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Journal of Clinical Investigation, ISSN 0021-9738, 08/2012, Volume 122, Issue 8, pp. 2884 - 2897
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 08/2012, Volume 122, Issue 8, pp. 2911 - 2915
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 08/2012, Volume 122, Issue 8, pp. 2871 - 2883
miR-122, an abundant liver-specific microRNA (miRNA), regulates cholesterol metabolism and promotes hepatitis C virus (HCV) replication. Reduced miR-122... 
MICRORNA BINDING | MEDICINE, RESEARCH & EXPERIMENTAL | HEPATOCELLULAR-CARCINOMA | LIPID-METABOLISM | IN-VIVO | GENE-EXPRESSION | MICE | STEATOHEPATITIS | DIFFERENTIATION | HEPATIC STEATOSIS | CANCER | MicroRNAs - therapeutic use | Liver - pathology | Cell Proliferation | MicroRNAs - antagonists & inhibitors | Fatty Liver - pathology | Humans | Cell Survival - genetics | MicroRNAs - metabolism | Liver Neoplasms - therapy | Monocytes - immunology | Genes, myc | Mice, 129 Strain | RNA, Neoplasm - metabolism | Liver Neoplasms, Experimental - metabolism | Liver - immunology | Base Sequence | Carcinoma, Hepatocellular - genetics | Lipids - blood | Monocytes - pathology | Lipid Metabolism - genetics | Liver Neoplasms, Experimental - genetics | 3' Untranslated Regions | Genes, Tumor Suppressor | Neutrophils - pathology | Fatty Liver - genetics | Gene Expression | Fatty Liver - metabolism | Liver Neoplasms - genetics | Liver - metabolism | Neutrophils - immunology | Liver Neoplasms, Experimental - etiology | Mice, Knockout | Animals | Liver Neoplasms - metabolism | RNA, Neoplasm - genetics | Carcinoma, Hepatocellular - therapy | Mice | MicroRNAs - genetics | Cytokines - biosynthesis | Carcinoma, Hepatocellular - metabolism | Fatty Liver - etiology | Liver diseases | MicroRNA | Anti-inflammatory drugs | Tumor necrosis factor | Physiological aspects | Hepatitis C virus | Cholesterol | Tumors
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 02/2013, Volume 123, Issue 2, pp. 630 - 645
Journal Article
Journal of Hepatology, ISSN 0168-8278, 2014, Volume 61, Issue 5, pp. 1088 - 1096
Background & Aims Although the growth suppressing Hippo pathway has been implicated in hepatocellular carcinoma (HCC) pathogenesis, it is unknown at which... 
Gastroenterology and Hepatology | Preneoplastic stages | HCC | Verteporfin | TCPOBOP | Hippo pathway | YES-ASSOCIATED PROTEIN | RAT | ORGAN SIZE CONTROL | REGENERATION | CELL-PROLIFERATION | LESIONS | CARCINOGENESIS | PATHWAY | HEPATOCELLULAR-CARCINOMA DEVELOPMENT | GASTROENTEROLOGY & HEPATOLOGY | TUMORIGENESIS | Adenoma, Liver Cell - drug therapy | Precancerous Conditions - etiology | Adenoma, Liver Cell - etiology | Humans | Middle Aged | Rats, Inbred F344 | Male | MicroRNAs - metabolism | Precancerous Conditions - metabolism | Phosphoproteins - metabolism | Liver Neoplasms - etiology | Precancerous Conditions - drug therapy | Young Adult | Liver Neoplasms, Experimental - metabolism | Carcinoma, Hepatocellular - drug therapy | Apoptosis Regulatory Proteins - genetics | Adult | Female | Antineoplastic Agents - pharmacology | Carcinoma, Hepatocellular - etiology | Protein-Serine-Threonine Kinases - metabolism | Gene Expression | Signal Transduction | Adenoma, Liver Cell - metabolism | Liver Neoplasms - drug therapy | Rats | Porphyrins - pharmacology | Liver Neoplasms, Experimental - etiology | Apoptosis Regulatory Proteins - metabolism | Animals | Liver Neoplasms, Experimental - drug therapy | Apoptosis Regulatory Proteins - antagonists & inhibitors | Liver Neoplasms - metabolism | Cell Line, Tumor | Aged | Cell Proliferation - drug effects | Mice | MicroRNAs - genetics | Adaptor Proteins, Signal Transducing - metabolism | Carcinoma, Hepatocellular - metabolism | Liver cancer | Health aspects | Index Medicus
Journal Article
International Journal of Cancer, ISSN 0020-7136, 10/2016, Volume 139, Issue 8, pp. 1764 - 1775
Journal Article
Cancer Cell, ISSN 1535-6108, 09/2014, Volume 26, Issue 3, pp. 331 - 343
Journal Article
Gastroenterology, ISSN 0016-5085, 08/2018, Volume 155, Issue 2, pp. 557 - 571.e14
MAF bZIP transcription factor G (MAFG) is activated by the farnesoid X receptor to repress bile acid synthesis. However, expression of MAFG increases during... 
FXR | Obeticholic Acid | S-Adenosylmethionine | Ursodeoxycholic Acid | CELLS | C-MYC | GLUTATHIONE SYNTHESIS | COLON-CANCER | DEOXYCHOLIC-ACID | MICE | GASTROENTEROLOGY & HEPATOLOGY | NF-KAPPA-B | METHIONINE | Diethylnitrosamine - toxicity | Up-Regulation | Carcinoma, Hepatocellular - mortality | Liver - pathology | Humans | Male | Cholangiocarcinoma - etiology | Liver Neoplasms - mortality | Gene Knockdown Techniques | Cholestasis - etiology | Carcinoma, Hepatocellular - genetics | Liver Neoplasms - pathology | Liver Neoplasms, Experimental - genetics | Bile Duct Neoplasms - genetics | Gene Expression Regulation, Neoplastic - drug effects | Liver Neoplasms, Experimental - pathology | MafG Transcription Factor - metabolism | MafG Transcription Factor - genetics | Repressor Proteins - metabolism | Liver Neoplasms - virology | Liver Neoplasms - genetics | Mice, Inbred C57BL | Repressor Proteins - genetics | Carcinoma, Hepatocellular - virology | Receptors, Cytoplasmic and Nuclear - agonists | S-Adenosylmethionine - pharmacology | Liver Neoplasms, Experimental - etiology | Bile Duct Neoplasms - etiology | Xenograft Model Antitumor Assays | Cholestasis - pathology | Cholic Acids - pharmacology | Cholangiocarcinoma - pathology | Animals | Mice, Nude | Bile Duct Neoplasms - mortality | Carcinoma, Hepatocellular - pathology | Cell Line, Tumor | Cholangiocarcinoma - genetics | Mice | Mice, Inbred BALB C | Bile Duct Neoplasms - pathology | RNA, Small Interfering - metabolism | Liver cancer | Analysis | obeticholic acid | ursodeoxycholic acid | S-adenosylmethionine
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 1/2015, Volume 112, Issue 2, pp. 542 - 547
Journal Article