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Journal Article
The Journal of clinical investigation, ISSN 0021-9738, 2013, Volume 123, Issue 7, pp. 2907 - 2920
Metabolic reprogramming is an important driver of tumor progression; however, the metabolic regulators of tumor cell motility and metastasis are not... 
CROSS-TALK | SURVIVAL | MEDICINE, RESEARCH & EXPERIMENTAL | HOMEOSTASIS | PROTEIN | PHOSPHORYLATION | KINASE | GROWTH | AMPK | AUTOPHAGY | LKB1 | Liver Neoplasms, Experimental - secondary | NIH 3T3 Cells | Neoplasm Transplantation | RNA, Small Interfering - genetics | Phosphorylation | Lung Neoplasms - mortality | Humans | Lung Neoplasms - metabolism | Stress, Physiological | Bone Neoplasms - secondary | Guanidines - pharmacology | Lung Neoplasms - pathology | Intracellular Signaling Peptides and Proteins - metabolism | Autophagy-Related Protein-1 Homolog | Bone Neoplasms - metabolism | Gene Knockdown Techniques | Liver Neoplasms, Experimental - metabolism | Carcinoma, Non-Small-Cell Lung - secondary | Adenylate Kinase - metabolism | Female | Antineoplastic Agents - pharmacology | HSP90 Heat-Shock Proteins - genetics | Mitochondrial Membranes - enzymology | Intracellular Signaling Peptides and Proteins - genetics | Protein-Serine-Threonine Kinases - metabolism | Carcinoma, Non-Small-Cell Lung - metabolism | Kaplan-Meier Estimate | Protein-Serine-Threonine Kinases - genetics | Mitochondria - metabolism | Lactams, Macrocyclic - pharmacology | Mice, SCID | Carcinoma, Non-Small-Cell Lung - mortality | Animals | Cell Line, Tumor | Cytoskeleton - metabolism | HSP90 Heat-Shock Proteins - metabolism | Mice | Protein Processing, Post-Translational | Hexokinase - metabolism | Cell Movement | Cancer cells | Physiological aspects | Cytoskeleton | Stress (Physiology) | Metastasis | Research | Health aspects | Motility | Kinases | Experiments | Patients | Cell adhesion & migration | Cell growth | Mitochondria | Microscopy | Protein folding | Laboratory animals | Tumors | Cancer
Journal Article
by Fu, X and Meng, Z and Liang, W and Tian, Y and Wang, X and Han, W and Lou, G and Lou, F and Yen, Y and Yu, H and Jove, R and Huang, W
Oncogene, ISSN 1476-5594, 2013, Volume 33, Issue 34, pp. 4296 - 4306
Journal Article
The Journal of clinical investigation, ISSN 0021-9738, 08/2012, Volume 122, Issue 8, pp. 2871 - 2883
miR-122, an abundant liver-specific microRNA (miRNA), regulates cholesterol metabolism and promotes hepatitis C virus (HCV) replication... 
MICRORNA BINDING | MEDICINE, RESEARCH & EXPERIMENTAL | HEPATOCELLULAR-CARCINOMA | LIPID-METABOLISM | IN-VIVO | GENE-EXPRESSION | MICE | STEATOHEPATITIS | DIFFERENTIATION | HEPATIC STEATOSIS | CANCER | MicroRNAs - therapeutic use | Liver - pathology | Cell Proliferation | MicroRNAs - antagonists & inhibitors | Fatty Liver - pathology | Humans | Cell Survival - genetics | MicroRNAs - metabolism | Liver Neoplasms - therapy | Monocytes - immunology | Genes, myc | Mice, 129 Strain | RNA, Neoplasm - metabolism | Liver Neoplasms, Experimental - metabolism | Liver - immunology | Base Sequence | Carcinoma, Hepatocellular - genetics | Lipids - blood | Monocytes - pathology | Lipid Metabolism - genetics | Liver Neoplasms, Experimental - genetics | 3' Untranslated Regions | Genes, Tumor Suppressor | Neutrophils - pathology | Fatty Liver - genetics | Gene Expression | Fatty Liver - metabolism | Liver Neoplasms - genetics | Liver - metabolism | Neutrophils - immunology | Liver Neoplasms, Experimental - etiology | Mice, Knockout | Animals | Liver Neoplasms - metabolism | RNA, Neoplasm - genetics | Carcinoma, Hepatocellular - therapy | Mice | MicroRNAs - genetics | Cytokines - biosynthesis | Carcinoma, Hepatocellular - metabolism | Fatty Liver - etiology | Liver diseases | MicroRNA | Anti-inflammatory drugs | Tumor necrosis factor | Physiological aspects | Hepatitis C virus | Cholesterol | Tumors
Journal Article
The Journal of clinical investigation, ISSN 0021-9738, 2012, Volume 122, Issue 8, pp. 2884 - 2897
Journal Article
European Urology, ISSN 0302-2838, 2016, Volume 71, Issue 3, pp. 313 - 316
Abstract STAT3 and its upstream activator IL6R have been implicated in the progression of prostate cancer and are possible future therapeutic targets. We... 
Urology | Tissue microarray | Castration-resistant | Prostate cancer | STAT3 | Metastases | CELLS | ACTIVATION | UROLOGY & NEPHROLOGY | RECEPTOR | BONE | Immunohistochemistry | Prostatic Neoplasms - metabolism | Humans | Transcriptome | Benzamides - metabolism | Bone Neoplasms - secondary | Male | Phosphoproteins - metabolism | Prostatic Neoplasms, Castration-Resistant - genetics | Prostatic Neoplasms, Castration-Resistant - pathology | Bone Neoplasms - metabolism | Neoplasm Metastasis | Adenocarcinoma - metabolism | Prostatic Neoplasms - genetics | Autopsy | Adenocarcinoma - genetics | Bone Neoplasms - genetics | Liver Neoplasms - secondary | STAT3 Transcription Factor - genetics | STAT3 Transcription Factor - metabolism | Prostatic Neoplasms - pathology | Liver Neoplasms - genetics | Piperidines - metabolism | Receptors, Interleukin-6 - genetics | Lymph Nodes - metabolism | Lymphatic Metastasis | Prostatic Neoplasms, Castration-Resistant - metabolism | Adenocarcinoma - secondary | Liver Neoplasms - metabolism | Receptors, Interleukin-6 - metabolism | RNA | Analysis | Genes | Genetic research | Development and progression | Metastasis | Gene expression | Proteins | Medical research | Medicine, Experimental | Urologi och njurmedicin | Medical and Health Sciences | Medicin och hälsovetenskap | Klinisk medicin | Clinical Medicine | Cancer and Oncology | Urology and Nephrology | Cancer och onkologi
Journal Article
JNCI : Journal of the National Cancer Institute, ISSN 0027-8874, 06/2012, Volume 104, Issue 12, pp. 906 - 922
Background A disintegrin and metalloproteinase 28 (ADAM28) is implicated in tumor growth and metastasis in human breast and non-small cell lung carcinomas. We... 
VONWILLEBRAND-FACTOR | A1 DOMAIN | ANGIOGENESIS | ONCOLOGY | DISINTEGRIN | FACTOR BINDING PROTEIN-3 | TUMOR-CELLS | PROLIFERATION | INTEGRIN ALPHA(V)BETA | GROWTH-FACTOR | METALLOPROTEINASES | Immunohistochemistry | Neoplasms - metabolism | Humans | Lung Neoplasms - metabolism | Gene Expression Regulation, Neoplastic | Immunoblotting | Lung Neoplasms - pathology | Transplantation, Heterologous | Kidney Neoplasms - metabolism | von Willebrand Factor - genetics | Luminescent Measurements | Breast Neoplasms - metabolism | Gene Knockdown Techniques | Neoplasms, Experimental - pathology | Liver Neoplasms - pathology | von Willebrand Factor - metabolism | Carcinoma, Non-Small-Cell Lung - pathology | Carcinoma, Renal Cell - pathology | Carcinoma, Non-Small-Cell Lung - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Gene Expression Regulation, Enzymologic | Carcinoma, Renal Cell - metabolism | Two-Hybrid System Techniques | ADAM Proteins - metabolism | Animals | Breast Neoplasms - pathology | Carcinoma, Hepatocellular - pathology | Liver Neoplasms - metabolism | Cell Line, Tumor | Kidney Neoplasms - pathology | Mice | Neoplasms, Experimental - metabolism | ADAM Proteins - genetics | Neoplasms - pathology | RNA, Small Interfering - administration & dosage | Apoptosis | Carcinoma, Hepatocellular - metabolism | RNA, Small Interfering - metabolism | Physiological aspects | Breast cancer | Metastasis | Research | Lung cancer, Non-small cell | Von Willebrand factor
Journal Article
Journal of hepatology, ISSN 0168-8278, 2013, Volume 59, Issue 3, pp. 518 - 527
Journal Article
Journal of Hepatology, ISSN 0168-8278, 2009, Volume 51, Issue 4, pp. 725 - 733
... of sorafenib in combination with rapamycin, an inhibitor of mTOR pathway, in experimental models... 
Gastroenterology and Hepatology | Molecular therapies | mTOR | Liver cancer | HCV | Signaling pathway | Ras | PROMOTER HYPERMETHYLATION | SUPPRESSOR | SIGNALING PATHWAYS | RASSF1A | ABERRANT METHYLATION | INACTIVATION | MOLECULAR CLASSIFICATION | GENE | HIGH-FREQUENCY | GASTROENTEROLOGY & HEPATOLOGY | NORE1A | Niacinamide - analogs & derivatives | Neoplasm Transplantation | Antineoplastic Combined Chemotherapy Protocols - administration & dosage | Humans | ras Proteins - metabolism | Transplantation, Heterologous | Phenylurea Compounds | Benzenesulfonates - administration & dosage | Promoter Regions, Genetic - drug effects | RNA, Messenger - metabolism | RNA, Neoplasm - metabolism | Liver Neoplasms, Experimental - metabolism | Gene Dosage - drug effects | Carcinoma, Hepatocellular - drug therapy | Carcinoma, Hepatocellular - genetics | Female | Liver Neoplasms - pathology | Genes, ras - drug effects | Liver Neoplasms, Experimental - genetics | Liver Neoplasms, Experimental - pathology | Protein Kinases - drug effects | Pyridines - administration & dosage | Liver Neoplasms - genetics | RNA, Messenger - genetics | Liver Neoplasms - drug therapy | Drug Synergism | Sirolimus - administration & dosage | Animals | Liver Neoplasms, Experimental - drug therapy | Signal Transduction - drug effects | Mice, Nude | Carcinoma, Hepatocellular - pathology | Liver Neoplasms - metabolism | Cell Line, Tumor | RNA, Neoplasm - genetics | Cell Proliferation - drug effects | Mice | TOR Serine-Threonine Kinases | DNA Methylation - drug effects | Carcinoma, Hepatocellular - metabolism | Antimitotic agents | Oncology, Experimental | Analysis | Genetic research | Transplantation of organs, tissues, etc | Hepatoma | Research | Hepatitis C | Antineoplastic agents | Cancer | Index Medicus
Journal Article
Nature communications, ISSN 2041-1723, 2019, Volume 10, Issue 1, pp. 83 - 13
Mitochondrial topoisomerase IB (TOP1MT) is a nuclear-encoded topoisomerase, exclusively localized to mitochondria, which resolves topological stress generated... 
CHAIN | RNA GRANULES | REGULATOR | HEPATOCELLULAR-CARCINOMA | ROLES | PROMOTION | MULTIDISCIPLINARY SCIENCES | TRANSCRIPTION | STEM-CELL | DNA TOPOISOMERASES | CANCER | Carcinoma, Hepatocellular - mortality | Liver - pathology | Protein Biosynthesis | Cell Proliferation | Prognosis | Humans | Gene Expression Regulation, Neoplastic | Liver Neoplasms, Experimental - chemically induced | Male | Gene Expression Profiling | Liver Neoplasms - mortality | Carcinogens - toxicity | DNA, Mitochondrial - isolation & purification | Liver Neoplasms, Experimental - metabolism | Cell Nucleus - metabolism | DNA, Mitochondrial - genetics | Carcinoma, Hepatocellular - genetics | Fibroblasts | Female | Liver Neoplasms - pathology | Liver Neoplasms, Experimental - genetics | Liver Neoplasms, Experimental - pathology | Datasets as Topic | DNA Topoisomerases, Type I - metabolism | Liver Neoplasms - genetics | HCT116 Cells | Liver - metabolism | DNA Topoisomerases, Type I - genetics | Mitochondria - metabolism | Mitochondria - pathology | Mice, Knockout | Carcinogenesis - pathology | Xenograft Model Antitumor Assays | Animals | Energy Metabolism | Mice, Nude | Carcinoma, Hepatocellular - pathology | Liver Neoplasms - metabolism | Survival Analysis | Glycolysis | Liver - cytology | Mice | Carcinoma, Hepatocellular - metabolism | Cell proliferation | Phosphorylation | Animal models | Translation | Transcription | Liver | Hepatocellular carcinoma | Mitochondrial DNA | Drug development | Carcinogenesis | Liver cancer | Mitochondria | Carcinogens | Colon cancer | Oxidative phosphorylation | Colon | Cancer | Tumors | Life Sciences | Human health and pathology
Journal Article
Journal Article
Journal of hepatology, ISSN 0168-8278, 2014, Volume 61, Issue 5, pp. 1088 - 1096
... of the transcriptional co-activator Yes-associated protein (YAP) is an early event. Methods The experimental model used is the resistant-hepatocyte (R-H) rat model... 
Gastroenterology and Hepatology | Preneoplastic stages | HCC | Verteporfin | TCPOBOP | Hippo pathway | YES-ASSOCIATED PROTEIN | RAT | ORGAN SIZE CONTROL | REGENERATION | CELL-PROLIFERATION | LESIONS | CARCINOGENESIS | PATHWAY | HEPATOCELLULAR-CARCINOMA DEVELOPMENT | GASTROENTEROLOGY & HEPATOLOGY | TUMORIGENESIS | Adenoma, Liver Cell - drug therapy | Precancerous Conditions - etiology | Adenoma, Liver Cell - etiology | Humans | Middle Aged | Rats, Inbred F344 | Male | MicroRNAs - metabolism | Precancerous Conditions - metabolism | Phosphoproteins - metabolism | Liver Neoplasms - etiology | Precancerous Conditions - drug therapy | Young Adult | Liver Neoplasms, Experimental - metabolism | Carcinoma, Hepatocellular - drug therapy | Apoptosis Regulatory Proteins - genetics | Adult | Female | Antineoplastic Agents - pharmacology | Carcinoma, Hepatocellular - etiology | Protein-Serine-Threonine Kinases - metabolism | Gene Expression | Signal Transduction | Adenoma, Liver Cell - metabolism | Liver Neoplasms - drug therapy | Rats | Porphyrins - pharmacology | Liver Neoplasms, Experimental - etiology | Apoptosis Regulatory Proteins - metabolism | Animals | Liver Neoplasms, Experimental - drug therapy | Apoptosis Regulatory Proteins - antagonists & inhibitors | Liver Neoplasms - metabolism | Cell Line, Tumor | Aged | Cell Proliferation - drug effects | Mice | MicroRNAs - genetics | Adaptor Proteins, Signal Transducing - metabolism | Carcinoma, Hepatocellular - metabolism | Liver cancer | Health aspects | Index Medicus
Journal Article