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Gastroenterology, ISSN 0016-5085, 2013, Volume 144, Issue 5, pp. 1024 - 1030
Journal Article
Journal of Hepatology, ISSN 0168-8278, 2011, Volume 55, Issue 4, pp. 906 - 912
Journal Article
Journal of Hepatology, ISSN 0168-8278, 2010, Volume 54, Issue 4, pp. 716 - 722
Background & Aims A single nucleotide polymorphism (rs12979860 C/T) 3 kb upstream of the interleukin 28B ( IL-28B ) gene was shown to be associated with... 
Gastroenterology and Hepatology | Interleukin 28B | Hepatocellular carcinoma | Hepatitis C | Liver cirrhosis | Genetic polymorphisms | IFN-LAMBDA | III INTERFERONS | ANTITUMOR-ACTIVITY | GENETIC-VARIATION | IL28B | HEPATOCELLULAR-CARCINOMA | POTENT ANTIVIRAL ACTIVITY | IN-VIVO | C VIRUS | INFECTION | GASTROENTEROLOGY & HEPATOLOGY | Liver Cirrhosis - immunology | Liver Transplantation | Hepatitis B, Chronic - immunology | Humans | Middle Aged | Male | DNA Primers - genetics | Hepatitis C, Chronic - complications | Liver Neoplasms - etiology | Young Adult | Interleukins - genetics | Hepatitis B, Chronic - genetics | Base Sequence | Carcinoma, Hepatocellular - genetics | Aged, 80 and over | Adult | Female | Liver Cirrhosis, Alcoholic - immunology | Carcinoma, Hepatocellular - etiology | Carcinoma, Hepatocellular - immunology | Liver Cirrhosis - genetics | Liver Cirrhosis, Alcoholic - complications | Liver Neoplasms - genetics | Liver Cirrhosis - complications | Gene Frequency | Risk Factors | Liver Neoplasms - immunology | Liver Cirrhosis, Alcoholic - genetics | Hepatitis B, Chronic - complications | Alleles | Hepatitis C, Chronic - immunology | Liver Cirrhosis - surgery | Aged | Polymorphism, Single Nucleotide | Hepatitis C, Chronic - genetics | Cohort Studies | Care and treatment | Interleukins | Genes | Transplantation of organs, tissues, etc | Biological response modifiers | Hepatitis C virus | Index Medicus
Journal Article
Genes and Development, ISSN 0890-9369, 04/2014, Volume 28, Issue 7, pp. 708 - 722
Journal Article
Gastroenterology, ISSN 0016-5085, 2016, Volume 150, Issue 5, pp. 1219 - 1230.e6
Background & Aims Nonalcoholic fatty liver disease (NAFLD) is a leading cause of liver damage and is characterized by steatosis. Genetic factors increase risk... 
Gastroenterology and Hepatology | PNPLA3 | NASH | Arachidonic Acid | TM6SF2 | ETHNIC-DIFFERENCES | FIBROSIS PROGRESSION | CONFERS SUSCEPTIBILITY | GENE VARIANT | STEATOHEPATITIS | WIDE ASSOCIATION | GASTROENTEROLOGY & HEPATOLOGY | HEPATIC STEATOSIS | SEVERITY | Liver Cirrhosis - ethnology | Acetyltransferases - metabolism | Liver - pathology | Humans | Male | Non-alcoholic Fatty Liver Disease - ethnology | Acyltransferases - metabolism | Acetyltransferases - genetics | Acyltransferases - genetics | Case-Control Studies | Proton Magnetic Resonance Spectroscopy | Liver Cirrhosis - metabolism | Female | Phosphatidylinositols - metabolism | Membrane Proteins - metabolism | Liver Cirrhosis - genetics | Texas - epidemiology | Severity of Illness Index | European Continental Ancestry Group - genetics | Genetic Predisposition to Disease | Genome-Wide Association Study | Liver Cirrhosis - diagnosis | Cross-Sectional Studies | Non-alcoholic Fatty Liver Disease - genetics | Membrane Proteins - genetics | Liver - metabolism | Risk Factors | Europe - epidemiology | Non-alcoholic Fatty Liver Disease - metabolism | Polymorphism, Genetic | Triglycerides - metabolism | Phenotype | Biopsy | Non-alcoholic Fatty Liver Disease - diagnosis | Disease susceptibility | Fatty liver | Epidemiology | Liver cirrhosis | Genomics | Index Medicus | Abridged Index Medicus | Kardiologi | Cardiac and Cardiovascular Systems
Journal Article
Proceedings of the National Academy of Sciences, ISSN 0027-8424, 11/2012, Volume 109, Issue 46, pp. E3186 - E3195
Although macrophages are widely recognized to have a profibrotic role in inflammation, we have used a highly tractable CCl 4 -induced model of reversible... 
Degradation | Kupffer cell | Proliferation | Collagen | Myofibroblast | Kupffer Cell | LUNG FIBROSIS | proliferation | myofibroblast | MULTIDISCIPLINARY SCIENCES | collagen | degradation | HEPATIC-FIBROSIS | RENAL FIBROSIS | IMMUNE-RESPONSES | APOPTOTIC CELLS | IN-VIVO | MONOCYTE SUBSETS | GENE-EXPRESSION | PULMONARY-FIBROSIS | T-CELLS | Liver Cirrhosis - immunology | Antigens, Ly - genetics | Matrix Metalloproteinase 9 - immunology | Matrix Metalloproteinase 12 - genetics | Insulin-Like Growth Factor I - genetics | Monocytes - immunology | MAP Kinase Signaling System - immunology | Liver Cirrhosis - chemically induced | Insulin-Like Growth Factor I - immunology | CD11b Antigen - genetics | MAP Kinase Signaling System - genetics | Matrix Metalloproteinase 9 - genetics | Monocytes - pathology | Liver Cirrhosis - genetics | Macrophages - immunology | CD11b Antigen - immunology | Macrophages - pathology | Gene Expression Regulation - genetics | Gene Expression Regulation - immunology | Matrix Metalloproteinase 12 - immunology | Mice, Transgenic | Carbon Tetrachloride - toxicity | Carbon Tetrachloride Poisoning - pathology | Gene Expression Regulation - drug effects | Animals | MAP Kinase Signaling System - drug effects | Antigens, Ly - immunology | Carbon Tetrachloride Poisoning - immunology | Liver Cirrhosis - pathology | Carbon Tetrachloride Poisoning - genetics | Mice | Proteins | Genotype & phenotype | Signal transduction | Liver diseases | Gene expression | Rodents | Index Medicus | Biological Sciences | PNAS Plus
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2011, Volume 6, Issue 11, pp. e27087 - e27087
Background: An isoleucine. methionine mutation at position 148 in the PNPLA3 gene (p. I148M, rs738409) has recently been identified as a susceptibility factor... 
FIBROSIS | HEPATOCELLULAR-CARCINOMA | GENE VARIANT | MULTIDISCIPLINARY SCIENCES | DISEASE | I148M | DOMAIN-CONTAINING 3 | TRENDS | SEVERITY | ADIPONUTRIN | CHILDREN | Humans | Middle Aged | Male | Case-Control Studies | Liver Neoplasms - etiology | Young Adult | Carcinoma, Hepatocellular - genetics | Aged, 80 and over | Adult | Female | Hepatitis C - complications | Carcinoma, Hepatocellular - etiology | Liver Cirrhosis - genetics | Liver Cirrhosis, Alcoholic - complications | Genetic Predisposition to Disease | Liver Neoplasms - genetics | Liver Cirrhosis - complications | Membrane Proteins - genetics | Risk Factors | European Continental Ancestry Group | Genotype | Hepatitis C - genetics | Liver Cirrhosis, Alcoholic - genetics | Aged | Lipase - genetics | Type 2 diabetes | Obesity | Genes | Development and progression | Amino acids | Risk factors | Infection | Genetic aspects | Hepatitis C virus | Health aspects | Liver cirrhosis | Cancer | Diabetes therapy | Hepatitis B | Pediatrics | Pathogenesis | Liver | Viruses | Lipids | Hepatocellular carcinoma | Alcohol | Homozygosity | Gene polymorphism | Alcohol use | Proteins | Confidence intervals | Hepatitis | Liver cancer | Genotype & phenotype | Rodents | Hepatology | Gastroenterology | Genotypes | Isoleucine | Liver diseases | Statistical analysis | Hematology | Internal medicine | Diabetes mellitus | Cloning | Health risks | Methionine | Patients | Disease control | Medicine | Cirrhosis | Hospitals | Damage detection | Hepatitis C | Polymorphism | Index Medicus
Journal Article