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Journal of the American Academy of Dermatology, ISSN 0190-9622, 2008, Volume 60, Issue 3, pp. 379 - 387
Background Previous reports regarding the rates at which various internal tumors metastasize to the skin have been limited and have only included the Caucasian... 
Dermatology | LUNG-CANCER | PROGNOSTIC-FACTORS | ASIAN ETHNICITY | BREAST-CANCER METASTASIS | SKIN-ASSOCIATED CHEMOKINE | BASAL-CELL CARCINOMA | WHITE WOMEN | DRINKING-WATER | AFRICAN-AMERICAN | CHINESE WOMEN | DERMATOLOGY | Immunohistochemistry | Lung Neoplasms - ethnology | Academic Medical Centers - statistics & numerical data | Carcinoma, Squamous Cell - metabolism | Humans | Lung Neoplasms - metabolism | Carcinoma, Transitional Cell - secondary | Lung Neoplasms - pathology | Mouth Neoplasms - metabolism | Carcinoma, Hepatocellular - secondary | Carcinoma, Transitional Cell - ethnology | Breast Neoplasms - metabolism | Carcinoma, Squamous Cell - ethnology | Gastrointestinal Neoplasms - pathology | Mouth Neoplasms - ethnology | Adenocarcinoma - metabolism | Urinary Bladder Neoplasms - pathology | Liver Neoplasms - pathology | Retrospective Studies | Adenocarcinoma - ethnology | Breast Neoplasms - ethnology | Urinary Bladder Neoplasms - metabolism | Carcinoma, Hepatocellular - ethnology | Gastrointestinal Neoplasms - metabolism | Receptors, CCR10 - metabolism | Skin Neoplasms - ethnology | Carcinoma, Transitional Cell - metabolism | Adenocarcinoma - secondary | Receptors, CXCR4 - metabolism | Skin Neoplasms - metabolism | Liver Neoplasms - ethnology | Breast Neoplasms - pathology | Taiwan - epidemiology | Liver Neoplasms - metabolism | Skin Neoplasms - secondary | Mouth Neoplasms - pathology | Carcinoma, Squamous Cell - secondary | Gastrointestinal Neoplasms - ethnology | Asian Continental Ancestry Group - statistics & numerical data | Carcinoma, Hepatocellular - metabolism | Urinary Bladder Neoplasms - ethnology | Medical colleges | Hospitals | Metastasis | Medical centers | Cancer
Journal Article
Journal Article
American Journal of Pathology, ISSN 0002-9440, 2010, Volume 177, Issue 4, pp. 1647 - 1656
Phosphatase and tensin homolog (PTEN) is a key modulator of trastuzumab sensitivity in HER2-overexpressing breast cancer. Because PTEN opposes the downstream... 
ADJUVANT CHEMOTHERAPY | INHIBITION | THERAPY | PROTEIN | HUMAN-BREAST-CANCER | PATHWAY | MUTATIONS | PATHOLOGY | RECEPTORS | TRASTUZUMAB RESISTANCE | EXPRESSION | Lung Neoplasms - drug therapy | Receptors, Estrogen - metabolism | Receptor, ErbB-2 - genetics | Lung Neoplasms - mortality | Humans | Middle Aged | Bone Neoplasms - secondary | Antibodies, Monoclonal - therapeutic use | Immunoenzyme Techniques | Receptors, Progesterone - metabolism | Young Adult | Carcinoma, Ductal, Breast - drug therapy | Brain Neoplasms - mortality | Liver Neoplasms - secondary | Proto-Oncogene Proteins c-akt - metabolism | Carcinoma, Ductal, Breast - metabolism | PTEN Phosphohydrolase - genetics | Ribosomal Protein S6 Kinases, 70-kDa - metabolism | Survival Rate | Lymphatic Metastasis | Brain Neoplasms - drug therapy | Mutation - genetics | Breast Neoplasms - drug therapy | Class I Phosphatidylinositol 3-Kinases | Carcinoma, Lobular - metabolism | Trastuzumab | Bone Neoplasms - mortality | Phosphorylation | Prognosis | Receptor, ErbB-2 - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Liver Neoplasms - mortality | Carcinoma, Ductal, Breast - mortality | Carcinoma, Lobular - mortality | Breast Neoplasms - metabolism | Antibodies, Monoclonal, Humanized | Brain Neoplasms - secondary | In Situ Hybridization | Lung Neoplasms - secondary | Adult | Female | Bone Neoplasms - drug therapy | Neoplasm Invasiveness | Liver Neoplasms - drug therapy | PTEN Phosphohydrolase - metabolism | Phosphatidylinositol 3-Kinases - genetics | Breast Neoplasms - mortality | Aged | Carcinoma, Lobular - drug therapy | Regular
Journal Article
Breast Cancer Research, ISSN 1465-5411, 09/2011, Volume 13, Issue 5, pp. R87 - R87
Journal Article
Metabolism, ISSN 0026-0495, 2015, Volume 64, Issue 2, pp. 182 - 189
Journal Article
Nature communications, ISSN 2041-1723, 2017, Volume 8, Issue 1, p. 13923
Tumour metastasis, the spread of cancer cells from the original tumour site followed by growth of secondary tumours at distant organs, is the primary cause of... 
EPITHELIAL-MESENCHYMAL TRANSITION | KINASE 4/6 INHIBITOR | PHOSPHORYLATION | MULTIDISCIPLINARY SCIENCES | TUMOR-METASTASIS | TRANSCRIPTION FACTOR SNAIL | HUMAN BREAST-CANCER | CELL | E-CADHERIN REPRESSION | PROGRESSION | PLASTICITY | Cyclin-Dependent Kinase 6 - antagonists & inhibitors | RNA, Small Interfering - genetics | Cyclin-Dependent Kinase 4 - genetics | Humans | Lung Neoplasms - metabolism | Gene Expression Regulation, Neoplastic | Ovarian Neoplasms - genetics | Ovarian Neoplasms - secondary | Snail Family Transcription Factors - genetics | Lung Neoplasms - secondary | MCF-7 Cells | Triple Negative Breast Neoplasms - pathology | Female | Antineoplastic Agents - pharmacology | Ovarian Neoplasms - metabolism | Cyclin-Dependent Kinase 4 - antagonists & inhibitors | Liver Neoplasms - secondary | Disease Models, Animal | Liver Neoplasms - prevention & control | Lung Neoplasms - genetics | Endopeptidases - metabolism | Snail Family Transcription Factors - metabolism | Liver Neoplasms - genetics | Ovarian Neoplasms - prevention & control | Signal Transduction | Cyclin-Dependent Kinase 6 - genetics | Cyclin-Dependent Kinase 6 - metabolism | Cyclin-Dependent Kinase 4 - metabolism | Piperazines - pharmacology | Xenograft Model Antitumor Assays | Leupeptins - pharmacology | Animals | Endopeptidases - genetics | Triple Negative Breast Neoplasms - genetics | Lung Neoplasms - prevention & control | Triple Negative Breast Neoplasms - metabolism | Liver Neoplasms - metabolism | Cell Line, Tumor | Mice | Pyridines - pharmacology | Cell Movement | RNA, Small Interfering - metabolism
Journal Article
Blood, ISSN 1528-0020, 2012, Volume 119, Issue 24, pp. 5795 - 5806
The pathogenesis of hepatosplenic T-cell lymphoma (HSTL), a rare entity mostly derived from γδ T cells and usually with a fatal outcome, remains largely... 
GAMMA-DELTA | TYROSINE KINASE | ALPHA-BETA | NON-HODGKINS-LYMPHOMA | NK-CELL | PI EXPRESSION | RECEPTOR | DRUG-RESISTANCE | HEMATOLOGY | NF-KAPPA-B | CLINICOPATHOLOGICAL ENTITY | Crystallins - metabolism | Protein-Tyrosine Kinases - metabolism | Humans | Middle Aged | Gene Expression Regulation, Neoplastic | Molecular Sequence Data | Male | Receptors, Antigen, T-Cell, gamma-delta - genetics | Gene Expression Profiling | Intracellular Signaling Peptides and Proteins - metabolism | Molecular Targeted Therapy | Young Adult | Base Sequence | Biomarkers, Tumor - metabolism | Adult | Female | Liver Neoplasms - pathology | Membrane Proteins - metabolism | Genes, Neoplasm - genetics | Syk Kinase | Cell Lineage - genetics | Splenic Neoplasms - drug therapy | Liver Neoplasms - genetics | Intracellular Signaling Peptides and Proteins - antagonists & inhibitors | Splenic Neoplasms - genetics | Liver Neoplasms - drug therapy | Lymphoma, T-Cell - genetics | Receptors, Antigen, T-Cell, alpha-beta - genetics | Isochromosomes - genetics | Drug Resistance, Neoplasm - genetics | Lymphoma, T-Cell - drug therapy | Splenic Neoplasms - pathology | Chromosome Aberrations | Lymphoma, T-Cell - pathology | Aged | Biomarkers, Tumor - genetics | Cluster Analysis | Protein-Tyrosine Kinases - antagonists & inhibitors | pathology | Lymphoma, T-Cell | Genes, Neoplasm | Drug Resistance, Neoplasm | Isochromosomes | Liver Neoplasms | genetics | Crystallins | Receptors, Antigen, T-Cell, alpha-beta | drug therapy | Tumor Markers, Biological | Intracellular Signaling Peptides and Proteins | antagonists & inhibitors | Membrane Proteins | Cell Lineage | Receptors, Antigen, T-Cell, gamma-delta | metabolism | Protein-Tyrosine Kinases | Splenic Neoplasms
Journal Article
Journal of Clinical Oncology, ISSN 0732-183X, 02/2010, Volume 28, Issue 6, pp. 976 - 983
Purpose To evaluate trastuzumab (H) and docetaxel (T) with or without capecitabine (X) as first-line combination therapy for human epidermal growth factor... 
SURVIVAL | COMBINATIONS | MULTICENTER | AMPLIFICATION | THERAPY | EFFICACY | ONCOLOGY | SAFETY | PRETREATED PATIENTS | ANTIBODY | PACLITAXEL | Prognosis | Receptor, ErbB-2 - genetics | Capecitabine | Humans | Middle Aged | Receptor, ErbB-2 - metabolism | Bone Neoplasms - secondary | Soft Tissue Neoplasms - drug therapy | Bone Neoplasms - metabolism | Breast Neoplasms - metabolism | Feasibility Studies | Young Adult | Antibodies, Monoclonal, Humanized | Fluorouracil - administration & dosage | Aged, 80 and over | Adult | Female | Bone Neoplasms - drug therapy | Liver Neoplasms - secondary | Fluorouracil - analogs & derivatives | Soft Tissue Neoplasms - secondary | Deoxycytidine - administration & dosage | Liver Neoplasms - drug therapy | Soft Tissue Neoplasms - metabolism | In Situ Hybridization, Fluorescence | Survival Rate | Treatment Outcome | Breast Neoplasms - drug therapy | Taxoids - administration & dosage | International Agencies | Antibodies, Monoclonal - administration & dosage | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Breast Neoplasms - pathology | Liver Neoplasms - metabolism | Aged | Neoplasm Staging | Deoxycytidine - analogs & derivatives | Trastuzumab | Liver Neoplasms | Breast Neoplasms | Life Sciences | Immunology | Taxoids | Fluorouracil | Antineoplastic Combined Chemotherapy Protocols | Antibodies, Monoclonal | Bone Neoplasms | Receptor, erbB-2 | Deoxycytidine | Soft Tissue Neoplasms
Journal Article
Nature communications, ISSN 2041-1723, 2015, Volume 6, Issue 1, p. 8760
Circulating tumour DNA analysis can be used to track tumour burden and analyse cancer genomes non-invasively but the extent to which it represents metastatic... 
HETEROGENEITY | ACTIVATION | VARIANTS | PATHWAY | MULTIDISCIPLINARY SCIENCES | KINASE | ACQUIRED-RESISTANCE | SEQUENCING DATA | Carcinoma, Ductal, Breast - genetics | Receptors, Estrogen - metabolism | Humans | Receptor, ErbB-2 - metabolism | Clonal Evolution - genetics | Tamoxifen - administration & dosage | Case-Control Studies | Breast Neoplasms - metabolism | Brain Neoplasms - secondary | Carcinoma, Ductal, Breast - drug therapy | Neoplasm Metastasis | Lung Neoplasms - secondary | Adult | Carcinoma, Ductal, Breast - pathology | Female | Quinazolines - administration & dosage | Lapatinib | Liver Neoplasms - secondary | Carcinoma, Ductal, Breast - metabolism | Lung Neoplasms - genetics | Liver Neoplasms - genetics | Deoxycytidine - administration & dosage | Brain Neoplasms - genetics | Trastuzumab - administration & dosage | Breast Neoplasms - drug therapy | Sequence Analysis, DNA | Spinal Neoplasms - secondary | Breast Neoplasms - genetics | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Breast Neoplasms - pathology | Spinal Neoplasms - genetics | Bayes Theorem | DNA, Neoplasm - genetics | Mutation | Deoxycytidine - analogs & derivatives | Biological evolution | Breast cancer | Genomes | Metastasis | ErbB-2 protein | Patients | Metastases | Heterogeneity | Biopsy | Evolution | Deoxyribonucleic acid--DNA | Cancer | Tumors
Journal Article
Annals of the Royal College of Surgeons of England, ISSN 0035-8843, 03/2017, Volume 99, Issue 3, pp. 193 - 197
INTRODUCTION Neuroendocrine tumours (NETs) are a heterogeneous group of tumours with a highly variable presentation and prognosis. Management decisions are... 
Ki-67 | Pathology | Neuroendocrine tumours | Diagnostic techniques and procedures | General surgery | SURVIVAL | SURGERY | PROGNOSTIC-FACTORS | NEOPLASMS | ENDOCRINE TUMORS | RGETNE | DISEASE | REGISTRY | Colorectal Neoplasms - surgery | Digestive System Neoplasms - metabolism | Pancreatic Neoplasms - metabolism | Prognosis | Humans | Lung Neoplasms - metabolism | Middle Aged | Stomach Neoplasms - metabolism | Ki-67 Antigen - metabolism | Lung Neoplasms - pathology | Male | Stomach Neoplasms - pathology | Neoplasm Grading | Digestive System Neoplasms - pathology | Adult | Female | Liver Neoplasms - pathology | Retrospective Studies | Biopsy, Large-Core Needle | Colorectal Neoplasms - metabolism | Ileal Neoplasms - pathology | Neuroendocrine Tumors - pathology | Ileal Neoplasms - surgery | Neuroendocrine Tumors - metabolism | Pancreatic Neoplasms - pathology | Pancreatic Neoplasms - surgery | Ileal Neoplasms - metabolism | Liver Neoplasms - surgery | Biopsy, Fine-Needle | Digestive System Neoplasms - surgery | Liver Neoplasms - metabolism | Lung Neoplasms - surgery | Neuroendocrine Tumors - surgery | Aged | Colorectal Neoplasms - pathology | Stomach Neoplasms - surgery | Cohort Studies | Ultrasonic imaging | Laboratories | Medical prognosis | Cell cycle | Hormones | Metastasis | Endoscopy | Epidemiology | Cancer therapies | Tumors | Oncology
Journal Article
Nature communications, ISSN 2041-1723, 2018, Volume 9, Issue 1, pp. 2612 - 11
The C-X-C chemokine receptor type 4 (CXCR4, CD184) pathway is a key regulator of cancer metastasis. Existing therapeutics that block CXCR4 signaling are... 
MIGRATION | D-AMINO ACIDS | CELLS | IN-VITRO | PROTEIN | RECOGNITION | CHOLESTEROL | MULTIDISCIPLINARY SCIENCES | DIMERIZATION | EXPRESSION | CXCR4 RECEPTOR | Luciferases - metabolism | Membrane Microdomains - metabolism | Humans | Lung Neoplasms - metabolism | Polyethylene Glycols - chemistry | Triple Negative Breast Neoplasms - drug therapy | Brain Neoplasms - metabolism | Luciferases - genetics | Antineoplastic Agents - metabolism | Brain Neoplasms - secondary | Peptides - metabolism | Lung Neoplasms - secondary | Triple Negative Breast Neoplasms - pathology | Female | Receptors, CXCR4 - genetics | Antineoplastic Agents - pharmacology | Liver Neoplasms - secondary | Genes, Reporter | Binding, Competitive | Liver Neoplasms - prevention & control | Lung Neoplasms - genetics | Gene Expression | Liver Neoplasms - genetics | Peptides - chemistry | Brain Neoplasms - genetics | Phosphatidylcholines - chemistry | Antineoplastic Agents - chemistry | Peptides - pharmacology | Receptors, CXCR4 - metabolism | Brain Neoplasms - prevention & control | Xenograft Model Antitumor Assays | Liposomes - chemistry | Animals | Triple Negative Breast Neoplasms - genetics | Lung Neoplasms - prevention & control | Membrane Microdomains - drug effects | Mice, Nude | Triple Negative Breast Neoplasms - metabolism | Liver Neoplasms - metabolism | Cell Line, Tumor | Protein Binding | Liposomes - metabolism | Mice | Phosphatidylethanolamines - chemistry | Cell Movement | Binding | Peptides | Gene regulation | Rafts | Lipid rafts | Breast cancer | Metastasis | Gene expression | Density | CXCR4 protein | Molecular chains | Cell adhesion & migration | Metastases | Signaling | CD18 antigen | Dimers | Liposomes | Cell migration | Cancer
Journal Article