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The Journal of Physiology, ISSN 0022-3751, 07/2011, Volume 589, Issue 13, pp. 3383 - 3393
Non‐technical summary  Energy sources for the brain include not only blood glucose, but also astrocytic glycogen, especially when the blood‐born glucose supply... 
TREADMILL EXERCISE | MICRODIALYSIS | PHYSIOLOGY | RECURRENT HYPOGLYCEMIA | METABOLISM | LACTATE | RAT HIPPOCAMPUS | GLUCOSE-UTILIZATION | RUNNING STRESS | ASTROCYTES | NEUROSCIENCES | HYPOTHALAMUS | Carbohydrates | Hypotheses | Glucose | Blood glucose | Cerebellum | Muscles (exercise effects) | Brain | Energy cost | Physical training | Brain stem | Glycogen | Liver | Cortex | Muscles | Fatigue | Metabolism | Blood | Treadmill ergometry | Energy | Exercise (duration) | Running | Hippocampus | monoamines | Integrative
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2013, Volume 8, Issue 7, p. e69420
Glycogen phosphorylase (GP) catalyzes the breakdown of glycogen and largely contributes to hepatic glucose production making GP inhibition an attractive target... 
CELLS | MULTIDISCIPLINARY SCIENCES | LIVER | MUSCLE | MICE | PROLIFERATION | UNCOUPLING PROTEIN-2 | GLUCOPYRANOSYLIDENE-SPIRO-THIOHYDANTOIN | CANCER | MTOR | REVEALS | Diabetes Mellitus, Experimental - drug therapy | Gene Expression - drug effects | TOR Serine-Threonine Kinases - metabolism | Diabetes Mellitus, Experimental - enzymology | Glucose - analogs & derivatives | Ion Channels - genetics | Male | Multiprotein Complexes - genetics | Mitochondrial Proteins - genetics | Mitochondrial Proteins - agonists | Enzyme Inhibitors - chemical synthesis | Mechanistic Target of Rapamycin Complex 2 | Urea - chemical synthesis | Multiprotein Complexes - metabolism | Glucose - chemical synthesis | Liver Glycogen - metabolism | TOR Serine-Threonine Kinases - genetics | Liver - drug effects | Liver Glycogen - antagonists & inhibitors | Mitochondrial Proteins - metabolism | Urea - analogs & derivatives | Diabetes Mellitus, Experimental - physiopathology | Glucose Tolerance Test | Liver - metabolism | Enzyme Inhibitors - pharmacology | Glucose - pharmacology | Glycogen Phosphorylase - metabolism | Animals | Ion Channels - metabolism | Oxygen Consumption - drug effects | Ion Channels - agonists | Mice, Obese | Multiprotein Complexes - agonists | Glycogen Phosphorylase - antagonists & inhibitors | Mice | Uncoupling Protein 2 | Urea - pharmacology | TOR protein | Mitochondrial uncoupling protein 2 | Animal models | Liver | Science | Biology | Glucose | Kinases | Proteins | Signal transduction | Mitochondria | Rodents | Hepatology | Oxidation | Inhibition | Glycogen | Diabetes mellitus | Oxygen consumption | Rapamycin | Metabolism | Phosphorylase | Glucose tolerance | Urea | Organic chemistry | Musculoskeletal system | Signaling | Sensitivity | Inhibitors | Glycogen phosphorylase | Sensitivity enhancement | Accommodation | Diabetes | Cancer
Journal Article
Journal Article
International Journal of Biochemistry and Cell Biology, ISSN 1357-2725, 01/2016, Volume 70, pp. 149 - 160
MicroRNAs (miRNAs) play important roles in epithelial-to-mesenchymal transition (EMT). Moreover, hyperglycaemia induces damage to renal tubular epithelial... 
Diabetic nephropathy | Renal tubular epithelial cell | HMGA2 | Mir-23b | Epithelial-to-mesenchymal transition (EMT) | ACTIVATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | LIVER FIBROSIS | PROLIFERATION | FIBROBLASTS | CELL BIOLOGY | RENAL FIBROSIS | IN-VITRO | HEPATIC STELLATE CELLS | DISEASE | IDIOPATHIC PULMONARY-FIBROSIS | EXPRESSION | Epithelial Cells - metabolism | MicroRNAs - antagonists & inhibitors | Epithelial Cells - drug effects | Humans | MicroRNAs - metabolism | Epithelial-Mesenchymal Transition - genetics | Proto-Oncogene Proteins c-akt - genetics | Glycogen - antagonists & inhibitors | HMGA2 Protein - metabolism | Oligoribonucleotides - metabolism | Glycogen - biosynthesis | HMGA2 Protein - genetics | MicroRNAs - agonists | Chromones - pharmacology | Proto-Oncogene Proteins c-akt - metabolism | Disease Models, Animal | Cell Line | Diabetic Nephropathies - pathology | Kidney Tubules, Proximal - pathology | Signal Transduction | Diabetic Nephropathies - metabolism | Gene Expression Regulation | Morpholines - pharmacology | Diabetic Nephropathies - genetics | Epithelial Cells - pathology | Mice, Transgenic | Glucose - pharmacology | Phosphatidylinositol 3-Kinases - genetics | Animals | Kidney Tubules, Proximal - metabolism | Mice | MicroRNAs - genetics | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Kidney Tubules, Proximal - drug effects | Oligoribonucleotides - genetics | Hyperglycemia | MicroRNA | Glycogen | Stem cells | Diabetic nephropathies | Glucose | Dextrose
Journal Article
Pathogens and Disease, ISSN 2049-632X, 06/2018, Volume 76, Issue 4
This study aimed to investigate how Akkermansia muciniphila can implicate type 2 diabetes mellitus and the mechanisms underlying the effects A. muciniphila on... 
Intestine microbiota | Oxidative stress | Inflammation | Akkermansia muciniphila | Type 2 diabetes mellitus | Gluco/lipotoxicity | INFECTIOUS DISEASES | intestine microbiota | HOST GENOTYPE | PERMEABILITY | MICROBIOLOGY | GUT MICROBIOTA | IMMUNOLOGY | DIET-INDUCED OBESITY | FIBRINOLYSIS | METABOLISM | inflammation | DISEASE | type 2 diabetes mellitus | PAI-1 | MICE | gluco/lipotoxicity | oxidative stress | ADIPOSE-TISSUE | Intestines - drug effects | Streptozocin | Tumor Necrosis Factor-alpha - genetics | Lipopolysaccharides - metabolism | Male | Intestines - metabolism | Lipopolysaccharides - antagonists & inhibitors | Lipopolysaccharides - immunology | Glucagon-Like Peptide 1 - genetics | Glycogen - immunology | Intestines - immunology | Verrucomicrobia - physiology | Diabetes Mellitus, Experimental - diet therapy | Cholesterol, HDL - immunology | Feces - microbiology | Glucagon-Like Peptide 1 - immunology | Liver - immunology | Glycogen - antagonists & inhibitors | Glycogen - metabolism | Liver - drug effects | Tumor Necrosis Factor-alpha - immunology | Diabetes Mellitus, Experimental - chemically induced | Diabetes Mellitus, Experimental - microbiology | Plasminogen Activator Inhibitor 1 - genetics | Malondialdehyde - metabolism | Cholesterol, HDL - metabolism | Liver - metabolism | Rats | Probiotics - pharmacology | Rats, Sprague-Dawley | Hypoglycemic Agents - pharmacology | Diabetes Mellitus, Experimental - immunology | Gene Expression Regulation - drug effects | Animals | Gastrointestinal Microbiome - drug effects | Cholesterol, HDL - agonists | Malondialdehyde - immunology | Gastrointestinal Microbiome - immunology | Oxidative Stress - drug effects | Malondialdehyde - antagonists & inhibitors | Plasminogen Activator Inhibitor 1 - immunology | Glucagon | Glycogen | Diabetes mellitus | Oral administration | Pasteurization | Tumor necrosis factor-α | Malondialdehyde | Blood levels | Lipopolysaccharides | Probiotics | Species diversity | Microbiota | Intestine | Rodents | Plasminogen activator inhibitors | Diabetes | Diabetes mellitus (non-insulin dependent) | Glucagon-like peptide 1
Journal Article
Journal Article
British Journal of Pharmacology, ISSN 0007-1188, 01/2018, Volume 175, Issue 2, pp. 301 - 319
Journal Article
Journal Article
Food and Function, ISSN 2042-6496, 09/2018, Volume 9, Issue 9, pp. 4926 - 4935
Silibinin, a flavonolignan derived from milk thistle (Silybum marianum), has been revealed to have a beneficial effect on improving diabetes-impaired glycemic... 
METABOLIC SYNDROME | HOMEOSTASIS | PATHWAY | STEATOSIS | INSULIN-RESISTANCE | BIOCHEMISTRY & MOLECULAR BIOLOGY | FOOD SCIENCE & TECHNOLOGY | ANIMAL-MODEL | SILYMARIN | HEPATOTOXICITY | HYPERTENSION | PROGRESSION | Duodenum - innervation | Neurons - pathology | Liver - pathology | Solitary Nucleus - pathology | Diet, High-Fat - adverse effects | Male | Diabetes Mellitus, Type 2 - metabolism | Liver - physiopathology | Gastrointestinal Tract - pathology | Brain - metabolism | Liver - innervation | Glucagon-Like Peptide-1 Receptor - metabolism | Liver Glycogen - metabolism | Hypoglycemic Agents - administration & dosage | Liver Glycogen - antagonists & inhibitors | Diabetes Mellitus, Type 2 - therapy | Duodenum - metabolism | Duodenum - physiopathology | Obesity - etiology | Neurons - metabolism | Gastrointestinal Tract - innervation | Solitary Nucleus - metabolism | Diabetes Mellitus, Type 2 - complications | Solitary Nucleus - physiopathology | Gluconeogenesis | Hypoglycemic Agents - therapeutic use | Hyperglycemia - prevention & control | Specific Pathogen-Free Organisms | Obesity - complications | Brain - physiopathology | Liver - metabolism | Gastrointestinal Tract - physiopathology | Rats, Sprague-Dawley | Vagotomy | Gastrointestinal Tract - metabolism | Rats, Zucker | Silybin - administration & dosage | Animals | Duodenum - pathology | Silybin - therapeutic use | Brain - pathology | Diabetes Mellitus, Type 2 - pathology | Dietary Supplements | Glucagon-Like Peptide-1 Receptor - agonists
Journal Article
Journal of Neuroscience Research, ISSN 0360-4012, 11/2011, Volume 89, Issue 11, pp. 1829 - 1839
Journal Article