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Gastroenterology, ISSN 0016-5085, 2012, Volume 143, Issue 3, pp. 765 - 776.e3
Background & Aims Interleukin (IL)-17 signaling has been implicated in lung and skin fibrosis. We examined the role of IL-17 signaling in the pathogenesis of... 
Gastroenterology and Hepatology | Immune Response | Myofibroblast | Mouse Model | Bone Marrow-Derived Macrophages | RAT-LIVER | TARGET | IL-17 RECEPTOR | HEPATOCYTES | FAMILY-MEMBERS | STAT3 | TRANSDUCTION | PATHWAY | DISEASE | GENE-EXPRESSION | GASTROENTEROLOGY & HEPATOLOGY | Kupffer Cells - pathology | Liver - pathology | Carbon Tetrachloride | Humans | Transforming Growth Factor beta1 - metabolism | Hepatic Stellate Cells - metabolism | Receptors, Interleukin-17 - genetics | Interleukin-23 - genetics | Receptors, Interleukin-17 - deficiency | Interleukins - genetics | Liver Cirrhosis, Experimental - prevention & control | Liver - immunology | Time Factors | Bone Marrow Transplantation | Liver Cirrhosis, Alcoholic - immunology | Interleukin-6 - metabolism | STAT3 Transcription Factor - genetics | Interleukin-17 - administration & dosage | Liver Cirrhosis, Experimental - immunology | Signal Transduction | Liver Cirrhosis, Alcoholic - pathology | Liver - metabolism | Interleukin-17 - genetics | Genotype | Bile Ducts - surgery | Disease Progression | Mice, Knockout | Interleukin-17 - metabolism | Phenotype | Interleukins - deficiency | Mice | Liver Cirrhosis, Experimental - metabolism | Tumor Necrosis Factor-alpha - metabolism | Inflammation Mediators - administration & dosage | Liver Cirrhosis, Experimental - etiology | Interleukin-23 - deficiency | Ligation | Hepatic Stellate Cells - immunology | Inflammation Mediators - metabolism | STAT3 Transcription Factor - deficiency | Liver Cirrhosis, Experimental - pathology | Kupffer Cells - metabolism | Hepatic Stellate Cells - pathology | Cell Line | Interleukin-1 - metabolism | Collagen Type I - metabolism | Mice, Inbred C57BL | Gene Expression Regulation | Interleukin-17 - deficiency | Liver Cirrhosis, Experimental - genetics | Animals | Interleukins - administration & dosage | Kupffer Cells - immunology | Medical colleges | Liver diseases | Liver | Carbon tetrachloride | Transforming growth factors | Muscle proteins | Macrophages | Endothelium | Messenger RNA | Interleukins | Actin | Analysis | Collagen | Fibrosis | Liver cirrhosis | Index Medicus | Abridged Index Medicus
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 08/2012, Volume 122, Issue 8, pp. 2884 - 2897
Journal Article
Gastroenterology, ISSN 0016-5085, 2014, Volume 146, Issue 5, pp. 1339 - 1350.e1
Background & Aims Vascular endothelial growth factor (VEGF)−induced angiogenesis is implicated in fibrogenesis and portal hypertension. However, the function... 
Gastroenterology and Hepatology | Extracellular Matrix | Sinusoidal Endothelial Cell | Liver Damage | Hepatic Sinusoid | Keywords | FACTOR VEGF | ANGIOGENESIS | MACROPHAGES | LIVER FIBROSIS | FIBROGENESIS | PATHOGENESIS | MATRIX METALLOPROTEINASES | HEPATIC STELLATE CELLS | INFLAMMATION | NEOVASCULARIZATION | GASTROENTEROLOGY & HEPATOLOGY | Antibodies, Neutralizing - administration & dosage | Liver - pathology | Capillary Permeability | Human Umbilical Vein Endothelial Cells - metabolism | Carbon Tetrachloride | Coculture Techniques | Humans | Monocytes - metabolism | Monocytes - immunology | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - antagonists & inhibitors | Vascular Endothelial Growth Factor A - genetics | fas Receptor - metabolism | Liver Cirrhosis, Experimental - prevention & control | Liver - immunology | Ligation | Cholecystostomy | Adenoviridae - genetics | Liver Cirrhosis, Experimental - pathology | Macrophages - immunology | Tacrolimus Binding Protein 1A - genetics | Gene Transfer Techniques | Promoter Regions, Genetic | Liver Regeneration | Liver Cirrhosis, Experimental - immunology | Jejunostomy | Liver - metabolism | Mice, Inbred C57BL | Cells, Cultured | Receptor, Macrophage Colony-Stimulating Factor - genetics | Mice, Transgenic | Vascular Endothelial Growth Factor A - immunology | Bile Ducts - surgery | Liver Cirrhosis, Experimental - genetics | Remission Induction | Matrix Metalloproteinase 13 - metabolism | Injections | Macrophages - metabolism | Animals | Chemokine CXCL9 - metabolism | Liver Cirrhosis, Experimental - chemically induced | Tacrolimus Binding Protein 1A - metabolism | Mice | Genetic Vectors | Liver Cirrhosis, Experimental - metabolism | Apoptosis | Hypertension | Medical colleges | Vascular endothelial growth factor | Health aspects | Fibrosis | Index Medicus | Abridged Index Medicus
Journal Article
Molecular and cellular biology, ISSN 0270-7306, 2014, Volume 34, Issue 17, pp. 3305 - 3320
Journal Article
Gastroenterology, ISSN 0016-5085, 04/2018, Volume 154, Issue 5, pp. 1465 - 1479.e13
Cirrhosis results from accumulation of myofibroblasts derived from quiescent hepatic stellate cells (Q-HSCs); it regresses when myofibroblastic HSCs are... 
Metabolic Reprogramming | Liver Diseases | Hippo Pathway | Fibrogenesis | FIBROSIS | TRANSCRIPTIONAL ACTIVITY | PULMONARY-HYPERTENSION | FATTY LIVER-DISEASE | MECHANISMS | CANCER | METABOLISM | GLUCOSE | GROWTH | Fibro-genesis | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | Liver - pathology | Cell Proliferation | Mitochondria, Liver - metabolism | Humans | Glutamine - metabolism | Hedgehog Proteins - metabolism | Hepatic Stellate Cells - metabolism | Smoothened Receptor - metabolism | Phosphoproteins - metabolism | Smoothened Receptor - genetics | Case-Control Studies | Cellular Reprogramming | Myofibroblasts - metabolism | Transfection | Hedgehog Proteins - genetics | RNA Interference | Time Factors | Liver Cirrhosis - metabolism | Liver Cirrhosis, Experimental - pathology | Cell Transdifferentiation | Ketoglutaric Acids - metabolism | Liver Cirrhosis - genetics | Hepatic Stellate Cells - pathology | Myofibroblasts - pathology | Mitochondria, Liver - pathology | Signal Transduction | Liver - metabolism | Mice, Inbred C57BL | Cells, Cultured | Gene Expression Regulation | Rats | Glutaminase - metabolism | Liver Cirrhosis, Experimental - genetics | Phosphoproteins - genetics | Mice, Knockout | Phenotype | Animals | Energy Metabolism | Adaptor Proteins, Signal Transducing - genetics | Liver Cirrhosis - pathology | Adaptor Proteins, Signal Transducing - metabolism | Liver Cirrhosis, Experimental - metabolism | Glucose metabolism | Metabolites | RNA | Analysis | Liver | Physiological aspects | Genetic research | Verteporfin | Development and progression | Mice | Liver cirrhosis | Glutamine | Index Medicus | Abridged Index Medicus
Journal Article
Gastroenterology, ISSN 0016-5085, 2012, Volume 142, Issue 4, pp. 938 - 946
Background & Aims The pathogenesis of liver fibrosis involves activation of hepatic stellate cells, which is associated with depletion of intracellular lipid... 
Gastroenterology and Hepatology | Inflammation | Myofibroblasts | Energy Depletion | Mouse Model | RAT-LIVER | FIBROSIS | PATHWAY | DISEASE | MECHANISMS | RETINOL | GASTROENTEROLOGY & HEPATOLOGY | Liver - pathology | Kidney - pathology | Microtubule-Associated Proteins - genetics | Microtubule-Associated Proteins - metabolism | Carbon Tetrachloride | Humans | Hepatic Stellate Cells - metabolism | Autophagy - drug effects | Idiopathic Pulmonary Fibrosis - metabolism | Kidney - metabolism | Epoxy Compounds - pharmacology | Liver - drug effects | RNA Interference | Adenosine Triphosphate - metabolism | Liver Cirrhosis, Experimental - pathology | Lung - metabolism | Autophagy - genetics | Microtubule-Associated Proteins - deficiency | Hepatic Stellate Cells - pathology | Fibroblasts - metabolism | Hepatic Stellate Cells - drug effects | Cell Line | Lung - pathology | Oleic Acid - metabolism | Adenine - analogs & derivatives | Liver - metabolism | Mice, Inbred C57BL | Adenine - pharmacology | Liver Cirrhosis, Experimental - genetics | Fibroblasts - pathology | Mice, Knockout | Thioacetamide | Autophagy-Related Protein 7 | Animals | Autophagy-Related Protein 5 | Energy Metabolism | Fibroblasts - drug effects | Liver Cirrhosis, Experimental - chemically induced | Lipid Metabolism - drug effects | Idiopathic Pulmonary Fibrosis - pathology | Mice | Liver Cirrhosis, Experimental - metabolism | Phosphates | Medical colleges | Neurosciences | Platelet-derived growth factor | Liver diseases | Albumin | Lipids | Muscle proteins | Fatty acids | Cells | Monosaccharides | Unsaturated fatty acids | Monounsaturated fatty acids | Actin | Analysis | Intermediate filament proteins | Sugars | Index Medicus | Abridged Index Medicus
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 12/2013, Volume 288, Issue 52, pp. 37082 - 37093
Sustained activation of hepatic stellate cells (HSCs) leads to hepatic fibrosis, which is characterized by excessive collagen production, and for which there... 
ACTIVATION | RAT | BIOCHEMISTRY & MOLECULAR BIOLOGY | LIVER FIBROSIS | STELLATE CELLS | AP-1 | Liver Injury | MicroRNA | Hepatic Stellate Cells | Fibrosis | IN-VIVO | Transforming Growth Factor (TGF) | PDCD4 | MIR-21 | MICRORNA EXPRESSION | UP-REGULATION | Carbon Tetrachloride Poisoning - drug therapy | RNA-Binding Proteins - genetics | MicroRNAs - antagonists & inhibitors | Transcription Factor AP-1 - genetics | Humans | Middle Aged | Hepatic Stellate Cells - metabolism | Male | MicroRNAs - metabolism | Liver Cirrhosis, Experimental - drug therapy | Transcription Factor AP-1 - metabolism | Carbon Tetrachloride Poisoning - metabolism | Apoptosis Regulatory Proteins - genetics | Liver Cirrhosis, Experimental - pathology | Adult | Female | Hepatic Stellate Cells - pathology | Signal Transduction | Liver Cirrhosis, Experimental - genetics | Carbon Tetrachloride - toxicity | Carbon Tetrachloride Poisoning - pathology | Mice, Inbred ICR | Apoptosis Regulatory Proteins - metabolism | Animals | Thioacetamide - toxicity | Transforming Growth Factor beta - genetics | Liver Cirrhosis, Experimental - chemically induced | Carbon Tetrachloride Poisoning - genetics | Aged | Mice | MicroRNAs - genetics | Transforming Growth Factor beta - metabolism | Liver Cirrhosis, Experimental - metabolism | RNA-Binding Proteins - metabolism | Index Medicus | Molecular Bases of Disease | Transforming Growth Factor β (TGFβ)
Journal Article
Toxicology and Applied Pharmacology, ISSN 0041-008X, 12/2015, Volume 289, Issue 2, pp. 163 - 176
SIRT1 (silent information regulator 1), a conserved NAD +-dependent histone deacetylase, is closely related with various biological processes. Moreover, the... 
Reversion | MALAT1 | Liver fibrosis | SIRT1 | Apoptosis | RESVERATROL | METASTASIS | MECHANISMS | CANCER | HEPATIC-FIBROSIS | RENAL FIBROSIS | LONG NONCODING RNA | MALAT-1 | PHARMACOLOGY & PHARMACY | TOXICOLOGY | EXPRESSION | PULMONARY-FIBROSIS | Sirtuin 1 - metabolism | Liver - pathology | Liver - enzymology | Apoptosis - drug effects | Carbon Tetrachloride | Humans | Actins - metabolism | Myofibroblasts - enzymology | Male | RNA, Messenger - metabolism | Xanthines - pharmacology | Sirtuin 1 - genetics | Actins - genetics | Dose-Response Relationship, Drug | Liver Cirrhosis, Experimental - prevention & control | Liver Cirrhosis, Experimental - enzymology | Collagen Type I - genetics | Dexamethasone - pharmacology | Transfection | Liver - drug effects | RNA Interference | Time Factors | Liver Cirrhosis, Experimental - pathology | Female | Chemical and Drug Induced Liver Injury - pathology | Chemical and Drug Induced Liver Injury - enzymology | Chemical and Drug Induced Liver Injury - etiology | Hepatic Stellate Cells - pathology | Transforming Growth Factor beta1 - pharmacology | Hepatic Stellate Cells - drug effects | Cell Line | Chemical and Drug Induced Liver Injury - prevention & control | Insulin - pharmacology | Myofibroblasts - pathology | Collagen Type I - metabolism | Signal Transduction | Mice, Inbred C57BL | Gene Expression Regulation | Hepatic Stellate Cells - enzymology | Chemical and Drug Induced Liver Injury - genetics | Liver Cirrhosis, Experimental - genetics | Phenotype | Animals | Liver Cirrhosis, Experimental - chemically induced | RNA, Long Noncoding - metabolism | Corticosteroids | Liver | Analysis | Fibrosis | Bone morphogenetic proteins | Transforming growth factors | Liver cirrhosis | Index Medicus | APOPTOSIS | DEXAMETHASONE | COLLAGEN | RNA | LUNGS | METASTASES | 60 APPLIED LIFE SCIENCES | HISTONES | MICE | VIRUSES | FIBROSIS | ONCOGENES | TRANSCRIPTION FACTORS | ACTIN | MATERIALS RECOVERY | STEROLS | TRANSCRIPTION | PHENOTYPE | INSULIN | NAD | GROWTH FACTORS | CARBON TETRACHLORIDE | LIVER | CARCINOMAS | RECEPTORS | MUSCLES
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 04/2015, Volume 125, Issue 4, pp. 1533 - 1544
The cause of organ failure is enigmatic for many degenerative diseases, including end-stage liver disease. Here, using a CCl4-induced rat model of irreversible... 
LONG-TERM | PATHOGENESIS | MEDICINE, RESEARCH & EXPERIMENTAL | NUCLEAR | MECHANISM | CHRONIC LIVER-DISEASE | CIRRHOSIS | RATS | HEPATOCYTE | EXPRESSION | TRANSPLANTATION | Liver Cirrhosis, Experimental - therapy | Liver Failure - etiology | Dependovirus - genetics | Genetic Therapy | Hepatocyte Nuclear Factor 1-alpha - genetics | Hepatocyte Nuclear Factor 3-beta - genetics | Rats, Inbred Lew | Transcriptome | Hepatocyte Nuclear Factor 1-alpha - biosynthesis | Liver Failure - pathology | Hepatocytes - pathology | Male | Gene Expression Profiling | Genetic Vectors - therapeutic use | Hepatocytes - metabolism | Gene Regulatory Networks | Hepatocyte Nuclear Factor 4 - physiology | Recombinant Fusion Proteins - metabolism | CCAAT-Enhancer-Binding Protein-alpha - biosynthesis | Hepatocyte Nuclear Factor 4 - biosynthesis | Liver Cirrhosis, Experimental - pathology | Liver Cirrhosis, Experimental - complications | Hepatocyte Nuclear Factor 3-beta - biosynthesis | Transcription Factors - physiology | Transduction, Genetic | PPAR alpha - biosynthesis | Down-Regulation | Cells, Cultured | Gene Expression Regulation | Rats | PPAR alpha - genetics | Hepatocyte Nuclear Factor 4 - genetics | Liver Cirrhosis, Experimental - genetics | Liver Failure - genetics | Disease Progression | Animals | Carbon Tetrachloride Poisoning - therapy | Liver Failure - therapy | CCAAT-Enhancer-Binding Protein-alpha - genetics | Carbon Tetrachloride Poisoning - genetics | Cell Dedifferentiation - genetics | Transcription factors | Liver diseases | Genetic research | Development and progression | Genetic aspects | Genetic transcription | Research | Properties | Hypertension | Cytochrome | Transplants & implants | Rodents | Stem cells | Metabolism | Laboratory animals | Liver cirrhosis | Index Medicus | Abridged Index Medicus | Hepatology | Gastroenterology
Journal Article
American Journal of Physiology - Gastrointestinal and Liver Physiology, ISSN 0193-1857, 12/2009, Volume 297, Issue 6, pp. G1093 - G1106
Choi SS, Omenetti A, Witek RP, Moylan CA, Syn W, Jung Y, Yang L, Sudan DL, Sicklick JK, Michelotti GA, Rojkind M, Diehl AM. Hedgehog pathway activation and... 
Regeneration | Proliferation | Bone morphogenetic protein-7 | Cyclopamine | Fibrosis | HEPATOCYTES | PHYSIOLOGY | TGF-BETA | proliferation | regeneration | LIVER FIBROSIS | STELLATE CELLS | cyclopamine | GROWTH-FACTOR-BETA | ORIGIN | bone morphogenetic protein-7 | LHX2 | SUBMESOTHELIAL CELLS | GENE-EXPRESSION | fibrosis | GASTROENTEROLOGY & HEPATOLOGY | SONIC-HEDGEHOG | Liver - pathology | Cell Proliferation | Epithelial Cells - metabolism | Carbon Tetrachloride | Epithelial Cells - drug effects | Humans | Hedgehog Proteins - metabolism | Hepatic Stellate Cells - metabolism | Male | Cell Transdifferentiation - genetics | RNA, Messenger - metabolism | Hedgehog Proteins - genetics | Liver - drug effects | Time Factors | Cell Transdifferentiation - drug effects | Veratrum Alkaloids - pharmacology | Liver Cirrhosis, Experimental - pathology | Hepatic Stellate Cells - pathology | Fibroblasts - metabolism | Hepatic Stellate Cells - drug effects | Liver Regeneration | Liver - metabolism | Mice, Inbred C57BL | Cells, Cultured | Gene Expression Regulation | Rats | Epithelial Cells - pathology | Signal Transduction - genetics | Hedgehog Proteins - antagonists & inhibitors | Liver Cirrhosis, Experimental - genetics | Fibroblasts - pathology | Rats, Sprague-Dawley | Animals | Signal Transduction - drug effects | Fibroblasts - drug effects | Liver Cirrhosis, Experimental - chemically induced | Mice | Liver Cirrhosis, Experimental - metabolism | Physiological aspects | Development and progression | Genetic aspects | Cellular signal transduction | Research | Liver cirrhosis | Liver cells | Proteins | Molecules | Transitions | Rodents | Liver | Ribonucleic acid--RNA | Cells | Index Medicus | Liver and Biliary Tract
Journal Article