular signal transduction | Lithium compounds | Signal transduction | Cellular biology | Dietary supplements | Index Medicus
Journal Article