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Science, ISSN 0036-8075, 2/2011, Volume 331, Issue 6017, pp. 593 - 596
Satellite repeats in heterochromatin are transcribed into noncoding RNAs that have been linked to gene silencing and maintenance of chromosomal integrity.... 
Tumor cell line | Artificial satellites | RNA | Lungs | Genes | Liver | REPORTS | Cell lines | Pancreas | Tumors | Cancer | ELEMENTS | CELLS | SMALL RNAS | HUMAN GENOMES | PERVASIVE TRANSCRIPTION | MULTIDISCIPLINARY SCIENCES | MOUSE | DIFFERENTIATION | Colonic Neoplasms - genetics | RNA, Untranslated - metabolism | Humans | Ovarian Neoplasms - pathology | Lung Neoplasms - pathology | Male | Gene Expression Profiling | RNA, Neoplasm - metabolism | RNA, Untranslated - genetics | Heterochromatin - chemistry | Carcinoma, Pancreatic Ductal - genetics | Ovarian Neoplasms - genetics | DNA Methylation | Carcinoma in Situ - genetics | Prostatic Neoplasms - genetics | Neoplasms - genetics | Neurosecretory Systems - metabolism | Female | Transcription, Genetic | Lung Neoplasms - genetics | Prostatic Neoplasms - pathology | Gene Expression | Carcinoma in Situ - pathology | Pancreatic Neoplasms - pathology | Pancreatic Neoplasms - genetics | Carcinoma, Pancreatic Ductal - pathology | Animals | Heterochromatin - genetics | Long Interspersed Nucleotide Elements | Mice, Nude | Colonic Neoplasms - pathology | RNA, Neoplasm - genetics | DNA, Satellite - genetics | Mice | DNA, Neoplasm - genetics | Neoplasms - pathology | Epithelial cells | Analysis | Microsatellites (Genetics) | Genetic aspects | Gene expression | Health aspects | Biomarkers | Chromatin | Pancreatic cancer | Deoxyribonucleic acid--DNA | Index Medicus | Human | Cellular | Satellites | Insertion | Aberration | Colon
Journal Article
Nature Communications, ISSN 2041-1723, 11/2015, Volume 6, Issue 1, pp. 8760 - 8760
Circulating tumour DNA analysis can be used to track tumour burden and analyse cancer genomes non-invasively but the extent to which it represents metastatic... 
HETEROGENEITY | ACTIVATION | VARIANTS | PATHWAY | MULTIDISCIPLINARY SCIENCES | KINASE | ACQUIRED-RESISTANCE | SEQUENCING DATA | Carcinoma, Ductal, Breast - genetics | Receptors, Estrogen - metabolism | Humans | Receptor, ErbB-2 - metabolism | Clonal Evolution - genetics | Tamoxifen - administration & dosage | Case-Control Studies | Breast Neoplasms - metabolism | Brain Neoplasms - secondary | Carcinoma, Ductal, Breast - drug therapy | Neoplasm Metastasis | Lung Neoplasms - secondary | Adult | Carcinoma, Ductal, Breast - pathology | Female | Quinazolines - administration & dosage | Liver Neoplasms - secondary | Carcinoma, Ductal, Breast - metabolism | Lung Neoplasms - genetics | Liver Neoplasms - genetics | Deoxycytidine - administration & dosage | Brain Neoplasms - genetics | Trastuzumab - administration & dosage | Breast Neoplasms - drug therapy | Sequence Analysis, DNA | Spinal Neoplasms - secondary | Breast Neoplasms - genetics | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Breast Neoplasms - pathology | Spinal Neoplasms - genetics | Bayes Theorem | DNA, Neoplasm - genetics | Mutation | Deoxycytidine - analogs & derivatives | Biological evolution | Breast cancer | Genomes | Metastasis | ErbB-2 protein | Patients | Metastases | Heterogeneity | Biopsy | Breast | Evolution | Deoxyribonucleic acid--DNA | Cancer | Tumors | Index Medicus
Journal Article
Journal Article
Journal of Thoracic Oncology, ISSN 1556-0864, 03/2013, Volume 8, Issue 3, pp. 346 - 351
Journal Article
Cell Stem Cell, ISSN 1934-5909, 2010, Volume 6, Issue 6, pp. 603 - 615
Recent evidence suggests that a subpopulation of cancer cells, cancer stem cells (CSCs), is responsible for tumor growth in colorectal cancer. However,... 
STEMCELL | CELLCYCLE | BREAST-CANCER | INITIATING CELLS | DIPEPTIDYL-PEPTIDASE-IV | CARCINOMA CELLS | EXTRACELLULAR-MATRIX | ACUTE MYELOID-LEUKEMIA | E-CADHERIN | IDENTIFICATION | TUMOR-GROWTH | HUMAN PANCREATIC-CANCER | CELL & TISSUE ENGINEERING | CELL BIOLOGY | Neoplasm Transplantation | RNA, Small Interfering - genetics | Prognosis | Follow-Up Studies | Apoptosis - drug effects | Neoplastic Stem Cells - drug effects | Colorectal Neoplasms - genetics | Humans | Apoptosis - genetics | Gene Expression Profiling | Colorectal Neoplasms - diagnosis | Organoplatinum Compounds - pharmacology | Neoplastic Stem Cells - metabolism | Cell Transformation, Neoplastic - genetics | Colorectal Neoplasms - drug therapy | Liver Neoplasms - physiopathology | Neoplastic Stem Cells - pathology | Tumor Burden - genetics | Tumor Cells, Cultured | Liver Neoplasms - secondary | Colorectal Neoplasms - metabolism | Carcinoma - secondary | Carcinoma - drug therapy | Liver Neoplasms - genetics | Dipeptidyl Peptidase 4 - biosynthesis | Liver Neoplasms - drug therapy | Carcinoma - diagnosis | Dipeptidyl Peptidase 4 - genetics | Mice, SCID | Carcinoma - physiopathology | Disease Progression | Colorectal Neoplasms - physiopathology | Drug Resistance, Neoplasm - genetics | Animals | Liver Neoplasms - diagnosis | Tumor Burden - drug effects | Liver Neoplasms - metabolism | Carcinoma - genetics | Biomarkers, Tumor - genetics | Fluorouracil - pharmacology | Mice | Carcinoma - metabolism | Colorectal Neoplasms - pathology | Neoplasm Invasiveness - genetics | Cell Migration Assays | Biomarkers, Tumor - biosynthesis | Index Medicus
Journal Article
Nature, ISSN 0028-0836, 01/2012, Volume 481, Issue 7380, pp. 190 - 196
Metastatic progression of cancer is a complex and clinically daunting process(1-4). We previously identified a set of human microRNAs ( miRNAs) that robustly... 
MULTIDISCIPLINARY SCIENCES | GROWTH | Human Umbilical Vein Endothelial Cells | Neoplasm Transplantation | RNA-Binding Proteins - genetics | Receptor, IGF Type 1 - metabolism | Cell Proliferation | Humans | Gene Expression Regulation, Neoplastic | Lung Neoplasms - pathology | Neovascularization, Pathologic - pathology | Intercellular Signaling Peptides and Proteins - metabolism | Lung Neoplasms - secondary | Membrane Transport Proteins - genetics | c-Mer Tyrosine Kinase | Female | Liver Neoplasms - pathology | Membrane Transport Proteins - metabolism | Liver Neoplasms - secondary | Lung Neoplasms - blood supply | Proto-Oncogene Proteins - metabolism | Regulon - genetics | Signal Transduction | Proto-Oncogene Proteins - genetics | Breast Neoplasms - blood supply | Mice, SCID | Receptor Protein-Tyrosine Kinases - metabolism | Cell Adhesion | Neoplasm Metastasis - genetics | Animals | Breast Neoplasms - genetics | Liver Neoplasms - blood supply | Receptor Protein-Tyrosine Kinases - genetics | Breast Neoplasms - pathology | Epistasis, Genetic | Neoplasm Metastasis - pathology | Survival Analysis | Cell Line, Tumor | Endothelium, Vascular - pathology | Mice, Inbred NOD | Neovascularization, Pathologic - genetics | Mice | MicroRNAs - genetics | Insulin-Like Growth Factor I - metabolism | RNA-Binding Proteins - metabolism | Cell Movement | Proteins | Angiogenesis | Genotype & phenotype | Breast cancer | Kinases | Recruitment | Index Medicus
Journal Article
Journal of Thoracic Oncology, ISSN 1556-0864, 05/2015, Volume 10, Issue 5, pp. 768 - 777
Journal Article
Blood, ISSN 0006-4971, 06/2012, Volume 119, Issue 24, pp. 5795 - 5806
The pathogenesis of hepatosplenic T-cell lymphoma (HSTL), a rare entity mostly derived from gamma delta T cells and usually with a fatal outcome, remains... 
GAMMA-DELTA | TYROSINE KINASE | ALPHA-BETA | NON-HODGKINS-LYMPHOMA | NK-CELL | PI EXPRESSION | RECEPTOR | DRUG-RESISTANCE | HEMATOLOGY | NF-KAPPA-B | CLINICOPATHOLOGICAL ENTITY | Crystallins - metabolism | Protein-Tyrosine Kinases - metabolism | Humans | Middle Aged | Gene Expression Regulation, Neoplastic | Molecular Sequence Data | Male | Receptors, Antigen, T-Cell, gamma-delta - genetics | Gene Expression Profiling | Intracellular Signaling Peptides and Proteins - metabolism | Molecular Targeted Therapy | Young Adult | Base Sequence | Biomarkers, Tumor - metabolism | Adult | Female | Liver Neoplasms - pathology | Membrane Proteins - metabolism | Genes, Neoplasm - genetics | Syk Kinase | Cell Lineage - genetics | Splenic Neoplasms - drug therapy | Liver Neoplasms - genetics | Intracellular Signaling Peptides and Proteins - antagonists & inhibitors | Splenic Neoplasms - genetics | Liver Neoplasms - drug therapy | Lymphoma, T-Cell - genetics | Receptors, Antigen, T-Cell, alpha-beta - genetics | Isochromosomes - genetics | Drug Resistance, Neoplasm - genetics | Lymphoma, T-Cell - drug therapy | Splenic Neoplasms - pathology | Chromosome Aberrations | Lymphoma, T-Cell - pathology | Aged | Biomarkers, Tumor - genetics | Cluster Analysis | Protein-Tyrosine Kinases - antagonists & inhibitors | Index Medicus | Abridged Index Medicus | pathology | Lymphoma, T-Cell | Genes, Neoplasm | Drug Resistance, Neoplasm | Isochromosomes | Liver Neoplasms | genetics | Crystallins | Receptors, Antigen, T-Cell, alpha-beta | drug therapy | Tumor Markers, Biological | Intracellular Signaling Peptides and Proteins | antagonists & inhibitors | Membrane Proteins | Cell Lineage | Receptors, Antigen, T-Cell, gamma-delta | metabolism | Protein-Tyrosine Kinases | Splenic Neoplasms
Journal Article
Nature Medicine, ISSN 1078-8956, 03/2016, Volume 22, Issue 3, pp. 298 - 305
Journal Article